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1 increase its energy barrier through four-way branch migration.
2 cally bind to Holliday junctions and promote branch migration.
3 ed, the acceptor accessed the cDNA 3' end by branch migration.
4 ical approach to analyze the mechanism of HJ branch migration.
5  exchange of base pairs known as spontaneous branch migration.
6 n, and then recruits two RuvB pumps to power branch migration.
7 s around the junction remain parallel during branch migration.
8 , presumably by blocking strand exchange and branch migration.
9  DNA, promoting DNA annealing, and promoting branch migration.
10 en propagates towards the primer terminus by branch migration.
11 er by promoting strand exchange invasion and branch migration.
12 accomplish additional activities such as DNA branch migration.
13 day junctions, using ATP hydrolysis to drive branch migration.
14     DnaB binds to just one DNA strand during branch migration.
15 strand annealing, and Holliday junction (HJ) branch migration.
16  cooperate to promote homologous pairing and branch migration.
17 ese joint molecules to promote ATP-dependent branch migration.
18 tails that equilibrate to many structures by branch migration.
19 on, because a symmetric junction can undergo branch migration.
20 ining protein required for specific tracheal branch migration.
21 ancing and receding duplexes of an HJ during branch migration.
22 evidence that RuvA has a mechanistic role in branch migration.
23 e found that the crosslink failed to inhibit branch migration.
24 y, possibly by Rqh1 catalysing their reverse branch migration.
25 ating joint molecules and in the polarity of branch migration.
26 s based on the inhibition of spontaneous DNA branch migration.
27  defines the boundaries of Holliday junction branch migration.
28 esolution occurs as the resolvasome promotes branch migration.
29 ng specificity and RuvB drives ATP-dependent branch migration.
30  investigate the thermodynamics of three-way branch migration.
31 sed the role of DNA unwinding in relation to branch migration.
32 g over by interfering with Holliday junction branch migration.
33 n relocate through an isomerization known as branch migration.
34 ence heterology to estimate the step size of branch migration.
35 junction such that RuvBC complexes catalysed branch migration.
36 ge of a Holliday junction during spontaneous branch migration.
37 omologous DNA molecules and subsequent polar branch migration.
38 he Holliday junction in vitro by spontaneous branch migration.
39 e conformational changes in SisPINA to drive branch migration.
40 lacement, strand separation (unwinding), and branch migration.
41 A strands, effectively halting RecA-mediated branch migration.
42 so mismatches almost terminate a spontaneous branch migration.
43          Unfolding of the HJ is required for branch migration.
44 cation fork regression and Holliday junction branch migration.
45 ere that the enzyme efficiently promotes DNA branch migration.
46  as little as a single base stalls catalysed branch migration.
47 of the significance of hybrid propagation by branch migration.
48 late-guided alignment proceeding through DNA branch migration.
49 141R, is unable to promote Holliday junction branch migration.
50 ly replaced by a distinct mode of migration: branched migration.
51 7] [8], a hexameric ring protein that drives branch migration [9] [10] [11].
52 e of this mutant reveals that the ATPase and branch migration activities of RecA are not necessarily
53 possesses exonuclease, strand annealing, and branch migration activities.
54    However, only RPA robustly stimulates WRN branch migration activity and increases the percentage o
55 four-way junction DNA and no DNA helicase or branch migration activity could be detected.
56 erodimer that binds DNA and enhances the DNA branch migration activity of FANCM.
57 ts demonstrate a functional link between the branch migration activity of hRad54 and the structure-sp
58  Rad51 (hRad51) significantly stimulates the branch migration activity of hRad54.
59 onserved, as yeast Rad51 also stimulates the branch migration activity of yeast Rad54.
60                                     However, branch migration activity requires a significantly highe
61  collar at Holliday junctions, promoting DNA branch migration activity while blocking other DNA remod
62 ation of model replication forks through its branch migration activity, but shows strong bias toward
63 rmine whether RPA and POT1 also modulate WRN branch migration activity, we examined biologically rele
64 l requirements for optimal hRad54 ATPase and branch migration activity.
