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1 mor virus and a human retrovirus cloned from breast cancer tissue.
2 r suppressor and has oncogenic capacities in breast cancer tissue.
3 that control the expression of aromatase in breast cancer tissue.
4 thought that SF1 protein is not expressed in breast cancer tissue.
5 the nuclear proteins interacting with S1 in breast cancer tissue.
6 metal-tagged biomarker in a thin section of breast cancer tissue.
7 ers I.3/II, which are preferentially used in breast cancer tissue.
8 related with beta-catenin and PKM2 levels in breast cancer tissues.
9 ethylation in human breast cell lines and T1 breast cancer tissues.
10 d from as low as 50-100 pg of total RNA from breast cancer tissues.
11 ated using 6 breast cancer cell lines and 10 breast cancer tissues.
12 EF (hSEF), is also expressed in a variety of breast cancer tissues.
13 ence of a variant phosphorolytic activity in breast cancer tissues.
14 er characterized by in situ hybridization to breast cancer tissues.
15 gene in breast cancer cell lines and primary breast cancer tissues.
16 UC-1 and erbB-2 were performed on 67 primary breast cancer tissues.
17 locus in a different region of the genome of breast cancer tissues.
18 ressed in three breast cancer cell lines and breast cancer tissues.
19 RNA, is significantly overexpressed in human breast cancer tissues.
20 s increased levels of both KLF4 and PRMT5 in breast cancer tissues.
21 candidate for detection of MGA expression in breast cancer tissues.
22 was selectively and significantly higher in breast cancer tissues.
23 ene signature" that is distinct from primary breast cancer tissues.
24 of breast cancer cell lines as well as human breast cancer tissues.
25 ntration of pVHL and accumulation of KLF4 in breast cancer tissues.
26 negatively correlated with CD8(+) T cells in breast cancer tissues.
27 miR-155 were frequently detected in invasive breast cancer tissues.
28 but not EGFR, was observed in HER2-amplified breast cancer tissues.
29 lates with centrosome amplification in human breast cancer tissues.
31 tochemically) was significantly decreased in breast cancer tissues (although the transcript levels we
33 ssion of RORalpha was downregulated in human breast cancer tissue and cell lines, and that reduced mR
34 correlate with ErbB3 levels in primary human breast cancer tissue and in a mouse model of ErbB2 mamma
36 tological studies of BRCA1 protein in frozen breast cancer tissue and MCF7 and HeLa cell lines reveal
39 to further characterize metabolic changes in breast cancer tissue and the tumor microenvironment.
40 coexpression patterns of hormonal markers in breast cancer tissue and their relationship with patholo
42 icantly, we also found that, out of 51 human breast cancer tissues and 10 normal controls examined, p
44 -1 was also expressed at high levels in both breast cancer tissues and breast cancer cells when compa
45 and protein expression are down-regulated in breast cancer tissues and cell lines compared with adjac
50 sine kinase is absent or reduced in invasive breast cancer tissues and cell lines; its loss in breast
51 differentially expressed as a 1.35 kb RNA in breast cancer tissues and cell-lines, and in several nor
52 the predicted E-cadherin-targeting miRNAs in breast cancer tissues and cells showed that miR-221 was
54 ase polymerase chain reaction in 915 primary breast cancer tissues and correlated with known clinicop
55 man glioblastoma cell line U87MG and primary breast cancer tissues and found that 26-45% of all genes
57 maging signal was increased significantly in breast cancer tissues and highly correlated with ex vivo
58 east cancer, demonstrable with primary human breast cancer tissues and human breast cancer cell lines
59 d that DNMT1 protein levels were elevated in breast cancer tissues and in MCF-7 breast cancer cells r
60 overexpression is found in 46 of 79 primary breast cancer tissues and is associated with high tumor
61 f ERalpha in breast cancer cell lines, human breast cancer tissues and Runx3(+/-) mouse mammary tumor
62 ficant difference in hGH mRNA levels between breast cancer tissues and their normal counterparts, alt
63 we investigated whether CHK is expressed in breast cancer tissues and whether it participates in the
64 matase (estrogen synthetase) is expressed in breast cancer tissue, and in situ expression of the enzy
65 essed in both cancer cell lines and advanced breast cancer tissues, and the levels of TRIM28 and TWIS
66 ariable in different breast cancer cells and breast cancer tissues, and was found to be localized in
68 sis showed that higher levels of sGCbeta1 in breast cancer tissues are correlated with greater surviv
71 protein p54(nrb)/Nono is highly expressed in breast cancer tissues as compared with the adjacent norm
72 expression in a significant number of human breast cancer tissues as well as in many established bre
75 test to identify truncated BRCA2 proteins in breast cancer tissue biopsies in vivo that does not use
76 trophil elastase was expressed by TAN within breast cancer tissues but not by breast cancer cells.
