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1 addition to the activation that accompanies brain pathology.
2 s in the brains of transgenic animals causes brain pathology.
3 ction in TNF-alpha levels, leukocytosis, and brain pathology.
4 life for their memory, learning ability and brain pathology.
5 underlying adaptation, stress response, and brain pathology.
6 creased blood-brain barrier permeability and brain pathology.
7 or risk adjustment, and how this relates to brain pathology.
8 group A PfEMP1 may be effective at reducing brain pathology.
9 tant role for monocytes/microglia in causing brain pathology.
10 meningeal-located immune cells in modulating brain pathology.
11 significantly associated with all markers of brain pathology.
12 it may provide a noninvasive way to examine brain pathology.
13 sy development, even in the absence of major brain pathology.
14 population but do not usually contribute to brain pathology.
15 n levels being associated with more advanced brain pathology.
16 thologically verified AD and correlates with brain pathology.
17 ese systems and the degree of Alzheimer type brain pathology.
18 equire greater recognition as a mechanism of brain pathology.
19 at oral intake of DHA reduces Alzheimer-like brain pathology.
20 tween neuronal vimentin expression and local brain pathology.
21 d in the Tg2576 mouse model of AD-like Abeta brain pathology.
22 ven before the development of substantial AD brain pathology.
23 ausal TSE virions that induce late-stage PrP brain pathology.
24 s with HS and 12 patients without structural brain pathology.
25 ain yet have different disease latencies and brain pathologies.
26 of biological data across a growing list of brain pathologies.
27 lia, potentially implicating Cyp1b1 in other brain pathologies.
28 k and vasculature are ubiquitous in numerous brain pathologies.
29 ell cycle checkpoint control with a focus on brain pathologies.
30 ledged as prime regulators involved in human brain pathologies.
31 ons have been extensively studied in various brain pathologies.
32 NA editing process itself is dysregulated by brain pathologies.
33 system and is important in stroke and other brain pathologies.
34 ype could be useful for treatment of various brain pathologies.
35 Hcy-thiolactone and lead to vascular and/or brain pathologies.
36 eficial role in neuroprotection from various brain pathologies.
37 ring stroke, brain injury, and certain other brain pathologies, anaerobic glycolysis produces severe
38 perinatal period to block the progression of brain pathologies and allow normal brain development to
40 ies have demonstrated a relationship between brain pathology and AD progression, the Alzheimer's path
41 rozygous TSC animal models do not show gross brain pathology and are seizure-free, suggesting that su
42 ssion were assessed at 15 and 24 months, and brain pathology and biochemistry were assessed at 24 mon
43 ationship between neocortical Alzheimer-type brain pathology and dementia in consecutive autopsies fr
45 have been proposed based on genetic studies, brain pathology and imaging, but a major impediment to t
46 dvances understanding of Alzheimer's disease brain pathology and provides new insights into the subce
47 use of the many-to-many relationship between brain pathology and psychiatric symptoms, true natural d
48 r a quarter of a century despite severe host brain pathology and with no evidence of immune response.
49 hagocytosis of viable neurons contributes to brain pathology and, surprisingly, blocking this process
50 ute to the development of lactic acidosis in brain pathologies, and suggest targeting it as a novel t
52 showed viral persistence in the CNS, chronic brain pathology, and demyelination in the spinal cord wh
53 elates, in fatal cases, with the severity of brain pathology, and has diagnostic and prognostic signi
54 hose aged 90 and older, other ageing-related brain pathologies are prevalent in addition to Alzheimer
55 studies have linked particulate exposures to brain pathologies associated with Parkinson's and Alzhei
56 tudies suggest that antibodies contribute to brain pathology associated with autoimmune diseases such
58 hed exosomes appear to exacerbate AD-related brain pathology by promoting the aggregation of Abeta.
59 t R47H and evaluated the variant's effect on brain pathology, cellular morphology, and expression of
60 severe weight loss and hypoventilation, with brain pathology characterized by TDP-43 immunostaining.
61 markedly reduced meningeal inflammation and brain pathology compared with wild type, despite similar
63 tenuated Bartha strain does not cause severe brain pathology despite viral replication and spread thr
64 These results suggest that expression of brain pathology differs between left-handed and right-ha
69 lack of correlation between serum titers and brain pathology have, no doubt, contributed to a delayed
72 knockout of Sirt1 results in exacerbation of brain pathology in a mouse model of Huntington's disease
73 gyrus mean diffusivity as an early marker of brain pathology in children with OSAS.SIGNIFICANCE STATE
75 to the generation of recurrent seizures and brain pathology in patients with medically intractable m
76 function associated with Alzheimer's disease brain pathology in terms of functional network organizat
77 te specific cognitive abnormalities to focal brain pathology in the presence of widespread disease su
82 brain edema are hallmark features of several brain pathologies, including traumatic brain injuries (T
87 acid (GABA)-A receptor during progression of brain pathology is challenging because of the lack of im
90 re birth is a major risk factor for multiple brain pathologies, notably periventricular leukomalacia
94 neurodegenerative disorder characterized by brain pathology of intracellular neurofibrillary tangles
95 evidence for the involvement of microglia in brain pathology of mouse models of the two diseases.
98 ever, L-DOPS treatment did not arrest global brain pathology or improve somatic growth, as gene thera
100 ces can be revealed by the fact that certain brain pathologies render inhibitory processing ineffecti
102 Tourette's syndrome, the absence of evident brain pathology resulted in vacillation between organic
105 in Alzheimer's disease is a manifestation of brain pathologies such as accumulation of amyloid-beta p
106 tion is an important pathogenetic factor for brain pathologies such as Parkinson's disease, ADHD, sch
107 and the influence of aging and co-occurring brain pathologies that commonly arise in the elderly.
108 users show early Alzheimer's disease-related brain pathology that may be the basis for cognitive impa
109 wakefulness, and specific patterns of local brain pathology-thereby possibly providing a mechanistic
110 "DC8" and "DC13" have been proposed to cause brain pathology through interactions with endothelial pr
111 y are frequently used in patients with acute brain pathologies to facilitate sedation or surgical and
112 results suggest that TDP-43 is an important brain pathology underlying cognitive decline and dementi
114 ical scrapie, but it was unclear whether the brain pathology was caused by damage to astrocytes, neur
116 or-beta/Smad2/3 activation and hydrocephalic brain pathology were evaluated at Day 14 and the inflamm
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