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1 functions, including neural integration and brain metabolism.
2 closely to the magnitude of the reduction in brain metabolism.
3 nts with insomnia is associated with greater brain metabolism.
4 ave long been held to play a special role in brain metabolism.
5 ow that this metric correlates strongly with brain metabolism.
6 influences on alcohol-associated changes in brain metabolism.
7 cant and should be considered when assessing brain metabolism.
8 ciated with alterations in the regulation of brain metabolism.
9 ing peptide produced at low levels by normal brain metabolism.
10 an anabolic, insulin-like role in developing brain metabolism.
11 brain metabolism found no sex differences in brain metabolism.
12 or accurate prediction of regional pediatric brain metabolism.
13 l conditions to precisely quantify and model brain metabolism.
14 ated with progressive impairment in PbO2 and brain metabolism.
15 g and may reflect abnormalities in pH and/or brain metabolism.
16 these mRNA levels and both AT expression and brain metabolism.
17 he interaction between neuronal dynamics and brain metabolism.
18 ood-brain barrier and inadequate glucose for brain metabolism.
19 years and can be associated with changes in brain metabolism.
20 due to apoE isoform-specific differences in brain metabolism.
21 not typically thought to be associated with brain metabolism.
22 of HIV-1 infection and IDU on (18)F-FDG PET brain metabolism.
23 to the relative roles of neurons and glia in brain metabolism.
24 a show that a calorie-restricted KD enhances brain metabolism.
27 lysis, we predict the behavioural diagnosis, brain metabolism and 1-year clinical outcome of individu
28 we demonstrate that the relationship between brain metabolism and aggression is causal, conserved ove
29 edication use and neuroimaging biomarkers of brain metabolism and atrophy as a proxy for understandin
32 nds peak during childhood, and evidence that brain metabolism and body growth rate covary inversely a
35 mplicates global and regional disruptions in brain metabolism and energetics in the pathogenesis of c
39 en resting state functional connectivity and brain metabolism and how this results in a distinctive p
42 changes in the activity of afferent inputs, brain metabolism and neurotransmitter uptake that are ty
43 racranial pressure, brain electrophysiology, brain metabolism and oxygenation, and cerebral blood flo
44 Also, investigations of the relationship of brain metabolism and specific neurotransmitter systems a
47 differences in baseline measures of regional brain metabolism and to assess their reproducibility.
48 e extracellular concentration increases with brain metabolism and which, in vitro, inhibits basal for
49 ces existed in the effect of age on regional brain metabolism, and asymmetry of metabolism, in the te
50 stained improvement in arterial ammonia, its brain metabolism, and brain water in cirrhotic rats.
51 s were associated with decreases in regional brain metabolism, and the correlations were strongest in
53 of cannabis use whereby global and regional brain metabolism are altered in those with prolonged can
54 Changes in synaptic excitability and reduced brain metabolism are among the earliest detectable alter
55 cur and subsequent developmental declines in brain metabolism are matched by proportionate increases
59 effects by separated but also differences in brain metabolism as depicted by proton magnetic resonanc
61 mer's disease, brain amyloid deposition, and brain metabolism as well as progressive cognitive impair
62 was to examine associations between regional brain metabolism, as measured by (18)F-FDG PET, and neur
63 associated with reductions in resting-state brain metabolism, as measured by PET, progressing with d
65 To identify specific regional patterns of brain metabolism associated with abnormal cognitive and
69 In line with an age-dependent decline in brain metabolism, both ROI and SPM-based methods reveale
70 Methylphenidate induced variable changes in brain metabolism, but it consistently increased cerebell
71 his study was to characterize the changes in brain metabolism caused by antecedent recurrent hypoglyc
72 on in arterial ammonia concentration and its brain metabolism, cerebral blood flow, brain cytokine pr
73 Anesthetic-related manipulation of baseline brain metabolism demonstrates the significant contributi
77 ose as the main energy substrate for resting brain metabolism during intoxication (presumably shiftin
78 brain metabolism are uncommon, because local brain metabolism evolves significantly with age througho
79 etabolite in male rats, the present study of brain metabolism found no sex differences in brain metab
80 ior studies of brain metabolism which viewed brain metabolism from a unitary perspective that equated
81 lation of its overall energy contribution to brain metabolism: glucose (predominantly unlabeled) and
82 ity of anesthetics to change global baseline brain metabolism has created the opportunity to examine
84 y 11 of the 18 patients and led to decreased brain metabolism in a number of cortical areas, with the
85 he interrelationships between microbiota and brain metabolism in childhood undernutrition could provi
87 xyglucose PET to detect abnormal patterns of brain metabolism in patients with neurodegenerative diso
90 er support for the hypothesis of an abnormal brain metabolism in the hippocampal and parahippocampal
93 yptophan depletion resulted in a decrease in brain metabolism in the middle frontal gyrus (dorsolater
98 mography and fludeoxyglucose F 18 to measure brain metabolism in Vietnam combat veterans with PTSD (n
99 e morphology, and altered liver, muscle, and brain metabolism in ways indicative of a greater ability
100 sed "relative" (region relative to the whole brain) metabolism in the cerebellum and decreased it in
102 potential contribution of plasma lactate to brain metabolism is 10% under basal plasma lactate condi
108 theory of visual attention, were related to brain metabolism, measured by (18)F-fluorodeoxyglucose p
109 imagery paradigms (26 patients) and resting brain metabolism measurement using (18) fluorodeoxygluco
111 e investigated another mechanism critical to brain metabolism, mitochondrial morphology, in BTICs.
113 ume is highly heritable early in life, it is brain metabolism-not brain structure-that is the critica
115 y be associated with modulations in regional brain metabolism occurring remote from the lesion site.
117 f so whether this is associated with altered brain metabolism of ammonia or anatomical disturbance of
122 tracers that provide information on regional brain metabolism or blood flow as well as neurochemicall
124 tical seizure-associated increases in global brain metabolism, partial brain tissue oxygenation, and
127 cits in AD by combining measures of regional brain metabolism (resting state fluorodeoxyglucose posit
130 entire cohort, EEG organization and overall brain metabolism showed no significant association with
131 These turnover rates can provide measures of brain metabolism similar to radioisotope techniques.
132 current hypoglycemia leads to adaptations of brain metabolism so that modest increments in circulatin
133 Methylphenidate induced variable changes in brain metabolism: subjects with the higher D2 measures t
136 ry (TBI) results in persistent disruption of brain metabolism that has yet to be mechanistically defi
137 to identify significant changes in regional brain metabolism that occurred with this intervention.
138 ntify and validate alternative ways to image brain metabolism, through which the efficacy of new ther
139 ed a doubling of the glucose contribution to brain metabolism under hypoglycemic conditions that rest
141 velop mathematic models of regional relative brain metabolism using pediatric (18)F-FDG PET with CT d
142 ith cognitive symptoms of dementia, regional brain metabolism was a sensitive indicator of AD and of
149 of age, optimal models of regional relative brain metabolism were linear for 9 regions, quadratic fo
151 jects, lorazepam-induced decrements in whole brain metabolism were significantly greater in cocaine-a
153 ron emission tomography (PET) measurement of brain metabolism with 18fluorodeoxyglucose to show that
154 RI correlate islands of relatively preserved brain metabolism with isolated fragments of behaviour.
155 ssing techniques to measure changes in mouse brain metabolism with preclinical (18)F-FDG PET/CT.
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