ライフサイエンスコーパス: brain MRI

1 from 135 high risk patients with CIS (>/= 2 brain MRI lesions and >/= 2 oligoclonal bands) enrolled

2 A total of 2,362 3T brain MRI scans were acquired from 469 subjects.

3 T1-weighted 3T brain MRI data of 15 patients suffering from motor CD (n

4 sual attention task (n = 35; phase 2b) and a brain MRI after traumatic brain injury (n = 23; phase 2c

5 Each patient had a brain MRI scan at entry and 6 months later using a stand

6 ldren were clinically assessed and 351 had a brain MRI scan.

7 nd in 5 of 29 patients (17%) who underwent a brain MRI.

8 volving the brain; 30% of cases had abnormal brain MRI at onset and 75% by follow up.

9 ns in relapse rate and development of active brain MRI lesions, measures considered to reflect CNS in

10 ma which was a radiological suggestion after brain MRI).

11 Although brain MRI findings are not characteristic for ephedrone

12 g multicenter neuroimaging data, we analyzed brain MRI scans from 2028 schizophrenia patients and 254

13 Anatomic brain MRI scans were obtained from 15 psychiatrically he

14 Thirteen healthy children for whom anatomic brain MRI scans were obtained every 2 years, for 8-10 ye

15 Anatomic brain MRIs for 57 boys with ADHD and 55 healthy matched

16 Anatomical brain MRI was performed at baseline and follow-up in 19

17 lgorithmically calculated for 108 anatomical brain MRI scans from 50 patients (20 of whom were female

18 In a cross-sectional design, 3 T anatomical brain MRI was acquired in 27 medication-free youth with

19 itive testing, stool microbiota analysis and brain MRI analysis.

20 , and time interval between abdominal CT and brain MRI.

21 underwent neuropsychological evaluation and brain MRI at exam 7.

22 the evaluation of cognition and memory, and brain MRI or FDG-PET abnormalities less frequently restr

23 Neuropsychological and brain MRI variables were related to cardiac MRI-assessed

24 Head CT scan and brain MRI showed a huge intra-axial right temporo-pariet

25 (t-tau), phosphorylated tau 181 (p-tau) and brain MRI examination.

26 MR angiography in TOF technique and brain MRI in T1- and T2-weighted images, FLAIR and DWI s

27 examination, neuropsychological testing and brain MRI.

28 f FBDS alone, patients had normal sodium and brain MRIs, but electroencephalography demonstrated icta

29 Patients with MS also underwent annual brain MRI scans.

30 rs completed neurocognitive testing, another brain MRI, and their parents completed neurobehavioural

31 ment, gait and balance assessment as well as brain MRI.

32 This was a retrospective study assessing brain MRI of 26 patients with natalizumab-associated PML

33 matoma evacuation, or autopsy) and available brain MRI sequences of adequate quality, including T2-we

34 ions sharing the "molar tooth sign" on axial brain MRI, together with cerebellar vermis hypoplasia, a

35 ischaemic stroke (n = 100) were assessed by brain MRI at 3 T including diffusion weighted imaging.

36 ite matter hyperintensities were assessed by brain MRI.

37 level, myopathic electrodiagnostic changes, brain MRI with cobblestone complex, and mutation in the

38 of childhood-onset chorea and characteristic brain MRI showing symmetrical bilateral striatal lesions

39 Clinical brain MRI is normal in the majority of patients with ant

40 s (161 females; 48 males) underwent clinical brain MRI and were genotyped for the BDNF rs6265 Val66Me

41 We collected brain MRI scans from 615 healthy young adult twins and s

42 blinded standardised review of conventional brain MRIs of 30 patients with MS, 31 patients with LHON

43 A blinded review of conventional brain MRIs shows that patients with LMS have a scan appe

44 All three subjects presented a distinctive brain MRI pattern characterized by cavitating leukodystr

45 normal admission head CT had abnormal early brain MRI.

46 In addition to admission head CT, early brain MRI was performed 12 +/- 3.9 days after injury.

47 test in routine practice, and gradient echo brain MRI will identify all symptomatic cases.

48 ctroencephalography (EEG), electromyography, brain MRI, CSF analysis, or a combination of these analy

49 s were also assessed by electroretinography, brain MRI and magnetic resonance spectroscopy (MRS), and

50 These patients also had gadolinium enhanced brain MRI scans and were divided into relapsing-remittin

51 Participants had clinical evaluations, brain MRI, and blood draws annually.

52 f this atlas and additional individual fetal brain MRI atlases for completely automatic multi-atlas s

53 automatic multi-atlas segmentation of fetal brain MRI.