65 manner, and associates with an ATP-dependent branch migration activity.
66 e, the first evidence that RPA can stimulate branch migration activity.
67                                    Novel DNA branch-migration activity is fully consistent with this
68 41 helicase has been shown to catalyze polar branch migration after the T4 gene 59 helicase assembly
69                We propose that RadA-mediated branch migration aids recombination by allowing the 3' i
70                                              Branch migration allows the exchange between homologous
71                      A HJ is able to undergo branch migration along DNA, generating increasing or dec
72 ion flips between conformations favorable to branch migration and conformations unfavorable to it.
73 del in which Sgs1 helicase catalyzes reverse branch migration and convergence of double HJs for noncr
74                             We combined both branch migration and direct dissociation into a model, w
75 gle base pair mismatch in the invader stalls branch migration and displacement occurs via direct diss
76  which UvsW is a DNA helicase that catalyzes branch migration and dissociation of RNA-DNA hybrids.
77 s hitherto uncharacterized protein possesses branch migration and DNA unwinding activity.
78 1 remodels these DNA substrates by promoting branch migration and fork regression.
79       Human RAD51C (hRAD51C) participates in branch migration and Holliday junction resolution and th
80 fication of a protein complex that catalyses branch migration and Holliday junction resolution argues
81 k, the trachea, and show that dVHL regulates branch migration and lumen formation via its endocytic f
82 section of the nascent DNA by RecJ and RecQ, branch migration and processing of the fork DNA surround
83 substitution, deletion or insertion inhibits branch migration and produces stable cruciform structure
84 fractionated human extracts caused a loss of branch migration and resolution activity, but these func
85 enetic and biochemical studies indicate that branch migration and resolution are coupled by direct in
86 al experiments suggest that the processes of branch migration and resolution are linked, coimmunoprec
87 ey also provide insight into the coupling of branch migration and resolution by the RuvABC resolvasom
88 vABC complex that is capable of facilitating branch migration and resolution of Holliday junctions vi
89            RuvABC is a complex that promotes branch migration and resolution of Holliday junctions.
90                                              Branch migration and resolution of these crossovers, or
91 ore process Holliday junctions via uncoupled branch migration and resolution reactions.
92 vided into three key steps: strand exchange, branch migration and resolution.
93  the effect of nicks on the efficiency of HJ branch migration and the dynamics of the HJ.
94 on by degrading the displaced strands during branch migration and thereby causing strand exchange to
95  models using a Holliday junction undergoing branch migration and time-lapse atomic force microscopy,
96  those that can undergo a number of steps of branch migration, and confirm that the enzyme exhibits a
97 volving RNase H cleavage, acceptor invasion, branch migration, and finally primer terminus transfer.
98 ing on DNA to catalyze DNA strand annealing, branch migration, and fork regression.
99 ion unfolding, which accelerates spontaneous branch migration, and individual time traces reveal comp
100 nded DNA structure is capable of spontaneous branch migration, and is lost during standard DNA extrac
101 tion, displacement-loop (D-loop) processing, branch migration, and resolution of double Holliday junc
102 RuvA and RuvB proteins, which together drive branch migration, and RuvC endonuclease, which resolves
103               We demonstrate that p53 blocks branch migration, and that cleavage of the Holliday junc
104 nferred rates for hybridization, fraying and branch migration, and they provide a biophysical explana
105 ated using a sensitive assay for spontaneous branch migration, and was shown to preserve both artific
106 e that RecA, recombinational DNA repair, and branch migration are all important for H(2)O(2) resistan
107 theless, the mechanistic models proposed for branch migration are all predicated on a parallel alignm
108 this assay, alterations in end processing or branch migration are reflected by the frequency of co-co
109 lecule FRET experiments led to the model for branch migration as a stepwise stochastic process in whi
110 f p53 on both spontaneous and RuvAB promoted branch migration as well as the effect on resolution of
111 re, we present a single-molecule spontaneous branch migration assay with single-base pair resolution
112 ase has a defined substrate specificity: the branch migration-associated resolvase is highly specific
113 ce dependence of crossover isomerization and branch migration at symmetric sites has been established
114 imitation of inappropriate recombination and branch migration at telomeric ends.
