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1  extensive conventional testing, including a brain biopsy.
2 uld allow some patients to avoid the risk of brain biopsy.
3 on initial type of encephalitis diagnosed by brain biopsy.
4  performed high-throughput RNA sequencing on brain biopsy.
5 sy, 5 of them underwent surgery, and 1 had a brain biopsy.
6 phalopathy (PML) was ultimately confirmed by brain biopsy.
7  of samples is available as is the case with brain biopsy.
8 iagnoses, which emphasizes the importance of brain biopsy.
9  by microscopic examination and culture of a brain biopsy.
10 dmission and before a CT-guided stereotactic brain biopsy.
11 ction and recycling in human fibroblasts and brain biopsies.
12 nts and thus obviate the need for diagnostic brain biopsies.
13      None had primary angiitis of the CNS at brain biopsy (60% specificity).
14     We analyzed axonal pathology in archival brain biopsy and autopsy samples from 19 children with e
15 zed 37 full-length env genes from uncultured brain biopsy and blood samples from four patients with A
16                                              Brain biopsy and cerebrospinal fluid polymerase chain re
17 (n = 13; median age 43 years, range 5-67) on brain biopsy and/or autopsy, ascertained retrospectively
18 e present the clinical, imaging, laboratory, brain biopsy, and autopsy findings of a 57-year-old male
19 nopathologic patterns of MS as determined by brain biopsy, and we identified unique antibody patterns
20                                              Brain biopsies are often not feasible as a result of coa
21 sis in patients with lesions not amenable to brain biopsy, as well as provide improved surrogates of
22                                              Brain biopsy confirmed active, T-cell type MS.
23                                In 1 patient, brain biopsy demonstrated axonal spheroids and pigmented
24 letion (STED) super-resolution microscopy of brain biopsies from patients who died of pneumococcal me
25 died patients with encephalitis diagnosed by brain biopsy from January 1, 1983, through December 31,
26                                              Brain biopsy has an uncertain role in the diagnosis of d
27 and signs of hypomyelination, and gliosis on brain biopsy in another patient.
28 d compare the safety and diagnostic value of brain biopsy in HIV patients in the pre-highly active an
29                                              Brain biopsy in HIV patients is safe with high diagnosti
30 tic procedures that may obviate the need for brain biopsy in the future.
31                                  Findings of brain biopsy in the remaining eight patients were nondia
32                           Thus, a diagnostic brain biopsy is not warranted in these patients.
33 tion (qPCR) in cerebrospinal fluid (CSF), or brain biopsy, is required for probable or definite diagn
34 pheral blood, cerebrospinal fluid (CSF), and brain biopsy material derived from MS patients and contr
35                                        Early brain biopsy may be indicated in HIV patients with focal
36 cumstances, an important role for diagnostic brain biopsy may be required in some cases.
37                              Angiography and brain biopsy may complement each other when determining
38                                     Positive brain biopsies occurred in 10% of patients.
39                  Expression of CXCL13 in the brain biopsy of a patient with anti-NMDAR encephalitis w
40  immune responses, previously attainable via brain biopsy only.
41 llowing withdrawal, and PML was confirmed by brain biopsy or by identifying JC virus in the cerebrosp
42 101 consecutive patients with PCNSV based on brain biopsy or conventional angiography (or both) betwe
43 n vivo, which until recently required either brain biopsy or PET imaging with an on-site cyclotron an
44 id JC virus (JCV) polymerase chain reaction, brain biopsy, or autopsy, and who had MR images availabl
45                                              Brain biopsies revealed inflammatory encephalitis associ
46                             Skin, liver, and brain biopsies revealed vasculopathic changes characteri
47                                              Brain biopsy revealed typical features of PML as well as
48 ourth patient, still alive, was diagnosed by brain biopsy) revealed changes affecting predominantly t
49 d recurrent aseptic meningitis and underwent brain biopsy revealing a diagnosis of neurosarcoidosis.
50  be diagnosed, which obviates the need for a brain biopsy sample to be taken.
51  on a dataset that is comprised of 646 human brain biopsy samples from 60 different patients.
52 oluble aggregates in the cortical area of LD brain biopsy samples, and there is also a dramatic loss
53 essing FAK in vitro but not in nonneoplastic brain biopsy samples.
54                                        Early brain biopsy should be considered in patients without cl
55                  Immunohistochemistry of the brain biopsy showed positive neuronal staining.
56 any presence of CAA from routinely collected brain biopsy specimen, biopsy specimen at hematoma evacu
57 s not detected in normal human brain, all 24 brain biopsy specimens containing PCNSL were positive fo
58 emistry on formalin-fixed, paraffin-embedded brain biopsy specimens from 24 patients with PCNSL to in
59 rectly, we undertook a morphometric study of brain biopsy specimens from AD and control cases.
60 ransfected with human PS1 complementary DNA, brain biopsy specimens from demented patients, and postm
61           Cerebrospinal fluid analysis and 2 brain biopsy specimens showed no evidence of an infectio
62                                              Brain biopsy specimens that exhibit encephalitis without
63 AA were included: 52 with autopsies, 22 with brain biopsy specimens, and 31 with pathologic samples f
64                        Patient demographics, brain biopsy technique, histopathology and patient outco
65            Compared with nonneoplastic adult brain biopsies, the levels of FAK protein are elevated a
66 on sequencing of his cerebrospinal fluid and brain biopsy tissue was performed to identify a causativ
67 istochemistry for free-living amoebas on the brain biopsy tissue were positive.
68 ed the presence of Cache Valley virus in the brain biopsy tissue.
69  patients (of 109; 12%) had undergone repeat brain biopsy to confirm PCNSL.
70 al testing (conjunctival, transbronchial and brain biopsies) to search for causes of an inflammatory
71  symptoms, laboratory studies, neuroimaging, brain biopsy, treatment, and complications were recorded
72        Median time from onset of symptoms to brain biopsy was 66 days (interquartile range, 18-135 da
73                  Diagnostic workup including brain biopsy was unrevealing.
74              The most common indications for brain biopsy were diagnosis unlikely to be toxoplasmosis
75 useful in guiding the decision to proceed to brain biopsy where a treatable disease cannot be exclude

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