ライフサイエンスコーパス: brain microvascular endothelial cell

1 berculosis BBB infection using primary human brain microvascular endothelial cells.

2 (BBB), which is composed of tightly apposed brain microvascular endothelial cells.

3 to both human bronchial epithelial cells and brain microvascular endothelial cells.

4 m Escherichia coli K1 interacting with human brain microvascular endothelial cells.

5 notypic and functional properties similar to brain microvascular endothelial cells.

6 cterial uptake in epithelial cells and human brain microvascular endothelial cells.

7 okine with its cognate receptor, CCR2, along brain microvascular endothelial cells.

8 y involve oxidative injury or dysfunction of brain microvascular endothelial cells.

9 irus in both 3T6 mouse fibroblasts and mouse brain microvascular endothelial cells.

10 ical and morphological properties of in situ brain microvascular endothelial cells.

11 ntributes to E. coli K1 membrane invasion of brain microvascular endothelial cells.

12 Similar effects were observed in human brain microvascular endothelial cells.

13 l junctions and prevent virus transit across brain microvascular endothelial cells.

14 s were obtained using an in vitro model with brain microvascular endothelial cells.

15 ocaine-mediated induction of PDGF-B in human brain microvascular endothelial cells.

16 y properties of monocyte subsets using human brain microvascular endothelial cells.

17 bacteria on the surface of rodent and human brain microvascular endothelial cells.

18 ococcal NanA had increased invasion of human brain microvascular endothelial cells.

19 ansiently expressed in COS-7 cells and mouse brain microvascular endothelial cells.

20 PCR-array screening of human brain microvascular endothelial cells after ABCD1 silenc

21 creted BDNF protein were quantified in human brain microvascular endothelial cells after exposure to

22 migration across monolayers of primary human brain microvascular endothelial cells and diminished BBB

23 ism of deleterious effects induced by IL2 on brain microvascular endothelial cells and may inform the

24 hibit an enhanced ability to adhere to human brain microvascular endothelial cells as compared with m

25 e that WNV-MAD78 replicated in and traversed brain microvascular endothelial cells as efficiently as

26 Primary cultures of rat brain microvascular endothelial cells as well as the rat

27 e previously identified two types of E. coli-brain microvascular endothelial cell (BMEC) interactions

28 in vitro model systems constructed of human brain microvascular endothelial cells (BMEC) and EA.hy 9

29 Listeria monocytogenes invasion of human brain microvascular endothelial cells (BMEC) and its rol

30 Transcytosis of brain microvascular endothelial cells (BMEC) by E. coli

31 nfluence the ability of E. coli K1 to invade brain microvascular endothelial cells (BMEC) in vitro an

32 Invasion of brain microvascular endothelial cells (BMEC) is a prereq

33 o we found that B. turicatae associated with brain microvascular endothelial cells (BMEC) significant

34 of E. coli is necessary for invasion of the brain microvascular endothelial cells (BMEC) that consti

35 own that S-fimbriated Escherichia coli binds brain microvascular endothelial cells (BMEC) via a lecti

36 In vitro, brain microvascular endothelial cells (BMEC) were incuba

37 nce the capabilities of E. coli K1 to invade brain microvascular endothelial cells (BMEC), for exampl

38 res a high degree of bacteremia, invasion of brain microvascular endothelial cells (BMEC), host cell

39 oodstream but also invade and survive within brain microvascular endothelial cells (BMEC), the princi

40 because of a unique capacity to invade human brain microvascular endothelial cells (BMEC), the single

41 In this study, using rat primary brain microvascular endothelial cells (BMEC), we demonst

42 E. coli promotes the actin rearrangement of brain microvascular endothelial cells (BMEC), which cons

43 e than its parent strain in bovine and human brain microvascular endothelial cells (BMEC), which cons

44 d-brain barrier that constitutes a lining of brain microvascular endothelial cells (BMEC).

45 the interaction(s) of C. albicans with human brain microvascular endothelial cells (BMEC).

46 al locus, ibeB, which allows RS218 to invade brain microvascular endothelial cells (BMEC).

47 ain barrier, which is composed of a layer of brain microvascular endothelial cells (BMEC).

48 smigrate through monolayers of rat and human brain microvascular endothelial cells (BMEC).

49 mined for its role in E. coli K1 invasion of brain microvascular endothelial cells (BMEC).

