ライフサイエンスコーパス: bone tissue

1 and enhance, the formation of de novo mature bone tissue.

2 important cellular and molecular aspects of bone tissue.

3 collagens and is expressed predominantly in bone tissue.

4 ession directly correlated with proximity to bone tissue.

5 development of cerebellar Purkinje cells and bone tissue.

6 e, the major mineral component of vertebrate bone tissue.

7 cus that modulates the mechanosensitivity of bone tissue.

8 als must be large in order to be anabolic to bone tissue.

9 gher in several tissues, including tumor and bone tissue.

10 r histological analysis of the treated human bone tissue.

11 t some part of the implant is not covered by bone tissue.

12 ft material for residual particle content in bone tissue.

13 suffer from a general loss of fat, lean, and bone tissue.

14 activity was associated with the subchondral bone tissue.

15 been reported to affect the regeneration of bone tissue.

16 o calculate an estimated distribution of the bone tissue.

17 he low proton density and fast decay time of bone tissue.

18 ell driver mutations needed for invading the bone tissue.

19 ercome the challenge of forming vascularized bone tissue.

20 mechanical loads distributed throughout the bone tissue.

21 with the repair and regeneration of missing bone tissue.

22 itulate this stepwise differentiation toward bone tissue.

23 h and Bmp4 synergize to promote expansion of bone tissue.

24 was identified in CXCR4-deficient cells and bone tissues.

25 y PTH in several osteoblastic cell lines and bone tissues.

26 l cells and the invasion of tumor cells into bone tissues.

27 s and quantification errors in the lungs and bone tissues.

28 antly more labile than in the case of mammal bone tissues.

29 jaw) nor pathology (healthy vs necrotic jaw bone tissue) affected the averaged spectral shape of the

30 istent level of approximately 10(7) CFU/gram bone tissue after day 7.

31 to restore the mechanical environment of the bone tissue after it has been perturbed by ovariectomy.

32 he pediatric atlas showed a reduced error in bone tissue and better delineation of bone structure.

33 PC-3MM2 cells growing adjacent to bone tissue and endothelial cells within these lesions e

34 is characterized by the presence of necrotic bone tissue and increased osteoclast activity.

35 sease that is characterized by overgrowth of bone tissue and is linked to mutations in the gene encod

36 RIN3 was expressed in bone tissue and its expression level was approximately 1

37 phorus and calcium signals representing hard bone tissue and sulfur distribution representing soft ti

38 pid, simultaneous visualisation of calcified bone tissue and the vasculature within the calcified bon

39 hat possess estrogen agonist-like actions on bone tissues and serum lipids while displaying potent es

40 nitors in the fetal BM contribute to nascent bone tissues and transient stromal cells that are replac

41 tatin (ATV) has shown pleiotropic effects on bone tissue, and osteoporosis can aggravate periodontiti

42 hyrin I isomer accumulation in erythrocytes, bone, tissues, and excreta and had fluorescent erythrodo

43 tion coefficient of 0.143 or 0.151 cm(-1) to bone tissue appears to give the best trade-off between b

44 Vascular malformations that occur inside bone tissue are rare.

45 Bone tissue arises from mesenchymal cells induced into t

46 dy is to analyze the tension distribution on bone tissue around implants with different angulations (

47 Stress patterns in the bone tissue around the implant were analyzed qualitative

48 ing, invasion, and growth of cancer cells in bone tissue as well as genes important for osteolysis, i

49 regulation of cell proliferation within the bone tissue as well as properties of the extracellular m

50 The loss of alveolar bone tissue associated with periodontal disease appears

51 suggest that incorrectly accounting for the bone tissue attenuation can lead to large underestimatio

52 and how the gene defects impact on skin and bone tissues besides than on the haematological compartm

53 ape immune surveillance and metastasize into bone tissue by inducing osteoclastic bone resorption.

54 Destruction of bone tissue by osteoclasts represents a severe pathologi

55 tor cells (BM-MSPCs) maintain homeostasis of bone tissue by providing osteoblasts.

