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1 rsor of brassinolide (the most active of the brassinosteroids).
2 factors, such as the phytohormones auxin and brassinosteroid.
3 auxin, strigolactone, gibberellic acid, and brassinosteroids.
4 of responses to the plant hormones auxin and brassinosteroids.
5 5, two defective transmembrane receptors for brassinosteroids.
6 and in alterations in response to auxin and brassinosteroids.
7 into the regulatory mechanisms of bioactive brassinosteroids.
8 critical component of a receptor complex for brassinosteroids.
9 to three brassinosteroid-biosynthesis genes (BRASSINOSTEROID-6-OXIDASE, CONSTITUTIVE PHOTOMORPHOGENIC
14 isic acid (ABA), and hyposensitive to auxin, brassinosteroid and cytokinin, but normally responsive t
15 uxin affected the expression of a few genes; brassinosteroid and gibberellin had only modest effects.
16 tly, TT8 affects stress response, along with brassinosteroid and jasmonic acid biosynthesis, by direc
17 mutant displayed reduced responses to GA and brassinosteroid and showed decreased expression of sever
18 we report evidence that interactions between brassinosteroids and auxin signaling modulate phototropi
20 alyses also suggested an induction of auxin, brassinosteroid, and gibberellin signatures and the invo
23 Moreover, shoot growth is restricted when brassinosteroids are depleted from the epidermis and bra
28 OMATIC EMBRYOGENESIS RECEPTOR KINASE (SERK)3/brassinosteroid-associated kinase (BAK)1, a receptor-lik
29 fied a R2R3-MYB transcription factor, BRAVO (BRASSINOSTEROIDS AT VASCULAR AND ORGANIZING CENTER), act
30 ants, steroid hormone action occurs when the brassinosteroids bind a membrane tyrosine kinase recepto
32 id, carotenoid, oxylipin, glucosinolate, and brassinosteroid biosyntheses and have shown that both P4
34 ng sturdy culm, specific for deficiencies in brassinosteroid biosynthesis and signaling in semidwarf
37 1 (DWF1) is responsible for an early step in brassinosteroid biosynthesis that converts 24-methylenec
38 igned more than 20 historic mutants to three brassinosteroid-biosynthesis genes (BRASSINOSTEROID-6-OX
39 ression of the brassinosteroid receptor or a brassinosteroid biosynthetic enzyme in the epidermis, bu
40 studies have demonstrated that cytokinin and brassinosteroid (BR) act as regulatory inputs to elevate
41 to investigate the relationship between the brassinosteroid (BR) and gibberellin (GA) hormones acros
42 A paradigmatic case is the concurrence of brassinosteroid (BR) and gibberellin (GA) signaling in t
48 coding sterol C-22 hydroxylases that control brassinosteroid (BR) hormone levels using a promoter tha
52 BES1 functions as a master regulator in the brassinosteroid (BR) pathway that promotes plant growth.
53 onic acid (JA) pathway was induced while the brassinosteroid (BR) pathway was suppressed in infected
54 nase that functions as a coreceptor with the brassinosteroid (BR) receptor BRI1 and the flagellin rec
55 urally defective yet biochemically competent brassinosteroid (BR) receptor BRI1, resulted in identifi
56 urally imperfect yet biochemically competent brassinosteroid (BR) receptor bri1-9, causing its increa
57 EBS1 gene suppressed the growth defects of a brassinosteroid (BR) receptor mutant, bri1-9, in an alle
61 nome-wide screen for mutants showing altered brassinosteroid (BR) sensitivity or bri1-like phenotypes
64 s GSK3-like kinase that negatively regulates brassinosteroid (BR) signaling by phosphorylating BES1 (
71 s in plant immunity, cell-death control, and brassinosteroid (BR) signaling, all four functional SERK
72 NSITIVE2 (BIN2), a key negative regulator of brassinosteroid (BR) signaling, can phosphorylate Arabid
73 e pattern; instead, the detailed analysis of brassinosteroid (BR) signaling, innate immunity, and sym
74 PRs of either the negative regulators of the brassinosteroid (BR) signaling, the glycogen synthase ki
80 we show that the activity of two homologous brassinosteroid (BR) transcriptional effectors, BRASSINA
81 enous signals, including light, temperature, brassinosteroid (BR), and gibberellin (GA), regulate cel
82 monal signals, involving light, temperature, brassinosteroid (BR), and gibberellin, that inhibit the
83 coding predicted proteins involved in auxin, brassinosteroid (BR), gibberellic acid (GA), abscisic ac
85 nstrating the role of BRAVO in counteracting Brassinosteroid (BR)-mediated cell division in the QC ce
86 pattern (PAMP)-triggered immunity (PTI) and brassinosteroid (BR)-mediated growth was recently report
88 through TOR controls the accumulation of the brassinosteroid (BR)-signaling transcription factor BZR1
91 uxin, gibberellic acid (GA), cytokinin (CK), brassinosteroids (BR) and peptide hormones are also impl
94 well as intrinsic signals such as hormones (brassinosteroid [BR], auxin, cytokinin, ethylene) and nu
96 es have indicated that CYP734A1 binds active brassinosteroids, brassinolide and castasterone, as well
110 strong evidence that steroid hormones called brassinosteroids (BRs) are required to maintain position
117 ) is the main ligand-perceiving receptor for brassinosteroids (BRs) in Arabidopsis (Arabidopsis thali
118 to investigate a role for the phytohormones brassinosteroids (BRs) in specifying bristle identity an
134 at are coordinated by hormones including the brassinosteroids (BRs), a group of steroids with structu
137 terplay of light and plant hormones, such as brassinosteroids (BRs), in the regulation of plant growt
145 kinase, BRI1, is a cell-surface receptor for brassinosteroids (BRs), the steroid hormones of plants,
147 3 double mutant roots are insensitive toward brassinosteroids but have a phenotype different from bri
148 the largely differing response to zeatin and brassinosteroid by the metabolic pathways in chloroplast
151 nts show that the minimal binding domain for brassinosteroids consists of a 70-amino acid island doma
153 ctivities of downstream MAPKs are reduced in brassinosteroid-deficient mutants but increased by treat
156 iption and integrate the auxin response to a brassinosteroid-dependent molecular circuit that promote
157 nthesis of structural sterols from signaling brassinosteroid derivatives and are highly regulated.
