ライフサイエンスコーパス: brain abscess

1 remature infant with sepsis, meningitis, and brain abscess.

2 crophorum who developed a right frontal lobe brain abscess.

3 agent of CNS parenchymal infections, such as brain abscess.

4 e part of the anaerobic community that cause brain abscess.

5 tion between innate and adaptive immunity in brain abscess.

6 was unsuccessfully treated for a C. bantiana brain abscess.

7 ormed as the method of choice for confirming brain abscesses.

8 a prospective nationwide study on bacterial brain abscesses.

9 e establishment of spontaneous polymicrobial brain abscesses.

10 cerebrally infected with S. aureus to induce brain abscesses.

11 tis, 60 (4%) had hemorrhages, and 2 (1%) had brain abscesses.

12 n reveal inflammatory changes and developing brain abscesses.

13 munity-acquired pneumonia (CAP) and multiple brain abscesses.

14 uable method to identify bacterial agents of brain abscesses.

15 -resistant mold, was cultured from bilateral brain abscesses.

16 ich often progresses to establish multifocal brain abscesses.

17 delta T cells were also a source of IL-17 in brain abscesses.

18 c central nervous system infection including brain abscesses.

19 filled macrophages within the ventricles and brain abscesses.

20 ight frontal lobe, which could correspond to brain abscesses.

21 plantation, and who later developed multiple brain abscesses.

22 sions, malignancy, or non-Aspergillus fungal brain abscesses accounted for all such lesions.

23 nimals consistently demonstrated more severe brain abscesses and higher CNS bacterial burdens compare

24 ation, ciglitazone may effectively sequester brain abscesses and limit bacterial dissemination.

25 g to the anaerobic community responsible for brain abscess, and M. oralis may participate in the path

26 Brain abscesses are very rarely diagnosed in neonates, b

27 Brain abscesses arise following parenchymal infection wi

28 d a well-characterized model of experimental brain abscess as a tool to query effects of the CNS infl

29 of polymicrobial infections, using bacterial brain abscesses as an example.

30 e describe the recovery of M. hominis from a brain abscess associated with a postpartum infection.

31 rred in a left lobe recipient, from a fungal brain abscess at 90 days.

32 Evaluation of brain abscesses by immunohistochemistry and flow cytomet

33 In neonates, brain abscesses can be detected on transfontanelle ultra

34 sting that a population of cells forming the brain abscess capsule originate from a bone marrow precu

35 The case of a patient who presented with a brain abscess caused by Streptomyces infection following

36 rt the case of a previously healthy boy with brain abscesses caused by M/emm type 12 GAS and review t

37 Murine Staphylococcus aureus-mediated brain abscess comprises 2 major phases, an initial phase

38 sion of select inflammatory mediators during brain abscess development including inducible NO synthas

39 Brain abscess development is orchestrated by IL-12 at di

40 d for its ability to influence the course of brain abscess development when treatment was initiated 3

41 majority of the cellular infiltrate in early brain abscess development, subsequent analysis focused o

42 he contribution of virulence determinants in brain abscess development, the abilities of S. aureus st

43 CNS host response during the early stages of brain abscess development, whereas MyD88-independent pat

44 of acquiring fibrotic characteristics during brain abscess development.

45 l for limiting pathogen dissemination during brain abscess development.

46 -toxin is a central virulence determinant in brain abscess development.

47 We report the first documented case of brain abscess due to the dematiaceous fungus Microascus

48 ly restricted, since all previous reports of brain abscesses due to this organism have been for patie

49 is the extremely high propensity to initiate brain abscesses during neonatal meningitis.

50 Unexpectedly, ciglitazone also accelerated brain abscess encapsulation, which was typified by the h

51 Brain abscesses form in response to a parenchymal infect

52 e of central nervous system involvement with brain abscess formation in a patient with chronic granul

53 ammatory cytokine or chemokine expression or brain abscess formation in vivo.

54 continued innate responses during late-stage brain abscess formation is not known but is important, b

55 and elicits chronic infection, resulting in brain abscess formation.

56 ic resonance imaging technology to visualize brain abscess formation.

