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1 after treatment started (including data for bone mineral density).
2 n associated with renal toxicity and reduced bone mineral density.
3 libido, vasomotor instability, and decreased bone mineral density.
4 ion, and then develop anemia and a decreased bone mineral density.
5 omy-induced osteoporosis results in improved bone mineral density.
6 exposure were independent predictors of low bone mineral density.
7 evious aromatase inhibitor use, and baseline bone mineral density.
8 n IBD patients and to IBD-associated loss of bone mineral density.
9 sumab, however, results in rapidly declining bone mineral density.
10 t or blunt the effects of corticosteroids on bone mineral density.
11 at individuals with RTS have decreased areal bone mineral density.
12 ents with osteoporosis/osteopenia and normal bone mineral density.
13 nflammatory bowel disease-associated loss of bone mineral density.
14 ceived placebo, had a significant decline in bone mineral density.
15 ea, but there were no such associations with bone mineral density.
16 sa that is independent of that provided with bone mineral density.
17 e surface, accompanied by a dramatic loss of bone mineral density.
18 ut not CD8+ T cells significantly diminished bone mineral density.
19 ears in a Women's Health Initiative study of bone mineral density.
20 95% CI -0.01, 0.01]; p = 0.80; n = 127,587); bone mineral density (0.01 g/cm(2) [95% CI -0.01, 0.03];
21 %, 95% CI 1.54 to 5.89; p=0.26), nor did hip bone mineral density (2.09%, 95% CI -1.45 to 5.63 vs 0.0
22 al women, 55 to 85 years of age, who had low bone mineral density (a T score of -2.0 or less at the l
24 ary endpoint was the percent change in areal bone mineral density (aBMD) of the lumbar spine (LS), as
26 are variables that are not captured by areal bone mineral density (aBMD), and dietary protein intakes
27 erum type I collagen C-telopeptide), low hip bone mineral density, absence of urticaria pigmentosa, a
29 eal (P=0.001) and volumetric (P<0.001-0.006) bone mineral density and 1.5- to 1.8-fold increases in r
32 s, romosozumab was associated with increased bone mineral density and bone formation and with decreas
33 not dwarfed and had significantly increased bone mineral density and bone mineral content in femurs
34 ation and finite element analysis to measure bone mineral density and bone strength at the hip and sp
36 ning markers of bone turnover and whole-body bone mineral density and content were not affected by ei
37 Knockout (KO) (sost(-/-)) mice had increased bone mineral density and content, increased cortical and
38 f calcium supplements to prevent declines in bone mineral density and fractures is widespread in the
41 g from autism have been reported to have low bone mineral density and increased risk for fracture, ye
42 genetic factors with pleiotropic effects on bone mineral density and lean mass.Bone mineral density
43 ffects on bone mineral density and lean mass.Bone mineral density and lean skeletal mass are heritabl
45 mine if computed tomographic (CT) metrics of bone mineral density and muscle mass can improve the pre
46 L5HU and PsoasL4-5, which are surrogates for bone mineral density and muscle mass, respectively, were
47 ignificantly higher bone volume/total volume bone mineral density and number of osteoblasts in the ra
48 nce were observed as were reduced whole body bone mineral density and reduced trabecular bone mass.
51 istal radius was performed and evaluated for bone mineral density and trabecular and cortical bone mi
52 hese mice revealed a significant increase in bone mineral density and trabecular and cortical bone pa
54 ) levels and high bone turnover markers, low bone mineral density, and an increased risk of osteoporo
55 rception of health by a visual analog scale, bone mineral density, and body composition at baseline a
56 ice by adoptive transfer, and bone turnover, bone mineral density, and indices of bone structure and
57 Association of perfluoroalkyl substances, bone mineral density, and osteoporosis in the U.S. popul
63 ores of less than -2.0, mean change of spine bone mineral density at 2 years did not differ significa
65 mide had a significantly smaller decrease in bone mineral density at hip (mean change -0.10% [95% CI
66 ry end points included percentage changes in bone mineral density at other sites and in markers of bo
67 01), and a significantly smaller decrease in bone mineral density at spine (mean % change -1.30 vs -2
68 ry, ECSW was associated with preservation of bone mineral density at the central skeleton; however, i
69 t was the percentage change from baseline in bone mineral density at the lumbar spine at 12 months.
