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1 well as the only case that was treated with botulinum toxin A.
2 ridgewater, NJ) and periocular injections of botulinum toxin A.
3 or when vesicular release was inhibited with botulinum toxin A.
4 roposal as third line for strong opioids and botulinum toxin A.
5 adverse events associated with injection of botulinum toxin A.
6 light chain, the metalloprotease domain, of botulinum toxin A.
7 fficacy and safety of one-time injections of botulinum toxin A (200 to 240 units) in 126 subjects wit
8 nts were randomized to receive intralesional botulinum toxin A (5 U per 1 cm2) or equivalent volumes
10 ghly concentrated, unlicensed preparation of botulinum toxin A and may have received doses 2857 times
11 as no significant association between use of botulinum toxin A and reduction in the number of episodi
12 enterotoxins A and B, cholera toxin, ricin, botulinum toxin A, and heat labile toxin of E. coli).
13 staphylococcal enterotoxin A, cholera toxin, botulinum toxin A, and ricin in model buffer (PBS-BSA) a
14 ococcal enterotoxins A and B, cholera toxin, botulinum toxin A, and ricin increased 2- to 5-fold, whi
15 tidepressants, strong opioids, tramadol, and botulinum toxin A, and undetermined for lidocaine patche
19 ical trial to compare topical diltiazem with botulinum toxin A (BTA) in the treatment of chronic anal
21 bjects were pretreated, subcutaneously, with botulinum toxin A (BTX-A) to inhibit the release of neur
23 demonstrate that clinical use of unlicensed botulinum toxin A can result in severe, life-threatening
25 ion of the light chains of tetanus toxin and botulinum toxin A did not disrupt the restricted motion
26 oteins play a role in short-term plasticity, Botulinum toxins A, E, and F, were used to disrupt SNARE
31 d limb use by the intramuscular injection of botulinum toxin A into selected forelimb muscles to prod
32 oaded pocketed microneedle device to deliver botulinum toxin A into the human dermis with the aim of
33 ar chemodenervation agents (alcohol, phenol, botulinum toxin A), intrathecally administered drugs (ba
37 by treating organotypic cultures of SCN with botulinum toxin A or dynasore to block exocytosis and en
39 shown that proteolytic cleavage of SNAP25 by botulinum toxin A reduces the ability of Gbetagamma to c
41 rmed at P7 for WT, cKO, and muscle-unloaded (botulinum toxin A treated) attachments for quantitative
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