50 rain barrier (BBB), which mainly consists of brain microvascular endothelial cells (BMEC).

51 ) that is composed of a specialized layer of brain microvascular endothelial cells (BMEC).

52 the fibrin-Matrigel mixed gel by coculturing brain microvascular endothelial cells (BMECs) and human

53 Acute Krit1 gene inactivation in mouse brain microvascular endothelial cells (BMECs) changes ex

54 Invasion of brain microvascular endothelial cells (BMECs) is a key s

55 Escherichia coli K1 invasion of brain microvascular endothelial cells (BMECs) is a prere

56 ZD1 was significantly less invasive in human brain microvascular endothelial cells (BMECs) than the p

57 Migration and tube formation properties of brain microvascular endothelial cells (BMECs) were analy

58 Primary mouse brain microvascular endothelial cells (BMECs) were cultu

59 occludens-1 (ZO-1) following stimulation of brain microvascular endothelial cells (BMECs) with the c

60 of Trypanosoma brucei gambiense cross human brain microvascular endothelial cells (BMECs), a human b

61 eloped a human in vitro BBB model comprising brain microvascular endothelial cells (BMECs), pericytes

62 ess to the brain, HIV-1 must migrate through brain microvascular endothelial cells (BMECs), which com

63 barrier (BBB) is a network formed mainly by brain microvascular endothelial cells (BMECs).

64 Thrombin reduced brain microvascular endothelial cell (BMVEC) and perivas

65 egulation of adhesion molecule expression on brain microvascular endothelial cells (BMVEC) could perm

66 consisting of a matrix-coated membrane with brain microvascular endothelial cells (BMVEC) on one sid

67 -1alpha also decreases monocyte adherence to brain microvascular endothelial cells (BMVEC) that are a

68 wnregulating the expression of MMP-9 gene in brain microvascular endothelial cells (BMVEC) that const

69 orted in several BBB models of rat and human brain microvascular endothelial cells (BMVEC) using a re

70 in-5, using in vitro cultures of the primary brain microvascular endothelial cells (BMVEC).

71 Immune mediators and leukocyte engagement of brain microvascular endothelial cells (BMVECs) contribut

72 s subjected to sham or stroke surgery and in brain microvascular endothelial cells (BMVECs) from Wist

73 ed the integrity of tight junctions (TJs) of brain microvascular endothelial cells (BMVECs) in an in

74 Brain microvascular endothelial cells (BMVECs) present a

75 previously that GSK3beta inhibition in human brain microvascular endothelial cells (BMVECs) reduced m

76 Exposure of primary human brain microvascular endothelial cells (BMVECs) to CD40L

77 ack CCR5 and CXCR4, including astrocytes and brain microvascular endothelial cells (BMVECs), for natu

78 Using human brain tissues and primary human brain microvascular endothelial cells (BMVECs), we demon

79 e moieties in HIV-1 entry into primary human brain microvascular endothelial cells (BMVECs), which re

80 f VE-cadherin mediated by miRNA-101 in human brain microvascular endothelial cells (BMVECs).

81 ygdP, a gene associated with the invasion of brain microvascular endothelial cells by Escherichia col

82 NF1 from screening the cDNA library of human brain microvascular endothelial cells by the yeast two-h

83 infected individuals, as controls) and human brain microvascular endothelial cells by using quantitat

84 Silencing of ABCD1 in human brain microvascular endothelial cells causes accumulatio

85 ucose transporter 1 (GLUT-1) levels in human brain microvascular endothelial cells, causing disruptio

86 a significantly decreased ability to invade brain microvascular endothelial cells compared to the wi

87 at expression of recombinant CLN-5 protected brain microvascular endothelial cell cultures from a VEG

88 Recombinant proteinase-3 applied to human brain microvascular endothelial cells degraded both the

89 rum isolates with increased binding to human brain microvascular endothelial cells did not alter the

90 We found that both human and murine brain microvascular endothelial cells express constituen

91 omologue ABCD2, is highly expressed in human brain microvascular endothelial cells, far exceeding its

92 Given our recently described role for human brain microvascular endothelial cells (HBEC) in modulati

93 y cells with low Gb3 content (cultured human brain microvascular endothelial cells [HBEC]).