54 For example, shape measurements derived from brain MRI scans are multidimensional geometric descripti

55 We present detailed findings from brain MRI in three mutation-positive individuals.

56 ar bundle RNFL correlated with higher global brain MRI lesion burden index (R(2) = 0.35, P = .001) an

57 , 50 HE subjects, and 43 AD/MCI patients had brain MRI and PiB PET.

58 brain atrophy was found in 12 of 14 who had brain MRI.

59 ork obtained from tractography of 14 healthy-brain MRIs.

60 underwent (i) clinical evaluations; and (ii) brain MRI scans analysed using whole-brain voxel-based m

61 ssion of signal intensity changes visible in brain MRI is not associated with clinical condition impr

62 Initial brain MRI revealed multiple, disseminated lesions that w

63 Initial brain MRI was abnormal in 54 (67%).

64 article presents clinical symptoms, initial brain MRI findings and characteristics of changes observ

65 Wechsler Abbreviated Scale of Intelligence), brain MRI, medical records, and structured interviews wi

66 een 2002 and 2006, more than 26 years later, brain MRIs were performed.

67 year period, based on analyzing longitudinal brain MRIs from 12 COS patients and 12 healthy controls

68 ogressive multiple sclerosis who had monthly brain MRI studies for 4 months (one baseline and three f

69 nium-enhanced T1-weighted lesions on monthly brain MRI scans from weeks 8 to 24.

70 aging studies (consisting of whole-body MRI, brain MRI, breast MRI, mammography, abdominal and pelvic

71 The evolution of multimodal brain MRI demonstrates that neuroimaging findings of tis

72 eurodevelopmental outcome we used multimodal brain MRI to study a large cohort of preterm infants.

73 (infantile encephalopathy and largely normal brain MRI) to that of NALCN-related infantile encephalop

74 bellum as revealed by volumetric analysis of brain MRI scans.

75 tients were highly correlated with degree of brain MRI involvement.

76 Re-evaluation of brain MRI images of four individuals showed a shared dis

77 Results of brain MRI were abnormal in 18 (31.6%) of 57 patients: 8

78 avity, assessed by blinded central review of brain MRI scans by the study neuroradiologist in the mod

79 rom JXG with CNS involvement and the role of brain MRI including DWI and PWI in the evaluation of bra

80 Over the past two decades, thousands of brain MRI scans from healthy youth and those with neurop

81 Prevalence of abnormalities on brain MRI by category of finding (no referral necessary,

82 rmine the long-term risk of abnormalities on brain MRI for the development of multiple sclerosis and

83 anges and subtle structural abnormalities on brain MRI.

84 are inversely associated with MS activity on brain MRI.

85 ncluding ataxia and/or cerebellar atrophy on brain MRI.

86 Here, based on brain MRI, we apply a 3D surface-based morphometry metho

87 of microbleeds were assessed at baseline on brain MRI of 4759 participants aged >/=45 years.

88 ive impairment and a thin corpus callosum on brain MRI.

89 teral involvement of the internal capsule on brain MRI was associated with poorer survival [20 +/- 18

90 between groups and no significant effects on brain MRI measures were noted in either group (mean T2 l

91 We found that ependymitis on brain MRI was the best predictor of higher log(sCD27) le

92 rintensity (WMH) burden is commonly found on brain MRI among patients with atrial fibrillation (AF).

93 f imaging characteristics of inflammation on brain MRI scans of inflammatory NTZ-PML patients.

94 tural, hippocampal, or neocortical lesion on brain MRI is not always indicative of the site of seizur

95 he number of gadolinium-enhancing lesions on brain MRI scans for both RRMS studies.

96 gadolinium-enhancing T1-weighted lesions on brain MRI, clinical relapses, and disability (Expanded D

97 inium contrast-enhancing lesions measured on brain MRI scans every 4 weeks between weeks 8 and 24.

98 he highly characteristic molar tooth sign on brain MRI.

99 and two of the following criteria: positive brain MRI; positive spinal cord MRI; or positive cerebro

100 Pre- and postprocedural brain MRI was performed on each patient within a week of

101 tal heart disease who underwent preoperative brain MRI as part of 2 separate prospective studies.

102 mean number of lesions noted on pretreatment brain MRI scans.

103 We performed a comparative prospective brain MRI study in patients with COS and pediatric patie

104 nsity on fluid-attenuated inversion recovery brain MRI within a vascular distribution.

