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1 gnificant risk factor for the development of bronchiolitis obliterans syndrome.
2 rences were observed in the overall onset of bronchiolitis obliterans syndrome.
3 recipients is limited by the development of bronchiolitis obliterans syndrome.
4 y may improve lung function in patients with bronchiolitis obliterans syndrome.
5 the main risk factor for the development of bronchiolitis obliterans syndrome.
6 ection has been linked to the development of bronchiolitis obliterans syndrome.
7 fts due to acute rejection (AR) or developed bronchiolitis obliterans syndrome.
8 reduce the incidence of post-lung transplant bronchiolitis obliterans syndrome.
9 ociated with an increased risk of developing bronchiolitis obliterans syndrome.
10 n of epithelial to mesenchymal transition in bronchiolitis obliterans syndrome.
11 ortality after lung transplantation (LTX) is bronchiolitis obliterans syndrome.
12 ng transplant patients prior to diagnosis of bronchiolitis obliterans syndrome.
13 defined as idiopathic pneumonia syndrome or bronchiolitis obliterans syndrome.
14 th chronic lung allograft rejection known as bronchiolitis obliterans syndrome.
15 he surviving study subjects remain free from bronchiolitis obliterans syndrome.
16 d the impact of primary graft dysfunction on bronchiolitis obliterans syndrome.
18 antly elevated within 3 months of developing bronchiolitis obliterans syndrome (8.3 [1.4-25.1] vs. 3.
20 epatitis C viral RNA (HCV RNA), freedom from bronchiolitis obliterans syndrome, acute rejection, and
22 Thin-section CT studies in six patients with bronchiolitis obliterans syndrome (age range, 2 months t
23 reported risk factor for the development of bronchiolitis obliterans syndrome, an important cause of
24 smatch model of multiorgan system cGVHD with bronchiolitis obliterans syndrome and a minor MHC mismat
25 study was to investigate the development of bronchiolitis obliterans syndrome and graft loss after L
26 onic lung allograft rejection in the form of bronchiolitis obliterans syndrome and its histopathologi
28 valuates the current diagnostic criteria for bronchiolitis obliterans syndrome and reviews the epidem
29 rom lung transplant recipients who developed bronchiolitis obliterans syndrome and were compared to s
32 tervention in five patients with progressive bronchiolitis obliterans syndrome, anti-TNFalpha treatme
34 odel demonstrated that the increased risk of bronchiolitis obliterans syndrome associated with primar
36 emic steroids are the standard treatment for bronchiolitis obliterans syndrome (BOS) after allogeneic
38 genetic polymorphisms on the development of bronchiolitis obliterans syndrome (BOS) after lung trans
41 the internationally recognized definition of bronchiolitis obliterans syndrome (BOS) and longer follo
42 for chronic graft dysfunction manifested as bronchiolitis obliterans syndrome (BOS) and worse posttr
45 d for multiorgan system cGVHD and associated bronchiolitis obliterans syndrome (BOS) in a murine mode
46 plant operation on survival and the onset of bronchiolitis obliterans syndrome (BOS) in consecutive l
47 rentiation is associated with development of bronchiolitis obliterans syndrome (BOS) in human lung al
48 after lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
49 lowing lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
53 Chronic allograft rejection manifested as bronchiolitis obliterans syndrome (BOS) is the leading c
59 allograft dysfunction (CLAD), presenting as bronchiolitis obliterans syndrome (BOS) or restrictive a
60 n (AR) and development of chronic rejection, bronchiolitis obliterans syndrome (BOS) remain major lim
63 -obliteration of the allograft airway during bronchiolitis obliterans syndrome (BOS) that occurs afte
65 ement of six lung transplant recipients with bronchiolitis obliterans syndrome (BOS), a condition pre
66 s limited by infectious complications and by bronchiolitis obliterans syndrome (BOS), a form of chron
68 gnized, idiopathic pneumonia syndrome (IPS), bronchiolitis obliterans syndrome (BOS), and bronchiolit
69 ssion to chronic rejection that manifests as bronchiolitis obliterans syndrome (BOS), but no biomarke