94 m by which HIV-1 Tat protein modulates human brain microvascular endothelial cell (HBMEC) functions,

95 pA) of E. coli interacts with a 95-kDa human brain microvascular endothelial cell (HBMEC) glycoprotei

96 ing mechanisms by which VEGF regulates human brain microvascular endothelial cell (HBMEC) integrity a

97 ypical of high therapeutic dosage on a human brain microvascular endothelial cell (HBMEC) model of th

98 used a Transwell culture system of the human brain microvascular endothelial cell (HBMEC) monolayer a

99 ltaLF mutant resulted in disruption of human brain microvascular endothelial cell (hBMEC) monolayer i

100 permeability changes that occur in the human brain microvascular endothelial cell (HBMEC) monolayer,

101 tes to Escherichia coli K1 invasion of human brain microvascular endothelial cells (HBMEC) and intera

102 the physiological response of isolated human brain microvascular endothelial cells (HBMEC) and monocy

103 E. coli K1 binding to and invasion of human brain microvascular endothelial cells (HBMEC) are a prer

104 We used human brain microvascular endothelial cells (HBMEC) as the in

105 peptide, inhibited E. coli invasion of human brain microvascular endothelial cells (HBMEC) by more th

106 causes meningitis in neonates, invades human brain microvascular endothelial cells (HBMEC) by rearran

107 tory genes in an in vitro BBB model of human brain microvascular endothelial cells (HBMEC) cocultured

108 pha gene expression were determined in human brain microvascular endothelial cells (HBMEC) exposed to

109 ain RS218 activates Rac1 (GTP-Rac1) of human brain microvascular endothelial cells (HBMEC) in a time-

110 chanism for inflammatory activation of human brain microvascular endothelial cells (HBMEC) in respons

111 arrays, we found that GBS infection of human brain microvascular endothelial cells (HBMEC) induced a

112 ausing meningitis, and its invasion of human brain microvascular endothelial cells (HBMEC) is a prere

113 t Cryptococcus binding and invasion of human brain microvascular endothelial cells (HBMEC) is a prere

114 urvival in serum, bacterial entry into human brain microvascular endothelial cells (HBMEC) is governe

115 Coculturing human brain microvascular endothelial cells (hBMEC) or NOTCH l

116 Human brain microvascular endothelial cells (HBMEC) produced a

117 Exposure of human brain microvascular endothelial cells (HBMEC) to C. neof

118 in vitro blood-brain barrier model of human brain microvascular endothelial cells (HBMEC) to study t

119 profile of E. coli K1 associated with human brain microvascular endothelial cells (HBMEC) to the gen

120 ants for loss or decreased invasion of human brain microvascular endothelial cells (HBMEC) which comp

121 he process by which PV enters cultured human brain microvascular endothelial cells (HBMEC), an in vit

122 odstream and interact with specialized human brain microvascular endothelial cells (hBMEC), which con

123 ontributes to type III GBS invasion of human brain microvascular endothelial cells (HBMEC), which con

124 n shown to promote E. coli invasion of human brain microvascular endothelial cells (HBMEC), which con

125 ha) contributes to E. coli invasion of human brain microvascular endothelial cells (HBMEC), which con

126 the brain requires E. coli invasion of human brain microvascular endothelial cells (HBMEC), which con

127 hich is also a receptor for Pf-IRBC on human brain microvascular endothelial cells (HBMEC).

128 ole in Escherichia coli K1 invasion of human brain microvascular endothelial cells (HBMEC).

129 espite lack of productive infection of human brain microvascular endothelial cells (HBMEC).

130 developed the in vitro BBB model with human brain microvascular endothelial cells (HBMEC).

131 Invasion of human brain microvascular endothelial cells (HBMECs) and human

132 nd interferon-alpha-inducible genes in human brain microvascular endothelial cells (HBMECs) and that

133 their capacity to adhere to and invade human brain microvascular endothelial cells (hBMECs) and to pe

134 It is shown here that human brain microvascular endothelial cells (HBMECs) are relat

135 tegrins alphavbeta3 and alphavbeta5 in human brain microvascular endothelial cells (HBMECs) by the fu

136 its effects on chemokine expression in human brain microvascular endothelial cells (HBMECs) by using

137 that Escherichia coli K1 infection of human brain microvascular endothelial cells (HBMECs) induces t

138 eramide and decreased sphingomyelin in human brain microvascular endothelial cells (HBMECs) plated on

