ライフサイエンスコーパス: bronchial hyperresponsiveness

1 liferation and MMP-1 protein associated with bronchial hyperresponsiveness.

2 eral blood eosinophilia, high level of FENO, bronchial hyperresponsiveness.

3 mediated inflammation, tissue remodeling and bronchial hyperresponsiveness.

4 77 gene with asthma and with the severity of bronchial hyperresponsiveness.

5 se-dust-mite-induced airway inflammation and bronchial hyperresponsiveness.

6 There was some evidence of associations with bronchial hyperresponsiveness.

7 es, whereas others are shared with atopy and bronchial hyperresponsiveness.

8 osinophils, mucin levels in the airways, and bronchial hyperresponsiveness.

9 h could explain its genetic association with bronchial hyperresponsiveness.

10 ase and phosphodiesterase also contribute to bronchial hyperresponsiveness.

11 was >37.8ppb, 25 of 38 subjects (65.7%) had bronchial hyperresponsiveness.

12 novel therapeutic targets for the control of bronchial hyperresponsiveness.

13 erity of airway narrowing or the severity of bronchial hyperresponsiveness.

14 is of asthma, elevated serum IgE levels, and bronchial hyperresponsiveness.

15 e of beta2 agonists has been known to induce bronchial hyperresponsiveness.

16 was >37.8ppb, 29 of 43 subjects (67.4%) had bronchial hyperresponsiveness.

17 region for linkage to asthma, serum IgE, and bronchial hyperresponsiveness.

18 associated with clinical signs of atopy and bronchial hyperresponsiveness.

19 ascular lung inflammation, and inhibition of bronchial hyperresponsiveness 6 wk after administration

20 (IL9) as a determining factor in controlling bronchial hyperresponsiveness, a hallmark of the disease

21 e to allergen, and prevented the increase in bronchial hyperresponsiveness after allergen challenge.

22 symptoms, reversible airflow obstruction, or bronchial hyperresponsiveness after having all asthma me

23 Subjects had bronchial hyperresponsiveness, an FEV1 < or = 70% of pre

24 tant in the inflammatory response leading to bronchial hyperresponsiveness and asthma.

25 implicated in determining susceptibility to bronchial hyperresponsiveness and asthma.

26 n for genes that determine susceptibility to bronchial hyperresponsiveness and atopy in animal models

27 y of the Genetics and Environment of Asthma, bronchial hyperresponsiveness and atopy) (170 with and 1

28 Viral respiratory infections can cause bronchial hyperresponsiveness and exacerbate asthma.

29 assumed coughing occurs as a consequence of bronchial hyperresponsiveness and inflammation, but the

30 gnosed asthma and atopy at 7(1/2) years, and bronchial hyperresponsiveness and lung function at 8(1/2

31 In patients with moderate to severe bronchial hyperresponsiveness and nasal polyposis, the c

32 regression analysis, only moderate to severe bronchial hyperresponsiveness and nasal polyps were inde

33 s strongly associated with susceptibility to bronchial hyperresponsiveness and protection from allerg

34 ated with severe peribronchial eosinophilia, bronchial hyperresponsiveness, and augmented IL-13 and I

35 arly childhood is associated with asthma and bronchial hyperresponsiveness, and faster weight growth

36 cle growth, MMP-1 levels are associated with bronchial hyperresponsiveness, and MMP-1 activation are

37 CHI3L1 is a susceptibility gene for asthma, bronchial hyperresponsiveness, and reduced lung function

38 Transient bronchial hyperresponsiveness appears to be the predomin

39 Allergen-induced increases in serum IgE and bronchial hyperresponsiveness are exaggerated in periost

40 role for IL-9 in the complex pathogenesis of bronchial hyperresponsiveness as a risk factor for asthm

41 participants underwent lung function tests, bronchial hyperresponsiveness assessment and sputum indu

42 terized by reversible airway obstruction and bronchial hyperresponsiveness associated with T(H)2 cell

43 inkage to qualitative measures of asthma and bronchial hyperresponsiveness (BHR) (p > 0.10) or to qua

44 on with the key asthma-related phenotypes of bronchial hyperresponsiveness (BHR) and atopy.

45 Bronchial hyperresponsiveness (BHR) can be present in su

46 Bronchial hyperresponsiveness (BHR) describes the exagge

47 been shown to induce airway inflammation and bronchial hyperresponsiveness (BHR) in mice.