70 ere associated with significant increases in bone mineral density at the lumbar spine, including an i
72 , vs. 55.0 to 52.3 kg [5% decrease]), as did bone mineral density at the total hip (grams per square
73 was also associated with large increases in bone mineral density at the total hip and femoral neck,
74 ciation between serum PFAS concentration and bone mineral density at total femur (TFBMD), femoral nec
75 entage changes in lumbar spine and total hip bone mineral density at week 48, assessed by dual energy
77 rnib monotherapy treatment reveal additional bone mineral density benefit but likely no added cardiov
79 l risedronate for prevention of reduction in bone mineral density (BMD) after 3 years of follow-up in
81 ondition associated with progressive loss of bone mineral density (BMD) and compromised bone strength
83 ody mass, shortened body length, and reduced bone mineral density (BMD) and content (BMC) first evide
84 c skeletal disorder characterized by reduced bone mineral density (BMD) and disrupted bone architectu
86 ociation studies (GWASs) identified multiple bone mineral density (BMD) and fracture-associated loci.
87 hoblastic leukemia (ALL) are at risk for low bone mineral density (BMD) and frail health, outcomes po
88 have increased fracture risk, despite normal bone mineral density (BMD) and high BMI-factors that are
89 tary patterns that explain most variation in bone mineral density (BMD) and hip bone geometry are ass
90 tis C virus (HCV) is associated with reduced bone mineral density (BMD) and increased fracture rates,
91 g CAC progression, including measurements of bone mineral density (BMD) and novel bone markers in adu
95 ndependently predict fracture risk and, with bone mineral density (BMD) assessed by X-ray (DXA), may
97 one density contributing to lower volumetric bone mineral density (BMD) at both distal radius and tib
99 between clinical attachment level (CAL) and bone mineral density (BMD) at the lumbar spine and hip,
100 hip, and non-vertebral fractures as well as bone mineral density (BMD) at the lumbar spine, total hi
101 e mineral content (BMC), bone area (BA), and bone mineral density (BMD) at the total body, lumbar spi
102 ng the relationship between dairy intake and bone mineral density (BMD) because they are unable to co
103 ce imaging in 215 healthy army recruits, and bone mineral density (BMD) by Dual X-Ray Absorptiometry
107 ave a protective effect on lumbar spine (LS) bone mineral density (BMD) compared with lower protein i
110 endpoint was percentage change in total hip bone mineral density (BMD) from baseline to week 48 in t
113 an mass (ALM), quadriceps strength (QS), and bone mineral density (BMD) in 2986 men and women, aged 1
114 ed lumbar spine, total hip, and femoral neck bone mineral density (BMD) in 581 HIV-positive (94.7% re
115 identified more than 60 loci associated with bone mineral density (BMD) in adults but less is known a
120 anion study to MA.27, we compared changes in bone mineral density (BMD) in the lumbar spine and total
121 o be significantly associated with decreased bone mineral density (BMD) in two independent cohorts in
122 in osteoprotegerin correlate with decreased bone mineral density (BMD) in untreated HIV infection.
123 monoclonal antibody, versus teriparatide on bone mineral density (BMD) in women with postmenopausal
130 hrolithiasis, bone densitometry scoring, and bone mineral density (BMD) loss according to bone turnov
131 (HIV) disease before treatment contribute to bone mineral density (BMD) loss after ART initiation.