115 J constructs we were able to follow junction branch migration at the single-molecule level.
116 nge mediated by spontaneous renaturation and branch migration; beta imposed a polarity on the strand
117 eins are known to bind HJs and promote their branch migration (BM) by translocating along DNA at the
118 xample, the combination between toeholds and branch migration (BM) domains is 'hard wired' during DNA
119 Several proteins have been shown to catalyze branch migration (BM) of the Holliday junction, a key in
120            Consistent with the dependence of branch migration (BM) on the ATPase-dependent DNA transl
121  a critical role in modulating its helicase, branch migration (BM), or strand annealing [18, 19].
122 not only to accelerate the intrinsic rate of branch migration but also to facilitate the passage of t
123 opy data show that the nick does not prevent branch migration, but it does decrease the probability t
124 day junction structure during DnaB-catalyzed branch migration, but not during unwinding.
125 Holliday junction when the DNA is undergoing branch migration, but RuvA is immobile at the same junct
126  the limits of a previous approach to thwart branch migration, but the design of the 12-arm junction
127 lliday junctions and catalyses ATP-dependent branch migration, but the equivalent proteins in archaea
128 rate that the stimulation of hRad54-promoted branch migration by hRad51 is driven by specific protein
129                 On the other hand, extensive branch migration by RecA, where a completely unwound pro
130  Furthermore, we show that TWINKLE catalyzes branch migration by resolving homologous four-way juncti
131 ing binding to duplex DNA and also constrain branch migration by RuvAB in a manner critical for junct
132                                              Branch migration by RuvAB is mediated by RuvB, a hexamer
133 chastic process of the junction dynamics and branch migration by the variability of the time that the
134 and those of hRad54 are to promote efficient branch migration, bypass potential mismatches and facili
135 uble crossover molecules to demonstrate that branch migration can occur in antiparallel Holliday junc
136     Our kinetic studies of Holliday junction branch migration catalysed by a ring-shaped helicase, T7
137         This process involves two key steps: branch migration, catalysed by the RuvB protein that is
138 vestigated the interaction between the RuvAB branch migration complex and the Topo IV.quinolone.DNA t
139         These results suggest that the RuvAB branch migration complex can actively remove quinolone-i
140                                   RuvAB is a branch migration complex that stimulates heterologous st
141 aced single-stranded DNA tail that indicates branch migration could be observed.
142 tion with EcRuvB, it was unable to stimulate branch-migration-dependent resolution in a RuvABC comple
143 ons; these are junctions that cannot undergo branch migration, despite the fact that they are flanked
144           Strand exchange then propagates by branch migration, displacing the fragmented donor RNA, i
145 itting an input strand into an A-toehold and branch migration domain.
146  a DNA structure that brings a toehold and a branch-migration domain into close proximity can catalyz
147 ellular data that support RPA enhancement of branch migration during HR repair and, conversely, POT1
148 nt with approximately 0-1, 1-2 and 2-3 bp of branch migration during recombination reactions involvin
149  been proposed to act as a Holliday junction branch migration enzyme.
150 r, MlRuvA formed functional DNA helicase and branch-migration enzymes with EcRuvB, although the heter
151            The restricted molecule undergoes branch migration, even though it is constrained to an an
152 -cDNA hybrid is thought to then propagate by branch-migration, eventually catching up with the primer
153                                Comparison of branch migration experiments through a single base-pair
154 tic Holliday junction was used as substrate, branch migration facilitated by Sep1 could not be detect
155                                 Unlike other branch migration factors RecG and RuvAB, RadA stimulates
156 ons stabilize folded conformations and stall branch migration for a period considerably longer than t
157 ix; however, to date, their effect on the HJ branch migration has not been studied.