56 enzyme phosphoglycerate kinase (PGK) inside bone tissue cells as a function of temperature from 38 t

57 More donor cells were found in bone tissues compared with other organs (P < .001), with

58 Alterations in bone tissue composition during osteoporosis likely disru

59 tical for crosslinking is reduced in proband bone tissue, consistent with decreased lysyl hydroxylase

60 aside from the joint pannus, the subchondral bone tissue constitutes an essential element in the deve

61 stigate the effect of biodegradation rate on bone tissue development in vivo.

62 treatment of the defect site with autologous bone tissue did not improve bone formation or defect bri

63 Unirradiated bone tissue displayed the presence of Mn(2+) ions at bot

64 l Runx2 for the formation of intramembranous bone tissues during embryogenesis.

65 hybrid system (CM-ALs) for drug delivery and bone tissue engineering application.

66 lymer, has established a good reputation for bone tissue engineering applications due to its many uni

67 For bone tissue engineering applications, tissue collection

68 ) (PLAGA) sintered microsphere scaffolds for bone tissue engineering applications.

69 esents an exciting property for their use in bone tissue engineering applications.

70 of graphene/nano-58S composite scaffold for bone tissue engineering applications.

71 el allows for evaluation of biomaterials and bone tissue engineering approaches within a reproducible

72 e is a growing need for the investigation of bone tissue engineering approaches within contaminated o

73 Current approaches in bone tissue engineering are restricted by delayed vascul

74 This study instituted a unique approach to bone tissue engineering by combining effects of mechanic

75 Although most in vivo work in the area of bone tissue engineering focuses on bone regeneration wit

76 Substantial progress has been made in bone tissue engineering in recent years, based on the re

77 A key tenet of bone tissue engineering is the development of scaffold m

78 ial enabling technology to translate generic bone tissue engineering methods into specific solutions

79 ng exploited together with growth factors as bone tissue engineering scaffolds regulating cell behavi

80 SORS) for nondestructive characterization of bone tissue engineering scaffolds.

81 ition, which has relevance for the design of bone tissue engineering strategies and may inform clinic

82 be used to develop therapeutic strategies in bone tissue engineering with numerable clinical applicat

83 For bone tissue engineering, a number of small animal models

84 nces in segmental bone defect animal models, bone tissue engineering, and drug delivery with the goal

85 njugated NDs to develop a novel platform for bone tissue engineering.

86 ted on hUCMSC encapsulation in scaffolds for bone tissue engineering.

87 al processes for implant osseointegration or bone tissue engineering.

88 CM-ALs (10%) scaffolds for drug delivery and bone tissue engineering.

89 be considered as an appealing candidate for bone tissue engineering.

90 al-control release systems that are used for bone tissue engineering.

91 25(OH)D3 has great potential for cell-based bone tissue engineering.

92 -induced pluripotent stem cells (hiPSCs) for bone tissue engineering.

93 e interest for their use as cell carriers in bone tissue engineering.

94 lly allow us to achieve the ultimate goal of bone tissue engineering: to reconstruct entire bones wit

95 with applications in cementitious materials, bone-tissue engineering, drug delivery and refractory ma

96 iation of MSCs with profound implications on bone-tissue-engineering applications.

97 the human bone core biopsies revealed normal bone tissue formation identical to the surrounding nativ

98 hanism, suggesting a new strategy to promote bone tissue formation in osteoporotic patients.

99 est a central role for WWOX in regulation of bone tissue formation.

100 er to mimic the natural microenvironment for bone tissue formation.

101 erentiation of stem and progenitor cells and bone tissue formation.

102 elevancy of these findings in infected human bone tissue from patients with S. aureus-associated oste

103 rmine occupancy of Cathepsin K inhibitors in bone tissues harvested from rabbit femurs.

104 Bone tissue has the capacity to adapt to its functional

105 tural and therapeutic effects for example in bone tissue healing and ageing.