132 [control (CON)].RCE significantly attenuated bone mineral density (BMD) loss at the L2-L4 lumbar spin
133 -analysis examining isoflavone therapies and bone mineral density (BMD) loss in peri- and postmenopau
135 ted data on current anthropometric measures, bone mineral density (BMD) measured by dual-energy X-ray
139 g-reported parental hip fracture in a unique bone mineral density (BMD) registry linked to administra
140 lowing strategies: no intervention; one-time bone mineral density (BMD) screening and selective bisph
141 Manitoba, Canada at the time of their first bone mineral density (BMD) test posttransplant (mean 1.1
145 nd whole-body bone mineral content (BMC) and bone mineral density (BMD) were measured at age 20 y thr
147 ion between protein intake with fracture and bone mineral density (BMD) within the Women's Health Ini
149 ally relevant to osteoporosis, assessed from bone mineral density (BMD), as a new potential target of
150 content and density, cortical and trabecular bone mineral density (BMD), BMC, and bone area at the 4%
151 stigated their prospective associations with bone mineral density (BMD), bone area, and bone mineral
153 ially vegan diets, are associated with lower bone mineral density (BMD), but this does not appear to
154 o, usual care, or active control in terms of bone mineral density (BMD), fractures, and safety in pat
155 ions aimed at preventing fracture, improving bone mineral density (BMD), or preventing or delaying os
156 tion between B-vitamin status biomarkers and bone mineral density (BMD), risk of osteoporosis, and bi
157 bl-Wnt16 mice displayed increased total body bone mineral density (BMD), surprisingly caused mainly b
158 were accompanied by diminishing weight loss, bone mineral density (BMD), trabecular thickness, trabec
181 dual-energy x-ray absorptiometry scans (low bone mineral density [BMD], 23.2%), serum ferritin (iron
182 DXA (reference standard) to determine areal bone mineral densities (BMDs), and (c) quantitative CT w
183 1 y of age and in a subgroup at 2 y of age : Bone mineral density, bone mineral content (BMC), area-a
184 p-null (Bsp(-/-)) mice exhibit reductions in bone mineral density, bone turnover, osteoclast activati
185 d a smaller decrease in lumbar spine and hip bone mineral density but greater accumulation of limb an
186 ) infection has been associated with reduced bone mineral density, but its association with fracture
187 tients with chronic hepatitis B have reduced bone mineral density, but the reduction is limited to 1
189 micro-computed tomographic (CT) imaging and bone mineral density by peripheral quantitative CT scann
190 ect to their metabolic bone status including bone mineral density, calcium kinetics studies, and mark
192 deletion of Cx37 (Cx37(-/-)) exhibit higher bone mineral density, cancellous bone volume, and mechan
193 absorptiometry (DEXA) was used to determine bone mineral density changes in TDF-exposed patients.
194 these antibodies led to a marked increase in bone mineral density, consistent with inhibition of oste
195 en switching from teriparatide to denosumab, bone mineral density continued to increase, whereas swit
196 nce of pathogenic variants in RECQL4 and low bone mineral density correlate with the history of incre
199 her fracture genetic risk score (Fx-GRS) and bone mineral density genetic risk score (BMD-GRS) modify
200 rong patient-level risk factors included low bone mineral density (hazard ratio [HR], 0.53 per unit i
201 nd was associated with a smaller decrease in bone mineral density; however, greater resistance and ga
206 t testosterone replacement therapy increases bone mineral density in hypogonadal men, including men w
207 sorptive agents are clearly able to preserve bone mineral density in men on ADT, whereas other approa
208 restores reproductive capacity and increases bone mineral density in patients with hypothalamic ameno
209 tch study, we aimed to assess the changes in bone mineral density in postmenopausal osteoporotic wome
211 reater than -2.0 at baseline, mean change of bone mineral density in the spine at 2 years did not dif
212 n during flight, the greater the decrease in bone mineral density in the total hip (P = 0.031), troch
213 nes were significantly associated with spine bone mineral density, including BDNF, PDE4D, and SATB2,
214 48 months, the primary outcome of mean spine bone mineral density increased by 18.3% (95% CI 14.9-21.