158 A15 (SAUR15), a well-characterized auxin and brassinosteroid early response gene in Arabidopsis (Arab
159 the mechanism by which another phytohormone, brassinosteroid, elevates ethylene biosynthesis in etiol
160 Overexpression of the HBI1-related bHLHs brassinosteroid enhanced expression2 (BEE2) and cryptoch
161 helix (bHLH) transcription factor homolog of brassinosteroid enhanced expression2 interacting with IB
162 binding of either transcription factor, and brassinosteroid enhancement of MP/ARF5 binding required
163 osynthesis of plant hormones (e.g. ethylene, brassinosteroid, gibberellic acid) were significantly ch
164 growth-regulating signals, including auxin, brassinosteroid, gibberellin, light, and temperature.
165 tential to fine-tune the levels of different brassinosteroid hormones throughout plant growth and dev
167 , abscisic acid (ABA), cytokinins (CKs), and brassinosteroids in molding plant-pathogen interactions.
168 and CYP72C1 in Arabidopsis diverge more than brassinosteroid inactivating P450s in other plants, this
169 YP734A1 and CYP72C1, have been identified as brassinosteroid-inactivating enzymes important for stero
170 onsible for regulating the levels of several brassinosteroids, including typhasterol, castasterone an
173 s capability to specifically suppress a weak brassinosteroid insensitive 1 (bri1) allele, bri1-5, whe
174 Recently, two Arabidopsis receptor kinases--BRASSINOSTEROID INSENSITIVE 1 (BRI1) and BRI1-ASSOCIATED
175 ntaining two transmembrane receptor kinases, BRASSINOSTEROID INSENSITIVE 1 (BRI1) and BRI1-ASSOCIATED
177 ity of the dual-specificity receptor kinase, brassinosteroid insensitive 1 (BRI1), reflects the balan
178 ed in BR perception and signal transduction: brassinosteroid insensitive 1 (BRI1), which is the BR re
181 LLIN-SENSING 2 (FLS2) with their co-receptor BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED KINASE 1 (BAK1)
182 atic embryogenesis receptor kinase 3 (SERK3)/brassinosteroid insensitive 1-associated kinase 1 (BAK1)
183 brane-resident RLKs, flagellin-sensing 2 and brassinosteroid insensitive 1-associated kinase 1 (BAK1)
184 ll death was shown to require N. benthamiana Brassinosteroid insensitive 1-Associated Kinase 1 (NbBAK
187 ense responses required the known coreceptor BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED RECEPTOR KINASE
188 ctivation of the Arabidopsis thaliana kinase BRASSINOSTEROID INSENSITIVE 1-ASSOCIATED RECEPTOR-LIKE K
192 Arabidopsis thaliana receptor-like kinases, brassinosteroid-insensitive 1 (BRI1) and BRI1-associated
193 growth and development and require an active BRASSINOSTEROID-INSENSITIVE 1 (BRI1) receptor serine/thr
194 gh plasma membrane-localized receptor kinase brassinosteroid-insensitive 1 (BRI1), BRI1-associated re
196 lycogen synthase kinase 3 (GSK3)-like kinase brassinosteroid-insensitive 2 (BIN2), which has been wel
197 LYCOGEN SYNTHASE KINASE 3)-like kinase BIN2 (BRASSINOSTEROID-INSENSITIVE 2), a well established negat
199 ifferent ligand-perceiving receptors such as BRASSINOSTEROID INSENSITIVE1 (BRI1) and FLAGELLIN-SENSIT
204 plasma membrane through the receptor kinase BRASSINOSTEROID INSENSITIVE1 (BRI1) together with co-rec
205 of the leucine-rich repeat receptor kinase, brassinosteroid insensitive1 (BRI1), was shown to depend
209 avily glycosylated brassinosteroid receptor, BRASSINOSTEROID INSENSITIVE1, while MNS1 to MNS3 appear
211 exes between the membrane-localized IOS1 and BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE1 (BAK1)-d
212 the leucine-rich repeat receptor-like kinase BRASSINOSTEROID INSENSITIVE1-ASSOCIATED KINASE1 (BAK1)/S
213 flagellin sensing2 and its signaling partner brassinosteroid insensitive1-associated kinase1 is obser
215 dent on the regulatory receptor-like kinases brassinosteroid insensitive1-associated receptor kinase1
216 seedling lethal and cell death phenotypes of BRASSINOSTEROID INSENSITIVE1-associated receptor kinase1
