1 ult in sister chromatid fusion and prolonged
breakage/fusion/bridge (
B/F/B) cycles, leading to extens
2 duplication such as tandem duplications and
breakage/fusion/bridge (
B/F/B) cycles.
3 Breakage-fusion-bridge (
BFB) cycle is a series of chromo
4 DNA amplification, most notably seen in the
breakage-fusion-bridge (
BFB) cycle.
5 mechanism of DNA amplification involves the
breakage-fusion-bridge (
BFB) cycle.
6 seen in tumor cell lines that have undergone
breakage-fusion-bridge (
BFB) cycles leading to gene ampl
7 te, which subsequently undergoes a series of
breakage-fusion-bridge (
BFB) cycles.
8 Breakage-fusion-bridge (
BFB) is a mechanism of genomic i
9 It is thought to arise through a
breakage-fusion-bridge (
BFB) mechanism.
10 new dicentric undergoes the chromosome-type
breakage-fusion-bridge cycle and produces double bridges
11 Dicentric chromosomes undergo a
breakage-fusion-bridge cycle as a consequence of having
12 hase spindle elongation is delayed and a DNA
breakage-fusion-bridge cycle ensues that is dependent on
13 chromosomes derived from the chromosome type
breakage-fusion-bridge cycle was examined for the presen
14 n chromosomes undergoing the chromosome type
breakage-fusion-bridge cycle were examined by FISH.
15 usion, perhaps through the initiation of the
breakage-fusion-bridge cycle.
16 ric chromosomes, thus starting a devastating
breakage-fusion-bridge cycle.
17 that subsequently initiates a chromatid-type
breakage-fusion-bridge cycle.
18 plex chromosomal rearrangements initiated by
breakage-fusion-bridge cycles and completed by simultane
19 In sporadic iAMP21,
breakage-fusion-bridge cycles are typically the initiati
20 Breakage-fusion-bridge cycles contribute to chromosome i
21 Breakage-fusion-bridge cycles followed by chromothripsis
22 15;21)c to be constitutionally dicentric and
breakage-fusion-bridge cycles generate dicentric chromos
23 Perpetuation of
breakage-fusion-bridge cycles in CML progenitors was med
24 chromosomes that protect against chromosomal
breakage-fusion-bridge cycles in dividing cells.
25 nds, which initiate McClintock's chromosomal
breakage-fusion-bridge cycles in maize.
26 wed by chromosome instability resulting from
breakage-fusion-bridge cycles involving the sister chrom
27 equent than fragments in the second mitosis,
breakage-fusion-bridge cycles possibly occurred during g
28 result in fusions which initiate chromosomal
breakage-fusion-bridge cycles, causing genomic instabili
29 Formation of long palindromes, through
breakage-fusion-bridge cycles, is thought to play an ear
30 epeatedly generates palindromic DNA, such as
Breakage-Fusion-Bridge cycles.
31 they generate new DSBs downstream of IgH via
breakage-fusion-bridge cycles.
32 a rapid increase in DNA content and trigger
breakage-fusion-bridge cycles.
33 of telomeric fusions indicative of multiple
breakage-fusion-bridge cycles.
34 t seal end-to-end fusions, in the absence of
breakage-fusion-bridge cycles.
35 duplications have been attributed solely to
breakage-fusion-bridge cycles.
36 by de novo telomere synthesis, or multistep
breakage-fusion-bridge cycles.
37 e amplification, suggested the occurrence of
breakage-fusion-bridge cycles.
38 ht be the result of cytoskeletal defects and
breakage-fusion-bridge cycles.
39 original stocks developed in the 1940s, via
breakage-fusion-bridge cycles.
40 rearranged, and corroded through hundreds of
breakage-fusion-bridge cycles.
41 mplification in these regions reminiscent of
breakage/fusion/bridge cycles.
42 inute chromosome formation (MYC and MDM2) or
breakage-fusion-bridge (
KRAS, MDM2 and RFC3).
43 omosome damage, repair, and damage through a
breakage-fusion-bridge mechanism.
44 ases of amplifications are compatible with a
breakage-fusion-bridge mechanism.
45 in (IgH)/c-myc coamplification mediated by a
breakage-fusion-bridge mechanism.
46 -myc through an intra- or interchromosome 12
breakage-fusion-bridge mechanism.
47 ic chromosomes and c-myc amplification via a
breakage-fusion-bridge mechanism.
48 Cycles of
breakage-fusion-bridge result in amplification of IgH/c-
49 g that the terminal deletions may occur by a
breakage-fusion-bridge type mechanism.