105 able partial epilepsy, where high-resolution brain MRI disclosed a unilateral, focal, hippocampal, or

106 With high-resolution brain MRI scans, we created composite maps of cortical g

107 d onto a surface rendering of each subject's brain MRI.

108 Serial brain MRI scanning is widely used for assessing multiple

109 nts with childhood-onset schizophrenia share brain MRI abnormalities with the patients that may follo

110 High field structural brain MRI images were acquired in a 3T scanner and analy

111 ased on measurements derived from structural brain MRI scans.

112 brain imaging studies, including structural brain MRI, magnetoencephalography and transcranial magne

113 spaces, brain atrophy) as seen on structural brain MRI and of a global SVD score on the patients' per

114 er and gray matter on 10 different synthetic brain MRI images corrupted with 18 different combination

115 ative and quantitative neuroimaging with 3-T brain MRI and optical coherence tomography.

116 rial Fibrillation (MACPAF) study, serial 3-T brain MRIs and neuropsychological assessment were perfor

117 matched control subjects underwent a 7-Tesla brain MRI scan and a detailed cognitive assessment.

118 The brain MRI images of these patients were evaluated.

119 This study suggests that the brain MRI criteria for differentiating RRMS from NMOSD a

120 acute or subacute neurological symptoms; the brain MRIs indicating diffuse brain damage.

121 Thus, brain MRI findings were normal in the majority of patien

122 brain swelling or focal changes according to brain MRI.

123 hort (mean age 39.2 years, SD 8.4) underwent brain MRI between June, 2009 and June, 2010.

124 troke or transient ischemic attack underwent brain MRI in 1992 and 1993, 1 to 2 years before echocard

125 ons centered on 4 Canadian cities, underwent brain MRI and simple tests of cognition and gait as part

126 imia nervosa and 8 female controls underwent brain MRI followed by (11)C-carfentanil PET.

127 group of 16 healthy term newborns underwent brain MRI at term-equivalent age.

128 e symptoms within 6 hours of onset underwent brain MRI followed by noncontrast CT.

129 ation carriers and 10 NC relatives underwent brain MRI and clinical assessment.

130 All of them underwent brain MRI with the use of 1.5T scanners.

131 Material/Patients with IIH who underwent brain MRI and contrast-enhanced MR venography before mea

132 ed at age 11 and 79 years, and who underwent brain MRI.

133 (47% males; range, 1 to 11 yr) who underwent brain MRI.

134 ~70% of young carrier parents who underwent brain MRI.

135 participants, aged 65 to 84 years, underwent brain MRI including DWI at baseline.

136 We used brain MRI or post-mortem examination in 351 fullterm inf

137 ry and 30 age-matched healthy controls using brain MRI.

138 AT, and VAT, they had undergone a volumetric brain MRI scan with measurements of total brain volume (

139 SM-IV) based history of enuresis, volumetric brain MRI scans and neuropsychological testing were obta

140 dementia-free survivors underwent volumetric brain MRI between 1999 and 2005, approximately 7.7 years

141 ce models were extracted from 82 T1-weighted brain MRI scans (256 x 192 x 124 volumes) of 42 subjects

142 w-up on 81 such patients who had T2-weighted brain MRI at presentation.

143 nium-enhanced and proton density/T2-weighted brain MRI from months 0 to 6 and 18 to 24 to determine t

144 nt lesions of at least 3 mm on a T2-weighted brain MRI scan, and an Expanded Disability Status Scale

145 Patients also underwent T1- and T2-weighted brain MRI.

146 e determined the contribution of these whole brain MRI markers to cognitive impairment in SVD.

147 nts into the brain of mice for in vivo whole brain MRI.

148 We report a whole-brain MRI morphometric survey of asymmetry in children w

149 pting to correlate locomotor disability with brain MRI findings.

150 der individuals with WMH were evaluated with brain MRI and detailed clinical and neuropsychological a

151 This article addresses findings with brain MRI that may underlie cognitive dysfunction in dia

152 ted in 4 sites; 60 patients were imaged with brain MRI after TAVR.

153 tes with CHD were studied prospectively with brain MRI: before surgery, within 2 weeks of surgery, an

154 a term applied to asymptomatic subjects with brain MRI abnormalities highly suggestive of multiple sc

155 f two sets of CTX patients, with and without brain MRI evidence of DN involvement, with a set of heal

156 f prospective follow-up that included yearly brain MRI and biannual clinical visits (n = 219).