ライフサイエンスコーパス: bronchial asthma

1 valuate the efficacy of omalizumab in severe bronchial asthma.

2 ls had sensorineural deafness, and three had bronchial asthma.

3 lizumab, was studied in patients with severe bronchial asthma.

4 modality for treating allergic reactions and bronchial asthma.

5 ey may play a significant pathogenic role in bronchial asthma.

6 mask in two young children with acute severe bronchial asthma.

7 gy in chronic inflammatory diseases, such as bronchial asthma.

8 nosed pulmonary sarcoidosis complicated with bronchial asthma.

9 in human patients with allergic rhinitis and bronchial asthma.

10 ategy for finding more efficient therapy for bronchial asthma.

11 e., atherosclerosis, pulmonary fibrosis, and bronchial asthma.

12 ences associated with RSV infection, such as bronchial asthma.

13 cellular matrix proteins is a key feature in bronchial asthma.

14 ment and/or cure of allergic inflammation in bronchial asthma.

15 licated as a mediator of disease, especially bronchial asthma.

16 the inflammatory and neurogenic elements of bronchial asthma.

17 om human patients with allergic rhinitis and bronchial asthma.

18 ed diseases, such as allograft rejection, or bronchial asthma.

19 myocardial infarction, atherosclerosis, and bronchial asthma.

20 ne E4(LTE4)-are important mediators of human bronchial asthma.

21 steroid action in allergic diseases such as bronchial asthma.

22 l processes, may be an important mediator of bronchial asthma.

23 in the inflammatory response associated with bronchial asthma.

24 ffect of azelastine in patients with chronic bronchial asthma.

25 l to allergic inflammation and implicated in bronchial asthma.

26 ukotrienes implicated in the pathobiology of bronchial asthma.

27 n and pathology in allergic diseases such as bronchial asthma.

28 as being used to treat rheumatoid arthritis, bronchial asthma (6%), pemphigus (5%), or other processe

29 ence in diverse allergic disorders including bronchial asthma, allergic rhinitis, and cutaneous conta

30 ctions (LPRs) are characteristic features of bronchial asthma, although the pathogenetic mechanisms r

31 ys important roles in the pathophysiology of bronchial asthma and allergic disorders; however, this c

32 nflammatory diseases in humans, particularly bronchial asthma and allergic rhinitis.

33 airway remodeling are consistent features of bronchial asthma and are correlated with disease chronic

34 s of chronic inflammatory disorders, such as bronchial asthma and atherosclerosis.

35 Diagnosis of bronchial asthma and atopic dermatitis was made, but she

36 yl leukotrienes are established mediators of bronchial asthma and have agonist roles analogous to tho

37 led corticosteroids in patients with chronic bronchial asthma and not lead to a deterioration in pulm

38 tor blockers are effective in treating human bronchial asthma and the mouse is often used to model hu

39 athophysiology of allergic diseases, such as bronchial asthma, and in host immunity to certain organi

40 athophysiology of allergic diseases, such as bronchial asthma, and in host immunity to parasitic orga

41 ay hyperreactivity (AHR) is a key feature of bronchial asthma, and inhalation of irritants may facili

42 ons in the pathogenesis of diseases, such as bronchial asthma, atherosclerosis, and pulmonary fibrosi

43 Concomitant bronchial asthma, atopic dermatitis, and food allergies

44 gical disorders including allergic rhinitis, bronchial asthma, atopic dermatitis, food allergies, urt

45 w reports on the effects of immunotherapy on bronchial asthma (BA) and allergic rhinitis (AR) in Japa

46 y, the prevalence of allergic rhinitis among bronchial asthma (BA) patients was reported to be 67.3%

47 was initially developed for the treatment of bronchial asthma but which also has been used for cerebr

48 is, ulcerative colitis, Crohn's disease, and bronchial asthma, but the mechanism is poorly understood

49 In bronchial asthma, eosinophils (EOS) adhere to, and migra

50 ic rhinitis/rhinoconjunctivitis with/without bronchial asthma (Global Initiative for Asthma I/II) rec

51 The role of human basophils in bronchial asthma has been hard to define.

52 otential in adults with airway reactivity or bronchial asthma has been reported, data are lacking on

53 0% of diseases that include angina pectoris, bronchial asthma, herpes simplex, and duodenal ulcer.

54 cious for bronchoconstriction in humans with bronchial asthma; however, the clinical response to thes

55 V) infection is considered a risk factor for bronchial asthma; however, the synergy between allergen

56 xidative stress have both been implicated in bronchial asthma; however, there is no previous study th

57 All patients had a diagnosis of bronchial asthma inadequately controlled by at least a m

58 ed in different chronic disorders, including bronchial asthma, inflammatory bowel diseases, and skin

59 Differentiation from bronchial asthma is also important.

60 Bronchial asthma is associated with an inflammatory proc

61 r type 1 (Th1) mediated immune response, and bronchial asthma is characteristic of the Th2.

62 Bronchial asthma is characterized by episodic airway obs

63 f hypercitrullination in the pathogenesis of bronchial asthma is still unknown.

64 The merger rate of bronchial asthma of the patients seen in pediatrics/inte

65 oids, first-line treatment for patients with bronchial asthma, on mucosal tolerance remain unknown.

66 However, many features of bronchial asthma, such as smooth muscle contraction, muc

67 ing lung function in patients suffering from bronchial asthma, the most common chronic inflammatory l

68 Bronchial asthma, the most prevalent cause of significan

69 Although he was treated for bronchial asthma, the symptom did not improve.

70 the blood of adult and elderly patients with bronchial asthma to establish potential association of C

71 by controversial results in mouse models of bronchial asthma treated with anti-IL-5 Ab and the failu

72 ine, plays a pivotal role in pathogenesis of bronchial asthma via IL-13 receptor alpha1 (IL-13Ralpha1

73 ients were women, and were diagnosed to have bronchial asthma with the reversibility of airway contra