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1 sclerosis, a disease affecting primarily the brain white matter.
2 ioblastomas was compared with that of normal brain white matter.
3 cement of OX-42 microglial immunostaining in brain white matter.
5 nt T cells in post-mortem multiple sclerosis brain white matter active lesions confirmed their involv
7 microvascular disease predominantly affects brain white matter and deep grey matter, resulting in is
9 ociated with levels of CSF biomarkers across brain white matter and in areas preferentially affected
10 olecular weights of 60- and 65-kDa in normal brain white matter and in astrocytic tumors, with an add
13 eIF2B mutations predominantly affect the brain white matter, and the characteristic features of V
15 hat uses diffusion MRI to characterize whole-brain white matter architecture as a single local connec
19 NA was absent or barely detectable in normal brain white matter but was easily detectable in malignan
20 vel cationic lipid was separated from bovine brain white matter by a series of chromatographies on ca
21 MRI) reveals microstructural features of the brain white matter by quantifying the anisotropic diffus
22 ther the NTRK2 and BDNF polymorphisms impact brain white matter connections in major depressive disor
24 obabilistic tractography analyses calculated brain white matter connectivity (streamlines) as an esti
25 used as early and sensitive markers of human brain white matter connectivity, development, natural ag
26 es correlated with executive function; whole brain white matter correlated with episodic memory, proc
28 -galactosylsphingosine (psychosine) found in brain white matter, enhances p140trk (Trk A) phosphoryla
29 l FAD mutation carriers (n = 12), mean whole brain white-matter FA (P = 0.045), FA of the columns of
31 (i) to characterize the relationship between brain white matter fiber-tract properties and second-lan
32 ted with subtle structural properties of the brain white matter found in multiple sclerosis (P = 0.02
34 hological deficits with the volumes of whole brain white matter hyperintensities and gray and white m
35 ns correlated with executive function; whole brain white matter hyperintensities correlated with exec
36 n mobility, cognition, and mood; the role of brain white matter hyperintensities in mediating this as
37 vascular disease was quantified by measuring brain white matter hyperintensities on fluid attenuation
39 f CA and its relationship to the presence of brain white matter hyperintensity (WMH) in older adults,
40 on (CA) and determined its associations with brain white matter hyperintensity (WMH) in older adults.
41 of 24-hour systolic BP in the progression of brain white matter hyperintensity volume burden associat
42 hy and neurobehavioural, neurocognitive, and brain white matter imaging outcomes in long-term survivo
43 d xenon combined with hypothermia attenuates brain white matter injury in comatose survivors of out-o
44 erences, indicating widespread alteration in brain white matter integrity but not necessarily white m
45 onance imaging-based measurements of reduced brain white matter integrity in the 1-cm radius white ma
47 lationships between 1) delirium duration and brain white matter integrity, and 2) white matter integr
48 ated with subjective cognitive complaints or brain white-matter lesions 5 to 10 years after the hyper
51 ally studying the effects of hemodialysis on brain white matter microstructure and further examine if
52 tested whether there were coupled changes in brain white matter microstructure indexed by fractional
58 n, relative to comparison subjects, in whole-brain white matter, prefrontal and temporal white matter
59 y in the human brain, suggest that the adult brain white matter preserves dynamic characteristics and
60 al analyses conducted across the rest of the brain white matter revealed lower FA bilaterally in the
67 ved hemodynamic tolerability, and changes in brain white matter were associated with hemodynamic inst
69 ypernatraemic rats there was myelinolysis of brain white matter, with karyorrhexis and necrosis of ne
70 usion tensor MR measures of global damage to brain, white matter ( WM white matter ), and GM gray mat
73 genetic and molecular pathways driving human brain white matter (WM) development are only beginning t
75 ensive relations between healthy adult human brain white matter (WM) microstructure and gray matter (
76 difficult bimanual conditions, less optimal brain white matter (WM) microstructure, and a decreased
78 ittle is known about the association between brain white matter (WM) structure and motor function in
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