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1 gnificant risk factor for the development of bronchiolitis obliterans syndrome.
2 rences were observed in the overall onset of bronchiolitis obliterans syndrome.
3  recipients is limited by the development of bronchiolitis obliterans syndrome.
4 y may improve lung function in patients with bronchiolitis obliterans syndrome.
5  the main risk factor for the development of bronchiolitis obliterans syndrome.
6 ection has been linked to the development of bronchiolitis obliterans syndrome.
7 fts due to acute rejection (AR) or developed bronchiolitis obliterans syndrome.
8 reduce the incidence of post-lung transplant bronchiolitis obliterans syndrome.
9 ociated with an increased risk of developing bronchiolitis obliterans syndrome.
10 n of epithelial to mesenchymal transition in bronchiolitis obliterans syndrome.
11 ortality after lung transplantation (LTX) is bronchiolitis obliterans syndrome.
12 ng transplant patients prior to diagnosis of bronchiolitis obliterans syndrome.
13  defined as idiopathic pneumonia syndrome or bronchiolitis obliterans syndrome.
14 th chronic lung allograft rejection known as bronchiolitis obliterans syndrome.
15 he surviving study subjects remain free from bronchiolitis obliterans syndrome.
16 d the impact of primary graft dysfunction on bronchiolitis obliterans syndrome.
17      Of the 22 patients (5%) who experienced bronchiolitis obliterans syndrome, 15 (6%) were in the a
18 antly elevated within 3 months of developing bronchiolitis obliterans syndrome (8.3 [1.4-25.1] vs. 3.
19           Among lung transplant recipients, "bronchiolitis obliterans syndrome," a disorder with clin
20 epatitis C viral RNA (HCV RNA), freedom from bronchiolitis obliterans syndrome, acute rejection, and
21              Infants and young children with bronchiolitis obliterans syndrome after lung transplanta
22 Thin-section CT studies in six patients with bronchiolitis obliterans syndrome (age range, 2 months t
23  reported risk factor for the development of bronchiolitis obliterans syndrome, an important cause of
24 smatch model of multiorgan system cGVHD with bronchiolitis obliterans syndrome and a minor MHC mismat
25  study was to investigate the development of bronchiolitis obliterans syndrome and graft loss after L
26 onic lung allograft rejection in the form of bronchiolitis obliterans syndrome and its histopathologi
27          Because current literature suggests bronchiolitis obliterans syndrome and restrictive allogr
28 valuates the current diagnostic criteria for bronchiolitis obliterans syndrome and reviews the epidem
29 rom lung transplant recipients who developed bronchiolitis obliterans syndrome and were compared to s
30 e hypertension, renal dysfunction, diabetes, bronchiolitis obliterans syndrome, and malignancy.
31 nd the first 6 months, bacterial infections, bronchiolitis obliterans syndrome, and survival.
32 tervention in five patients with progressive bronchiolitis obliterans syndrome, anti-TNFalpha treatme
33                Emphysema, female gender, and bronchiolitis obliterans syndrome are risk factors for s
34 odel demonstrated that the increased risk of bronchiolitis obliterans syndrome associated with primar
35 condary end points were overall survival and bronchiolitis obliterans syndrome at 2 years.