48 The severity of bronchial hyperresponsiveness (BHR) is a fundamental fea

49 Bronchial hyperresponsiveness (BHR) is a phenotypic hall

50 Bronchial hyperresponsiveness (BHR) is an important, but

51 The bronchial hyperresponsiveness (BHR) test is useful to di

52 physiology of lung allergic inflammation and bronchial hyperresponsiveness (BHR) that characterize as

53 asthma is increased airway irritability, or bronchial hyperresponsiveness (BHR) which is still poorl

54 25 yr later, 38 subjects (21%) did not show bronchial hyperresponsiveness (BHR)(PC20 > 16 mg/ml), 45

55 inophil counts in relation to lung function, bronchial hyperresponsiveness (BHR), and asthma control

56 fraction of exhaled nitric oxide (Feno), low bronchial hyperresponsiveness (BHR), and low bronchodila

57 he associations of parental asthma severity, bronchial hyperresponsiveness (BHR), and total and speci

58 the lack of bronchoprotection is related to bronchial hyperresponsiveness (BHR), and whether the bro

59 inflammation; associated phenotypes include bronchial hyperresponsiveness (BHR), elevated total seru

60 of airway Mycoplasma pneumoniae infection on bronchial hyperresponsiveness (BHR), lung inflammation,

61 characteristic features of asthma, including bronchial hyperresponsiveness, bronchoconstriction, airw

62 he presence of nocturnal asthma, more severe bronchial hyperresponsiveness, exercise-induced asthma,

63 n, the importance of T cell costimulation in bronchial hyperresponsiveness had not been characterized

64 uch as high total serum immunoglobulin E and bronchial hyperresponsiveness, have been linked by numer

65 mplementary roles of FENO and FOT to predict bronchial hyperresponsiveness in adult stable asthmatic

66 R5 or R20 and FENO can predict the level of bronchial hyperresponsiveness in adult stable asthmatics

67 rphisms showing association with severity of bronchial hyperresponsiveness in asthma patients.

68 Adenosine has been suggested to induce bronchial hyperresponsiveness in asthmatics, which is be

69 Bronchial hyperresponsiveness in at-risk neonates preced

70 67 restored the sGC signaling and attenuated bronchial hyperresponsiveness in CS-exposed mice.

71 ease) is strongly associated with asthma and bronchial hyperresponsiveness in different populations.

72 Bronchial hyperresponsiveness in mild to moderate asthma

73 Bronchial hyperresponsiveness is a characteristic featur

74 ay smooth muscle function to the extent that bronchial hyperresponsiveness is ablated, consistent wit

75 10) of the likelihood ratio (LOD), 2.94) and bronchial hyperresponsiveness (LOD, 3.93).

76 mice, compared with WT mice, had diminished bronchial hyperresponsiveness (lung airway resistance);

77 s suggest that a gene controlling asthma and bronchial hyperresponsiveness maybe located in this regi

78 asthma through the assessment of nonspecific bronchial hyperresponsiveness (NSBH) is a key step in th

79 ; 95% CI, 1.37-3.85; P = 0.002), but not for bronchial hyperresponsiveness or atopy.

80 ither by the immunopathogenesis of asthma or bronchial hyperresponsiveness, or uncovered by the whole

81 as significantly associated with more severe bronchial hyperresponsiveness (P < .0001) and with curre

82 ide evidence for linkage between DXYS154 and bronchial hyperresponsiveness (P = 0.000057) or asthma (

83 levels (P=1.1 x 10(-13)), asthma (P=0.047), bronchial hyperresponsiveness (P=0.002), and measures of

84 Bronchial hyperresponsiveness-related symptoms were asso

85 nto health care ("reported asthma") and (2) "bronchial hyperresponsiveness-related symptoms," defined

86 es in support of HLA-G as a novel asthma and bronchial hyperresponsiveness susceptibility gene in the

87 we evaluated these patients before and after bronchial hyperresponsiveness to acetylcholine (ACh) or

88 ese correlations in patients with normalized bronchial hyperresponsiveness to ACh or Hist.

89 adult patients with asthma having normalized bronchial hyperresponsiveness to ACh or Hist.

90 his study was to assess the relation between bronchial hyperresponsiveness to dry, cold air at age 6

91 ypes [total and allergen-specific serum IgE, bronchial hyperresponsiveness to methacholine, forced ex

92 sGCalpha1(-/-) mice exposed to CS exhibited bronchial hyperresponsiveness to serotonin.

93 Because improvement in bronchial hyperresponsiveness was associated with a redu

94 The presence of bronchial hyperresponsiveness was measured with a methac