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1 tal discharge and none developed symptomatic brain hemorrhage.
2 not induce reperfusion and induced striking brain hemorrhage.
3 l artery stroke, without thrombocytopenia or brain hemorrhage.
4 th subsequent cerebral edema and devastating brain hemorrhage.
5 ith respiratory failure and intraparenchymal brain hemorrhage.
6 al signs of CM, associated with perivascular brain hemorrhage.
7 based study of the late sequelae of neonatal brain hemorrhage.
8 e observe hindbrain ventricular swelling and brain hemorrhage.
9 al vascular morphogenesis and do not exhibit brain hemorrhage.
10 iven 60 minutes after occlusion but provoked brain hemorrhage.
11 at 75 vol% reduces ischemic brain damage and brain hemorrhages.
12 tion of tissue plasminogen activator-induced brain hemorrhages.
13 survived the infection and had little or no brain hemorrhaging.
14 roprotective activities, blocks tPA-mediated brain hemorrhage after transient brain ischemia and embo
16 fusion-induced vascular injury and secondary brain hemorrhage and offers a unique opportunity to eval
17 s, 145 had prior infarct, 8 had hypertensive brain hemorrhage, and 164 admissions for PSR were identi
18 a-induced fl02k mutant showed heart failure, brain hemorrhage, and diminished cardiac and vessel lume
19 cularly in patients who are at high risk for brain hemorrhage, and thus provide a new approach for th
21 nifestations, including embryonic lethality; brain hemorrhage; and vasculogenic, craniofacial, and ne
22 botic ischemic stroke but is associated with brain hemorrhage; antiplatelet therapy has limited effic
23 lth, diabetes mellitus, history of stroke or brain hemorrhage, cognitive function, and self-reported
24 rate to severe ischemic stroke (HR 1.63) and brain hemorrhage (HR 1.73) were significantly associated
25 rium Borrelia turicatae rapidly succumb to a brain hemorrhage if they are unable to clear peak bacter
31 Exogenous NO provided protection against brain hemorrhages (mean, 1.4 vs 24.5 hemorrhagic foci pe
32 The primary end point was ischemic stroke, brain hemorrhage, or death from vascular causes other th
34 ated Tn antigen in these cells and developed brain hemorrhage that was uniformly fatal by embryonic d
35 erebral artery, tPA administration increased brain hemorrhage transformation, infarct volume, and ede
37 stigate the role of tPA and microglia during brain hemorrhage, we induced experimentally ICH by intra
39 ical dysfunction and reproducibly results in brain hemorrhages whose appearance is highly reminiscent
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