36 emic steroids are the standard treatment for bronchiolitis obliterans syndrome (BOS) after allogeneic
37                In past years, a diagnosis of bronchiolitis obliterans syndrome (BOS) after allogeneic
38  genetic polymorphisms on the development of bronchiolitis obliterans syndrome (BOS) after lung trans
39                                              Bronchiolitis obliterans syndrome (BOS) after lung trans
40            Using log-rank test, freedom from bronchiolitis obliterans syndrome (BOS) and graft surviv
41 the internationally recognized definition of bronchiolitis obliterans syndrome (BOS) and longer follo
42  for chronic graft dysfunction manifested as bronchiolitis obliterans syndrome (BOS) and worse posttr
43                                 Freedom from bronchiolitis obliterans syndrome (BOS) at three years w
44                               Development of bronchiolitis obliterans syndrome (BOS) following lung t
45 d for multiorgan system cGVHD and associated bronchiolitis obliterans syndrome (BOS) in a murine mode
46 plant operation on survival and the onset of bronchiolitis obliterans syndrome (BOS) in consecutive l
47 rentiation is associated with development of bronchiolitis obliterans syndrome (BOS) in human lung al
48  after lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
49 lowing lung transplantation fails to prevent bronchiolitis obliterans syndrome (BOS) in many patients
50                                     The term bronchiolitis obliterans syndrome (BOS) is a clinical su
51                                              Bronchiolitis obliterans syndrome (BOS) is a condition o
52                           Early diagnosis of bronchiolitis obliterans syndrome (BOS) is critical in u
53    Chronic allograft rejection manifested as bronchiolitis obliterans syndrome (BOS) is the leading c
54                                              Bronchiolitis obliterans syndrome (BOS) is the major lim
55                                              Bronchiolitis obliterans syndrome (BOS) is the major lim
56                                              Bronchiolitis obliterans syndrome (BOS) is the major obs
57                                              Bronchiolitis obliterans syndrome (BOS) is the most comm
58                                              Bronchiolitis obliterans syndrome (BOS) is the primary l
59  allograft dysfunction (CLAD), presenting as bronchiolitis obliterans syndrome (BOS) or restrictive a
60 n (AR) and development of chronic rejection, bronchiolitis obliterans syndrome (BOS) remain major lim
61                                              Bronchiolitis obliterans syndrome (BOS) remains the lead
62                                              Bronchiolitis obliterans syndrome (BOS) remains the main
63 -obliteration of the allograft airway during bronchiolitis obliterans syndrome (BOS) that occurs afte
64                                              Bronchiolitis obliterans syndrome (BOS), a condition of
65 ement of six lung transplant recipients with bronchiolitis obliterans syndrome (BOS), a condition pre
66 s limited by infectious complications and by bronchiolitis obliterans syndrome (BOS), a form of chron
67                                              Bronchiolitis obliterans syndrome (BOS), a process of fi
68 gnized, idiopathic pneumonia syndrome (IPS), bronchiolitis obliterans syndrome (BOS), and bronchiolit
69 ssion to chronic rejection that manifests as bronchiolitis obliterans syndrome (BOS), but no biomarke
70 tive (ELR(+)) CXC chemokines associated with bronchiolitis obliterans syndrome (BOS), but the effect
71 n after lung transplantation, manifesting as bronchiolitis obliterans syndrome (BOS), has become the
72 g to progressive airflow obstruction, termed bronchiolitis obliterans syndrome (BOS), is the major ca
73 man lung allograft rejection, represented by bronchiolitis obliterans syndrome (BOS), is the single m
74 ween these disorders and risk for subsequent bronchiolitis obliterans syndrome (BOS), mortality and g
75                                              Bronchiolitis obliterans syndrome (BOS), pathognomonic f
76                                              Bronchiolitis obliterans syndrome (BOS), the clinical co
77                                              Bronchiolitis obliterans syndrome (BOS), the major cause
78 n the fibro-obliterative lesion found during bronchiolitis obliterans syndrome (BOS), we hypothesized
79 mputed tomography morphology, mortality, and bronchiolitis obliterans syndrome (BOS)-free survival we
80 ejection or infection) (NORMAL POST) or with bronchiolitis obliterans syndrome (BOS).
81 mortality after lung transplantation (LT) is bronchiolitis obliterans syndrome (BOS).
82        The primary endpoint was freedom from bronchiolitis obliterans syndrome (BOS).
83 to play an important role in the etiology of bronchiolitis obliterans syndrome (BOS).
84 hether it correlated with the development of bronchiolitis obliterans syndrome (BOS).
85 irreversible decline in lung function termed bronchiolitis obliterans syndrome (BOS).
86 splantation may contribute to development of bronchiolitis obliterans syndrome (BOS).
87 al owing to chronic allograft failure termed bronchiolitis obliterans syndrome (BOS).
88 e development of chronic rejection, known as bronchiolitis obliterans syndrome (BOS).
89 all airway injury would increase the risk of bronchiolitis obliterans syndrome (BOS).
90 tation and is an established risk factor for bronchiolitis obliterans syndrome (BOS).
91 al outcomes, often due to the development of bronchiolitis obliterans syndrome (BOS).
92 tation remains limited by the development of bronchiolitis obliterans syndrome (BOS).
93 The per-protocol analysis shows incidence of bronchiolitis obliterans syndrome (BOS): 1/43 in the Eve
94 ce of all other causes (currently defined as bronchiolitis obliterans syndrome [BOS]) is considered t
95 ar that patients may develop an obstructive (bronchiolitis obliterans syndrome [BOS]) or a restrictiv
96            A diagnosis of chronic rejection (bronchiolitis obliterans syndrome [BOS]) was made in 191
97               Different clinical phenotypes (bronchiolitis obliterans syndrome [BOS]-neutrophilic BOS
98 transplants is limited by chronic rejection (bronchiolitis obliterans syndrome, BOS).