139 hate oxidase (GlpO), was cytotoxic for human brain microvascular endothelial cells (HBMECs) via gener

140 Here, we found that infection of human brain microvascular endothelial cells (hBMECs) with GBS

141 howed defects in the ability to invade human brain microvascular endothelial cells (HBMECs), and RDI

142 ause of neonatal meningitis, can enter human brain microvascular endothelial cells (hBMECs), but the

143 demonstrates that CnMVs can fuse with human brain microvascular endothelial cells (HBMECs), the cons

144 able to adhere, invade, and penetrate human brain microvascular endothelial cells (hBMECs), the sing

145 Using human brain microvascular endothelial cells (HBMECs), we found

146 of inter-endothelial cell junctions in human brain microvascular endothelial cells (HBMECs).

147 integrin expression and activation in human brain microvascular endothelial cells (HBMECs).

148 r (BBB), which consists of specialized human brain microvascular endothelial cells (hBMECs).

149 ent inhibitor of E. coli invasion into human brain microvascular endothelial cells (HBMECs).

150 e characterized for binding to primary human brain microvascular endothelial cells (HBMECs).

151 omote SPN adherence to and invasion of human brain microvascular endothelial cells (hBMECs).

152 with Tat activated the Ras pathway in human brain microvascular endothelial cells (HBMECs).

153 Human brain microvascular endothelial cells (hBMVECs) that con

154 hod for generating primary cultures of human brain microvascular endothelial cells (HBMVECs).

155 lysis using Heme-treated and untreated human brain microvascular endothelial cells (HBVEC), and deter

156 Exposure of human brain microvascular endothelial cells (hCMEC/D3 cell lin

157 enhanced adherence to and invasion of human brain microvascular endothelial cells in vitro, demonstr

158 In mouse brain microvascular endothelial cells in vitro, viruses

159 mination revealed binding of B. turicatae to brain microvascular endothelial cells in vivo.

160 ng that the GlcNAc beta 1-4GlcNAc epitope of brain microvascular endothelial cells indeed mediates th

161 wth factor (VEGF) in regulating integrins in brain microvascular endothelial cells is unknown.

162 nt (IT4var09) cytoadhere in vitro to a human brain microvascular endothelial cell line (HBEC-5i).

163 A mouse brain microvascular endothelial cell line infected with

164 e-bound OmpA and wheat germ agglutinin-bound brain microvascular endothelial cell membrane proteins i

165 125I-sAbeta1-40 binding to the brain microvascular endothelial cell monolayer was time

166 In these studies, rat brain microvascular endothelial cell monolayers exposed

167 Permeability of human brain microvascular endothelial cell monolayers was incr

168 ld not induce changes in the permeability of brain microvascular endothelial cell monolayers.

169 en S2 expression was down-regulated in mouse brain microvascular endothelial cells (MvEC), this inhib

170 reading, migration, and sprouting of primary brain microvascular endothelial cells (MVECs) in a dose-

171 V-1 Nef to induce apoptosis in primary human brain microvascular endothelial cells (MVECs).

172 The blood-brain barrier, mainly composed of brain microvascular endothelial cells, poses an obstacle

173 g via endothelial cell adhesion molecules in brain microvascular endothelial cell proliferation and a

174 n vitro BBB model based on co-culture of rat brain microvascular endothelial cells (RBMEC) and astroc

175 In brain microvascular endothelial cells, there was no chan

176 caine-mediated induction of PDGF-BB in human brain microvascular endothelial cells through the bindin

177 Cocaine-mediated induction of ALCAM in human brain microvascular endothelial cells through the transl

178 we used human brain microvascular endothelial cells to examine the rol

179 These exosomes alone can activate human brain microvascular endothelial cells to stimulate adhes

180 In cultures of brain microvascular endothelial cells, VEGF-A specifical

181 eport that this OmpA+ K1 E. coli invasion of brain microvascular endothelial cells was inhibited by w

182 d not occur, but invasion similar to that of brain microvascular endothelial cells was observed when

183 ), or ICAM-1(null)/ICAM-2(-/-) primary mouse brain microvascular endothelial cells, we demonstrate th

184 messenger annexin A1 (ANXA1) is expressed in brain microvascular endothelial cells, where it regulate

185 Treatment of brain microvascular endothelial cells with AGE caused a

186 rculation involves both binding and crossing brain microvascular endothelial cells, with significant