218 th these findings, the transcription factors BRASSINOSTEROID INSENSITIVE1-EMS SUPPESSOR1 and MONOPTER
220 ffectors, BRASSINAZOLE RESISTANT1 (BZR1) and BRASSINOSTEROID INSENSITIVE1-ETHYL METHANESULFONATE-SUPP
221 growth and development and require an active BRASSINOSTEROID-INSENSITIVE1 (BRI1) and BRI1-ASSOCIATED
222 The leucine-rich repeat receptor-like kinase BRASSINOSTEROID-INSENSITIVE1 (BRI1) is the main ligand-p
224 al transduction cascade, as is the case with BRASSINOSTEROID-INSENSITIVE1 Associated Kinase1 and CPK5
228 Here, we demonstrate that the protein kinase BRASSINOSTEROID INSENSITIVE2 (BIN2), a key negative regu
229 of which carry gain-of-function mutations in BRASSINOSTEROID-INSENSITIVE2 (BIN2), one of the three me
230 In addition, auxin, cytokinin, ethylene, brassinosteroids, jasmonates, and salicylic acid also co
231 pression data, the SUB1A genotype had higher brassinosteroid levels after submergence compared to the
235 of the mutant is not rescued by gibberellin, brassinosteroid, or indoleacetic acid application and is
239 haracterized roles in stamen development and brassinosteroid perception, SERK1 plays a unique role in
242 iterpenoids, which include membrane sterols, brassinosteroid phytohormones, and non-steroidal triterp
248 he auxin translocators PIN2 and AUX1 and the brassinosteroid receptor BRI1 into distinct endomembrane
249 d cell-death control by interacting with the brassinosteroid receptor BRI1, immune receptors, such as
250 ession of phenotypes caused by the defective brassinosteroid receptor bri1-9 strongly suggest that th
257 sfolded variants of the heavily glycosylated brassinosteroid receptor, BRASSINOSTEROID INSENSITIVE1,
258 ich are caused by ER retention and ERAD of a brassinosteroid receptor, BRASSINOSTEROID-INSENSITIVE 1
260 rmation with a second receptor kinase termed brassinosteroid receptor1 associated kinase1 (BAK1).
263 Here we demonstrate in Arabidopsis that brassinosteroid regulates stomatal development by activa
264 tudy provides insights into the mechanism of brassinosteroid regulation of root hair patterning.
267 tematically examined root hair phenotypes in brassinosteroid-related mutants, and found that brassino
268 use dwarf, brassinosteroid biosynthesis and brassinosteroid response mutants in conjunction with tis
269 teroid signaling components to attenuate the brassinosteroid response, thus illuminating one aspect o
271 sic acid, gibberellin, methyl jasmonic acid, brassinosteroid, salicylic acid), chemicals (clofibrate,
272 xpression of genes involved in GA, auxin and brassinosteroid signaling as well as cell elongation/exp
273 ein in rice directly interacts with critical brassinosteroid signaling components to attenuate the br
274 d gene expression demonstrate that auxin and brassinosteroid signaling function interdependently.
275 ssinosteroid-related mutants, and found that brassinosteroid signaling inhibits root hair formation t
282 s associated with cell wall modification and brassinosteroid signaling were induced under enriched FR
284 olved in auxin, abscisic acid, ethylene, and brassinosteroid signaling, peroxisome function, disease
285 rt through regulation of auxin transport and brassinosteroid signaling, two processes that are crucia
289 HOTOMORPHOGENIC DWARF, and DIMINUTO) and one brassinosteroid-signaling gene (BRASSINOSTEROID-INSENSIT
291 regulated transcription factor PIF4, and the brassinosteroid-signaling transcription factor BZR1, int
293 lyses indicate that receptor kinase-mediated brassinosteroid signalling inhibits stomatal development
295 members of the CYP734A subfamily inactivate brassinosteroids through C-26 hydroxylation, the biochem
296 ant MAPKKK to its upstream regulators and of brassinosteroid to a specific developmental output.
297 first evidence for direct binding of active brassinosteroids to BRI1 using a biotin-tagged photoaffi
300 ASE1 (BAK1), the receptor and coreceptor for brassinosteroids--were shown to autophosphorylate on tyr
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