ライフサイエンスコーパス: bronchi

1 in the respiratory epithelia of trachea and bronchi.

2 tal and acquired stenosis of the trachea and bronchi.

3 pithelium including nasopharynx, trachea and bronchi.

4 terol delivery in a model of the trachea and bronchi.

5 ute to the epithelial layer in more than 99% bronchi.

6 a prejunctional mechanism in human isolated bronchi.

7 y used in vivo model of human small-diameter bronchi.

8 teroids on mast cell localisation within the bronchi.

9 n, and saline in three distinct subsegmental bronchi.

10 ads to tracheal agenesis and ectopic primary bronchi.

11 d mean wall area percentage in six segmental bronchi.

12 P63 into the prospective trachea and primary bronchi.

13 (Golf and AC3) in the smooth muscle of human bronchi.

14 like structures in the trachea and main-stem bronchi.

15 number and shape of cartilage in trachea and bronchi.

16 is when lesions are located centrally in the bronchi.

17 roups in bacterial colonization of the lungs/bronchi.

18 had fluid in the subglottic trachea and main bronchi.

19 on and immunohistochemical analysis in human bronchi.

20 epithelium, glands, and endothelium of human bronchi.

21 ge pulmonary arteries and veins and mainstem bronchi.

22 ase stimulated contraction of isolated human bronchi.

23 e fields were located in the trachea or main bronchi.

24 terized by inflamed and dilated thick-walled bronchi.

25 vessels, bile ducts, esophagus, trachea, and bronchi.

26 rmal numbers of mast cells in their skin and bronchi.

27 er lobar bronchi 45%, left 55%; middle lobar bronchi 21%, lingula 26%; right lower lobar bronchi 28%,

28 bronchi 21%, lingula 26%; right lower lobar bronchi 28%, left 29%.

29 N THE TYPICAL ONES WAS IN: right upper lobar bronchi 45%, left 55%; middle lobar bronchi 21%, lingula

30 In human bronchi, a similar role of Rac1 was observed during bron

31 A parasitology exam from a bronchi alveolar lavage yielded an immature rhabditiform

32 ere common abnormalities such as hyparterial bronchi and absence of middle lobe fissure on CTscans su

33 lammatory changes and epithelial necrosis of bronchi and bronchiolar epithelium.

34 al passage of neutrophils into the pulmonary bronchi and bronchioles of lungs infected with P. haemol

35 ipheral lung samples (in epithelium of small bronchi and bronchioles or lymphoid aggregates).

36 More than 30% of both bronchi and bronchioles were obstructed by cast formatio

37 The conducting airways (bronchi and bronchioles) and peripheral gas exchange (al

38 bular septa, centrilobular region, and small bronchi and bronchioles), abnormal findings (reticulatio

39 gh the extensive extracellular matrix of the bronchi and bronchioles.

40 sia of neuroendocrine cells involving distal bronchi and bronchioles.

41 ient neutrophils into the walls and lumen of bronchi and bronchioles.

42 isotropic, and submillimeter imaging of the bronchi and lung parenchyma with high CNR and SNR and ma

43 binding model in membranes prepared from all bronchi and lung parenchyma.

44 efects in the smooth muscle component of the bronchi and major pulmonary vessels with decreased Fgf10

45 efects in the smooth muscle component of the bronchi and major pulmonary vessels.

46 ude the ciliated epithelial cells lining the bronchi and oviduct, as well as in the developing sperma

47 ng sites (Bmax) was reduced significantly in bronchi and parenchyma from rejecting lungs with or with

48 gned to characterize endothelin receptors in bronchi and parenchyma of transplanted lungs during acut

49 ssociation between structural alterations of bronchi and PH in COPD.

50 imulated submucosal gland secretion in human bronchi and smooth muscle contraction in mouse intestine

51 ontraction of isolated pulmonary vessels and bronchi and stimulates proliferation of smooth muscle ce

52 onchial smooth muscle and in human embryonic bronchi and surrounding mesenchyme, strongly suggesting

53 ial or endothelial cells that line the human bronchi and the aorta express nicotinic acetylcholine re

54 Main bronchi and tracheal ASM were significantly hyposensitiv

55 release a variety of signals that target the bronchi and vasculature and recruit other immune cells t

56 ory epithelium of the nasal cavity, trachea, bronchi, and bronchioles with accompanying inflammation.

57 ertrophy of epithelial cells of the trachea, bronchi, and bronchioles.

58 es, including neural tubules, gut, pulmonary bronchi, and hepatic cells.

59 , in airway smooth muscle of the trachea and bronchi, and in the smooth muscle layers of all abdomina

60 inal size reduction in the trachea, mainstem bronchi, and proximal airways.

61 y using visual scoring, numbering of visible bronchi, and quantitative measurement of the apparent co

62 ependent induction of MMCs in trachea, large bronchi, and small intestine provides numbers but does n

63 were taken from the trachea, the five lobar bronchi, and the five lobar parenchyma, for qualitative

64 arterial smooth muscle cells, the developing bronchi, and the urogenital ridge and bladder.

65 sets of epithelial cells lining the trachea, bronchi, and tracheal glands.

66 lineages of conducting airways (trachea and bronchi), as distinct from those of peripheral airways (

67 In freshly harvested human bronchi, ASL thickness was 55 +/- 5 microm, [Na+] was 10

68 ronchial tissue oximetry of native and donor bronchi at 0, 3, and 30 days after transplantation (n =

69 VF contents in the lower trachea, carina and bronchi at baseline and at LAP +10 mmHg were 52.1 +/- 1.

70 ion therapy-induced consolidation in ectatic bronchi at least 9 months after completion of radiation

71 The main-stem bronchi bifurcate from this common structure and connect

72 s present in basal epithelial cells of large bronchi, both histologically normal (26%) and hyperplast

73 ted, by immunostaining, in the epithelium of bronchi, bronchioles, and alveolar walls.