57 re unique in their frequent association with brain abscess formation.

58 iminated 2 distinct bacterial populations in brain abscess from dental and sinusal origin.

59 nervous system bacterial infections, such as brain abscess, has not yet been investigated.

60 ion, the influx of fibrocyte-like cells into brain abscesses immediately preceded the onset of fibrot

61 thogenesis of S. aureus-induced experimental brain abscess in TLR2 knockout (KO) and wild-type (WT) m

62 reus is one of the major etiologic agents of brain abscesses in humans, occasionally leading to focal

63 aureus, one of the main etiologic agents of brain abscesses in humans.

64 Empiric therapy of brain abscesses in liver transplant recipients should in

65 showed that C. koseri causes meningitis and brain abscesses in the neonatal rat model, and we utiliz

66 Streptococcus intermedius, a common cause of brain abscesses, in both CSF samples as well as in the f

67 cribe a case of polymicrobial infection in a brain abscess including two rapidly growing Mycobacteriu

68 s of Citrobacter spp. causing meningitis and brain abscess is not well characterized; however, as wit

69 The bacterial flora involved in brain abscess is often complex.

70 M. cinereus was isolated from brain abscess material from a bone marrow transplant rec

71 enesis of C. freundii causing meningitis and brain abscess may relate to invasion of and intracellula

72 We have developed a mouse brain abscess model by using Staphylococcus aureus, one

73 and chemokine expression in an experimental brain abscess model in the rat during the acute stage of

74 e modulatory effects in a mouse experimental brain abscess model, we found that minocycline significa

75 sis of S. aureus in the brain using a murine brain abscess model.

76 ed pneumonitis (n = 7), myocarditis (n = 5), brain abscesses (n = 5), chorioretinitis (n = 3), lymph

77 oprotein receptor-1 (LOX-1) was increased in brain abscesses of both TLR2 KO and WT mice compared to

78 n neutrophil and macrophage recruitment into brain abscesses of MyD88 KO animals.

79 crophages, and neutrophils isolated from the brain abscesses of MyD88 KO mice produced significantly

80 We previously reported that brain abscesses of TLR2 knockout (KO) mice exhibited ele

81 d CD8(+) T cell infiltrates were elevated in brain abscesses of TLR2 KO mice at days 3, 7, and 14 pos

82 y protein-2, was significantly attenuated in brain abscesses of TLR2 KO mice compared to WT mice duri

83 However, LOX-1 induction in brain abscesses of TLR2 KO mice was significantly attenu

84 unctions in a compensatory manner to control brain abscess pathogenesis, with cells other than glia a

85 unctional significance of these mediators in brain abscess pathogenesis.

86 e response leading to the establishment of a brain abscess remains poorly defined.

87 Brain abscess represents the infectious disease sequelae

88 Brain abscesses result from a pyogenic parenchymal infec

89 sed to detect specific archaeal sequences in brain abscess samples and controls.

90 ielded archaea, mostly methanogens, in 28/32 brain abscess samples, and no archaea in 71 negative con

91 Despite these differences, brain abscess severity in TLR2 KO and WT animals was sim

92 Antibiotic treatment of brain abscess should contain anti-archaeal compounds suc

93 ch correlated with a significant decrease in brain abscess size.

94 rchaea-specific qPCR yielded archaea in 8/18 brain abscess specimens and 1/27 controls (P < .003), an

95 All bacteria present in brain abscess specimens were identified, in view of the

96 ied 44 bacteria that had never been found in brain abscess specimens, including 22 uncultured bacteri

97 of tissue injury, with significantly larger brain abscesses typified by exaggerated edema and necros

98 the importance of MyD88-dependent signals in brain abscesses, we compared disease pathogenesis using

99 vents related to procedures, endocarditis or brain abscess were excluded.

100 All brain abscesses were fungal; 73% (8/11) of these patient

101 To address this issue, brain abscesses were induced in TCR alphabeta knockout (

102 Cells recovered from brain abscesses were shown to harbor S. aureus intracell

103 al hematoma, acute infarcts, and Aspergillus brain abscesses were the predominant etiologies during t