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1 tive spheroidal antigen in acute KD ciliated bronchial epithelium.
2 ic inclusion bodies in the acute KD ciliated bronchial epithelium.
3 ation of the RASSFIA was not detected in the bronchial epithelium.
4 lial site, or unmethylated in both tumor and bronchial epithelium.
5 fibroblasts, and primary cultures of normal bronchial epithelium.
6 ly involved in protection and maintenance of bronchial epithelium.
7 alveolar type II cells, and in tracheal and bronchial epithelium.
8 ons that have been examined in preneoplastic bronchial epithelium.
9 an brain tissue, non-SCLC tumors, and normal bronchial epithelium.
10 d found that IP-10 mRNA was expressed in the bronchial epithelium.
11 or establishment of the endodermally derived bronchial epithelium.
12 th the exception of the endodermally derived bronchial epithelium.
13 ch GSDMB induces 5-LO to induce TGF-beta1 in bronchial epithelium.
14 re also deficient in populating the layer of bronchial epithelium.
15 DSB marker gamma Histone 2AX (H2AX) foci in bronchial epithelium.
16 dysfunction and pro-inflammatory activity of bronchial epithelium.
17 levant cell line, Calu-3, derived from human bronchial epithelium.
18 ion of genes inducible by T(H)2 cytokines in bronchial epithelium.
19 ssed primarily in tumor cells along with the bronchial epithelium.
20 s only observed in ciliated cells, not basal bronchial epithelium.
21 vailable experimental data of cultured human bronchial epithelium.
22 ICAM-2, but not ICAM-3, is expressed on the bronchial epithelium.
23 lls but not in normal mucous NHTBE or normal bronchial epithelium.
24 n lungs and of interferon-sensitive genes in bronchial epithelium.
25 motility and colonization of differentiated bronchial epithelium.
26 ic inclusion bodies in the acute KD ciliated bronchial epithelium.
27 lesions, 47%) than in histologically normal bronchial epithelium (63 of 323 specimens, 20%; P < 0.00
28 of cells staining for mucus was seen in the bronchial epithelium, a feature more commonly associated
29 ew HIF2alpha-dependent mechanism involved in bronchial epithelium adaptation to oxygen fluctuations.
30 ted into human airway epithelial cells [both bronchial epithelium + adenovirus 12 - SV40 hybrid (BEAS
31 n with Mycobacterium paratuberculosis and in bronchial epithelium after acute infection with Pasteure
32 striking induction in pIgR expression by the bronchial epithelium and a subsequent increase in airway
33 ed DNA damage and cytokine production in the bronchial epithelium and apoptosis in the allergic airwa
34 edly increases in hypertrophic, hyperplastic bronchial epithelium and appears in type II alveolar pne
35 GS2 was highly expressed in human and murine bronchial epithelium and ASM and was markedly downregula
37 ociation seen between p16 methylation in the bronchial epithelium and corresponding primary tumor sub
38 n present in acute Kawasaki disease ciliated bronchial epithelium and in a subset of macrophages in a
39 ene protein, is uniformly found in the human bronchial epithelium and in non-small cell lung carcinom
40 enzymes were significantly increased in the bronchial epithelium and inflammatory immune cells infil
41 rom the interstitium of the lung, across the bronchial epithelium and into the airway lumen, is known
42 We found that SOCS1 was increased in vivo in bronchial epithelium and related to asthma severity.
45 heterozygosity, at 9p and p16 methylation in bronchial epithelium and the prevalence for methylation
46 ons, immunomodulatory cross-talk between the bronchial epithelium and tissue-resident immune cells co
47 be expressed by lung Clara-like cells in the bronchial epithelium and to be up-regulated in cystic fi
48 demonstrate that ICAM-2 is expressed on the bronchial epithelium and, together with ICAM-1, has an e
49 en tissue samples: 25 normal lung, 29 normal bronchial epithelium, and 20 preinvasive and 36 invasive
50 -alpha (GRO-alpha) protein expression in the bronchial epithelium, and an accompanying trend toward a
52 rferon-stimulated genes were detected in the bronchial epithelium, and significant modulation of the
53 ene is expressed in the normal thyroid, lung bronchial epithelium, and specific areas of the forebrai
54 n and motogen for both normal and neoplastic bronchial epithelium, and that expression of the HGF rec
55 sia of goblet cell, eosinophilic invasion to bronchial epithelium, and thickened basal membrane were
56 t overexpress PGI(2) synthase selectively in bronchial epithelium are protected against RSV-induced w
59 ked increases in PSSG reactivity both in the bronchial epithelium as well as in parenchymal regions.
60 he lungs that was primarily localized to the bronchial epithelium at 4 h, but was present in a variet
61 eased type III collagen deposition below the bronchial epithelium basement membrane, reminiscent of b
63 or trigger of asthma exacerbations, with the bronchial epithelium being the major site of HRV infecti
64 fferentially expressed between CF and non-CF bronchial epithelium but only 15 were differentially exp
66 that T cells can migrate across a disrupted bronchial epithelium, but we provide evidence that egres
67 of allergen-induced NF-kappaB activation in bronchial epithelium by GSTM1 in human atopic asthmatics
68 The antigen detected in acute KD ciliated bronchial epithelium by IHC with synthetic KD antibodies
71 l data and experimental studies suggest that bronchial epithelium could serve as a portal of entry fo
73 elevance of air-liquid interface cultures of bronchial epithelium derived from endobronchial biopsy s
74 evels of inflammatory mediator production in bronchial epithelium during the pathogenesis of inflamma
75 er, mir-218 expression is reduced in primary bronchial epithelium exposed to cigarette smoke condensa
78 arker gene expression did not persist in the bronchial epithelium following adeno-associated virus (A
79 e show that early induction of IL17 from the bronchial epithelium, following pathogenic encounter is
80 (up to 1.4 microns in diameter) in ciliated bronchial epithelium from 4 patients with acute KD exami
81 transversion in codon 245 was identified in bronchial epithelium from 7 of 10 sites in both lungs.