99 sinophilic BAL predisposed to development of bronchiolitis obliterans syndrome but particularly to re
100 s that CMVIG prophylaxis reduces the risk of bronchiolitis obliterans syndrome, but a controlled tria
101   Acute rejection is a major risk factor for bronchiolitis obliterans syndrome, but noninvasive bioma
102 d a shorter survival and an earlier onset of bronchiolitis obliterans syndrome compared with patients
103 pha as a potential new therapeutic target in bronchiolitis obliterans syndrome deserving of a randomi
104                               This resembles bronchiolitis obliterans syndrome developed following hu
105  Adjustment for clinical variables including bronchiolitis obliterans syndrome did not change this re
106 aft-vs-host disease affects the lung tissue, bronchiolitis obliterans syndrome ensues.
107     Secondary outcomes included freedom from bronchiolitis obliterans syndrome (fBOS) and rates of ac
108 nd 30-day mortality, follow-up survival, and bronchiolitis obliterans syndrome-free survival.
109    A trend, however, toward reduced onset of bronchiolitis obliterans syndrome grade 2 or 3 was obser
110  survival in a multivariable model including bronchiolitis obliterans syndrome grade and baseline FEV
111                                Patients with bronchiolitis obliterans syndrome had a higher risk of s
112                                              Bronchiolitis obliterans syndrome has been associated wi
113 o activate fibroblasts in the development of bronchiolitis obliterans syndrome has not been evaluated
114 ted rejection, acute cellular rejection, and bronchiolitis obliterans syndrome; however, the signific
115 enance macrolide therapy in the treatment of bronchiolitis obliterans syndrome in lung transplant rec
116 after heart transplantation, and potentially bronchiolitis obliterans syndrome in lung transplant rec
117 ges to the progress of medical management of bronchiolitis obliterans syndrome include difficulties a
118 tion is associated with an increased risk of bronchiolitis obliterans syndrome independent of acute r
119                                              Bronchiolitis obliterans syndrome is a fibrotic occlusio
120                                              Bronchiolitis obliterans syndrome is a major problem for
121                                              Bronchiolitis obliterans syndrome is caused by a fibropr
122                                              Bronchiolitis obliterans syndrome is characterized by fi
123                                              Bronchiolitis obliterans syndrome is the leading cause o
124                       Small airway fibrosis (bronchiolitis obliterans syndrome) is the primary obstac
125 ) chronic allograft dysfunction, manifest by bronchiolitis obliterans syndrome, is frequent and limit
126 and were divided into three groups: no CLAD (bronchiolitis obliterans syndrome level 0 [BOS 0]), earl
127 lantation imitate the in vivo development of bronchiolitis obliterans syndrome-like lesions and revea
128                     Recent data suggest that bronchiolitis obliterans syndrome may affect up to 6% of
129 o be important in obliterative bronchiolitis/bronchiolitis obliterans syndrome (OB/BOS), which severe
130 onic lung allograft dysfunction manifests as bronchiolitis obliterans syndrome or the recently descri
131 was not a risk factor for the development of bronchiolitis obliterans syndrome or worse overall survi
132 rvival (P = 0.09) and increased freedom from bronchiolitis obliterans syndrome (P = 0.03) was observe
133 sease (P=0.54) nor a subsequent diagnosis of bronchiolitis obliterans syndrome (P=0.70).
134                                              Bronchiolitis obliterans syndrome remains the leading ca
135 genesis of chronic lung allograft rejection (bronchiolitis obliterans syndrome) remains to be elucida
136 trated that respiratory viral infection is a bronchiolitis obliterans syndrome risk factor and virus-
137              The remaining four patients had bronchiolitis obliterans syndrome scores of 0 compared w
138 iated with a significantly increased risk of bronchiolitis obliterans syndrome stage 1 (grade 1: rela
139 acute rejection, lymphocytic bronchitis, and bronchiolitis obliterans syndrome stage 1, using univari
140 sponse to viruses and in the pathogenesis of bronchiolitis obliterans syndrome, the predominant manif
141                                 Freedom from bronchiolitis obliterans syndrome was lower, and mortali
142 the association of bronchial dilatation with bronchiolitis obliterans syndrome was significant (P = .
143 s in the six patients with clinically proved bronchiolitis obliterans syndrome were mosaic perfusion
144 hown to be implicated in the pathogenesis of bronchiolitis obliterans syndrome, which is considered t

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