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1 cid (1) was synthesized for potential use in boron neutron capture therapy.
2 eliver boronated EGF to EGFR (+) gliomas for boron neutron capture therapy.
3 o, and investigated as a potential agent for boron neutron capture therapy.
4 somal delivery and subsequent application in boron neutron capture therapy.
5 and BPA, and, subsequently, the efficacy of boron neutron capture therapy.
6 interesting templates as carriers of 10B for boron neutron capture therapy.
7 l-down, inhibition of hydrolytic enzymes and boron neutron capture therapy.
8 lored potential in traditional areas such as Boron Neutron Capture Therapy agents, but also as pharma
10 ligonucleotides are potential candidates for boron neutron capture therapy, antisense technology, and
11 e of sufficient amounts of 10B for effective boron neutron capture therapy ( approximately 10(8)-10(9
16 ntan-1-yl] thymidine, designated N5-2OH, for boron neutron capture therapy (BNCT) of brain tumors usi
21 rborane 2 was also tested as a candidate for boron neutron capture therapy, but showed poor tumor ret
22 ses an exceptional challenge, because viable boron neutron-capture therapy by this method will requir
23 udy was to determine whether the efficacy of boron neutron capture therapy could be enhanced by means
26 nophenylalanine has been applied in clinical boron neutron capture therapy for the treatment of high-
31 unoprotein-based targeting strategies to the boron neutron-capture therapy of cancer poses an excepti
33 ic boron cluster system, previously used for boron neutron capture therapy, was modified by the addit
34 use of boronated EGF as a delivery agent for boron neutron capture therapy, which is based on the cap
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