216 ineral density secondary outcomes, total hip bone mineral density increased more in the teriparatide
218 ion Combined assessment of bone strength and bone mineral density is a cost-effective strategy for os
220 uce fragility fractures in patients with low bone mineral density is beyond the scope of the guidelin
221 ar growth attenuation and adverse effects on bone mineral density is generally low but should be cons
224 years for altered BMB to produce changes in bone mineral density large enough to resolve by X-ray de
225 ovements in blood pressure, body mass index, bone mineral density, lipid levels, or quality-of-life m
226 s in eight loci, including seven established bone mineral density loci: WNT4, GALNT3, MEPE, CPED1/WNT
228 cant component of the pathophysiology of the bone mineral density loss associated with Inflammatory B
233 ctures in nontrauma patients at risk for low bone mineral density may go unreported at abdominal mult
234 osteoporotic fracture risk, with or without bone mineral density measurements obtained from dual-ene
235 splay skeletal alterations including reduced bone mineral density, modified bone structure and distin
238 f fluoride's effects showed some increase in bone mineral density of adolescents and young adults in
240 D status were demonstrated to reduce loss of bone mineral density on long-duration International Spac
244 e revealed increased remodelling and reduced bone mineral density portrayed by increased carbonate to
245 we show that Ppia(-/-) mice demonstrate low bone mineral density, reduced osteoblast numbers, and in
246 , and suppression of ectopic calcifications, bone mineral density reduction, pulmonary emphysema and
248 8-year-old woman consults you after a recent bone mineral density screening revealed osteopenia, tota
250 users should not routinely screen or monitor bone mineral density, serum creatinine, magnesium, or vi
252 revented the reduction in spinal and femoral bone mineral density, spinal bone volume/tissue volume,
253 ostin inhibition could be applied to enhance bone mineral density, stability, and regeneration in non
254 eplacement therapy has been shown to improve bone mineral density, studies have also linked bone loss
255 n increases in bone formation biomarkers and bone mineral density, suggesting that sclerostin inhibit
256 risk factors for osteoporotic fractures, and bone mineral density surveillance) originated from the q
257 commended in postmenopausal women who have a bone mineral density T score of -2.5 or less, a history
258 erate or one severe vertebral fracture and a bone mineral density T score of less than or equal to -1
259 The Mann-Whitney test was used to compare bone mineral density T scores and elastic moduli between
262 eak relationships between elastic moduli and bone mineral density T scores in patients with fractures
265 ient subgroups, including in patients with a bone mineral density T-score of -1 or higher at baseline
266 CI 0.31-0.64], p<0.0001) and in those with a bone mineral density T-score of less than -1 already at
267 y lean mass (TB-LM) and total-body less head bone mineral density (TBLH-BMD) regions in 10,414 childr
268 s associated with significantly less loss of bone mineral density than a standard regimen containing
269 urgery, the hind limb had significantly less bone mineral density than contralateral controls, confir
270 pids, and greater decreases from baseline in bone mineral density than did those who received placebo
271 ficantly smaller mean percentage declines in bone mineral density than those receiving tenofovir diso
272 hese mice displayed significant reduction in bone mineral density, trabecular bone volume, and cortic
273 ng bone disease that is characterised by low bone mineral density, typically assessed using dual-ener
274 ex, serum type I collagen C-telopeptide, hip bone mineral density, urticaria pigmentosa, and alcohol
276 oncentration, serum phosphate concentration, bone mineral density, vascular calcification, renal func
277 with a significant increase in femoral neck bone mineral density; vascular calcification remained un
290 t mass, lean mass, bone mineral content, and bone mineral density, was determined by dual-energy X-ra
291 rial which tested the effect of denosumab on bone mineral density, we assessed the impact of this dru
294 g/mL was significantly associated with lower bone mineral density, whereas an untransformed 25(OH)D c
295 the loss of total, trabecular, and cortical bone mineral density, whereas ST-SPI diet only reduced c
296 AS concentrations were associated with lower bone mineral density, which varied according to the spec
299 t model, there is a large loss of trabecular bone mineral density without apparent proportional chang
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