82 p16 and DAP kinase was seen as frequently in bronchial epithelium from current smokers as from former
83 Ex vivo cultures of fully differentiated bronchial epithelium from endobronchial biopsy specimens
85 on bodies in acute Kawasaki disease ciliated bronchial epithelium has provided direction for future K
86 a number of genes that are expressed in the bronchial epithelium have been linked to asthma suscepti
87 yocardiocytes, vascular smooth muscle cells, bronchial epithelium, hepatocytes, thymocytes, plasma ce
88 ays containing 323 samples, including normal bronchial epithelium, hyperplasia, squamous metaplasia,
90 ons are expressed similarly in the nasal and bronchial epithelium in CF, the consequences are differe
95 nduces a 127-fold increase in ORMDL3 mRNA in bronchial epithelium in WT mice, with lesser 15-fold inc
96 horcic (buccal and nasal) and intrathoracic (bronchial) epithelium in healthy current and never smoke
97 Overexpression of GSDMB in primary human bronchial epithelium increased expression of genes impor
98 city, whereas enforced SNORA42 expression in bronchial epitheliums increases cell growth and colony f
99 e antigen in acute Kawasaki disease ciliated bronchial epithelium indicate that the Kawasaki disease-
100 of cyclin D1 expression in the normal human bronchial epithelium, indicating an inverse relationship
102 w that egression of human T cells across the bronchial epithelium is a multistep process, driven in p
107 abnormalities in both NSCLC and premalignant bronchial epithelium is increase in chromosomal copy num
108 l expressed in alveolar, tracheal, and upper bronchial epithelium, is significantly down-regulated du
110 mall amounts of TGF-beta were present in the bronchial epithelium, macrophages, bronchial and vascula
112 ohistochemical expression patterns in normal bronchial epithelium (n = 36), squamous metaplasia (SM;
114 ir-liquid interface cultures of normal human bronchial epithelium (NHBE) exhibiting mucociliary activ
115 To determine whether the denudation of the bronchial epithelium observed in endobronchial biopsies
116 The correlation between p16 status in the bronchial epithelium obtained from lung lobes that did n
117 In addition, we analyzed brushes from the bronchial epithelium of 35 heavy smokers without cancer.
118 of miR-145, miR-223, and miR-494 in vivo in bronchial epithelium of individuals carrying the DeltaF5
119 odies (ICI) have been identified in ciliated bronchial epithelium of Kawasaki disease (KD) patients u
120 extent, DAP kinase, occurs frequently in the bronchial epithelium of lung cancer cases and cancer-fre
123 tify and investigate the role of PSCs in the bronchial epithelium of neonatal mice, we developed an e
124 viously characterized gene expression in the bronchial epithelium of never smokers and identified the
125 nt protein-3 was found to be elevated in the bronchial epithelium of these mice, which may have impor
129 PD correlates with disease severity, and the bronchial epithelium reconstituted in vitro from these p
130 Ormdl3 transcript levels specifically in the bronchial epithelium resulted in reinstatement of suscep
131 Concurrent activation of TLR2 and IL17R in bronchial epithelium results in the sequestration of MyD
132 neuroendocrine/neurosensory cells within the bronchial epithelium, revealing a targeted mode of cell
133 uired to form a morphologically recognizable bronchial epithelium, revealing an in vivo, cell type-sp
134 d gene expression in CF and non-CF nasal and bronchial epithelium samples using Illumina HumanRef-8 E
135 opsy samples and promotes oncogenesis in the bronchial epithelium, suggesting that strategies to inhi
136 imensional primary cell cultures from infant bronchial epithelium that reproduce several hallmarks of
138 cular density as well as the contribution of bronchial epithelium to produce vascular endothelial gro
140 unction and the pro-inflammatory response of bronchial epithelium upon HDM exposure and may thus have
141 nchi, we examined paraffin-embedded ciliated bronchial epithelium using light microscopy (LM) and tra
144 dition, PCNA immunostaining of the primitive bronchial epithelium was increased in the presence of TG
146 iency of transduction in the balloon-treated bronchial epithelium was low but reached 20% in some are
147 binding to a cytoplasmic antigen in proximal bronchial epithelium was observed in 10 of 13 patients w
148 Allergen-provoked NF-kappaB induction in bronchial epithelium was significantly greater in GSTM1+
150 and increased localization to the asthmatic bronchial epithelium, we investigated whether HRV infect
151 xpression of miR-629-3p was localized in the bronchial epithelium, whereas miR-223-3p and miR-142-3p
152 r absence of a three-gene "Th2 signature" in bronchial epithelium, which differ in terms of eosinophi
153 The specific tissue modeled is the human bronchial epithelium, which is of particular interest, a
154 tion to TGF-beta-driven reprogramming of the bronchial epithelium, which results in impaired lung IgA
155 hma where their numbers are increased in the bronchial epithelium with increasing disease severity.
156 way contributing to IL-8 secretion in the CF bronchial epithelium with KL functioning as an endocrine
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