158          Both DNA structural transitions and branch migration have been used as the basis for the ope
159     All attempts at modeling the kinetics of branch migration have relied on the assumption that bran
160      The data obtained support the model for branch migration having the extended conformation of the
161 s a stepwise stochastic process in which the branch migration hop is terminated by the folding of the
162  underlying molecular basis for the block to branch migration imposed by sequence heterology.
163 mediated DNA replication fork regression and branch migration in a model substrate.
164 model system has been developed for studying branch migration in a small synthetic four-arm junction.
165 ng duplex DNA while facilitating and biasing branch migration in a specific direction.
166 specificity and promotes their bidirectional branch migration in an ATPase-dependent manner.
167                                 Furthermore, branch migration in both directions is equally impeded b
168                   It also promotes efficient branch migration in combination with RuvA, and forms fun
169  the fully exchanged molecules resulted from branch migration in either direction depending on which
170 ese results, we conclude that Rad51-promoted branch migration in either direction occurs fundamentall
171 e we report that the rates of Rad51-mediated branch migration in either the 5'- to 3'- or 3'- to 5'-d
172 ere, we report direct real-time detection of branch migration in individual molecules.
173 that MutS,L directly inhibit RuvAB-dependent branch migration in the absence of RecA.
174 nirps-related rescues dorsal but not ventral branch migration in the breathless hypomorph.
175 ose formed by wild-type protein and promotes branch migration in the presence of RuvA.
176 ures and the ability to detect inhibition of branch migration in these structures.
177   The results imply that Sep1 cannot promote branch migration in vitro.
178              The results suggest a model for branch migration in which DNA is pumped out of the small
179 e basis of these data, we propose a model of branch migration in which the propensity of the junction
180               These data suggest a model for branch migration in which the sequence modulates the ove
181 ch-migration subunit.Whereas MlRuvA promoted branch-migration in combination with EcRuvB, it was unab
182 is not required for RuvA mobilization during branch migration, in contrast to previous proposals.
183 ng of a single base pair and (ii) initiating branch migration incurs a thermodynamic penalty, not cap
184  displacement processes (toehold-binding and branch migration) independently, and information can be
185                                Inhibition of branch migration indicates the presence of sequence alte
186       This mutation detection method, termed branch migration inhibition (BMI), is suitable for the d
187                                          DNA branch migration is a fundamental process in genetic rec
188 ce studies led to a model according to which branch migration is a stepwise process consisting of con
189 ce studies led to a model according to which branch migration is a stepwise process consisting of con
190                                   In E.coli, branch migration is catalysed by the RuvB protein, a hex
191                                              Branch migration is detected as a stepwise random proces
192     The free energy landscape of spontaneous branch migration is found to be highly nonuniform and go
193 e heterology suggests that the inhibition of branch migration is largely attributable to a thermodyna
194 pendently, we conclude that the step size of branch migration is quite small, of the order of one or
195                                              Branch migration is the process by which the exchange of
196 ase activity in vitro [12], the mechanism of branch migration is thought to involve DNA opening withi
197                                              Branched migration is cell-autonomous, associated with i
198 ns interact at Holliday junctions to promote branch migration leading to the formation of heteroduple
199                                              Branch migration leads to polydispersity, which makes it
200  a junction along DNA, by a process known as branch migration, leads to heteroduplex formation, where
201 The replication fork helicase DnaB catalyzes branch migration like RuvB but, unlike RuvB, is not depe
202 ndonuclease acting in concert with the RuvAB branch migration machinery.
203      Results suggest that both proximity and branch migration mechanisms contribute to transfers, wit
204                  However it does not support branch migration mediated by E. coli RuvB and when bound
205 migration have relied on the assumption that branch migration minima are sequence-independent.
