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1 surement of serum and urine telopeptides and bone-specific alkaline phosphatase.
2 markers of skeletal turnover osteocalcin and bone-specific alkaline phosphatase.
3 entrations of intact parathyroid hormone and bone-specific alkaline phosphatase also varied in a sinu
4 Biochemical markers of bone turnover such as bone-specific alkaline phosphatase and N-terminal telope
5                                        Serum bone-specific alkaline phosphatase and osteocalcin conce
6                                        Serum bone-specific alkaline phosphatase and osteocalcin decre
7 ts of biochemical markers of bone formation (bone-specific alkaline phosphatase and osteocalcin) and
8 ing markers, including serum osteocalcin and bone-specific alkaline phosphatase and urine N-telopepti
9 ing protein III, and bone formation markers, bone-specific alkaline phosphatase, and osteocalcin.
10 eukin (IL)-1beta, osteoprotegerin (OPG), and bone-specific alkaline phosphatase (BALP) serum levels w
11     Leukogram, liver and kidney enzymes, and bone-specific alkaline phosphatase (BALP) serum levels w
12     Blood samples were collected to evaluate bone-specific alkaline phosphatase (BALP), leukogram, an
13 treatment, urinary N:-telopeptides and serum bone-specific alkaline phosphatase (BAP) were measured.
14 vitamin D, parathyroid hormone, osteocalcin, bone-specific alkaline phosphatase (BAP), procollagen I
15 n, N:-telopeptides of type I collagen (NTx), bone-specific alkaline phosphatase (BSAP), and phylloqui
16 e of type 1 collagen, and 28.1% (P<.001) for bone-specific alkaline phosphatase, but after 5 years wi
17 %ucOC was positively associated with NTx and bone-specific alkaline phosphatase concentrations (P < 0
18  upfront group, mean serum N-telopeptide and bone-specific alkaline phosphatase concentrations decrea
19 T resulted in significant decreases in serum bone-specific alkaline phosphatase levels (mean change,
20                        Serum osteocalcin and bone specific alkaline phosphatase, markers of skeletal
21  affecting parathyroid hormone, osteocalcin, bone-specific alkaline phosphatase, or tartrate-resistan
22 %-15%), 18% (13%-27%), and 14% (10%-21%) for bone-specific alkaline phosphatase, osteocalcin, and uri
23 henytoin had significantly greater levels of bone-specific alkaline phosphatase (p = 0.007).
24 (2); P=0.30) and bone biomarker measurements-bone-specific alkaline phosphatase (soy-fed: 82.3 +/- 4.
25 itive for osteocalcin and cells positive for bone-specific alkaline phosphatase were detected in the
26 1 collagen (NTx) breakdown, osteocalcin, and bone-specific alkaline phosphatase were measured to refl
27 m markers of bone formation (osteocalcin and bone-specific alkaline phosphatase) were lower during re
28 ventions; however, a significant increase in bone-specific alkaline phosphatase, which is a bone-form

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