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1 tabolic diseases, including diabetes, and in bowel cancer.
2 nflammatory drugs have reduced risk of large bowel cancer.
3 d tumors which are now the most common small bowel cancer.
4 se patients continue to have a small risk of bowel cancer.
5 adenomas, which are precursors of most large-bowel cancers.
6 se, 23 subsequently died (disseminated large bowel cancer, 12; unrelated causes, 9; related causes, 2
7 was inversely related to incidence of large bowel cancer (adjusted relative risk 0.75 [95% CI 0.59-0
10 od supports the theory that FAP and sporadic bowel cancer follow the same genetic pathway and are sep
11 intakes of different PUFAs and distal large bowel cancer in a population-based case-control study of
12 association between NSAIDs and distal large bowel cancer in African Americans and whites, using data
15 associated with reduced risk of distal large bowel cancer in whites (multivariable odds ratios = 0.88
16 e was inversely associated with distal large bowel cancer in whites (odds ratio (OR) = 0.60, 95% conf
17 sociated with increased risk of distal large bowel cancer in whites, but not among African Americans
18 gent fluoropyrimidine adjuvant therapy after bowel cancer resection include intravenous fluorouracil
19 nd 95% confidence intervals for distal large bowel cancer risk in relation to quartiles of PUFA intak
20 acids was inversely related to distal large bowel cancer risk, whereas the ratio of omega-6 to long-
21 od samples were taken from patients from the Bowel Cancer Screening Programme (asymptomatic but faeca
23 ed incidence of colorectal cancer, uptake of bowel cancer screening varies across demographic groups.
24 The highest risks were found for male small bowel cancer (SIR, 251; 95% CI, 177 to 346; CR at 70 yea
27 ents with excluded bowel tumor died of large bowel cancer within 2.4 years; by contrast, the actuaria
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