74 mmatory cell infiltrates in the lungs around bronchi, bronchioles, and pulmonary arteries and veins;

75 However, only in the main bronchi, but not in the trachea, did the loss of SM or c

76 Trachea and main bronchi did not show significant differences in reactivi

77 blished place in diagnostics of tracheal and bronchi disorders and its potential has not been examine

78 THE FREQUENCY OF LOBAR BRONCHI DIVISIONS OTHER THAN THE TYPICAL ONES WAS IN: ri

79 vessels that supply the trachea and mainstem bronchi do not penetrate into the intraparenchymal airwa

80 astrointestinal system (GI), followed by the bronchi, endocrine glands-like C cells of the thyroid (m

81 mutant mice was also decreased and the large bronchi extended to the periphery.

82 ination of glutaraldehyde-fixed medium-sized bronchi from acute Kawasaki disease fatalities and analy

83 Third-order bronchi from autografted or allografted lungs were eithe

84 receptors) caused comparable contraction of bronchi from autotransplanted and allotransplanted rejec

85 in A and B receptors) caused contractions of bronchi from autotransplanted lungs which were not diffe

86 facto the contralateral lung or nonbleeding bronchi from blood aspiration.

87 Bronchi from COX-1(-/-) mice were hyperresponsive to bro

88 made from parasympathetic ganglia located on bronchi from human lungs in order to determine the level

89 tions caused by endothelin 3 were reduced in bronchi from rejecting allotransplanted lungs.

90 es of bronchial reactivity were performed on bronchi from smooth muscle (SM)-specific Rac1 knockout m

91 racheas (605/827 glands, 15 subjects) and in bronchi from subjects who were transplanted because of o

92 Bronchi from wild-type mice contained predominantly COX-

93 pithelium, and adventitia of bronchioles and bronchi in lungs of calves with BLAD compared to normal

94 Branching of the bronchi in lungs of mutant mice was also decreased and t

95 E(4) mediate antigen-induced constriction of bronchi in tissue obtained from subjects with asthma.

96 CF bronchi in vivo exhibited the same pattern of expression

97 Bronchi isolated from 10 of 16 lungs contracted in respo

98 The bronchi isolated from 8 of 16 lungs also exhibited relax

99 ryonic murine craniofacial cartilage, heart, bronchi, kidney and vertebral bodies.

100 We found that, in the trachea and main bronchi, loss of SM or cartilage resulted in an increase

101 st susceptible to airway obstruction (nose < bronchi << bronchioles).

102 Filling in of previously patent ectatic bronchi occurred in the 20 patients with recurrent disea

103 f a single somatic p53 point mutation in the bronchi of a smoker suggests that a single progenitor br

104 al similarities in the primary and secondary bronchi of birds and crocodilians suggest that these str

105 a subset of SCGB3A2-expressing cells within bronchi of both mouse and neonatal human lungs.

106 sion molecule (CEACAM) family members in the bronchi of patients with severe asthma.

107 Intrapulmonary bronchi of sensitized monkeys had focal mucus cell hyper

108 s also expressed strongly in the trachea and bronchi of the adult lung.

109 In the trachea and bronchi of the mouse, airway smooth muscle (SM) and cart

110 roid, and thymic epithelia, but not trachea, bronchi, or gastrointestinal tract occurred when mice we

111 tive stress (p = 0.002), airway obstruction (bronchi: p = 0.001, bronchioli: p = 0.013), parenchymal

112 n through most of the tubular gas-exchanging bronchi (parabronchi), whereas in the lungs of mammals a

113 rations of PGE2 (0.01-1 mumol/L) relaxed the bronchi precontracted by histamine.

114 r malacia that involved the trachea and main bronchi (reduction in cross-sectional area of more than

115 stable radiation-induced fibrosis and patent bronchi remained disease free.

116 DAC of vehicle-treated bronchi resulted in late-phase airway obstruction (appro

117 ammation that is detected in the trachea and bronchi (termed inflammatory airway disease [IAD]) is mo

118 Preliminary data showed that 90% of bronchi (up to third order) measureable on multiplanar C

119 d spacer on filter placed at the ends of the bronchi was measured by spectrophotometry (246 nm).

120 or 10 s every 100 s) of human isolated human bronchi was studied.

121 further characterize the antigen in acute KD bronchi, we examined paraffin-embedded ciliated bronchia

122 Because CXCL17 is strongly expressed in bronchi, we measured it in bronchoalveolar lavage fluids

123 Human trachea and main bronchi were dissected free of epithelium and connective

124 Sections containing bronchi were evaluated for epithelial cell proliferation

125 Specific bronchi were exposed to a single DAC on five consecutive

126 Subsuperior bronchus or bronchi were found on the right side in 44% and on the l

127 he total study population (in whom both main bronchi were imaged) included 25 men and 26 women (mean

128 Bronchi were visible consistently up to the fourth gener

129 tract, from the epiglottis to the secondary bronchi, were imaged.

130 lso holds for the lung CTMCs in the proximal bronchi, whereas the induced MMCs express only four prot

131 lung, toxic milk expression is restricted to bronchi, while mottled expression is diffuse.

132 nd relaxation were studied in isolated human bronchi with an inner diameter of 1 mm or less.

133 by treatment of both mouse trachea and human bronchi with specific SFK inhibitors.

134 The trachea, larynx and main bronchi with the right vagus nerve and nodose ganglion w

135 ed efficiently in explants from tracheas and bronchi, with limited replication in alveolar cells.