106 ntly, cartilage ECM could not generate frank bone tissue if devitalized by standard "freeze & thaw" (

107 de (FF-XANES) at the calcium K-edge on human bone tissue in healthy and diseased conditions and for d

108 are found in hyaline cartilage in the adult, bone tissue in newborn mice, and osteoblasts and associa

109 anipulation of cancer cell interactions with bone tissue in real time.

110 GE was expressed at higher levels in healing bone tissues in diabetic compared to control animals.

111 to identify late stages of the disease, when bone tissue is affected.

112 ect PET quantification in these regions when bone tissue is ignored.

113 A paradox in bone tissue is that tissue-level strains due to animal a

114 G laser, despite producing thermal damage to bone tissue, is comparable to that with conventional dri

115 eover, aP2-Cre-mediated ACC1 inactivation in bone tissue led to a decreased number of osteoblasts but

116 In vitro, co-culture of PC3 cells with bone tissue led to activation of pro-MMP-9 and increases

117 The total numbers of soft-tissue and bone-tissue lesions, in a site-by-site comparison, were

118 ifferences in the fracture resistance at the bone tissue level.

119 e report the presence of endosteally derived bone tissues lining the interior marrow cavities of port

120 e 10 were distinct from BW, lean tissue, and bone tissue loci.

121 to CT-based attenuation maps for regions of bone tissue, lungs, and soft tissue.

122 at: 1) the oblique load was more damaging to bone tissue, mainly when associated with external hexago

123 he bone glue that acts as a scaffold between bone tissues matrix composition to bind them together an

124 d yield critical information on cellular and bone tissue mechanisms and translate to new mechanistic

125 mal mineralization of the collagen matrix of bone, tissue-nonspecific alkaline phosphatase (TNAP) is

126 in was determined in articular cartilage and bone tissue obtained from mice, rats, and human subjects

127 model, efficiently remodeled to form de novo bone tissue of host origin, including mature vasculature

128 tified: participants with loss of supporting bone tissue of less than one third of the root length (B

129 in <30% of teeth (BL), or loss of supporting bone tissue of one third or more of the root length in >

130 of the root length (BL-), loss of supporting bone tissue of one third or more of the root length in <

131 l-1 is increased approximately 4-fold in the bone tissues of GILZ transgenic (Tg) mice, and this incr

132 ion of MSCs into osteoblasts laying down new bone tissue on orthopedic implants.

133 eural crest development: 1) disagreements in bone tissue origin within and across current model syste

134 In healthy bone tissue, osteocytes experience higher maximum strain

135 to assess chemical properties of Ca in human bone tissue our data suggest that neither the anatomical

136 ficantly higher stress concentrations in the bone tissue (P <0.05) compared with the tapered connecti

137 rved an almost complete normalization of all bone tissue parameters, using radiographic, microcompute

138 in single amino-acid position for particular bone tissue pathology.

139 Net MMP-9 activity in bone tissues peaked 2 weeks after injection, coinciding

140 rkca(-/-) female but not male mice, in which bone tissue progressively invades the medullary cavity i

141 hymal stromal cells (MSCs) could be used for bone tissue regeneration as tissue engineered periosteum

142 The goal of this study was to improve bone tissue regeneration by using targeted GNPs.

143 Biomaterials for bone tissue regeneration represent a major focus of orth

144 Although bone tissue regeneration strategies involve culture of b

145 ters and have the potential to inform future bone tissue regeneration strategies that can optimize th

146 special attention for their ability to guide bone tissue regeneration through structural and biologic

147 t that OA is a promising bioactive agent for bone tissue regeneration, and inhibition of Notch signal

148 ems can develop new strategies for improving bone tissue regeneration.

149 patterns (spatial) and trends (temporal) of bone tissue regeneration.

150 nthetic, off-the-shelf, cell-free option for bone tissue repair and restoration.

151 congenital defects that require large-scale bone tissue repair have few successful clinical therapie

152 ced the host cells to coordinate and promote bone tissue repair through paracrine effects.

