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1 onal innervation of the smooth muscle of the bowel wall.
2 long distances between the high tie and the bowel wall.
3 rated network of neurons and glia within the bowel wall.
4 s of neurite extension within the developing bowel wall.
6 expression than in carcinomas limited to the bowel wall (3.4 [2.1-5.9] vs 1.9 [1.7-2.4], p=0.007), an
7 ry findings (n = 4), mesenteric hematoma and bowel wall abnormality (n = 2), mesenteric hematoma only
10 orrelations between VAS score and MR imaging bowel wall arterial phase enhancement after contrast mat
13 not only of inflammatory involvement of the bowel wall but also in terms of how the bowel in its tur
14 g of maximal enhancement of the normal small-bowel wall by using contrast material-enhanced multi-det
15 uld differentiate inflammatory from fibrotic bowel wall changes in both animal models of colitis and
16 e (4.9 +/- 0.1 vs 4.6 +/- 0.1, P: <.005) and bowel wall conspicuity (4.6 +/- 0.2 vs 4.2 +/- 0.2, P: <
17 roved reader confidence in the assessment of bowel wall conspicuity and the ability of CT colonograph
19 from intraluminal bacterial gas entering the bowel wall due to increased mucosal permeability caused
21 P < .0001), arterial signal (P = .0005), and bowel wall echotexture (P < or = .0200) between patients
22 ssociated splanchnic circulation congestion, bowel wall edema, and impaired intestinal barrier functi
24 lation in the multivariate analysis: reduced bowel wall enhancement (odds ratio, 7.8; 95% confidence
26 oves the diagnostic performance of decreased bowel wall enhancement as a sign of ischemia complicatin
27 ild-reported VAS score and (a) the degree of bowel wall enhancement in the arterial phase at contrast
28 as between change in VAS score and change in bowel wall enhancement in the arterial phase at contrast
29 adding unenhanced images improved decreased bowel wall enhancement sensitivity (observer 1: 46.3% [1
30 lar contrast medium allowed visualization of bowel wall enhancement that was obscured by intraluminal
32 ore that combines three CT findings (reduced bowel wall enhancement, a closed-loop mechanism, and dif
36 s that include soft-tissue thickening of the bowel wall, free fluid, periintestinal soft-tissue stran
38 f intestinal epithelial cells may compromise bowel wall integrity and be a mechanism for bacterial or
39 n abnormally enhanced, thin mucosal layer of bowel wall involving fluid-filled, dilated, poorly opaci
40 l factor (SCF), but the source of SCF in the bowel wall is unclear and controversy exists about wheth
41 gnosed when the cancers are localized to the bowel wall, it is likely that widespread implementation
49 -selectin (31.2% +/- 25.7) in vessels in the bowel wall of segments with ileitis were higher than in
50 e hand, and of the muscularis propria of the bowel wall on the other, it might be valid to consider i
56 ysician in consensus evaluated the following bowel wall PET/MR enterography biomarkers: signal intens
57 tents that breach the mucosal barrier of the bowel wall, resulting in granuloma formation and chronic
63 mputed tomography scan results demonstrating bowel wall thickening (>5 mm) and/or fat stranding consi
64 ome PI vs percentage of benign): soft-tissue bowel wall thickening (51.2% vs 13.3%, P = .0167), free
65 appendageal fat stranding (n = 10), adjacent bowel wall thickening (n = 4) or compression (n = 2), an
66 signs of small bowel inflammation were fund: bowel wall thickening (n=21), submucosal edema (n=8), se
67 large amount of retained stool in the colon, bowel wall thickening and infiltration of peri-colonic f
68 Hospitalization for diarrhea associated with bowel wall thickening occurred in 5.5% of the patients r
69 should undergo evaluation of the abdomen for bowel wall thickening of >4 mm, the hallmark of NEC.
72 r risk of GIP was observed for patients with bowel wall thickening or bowel obstruction on CT scan.
73 itoneal thickening or enhancement, and small-bowel wall thickening or distortion demonstrated positiv
78 ed tomography of her abdomen showed diffused bowel wall thickening without evidence of bowel obstruct
80 imaging findings of these episodes included bowel wall thickening, lymphadenopathy, and focal masses
81 and associated findings such as soft-tissue bowel wall thickening, periintestinal soft-tissue strand
85 using the Limberg index on the basis of (a) bowel wall thickness and (b) size and extent of Doppler
86 P-selectin-targeted US imaging, by measuring bowel wall thickness and perfusion, and by using a clini
87 Patients with intestinal murmurs had greater bowel wall thickness of the sigmoid and descending colon
91 susception and the inner fat core, the outer bowel wall thickness, and the presence or absence of lym
94 difference between inflammatory and ischemic bowel wall thicknesses was not significant (P = .49).
95 e mean time to peak enhancement of the small-bowel wall was 49.3 seconds +/- 7.7 (standard deviation)
97 hat is, diverticulae) due to weakness in the bowel wall, which can become infected and inflamed causi
98 ctic acid level secondary to ischemia of the bowel wall with CT scan findings aid in establishing the
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