77 s were 37 kDa, whereas in a large portion of breast cancer tissues, but not normal control tissues, o
80 d HER2 are colocalized in plasma membrane of breast cancer tissue cells and breast cancer cell lines
81 to capture GPI-anchored proteins from human breast cancer tissues, cells, and serum for proteomic an
82 erences have been charted between normal and breast cancer tissues, changes in higher-order chromatin
83 e expression of IRF-1 and IRF-2 may occur in breast cancer tissue compared with normal breast tissue,
85 nd to have significantly lower expression in breast cancer tissues compared to paired normal breast t
86 c RNA) is upregulated in tamoxifen-resistant breast cancer tissues compared to their primary counterp
87 uctions and enhancements in the intensity in breast cancer tissues compared to uninvolved breast tiss
89 kinase, is significantly increased in human breast cancer tissues compared with normal and benign br
90 ession is up-regulated in distant metastatic breast cancer tissues compared with primary cancer tissu
93 ign Tissue Bank (BCCTB), a vital resource of breast cancer tissue for researchers to support and prom
94 Breast Cancer Project, and paraffin-embedded breast cancer tissues for 90 patients were available for
95 erse transcription-PCR analysis of 18 paired breast cancer tissues found that in 28% of the cancer sa
96 receptor and pSmad2 immunohistochemically in breast cancer tissue from 1,045 patients in the Shanghai
97 emistry was performed on adjacent normal and breast cancer tissues from 96 premenopausal women with k
98 ming immunohistochemical staining in primary breast cancer tissues from patients with different stage
99 lar effects are noted with in vitro cultured breast cancer tissues from patients, but not with normal
100 A reduction of the expression of SnaH in breast cancer tissue further suggests a cancer-protectiv
101 sues and their normal counterparts, although breast cancer tissues generally appeared to have heterog
102 on pathway in different pathologic grades of breast cancer tissue has not been described thoroughly,
103 (111In-MX-DTPA) BrE-3 to specifically target breast cancer tissue in patients, and the dosimetry deri
104 expression were also selectively induced in breast cancer tissues in transgenic mice expressing the
105 of p63 mRNA compared with other genotypes in breast cancer tissues, indicating that rs17506395 may be
110 AKT) expression by immunohistochemistry in a breast cancer tissue microarray (n = 377) with approxima
115 inhibitor (SLPI), were studied further with breast cancer tissue microarrays using a novel method of
116 Reclassification of breast cancer cells and breast cancer tissue microarrays with this system correl
119 analysis was not able to detect SF1 mRNA in breast cancer tissue or in SK-BR-3 cells, it is thought
120 niques to analyze expression of EI24/PIG8 in breast cancer tissue progression arrays and showed that
121 studies to be up-regulated in both lung and breast cancer tissues relative to normal adjacent tissue
122 n-2 protein expression was also decreased in breast cancer tissue samples as evaluated by immunohisto
123 ly, immunohistochemical staining of lung and breast cancer tissue samples demonstrated that increased
125 ance to our studies is our analysis of human breast cancer tissue samples that indicated association
126 indings suggest that the analysis of primary breast cancer tissue samples will be indispensable for t
127 ry-based proteomics, which allow analysis of breast cancer tissue samples, leading to the first large
128 overexpressed in approximately 50% of human breast cancer tissue samples, suggesting that regulation
129 rgeting 8 cancer proteins was performed on a breast cancer tissue section to illustrate the potential
132 ding is further confirmed from data in human breast cancer tissues showing that CNNM3 levels correlat
133 owed that elevated CDK11(p110) expression in breast cancer tissues significantly correlated with poor
134 approach by imaging Papanicolaou smears and breast cancer tissue slides over a large field-of-view o
135 stribution of MT1-MMP with Ln-5 in colon and breast cancer tissue specimens suggested a role for this
138 levels were significantly higher in invasive breast cancer tissues than in breast adenocarcinoma tiss
139 expression was significantly higher in most breast cancer tissues than in normal breast tissues.
141 mpare gene expression profiles from the same breast cancer tissue that had been either frozen or FPE
142 omprehensive metabolic phenotyping method in breast cancer tissue that uses desorption electrospray i
143 elevated in HER2/neu-positive primary human breast cancer tissues that are known to be resistant to
144 the AI activity of a dual AI/SERM, while in breast cancer tissue the antiestrogenic SERM activity of
148 alyzing 188 normal breast and 1247 malignant breast cancer tissues, we observed the loss of KLLN in m
150 This study examines HER2 testing of primary breast cancer tissue when performed with immunohistochem
151 el gene that is overexpressed in ovarian and breast cancer tissues when compared with normal tissues.
152 ided evidence of the expression of UGT1A1 in breast cancer tissue, where a positive signal was observ
153 esults showed that MUC16 is overexpressed in breast cancer tissues whereas not expressed in non-neopl
154 ive (HER2+) but not in HER2-negative (HER2-) breast cancer tissues, whereas both HER2+ and HER2- tumo
155 generating PIP2, is positively expressed in breast cancer tissues, which correlates intimately with
156 ine CSCs in formalin-fixed paraffin-embedded breast cancer tissue, with the goal of assessing the pro
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