153 opological optimization for designing facial bone tissue replacements might improve current clinical

154 aurs, and in light of evidence that dinosaur bone tissue resembles the histology in mammals, the hist

155 f interest inside the lung, soft tissue, and bone tissue, respectively.

156 Bone tissues respond to mechanical loading/unloading reg

157 d the HyA staining of osteocytes in cortical bone tissue sections to the extent that the lacunocanali

158 ing with decreased numbers of osteoclasts in bone tissue sections.

159 Rather, microscopic analyses of the long-bone tissues show that dinosaurs grew to their adult siz

160 Intravital two-photon imaging of bone tissues showed that a potent S1P(1) agonist, SEW287

161 ntal ligament with Sharpey's fibers, and new bone tissue similar to native periodontal tissues.

162 tors are key regulators of hematopoietic and bone tissue-specific gene expression.

163 lated transcriptional regulators to suppress bone tissue-specific genes during proliferative stages o

164 Developmental control of bone tissue-specific genes requires positive and negativ

165 3 (vitamin D)-induced transactivation of the bone tissue-specific osteocalcin gene.

166 Osteocalcin (OC) is known to be a bone tissue-specific protein, expression of which is bel

167 Bone tissue specimens were examined by light microscopy

168 antler, form and change rapidly, while other bone tissues, such as human tooth dentine, develop slowl

169 presence of apolipoprotein in demineralized bone tissue suggest the possibility that these particles

170 naling is adequate for mineralization of the bone tissue that does form.

171 erized by low bone mass and deterioration of bone tissue that leads to bone fragility and an increase

172 ed to distinct diseases involving adipose or bone tissue, the metabolic syndrome, and osteogenesis im

173 orders of magnitude below those that damage bone tissue, this anabolic, non-invasive stimulus may ha

174 The generation and homeostasis of bone tissue throughout development and maturity is contr

175 into the bone microenvironment, and destroy bone tissue to allow for tumor growth.

176 ations depends on the ability of surrounding bone tissue to integrate with the surface of the device,

177 ometry were used to determine lean, fat, and bone tissue traits in a F(2) mouse population from a C57

178 ion within bone and determines tumor-induced bone tissue transformation.

179 ed side effects and be clinically useful for bone tissue transplantation.

180 s studies have demonstrated that engineering bone tissue using mesenchymal stem cells (MSCs) is feasi

181 tic method to estimate the mu map, including bone tissue using only MR information, is presented.

182 ossible to estimate the anatomic location of bone tissue using UTE sequences.

183 ry subtle spectral variations related to the bone tissue variations itself.

184 the dynamic bone structure, showing reduced bone:tissue volume ratio and trabecular number in FVIIIK

185 New bone tissue was formed that grew in mass and cellularity

186 -16% and -11% occurred for methods in which bone tissue was ignored (SEG1 and SEG2).

187 Bone tissue was implanted into established pouches on BA

188 al ligament at the coronal aspect of the new bone tissue was similar in the smaller lesions between t

189 Sharpey's fibers, periodontal ligament, and bone tissue were formed far above the notch placed at th

190 Sharpey's fibers, periodontal ligament, and bone tissue were formed far coronal to the notch at the

191 that contained BMP-2, similar volumes of new bone tissue were formed; however, the faster degrading h

192 ability of the radiation-induced radicals in bone tissue were investigated by means of both isotherma

193 ntal ligament with Sharpey's fibers, and new bone tissue were observed.

194 of irradiated frog Limnonectes macrodon leg bones tissue were studied by electron paramagnetic reson

195 scaffolds can be modulated to form humanized bone tissue, which was also able to support human HSC en

196 ow bone mass and structural deterioration of bone tissue with an increased susceptibility to fracture

197 t means of obtaining high-resolution maps of bone tissue with sufficient anatomic accuracy for, for e

198 -expressing BMSSCs (BMSSC-Ts) generated more bone tissue, with a mineralized lamellar bone structure

199 r mesenchymal cells organize into trabecular bone tissue within the artery wall.