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1 leus accumbens (NAc), a key component of the brain reward system.
2 ggested to be linked to a dysfunction of the brain reward system.
3 at results from contingent activation of the brain reward system.
4  together lead to altered homeostasis of the brain reward system.
5 easing neurons appear to be important in the brain reward system.
6  that ADX does not affect sensitivity of the brain reward system.
7 for interaction between hypothalamus and the brain reward system.
8 ddition to during gestation, can program the brain reward system.
9  (ICSS) test is sensitive to the function of brain reward systems.
10 hibitory effects of higher nicotine doses on brain reward systems.
11 lecular manipulations affect the function of brain reward systems.
12  nucleus accumbens (NAc), a key component of brain reward systems.
13 ion of dopaminergic neurotransmission in the brain reward systems.
14 tering dopaminergic neurotransmission in the brain reward systems.
15 ral tegmental area (VTA) are the origin of a brain reward system and are critically involved in drug
16 esis that insulin modulates sensitivity of a brain reward system and that hypoinsulinemia may be the
17 pioid system is widely known to modulate the brain reward system and thus affect the behavior of huma
18 tualized as a cycle of decreased function of brain reward systems and recruitment of antireward syste
19                We review the neurobiology of brain reward systems and the impact of sleep and circadi
20 icotine dependence, long-term alterations in brain reward systems, and nicotine receptor regulation i
21  the central links between somatosensory and brain reward systems are not known.
22 mate signaling in pain states and define the brain reward system as an important region for the regul
23 thermore, similar genetic vulnerabilities in brain reward systems can increase predisposition to drug
24 cond) cellular and chemical responses in the brain reward system during drug-seeking and drug-taking
25                    The decreased function in brain reward systems during nicotine withdrawal is compa
26 hat addictive drugs often render an addict's brain reward system hypersensitive, leaving the individu
27 n order to evaluate the effect of ADX on the brain reward system in general, and cocaine reward in pa
28  partly from the inappropriate activation of brain reward systems in response to aggressive or violen
29 ed whether ADX decreases sensitivity of the 'brain reward system' in general, or its response to coca
30 nt mechanism by which normal function of the brain reward system may be impaired during substance abu
31 ce for fatty foods, regulated in part by the brain reward system, may contribute to the development o
32  a cycle of progressive dysregulation of the brain reward system that results in the compulsive use a
33 or miRNAs in drug-induced neuroplasticity in brain reward systems that drive the emergence of compuls
34 ed as a cycle of spiralling dysregulation of brain reward systems that progressively increases, resul
35 ltaFosB influences the responsiveness of the brain reward system to rewarding and aversive drugs.
36 evealing how social interactions can recruit brain reward systems to drive changes in affiliative beh
37 a) on the hedonic response and activation of brain-reward systems to food.
38 g psychostimulant-elicited plasticity in the brain reward system, we undertook a phenotype-driven app

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