206  of wild-type NDEL1 levels displayed diverse branched migration modes with multiple leading processes
207 sequence heterologies exert their effects on branch migration more or less independently, we conclude
208  resolvase does not interact directly with a branch migration motor, which simplifies analysis of its
209 nction via a mechanism that involves neither branch migration nor helical restacking.
210                                ATP-dependent branch migration occurs as duplex DNA is pumped out thro
211 e physical process by which a single step of branch migration occurs is significantly slower than the
212 trand exchange, allowing the moment at which branch migration occurs to be detected.
213                                              Branch migration of a DNA Holliday junction is a key ste
214 nd that RuvA does not inhibit DnaB-catalyzed branch migration of a homologous junction, even at high
215 e encircling two DNA strands, DnaB can drive branch migration of a synthetic Holliday junction with h
216 or 2D agarose gel analysis are favorable for branch migration of asymmetrically replicating nascent s
217 ve developed a rapid protocol that restrains branch migration of four-way DNA junctions.
218 a there are specialized enzymes that promote branch migration of HJs.
219 a junction-specific DNA helicase that drives branch migration of Holliday intermediates in genetic re
220                  The RuvAB proteins catalyze branch migration of Holliday junctions during DNA recomb
221 chia coli RuvA and RuvB proteins promote the branch migration of Holliday junctions during the late s
222 , we also demonstrate that UvsW promotes the branch migration of Holliday junctions efficiently throu
223        Two key steps in this process are the branch migration of Holliday junctions followed by their
224 ons, but the controversy on the mechanism of branch migration of Holliday junctions remains unsolved.
225 ults strongly support a role for UvsW in the branch migration of Holliday junctions that form during
226                        RuvA and RuvB promote branch migration of Holliday junctions, a process that e
227                     In vitro, Rad54 promotes branch migration of Holliday junctions, whereas the Mus8
228                            BLM also promotes branch migration of Holliday junctions.
229 vB protein complex can promote ATP-dependent branch migration of Holliday junctions.
230 -B-form DNA, such as G-quadruplexes, and the branch migration of Holliday junctions.
231 ng duplex DNA, enabling the protein to drive branch migration of Holliday junctions.
232 ights into how RuvB functions as a motor for branch migration of Holliday junctions.
233 bacterial RuvB DNA helicase, which catalyses branch migration of Holliday junctions.
234 51-mediated DNA strand exchange and promotes branch migration of Holliday junctions.
235 cilitate long-range strand exchanges through branch migration of Holliday junctions.
236     We previously showed that Rad54 promotes branch migration of Holliday junctions.
237  core complex component FANCM also catalyzes branch migration of model Holliday junctions and replica
238 NA), the MmsA protein appears to promote the branch migration of partially exchanged intermediates in
239  DNA helicase believed to be involved in the branch migration of recombinational intermediates.
240                        Propagation by simple branch migration of strands was limited to 24-32 nt.
241 rase partnered with a helicase by convergent branch migration of the HJs.
242   The E. coli RuvAB protein complex promotes branch migration of the Holliday junction recombination
243  as molecular motor proteins responsible for branch migration of the Holliday junction(s) and reversa
244                                              Branch migration of this four-stranded DNA structure is
245                 Both RuvAB and RecG catalyze branch migration of three- and four-stranded DNA junctio
246 acetyllactosamine (LacNAc) epitopes, induces branching migration of mammary epithelia in vivo, ex viv
247                             The movement, or branch migration, of this junction is necessary for reco
248 cinity of a bulge and reducing the impact of branch migration on primer extension.
249               These results demonstrate that branch-migration per se and the assembly of a RuvABC com
250        These results indicate that it is the branch migration phase (and not the initial pairing step
251  a DNA-binding protein and can stimulate the branch migration phase of RecA-mediated strand transfer
252 e of the folded conformations terminates the branch migration phase.
