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1 ll patients showed a new medullary lesion on brain magnetic resonance imaging.
2 e on the basis of core clinical features and brain magnetic resonance imaging.
3 lvement in pure AMN patients who have normal brain magnetic resonance imaging.
4 in patients with a gadolinium enhancement on brain magnetic resonance imaging.
5 ignificant decreases in enhancing lesions on brain magnetic resonance imaging.
6 lerosis or >1 new or enlarging T2 lesions on brain magnetic resonance imaging.
7 ve battery was administered; 1075 also had a brain magnetic resonance imaging.
8 solution dynamic [(11) C]FMZ PET imaging and brain magnetic resonance imaging.
9 nant axial rigidity, and midbrain atrophy on brain magnetic resonance imaging.
10 inical history and the hot cross bun sign on brain magnetic resonance imaging.
11 determination, [18F]MNI-659 PET imaging, and brain magnetic resonance imaging.
12 or disorder with underlying abnormalities on brain magnetic resonance imaging.
13 a subset of 1901 participants with available brain magnetic resonance imaging (1999-2005), we further
14 ith 1-year Psychomotor Development Index and brain magnetic resonance imaging abnormalities among inf
15 ed individuals aged between 45 and 98 years, brain magnetic resonance imaging and genotyping was perf
17 Subjects who agreed to participate had a brain magnetic resonance imaging and positron emission t
18 Thorough clinical evaluations, including brain magnetic resonance imaging and sensory evoked pote
19 and weeks 10 and 20, participants underwent brain magnetic resonance imaging and spectroscopy protoc
20 nts gave written informed consent to undergo brain magnetic resonance imaging and the Addenbrooke's C
21 presence of brain injury on the preoperative brain magnetic resonance imaging and the trajectory of p
22 tions, anti-CASPR2 antibodies specificities, brain magnetic resonance imaging, and CSF analyses, canc
23 and nerve morphology, longitudinal study of brain magnetic resonance imaging, and glycogen branching
24 and retinal function, had normal findings on brain magnetic resonance imaging, and had normal cerebro
25 isorders, Fourth Edition [DSM-IV] criteria), brain magnetic resonance imaging, and proton magnetic re
27 onset parkinsonism-dystonia with distinctive brain magnetic resonance imaging appearances and neurode
28 sability Status Scale score, spinal cord and brain magnetic resonance imaging, AQP4-ab titers, pain l
29 ll of lifetime smoking data and a structural brain magnetic resonance imaging at age 73 years from wh
30 motherapy underwent clinical examination and brain magnetic resonance imaging at the following three
31 presentation of tuberculosis and appeared on brain magnetic resonance imaging at the time of presenta
33 3 weeks, with standardized naming tests and brain magnetic resonance imaging before and after therap
34 f clinically definite multiple sclerosis and brain magnetic resonance imaging changes in subgroups ba
36 ess this, we meta-analyzed three-dimensional brain magnetic resonance imaging data from 1728 MDD pati
37 on auditory scene analysis tasks, structural brain magnetic resonance imaging data from the patient c
38 d 70 years of age as well as high-resolution brain magnetic resonance imaging data obtained at approx
40 urological examination, and abnormalities on brain magnetic resonance imaging did not differ between
42 and associated with clinical investigations (brain magnetic resonance imaging, electroencephalography
43 linical demyelinating event in patients with brain magnetic resonance imaging evidence of subclinical
44 first demyelinating event, 383 patients with brain magnetic resonance imaging evidence of subclinical
50 follow-up with neuropsychological tests and brain magnetic resonance imaging for 3 years, and 87 pat
51 optic glioma were twice as likely to undergo brain magnetic resonance imaging for visual symptoms and
54 omen, 17% blacks, 68% hypertension, 3600 had brain magnetic resonance imaging in 1992-1993, survival
56 lination with white matter hyperintensity on brain magnetic resonance imaging in one-third of the coh
57 velopment of new brain lesions observed with brain magnetic resonance imaging in relapsing-remitting
62 onset of fluctuating neurological signs and brain magnetic resonance imaging lesions simulating mult
70 (standard deviation, 4.7) years) with 1.5-T brain magnetic resonance imaging measures of infarct-lik
72 index, would be associated with preclinical brain magnetic resonance imaging (MRI) and neuropsycholo
74 ldhood-onset schizophrenia shows progressive brain magnetic resonance imaging (MRI) changes during ad
75 ly to have normal appearing basal ganglia on brain magnetic resonance imaging (MRI) compared to minor
76 th detailed placental histology and neonatal brain magnetic resonance imaging (MRI) data at term equi
78 This study attempts to determine which fetal brain magnetic resonance imaging (MRI) features might be
79 [RNFL] thickness and macular volume) and 3T brain magnetic resonance imaging (MRI) for posterior vis
81 T2 lesions or contrast-enhancing lesions on brain magnetic resonance imaging (MRI) in relapsing mult
85 s have discussed incidental disease found on brain magnetic resonance imaging (MRI) scans that had be
94 21 age-matched full-term controls underwent brain magnetic resonance imaging (MRI), as well as neuro
95 tients underwent neurocognitive evaluations, brain magnetic resonance imaging (MRI), echocardiograms,
96 py, optical imaging (multiphoton), and whole brain magnetic resonance imaging (MRI), facilitating mol
97 ssion of clinical features and serum sodium, brain magnetic resonance imaging (MRI), positron emissio
106 based on type of presenting event, baseline brain magnetic resonance imaging parameters, and demogra
107 also was seen in subgroups based on baseline brain magnetic resonance imaging parameters, gender, and
108 newborns with critical CHD were studied with brain magnetic resonance imaging preoperatively and post
110 esults of audiology testing, echocardiogram, brain magnetic resonance imaging, renal ultrasound, and
117 ented with headache, nausea, and vomiting; a brain magnetic resonance imaging scan showed left anteri
120 ), from an initial group of 58 who underwent brain magnetic resonance imaging scanning while in treat
123 er's Disease Neuroimaging Initiative who had brain magnetic resonance imaging scans at baseline and 2
124 examine cortical change, we used anatomical brain magnetic resonance imaging scans for 15 patients w
125 9 participants underwent repeated structural brain magnetic resonance imaging scans from September 17
126 ter thickness and surface area measures from brain magnetic resonance imaging scans of 6503 individua
127 nd JCV polymerase chain reaction testing and brain magnetic resonance imaging scans should be repeate
136 (n = 24) and control men (n = 25) received 2 brain magnetic resonance imaging scans, on average 4 yea
138 cALD demonstrated an inverse correlation to brain magnetic resonance imaging severity score (R2 = 0.
151 glia lesions were the most common finding in brain magnetic resonance imaging, usually associated wit
154 Ophthalmologic examination was normal, the brain magnetic resonance imaging was not suggestive of P
156 analysis of whole-brain 3-T high-resolution brain magnetic resonance imaging was used to determine t
159 it and approximately 7 years later underwent brain magnetic resonance imaging with automated volumetr
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