70 tive (ELR(+)) CXC chemokines associated with bronchiolitis obliterans syndrome (BOS), but the effect
71 n after lung transplantation, manifesting as bronchiolitis obliterans syndrome (BOS), has become the
72 g to progressive airflow obstruction, termed bronchiolitis obliterans syndrome (BOS), is the major ca
73 man lung allograft rejection, represented by bronchiolitis obliterans syndrome (BOS), is the single m
74 ween these disorders and risk for subsequent bronchiolitis obliterans syndrome (BOS), mortality and g
78 n the fibro-obliterative lesion found during bronchiolitis obliterans syndrome (BOS), we hypothesized
79 mputed tomography morphology, mortality, and bronchiolitis obliterans syndrome (BOS)-free survival we
93 The per-protocol analysis shows incidence of bronchiolitis obliterans syndrome (BOS): 1/43 in the Eve
94 ce of all other causes (currently defined as bronchiolitis obliterans syndrome [BOS]) is considered t
95 ar that patients may develop an obstructive (bronchiolitis obliterans syndrome [BOS]) or a restrictiv
99 sinophilic BAL predisposed to development of bronchiolitis obliterans syndrome but particularly to re
100 s that CMVIG prophylaxis reduces the risk of bronchiolitis obliterans syndrome, but a controlled tria
101 Acute rejection is a major risk factor for bronchiolitis obliterans syndrome, but noninvasive bioma
102 d a shorter survival and an earlier onset of bronchiolitis obliterans syndrome compared with patients
103 pha as a potential new therapeutic target in bronchiolitis obliterans syndrome deserving of a randomi
105 Adjustment for clinical variables including bronchiolitis obliterans syndrome did not change this re
107 Secondary outcomes included freedom from bronchiolitis obliterans syndrome (fBOS) and rates of ac
109 A trend, however, toward reduced onset of bronchiolitis obliterans syndrome grade 2 or 3 was obser
110 survival in a multivariable model including bronchiolitis obliterans syndrome grade and baseline FEV
113 o activate fibroblasts in the development of bronchiolitis obliterans syndrome has not been evaluated
114 ted rejection, acute cellular rejection, and bronchiolitis obliterans syndrome; however, the signific
115 enance macrolide therapy in the treatment of bronchiolitis obliterans syndrome in lung transplant rec
116 after heart transplantation, and potentially bronchiolitis obliterans syndrome in lung transplant rec
117 ges to the progress of medical management of bronchiolitis obliterans syndrome include difficulties a
118 tion is associated with an increased risk of bronchiolitis obliterans syndrome independent of acute r
125 ) chronic allograft dysfunction, manifest by bronchiolitis obliterans syndrome, is frequent and limit
126 and were divided into three groups: no CLAD (bronchiolitis obliterans syndrome level 0 [BOS 0]), earl
127 lantation imitate the in vivo development of bronchiolitis obliterans syndrome-like lesions and revea
129 o be important in obliterative bronchiolitis/bronchiolitis obliterans syndrome (OB/BOS), which severe
130 onic lung allograft dysfunction manifests as bronchiolitis obliterans syndrome or the recently descri
131 was not a risk factor for the development of bronchiolitis obliterans syndrome or worse overall survi
132 rvival (P = 0.09) and increased freedom from bronchiolitis obliterans syndrome (P = 0.03) was observe
135 genesis of chronic lung allograft rejection (bronchiolitis obliterans syndrome) remains to be elucida
136 trated that respiratory viral infection is a bronchiolitis obliterans syndrome risk factor and virus-
138 iated with a significantly increased risk of bronchiolitis obliterans syndrome stage 1 (grade 1: rela
139 acute rejection, lymphocytic bronchitis, and bronchiolitis obliterans syndrome stage 1, using univari
140 sponse to viruses and in the pathogenesis of bronchiolitis obliterans syndrome, the predominant manif
142 the association of bronchial dilatation with bronchiolitis obliterans syndrome was significant (P = .
143 s in the six patients with clinically proved bronchiolitis obliterans syndrome were mosaic perfusion
144 hown to be implicated in the pathogenesis of bronchiolitis obliterans syndrome, which is considered t
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