253 cules from any type of ends on the dsDNA and branch migration proceeds exclusively in the 5'- to 3'-d
254         Our present experiments confirm that branch migration proceeds in either direction, the polar
255 r 5' overhanging end of the linear dsDNA and branch migration proceeds in either direction.
256 n initiated with a 3' or 5' overhanging end, branch migration proceeds more rapidly when it is initia
257 iation of double D-loop DNA hybrids is a DNA branch migration process involving the rotation of both
258 ompetitive displacement technique mimics the branch migration process that occurs during DNA recombin
259 ilitate the invasion step and/or the ensuing branch migration process.
260 e sequence on Holliday junction dynamics and branch migration process.
261    These findings suggest that p53 can block branch migration promoted by proteins such as RuvAB and
262 is highly homologous to that of the Holliday branch migration protein RecG.
263 ong synergistic phenotypes with those in the branch migration protein RecG.
264 anner that requires the presence of the RecG branch migration protein.
265 tes the identity of homologous recombination branch-migration protein(s) has remained elusive.
266        To gain insight into the mechanism of branch migration, random mutations were introduced into
267                                         This branch migration reaction is inhibited by SSB, possibly
268 ularly, the concept of "toehold-mediated DNA branch migration reactions" has attracted considerable a
269  be used for subsequent toehold-mediated DNA branch migration reactions, e.g., DNA hybridization chai
270           Together, these activities promote branch migration/resolution reactions similar to those c
271                              Spontaneous DNA branch migration results in dissociation of these struct
272 ase to form complexes that catalyse junction branch migration (RuvAB) and resolution (RuvABC).
273 on Holliday junction DNA that drives coupled branch migration (RuvAB) and resolution (RuvC) reactions
274 re recombination intermediates--and promotes branch migration; RuvC is a junction-specific endonuclea
275        Characterization of the reconstituted branch migration substrates by micrococcal nuclease mapp
276            Similar results are obtained with branch migration substrates containing an octamer positi
277  RuvA homologue of M. leprae is a functional branch-migration subunit.Whereas MlRuvA promoted branch-
278 ation, strand-annealing, strand-exchange and branch migration suggest a dual role of TWINKLE in mitoc
279                           Kinetic studies of branch migration suggest an alternative model in which t
280         T7 phage gp4 protein also drives DNA branch migration, suggesting this activity generalizes t
281                    Models for RuvAB-mediated branch migration that invoke only limited DNA unwinding
282 ctions adopt an unfolded conformation during branch migration that is retained despite a broad range
283 ecules to undergo an isomerization, known as branch migration, that relocates the site of the branch
284 uld function during recombination to promote branch migration through chromatin.
285               We also devise a simulation of branch migration through mismatched base-pairs to arrive
286      In the presence of RuvAB and ATP, rapid branch migration through the nucleosome is observed.
287 s that breathless and pointed control dorsal branch migration through transcriptional regulation of t
288 rimer and acceptor strands then propagate by branch migration to catch the advancing primer terminus.
289 tween homologous DNA molecules but can drive branch migration to extend the region of heteroduplex DN
290 nor RNA, the cDNA-acceptor hybrid expands by branch migration until transfer of the primer terminus i
291              Two models for the mechanism of branch migration were suggested.
292 izing Holliday junctions against spontaneous branch migration when ATP is not present.
293 tudies have shown that RuvA and RuvB promote branch migration whereas RuvC resolves junctions by endo
294 54 and hRad51 during DNA strand exchange and branch migration, which are two core steps of homologous
295 ergo a conformational isomerization known as branch migration, which relocates the site of branching.
296 ECQ1 catalyzes unidirectional three-stranded branch migration with a 3' --> 5' polarity.
297  direction and catalyses strand exchange and branch migration with a 5'-3' polarity.
298 strates that simultaneously exploit four-way branch migration, with a high-energy barrier to minimize
299 y barrier to minimize leakage, and three-way branch migration, with a low-energy barrier to maximize
300 tion factors RecG and RuvAB, RadA stimulates branch migration within the context of the RecA filament

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