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1 ludes B. pertussis, B. parapertussis, and B. bronchiseptica.
2 strains of Bordetella hinzii and Bordetella bronchiseptica.
3 n to be required for optimal virulence of B. bronchiseptica.
4 ndent gene regulation would also occur in B. bronchiseptica.
5 ur1, one of two fur homologues carried by B. bronchiseptica.
6 erved among multiple clinical isolates of B. bronchiseptica.
7 h CyaA is not critical for the success of B. bronchiseptica.
8 rge in-frame deletion relative to batB of B. bronchiseptica.
9 development of cell-free vaccines against B. bronchiseptica.
10 racts of mice more rapidly than wild-type B. bronchiseptica.
11 ned nasal tissues from mice infected with B. bronchiseptica.
12 em regulates biofilm formation in Bordetella bronchiseptica.
13 rongly support nosocomial transmission of B. bronchiseptica.
14 fectively reduced ciliary binding by Bvg+ B. bronchiseptica.
15 gens Bordetella parapertussis and Bordetella bronchiseptica.
16 pared with BMDCs treated with heat-killed B. bronchiseptica.
17 jority of the transcriptional response to B. bronchiseptica.
18 as shown in a previous study with Bordetella bronchiseptica.
19 al biofilm formation in the Bvgi phase in B. bronchiseptica.
20 synthesis of Bps and biofilm formation by B. bronchiseptica.
21 es, did not silence expression of bfrD in B. bronchiseptica.
22 ica or B. pertussis inhibited shedding of B. bronchiseptica.
23 ation in F. tularensis as well as Bordetella bronchiseptica.
24 ation in the lungs of mice than wild-type B. bronchiseptica.
25 a felis, Chlamydophila felis, and Bordetella bronchiseptica.
26 lis, 5 for FCV, 1 for C. felis, and 0 for B. bronchiseptica.
28 In this study, we examined the effects of B. bronchiseptica ACT and TTSS on murine bone marrow-derive
32 athogens Bordetella pertussis and Bordetella bronchiseptica Although B. pertussis represents a pathog
36 erric enterobactin utilization by Bordetella bronchiseptica and Bordetella pertussis requires the Bfe
38 coding lipid A 3-O-deacylase from Bordetella bronchiseptica and by inactivation of the lgtB gene enco
39 nomes of Bordetella pertussis and Bordetella bronchiseptica and controls their infectious cycles.
40 to infection with relatively low doses of B. bronchiseptica and in vivo neutralization studies indica
42 e 1beta-ADP pathways operative in Bordetella bronchiseptica and Mesorhizobium loti and by the GmhB of
43 e terminal trisaccharide, while wild-type B. bronchiseptica and mutants lacking only the palmitoyl tr
44 t information obtained studying FHA using B. bronchiseptica and natural-host animal models should app
47 amplifying and disseminating vectors for B. bronchiseptica and reveal an important role for the Bvg-
48 ns with the respiratory bacterium Bordetella bronchiseptica and the gastrointestinal helminth Trichos
50 atory diseases in a long list of animals (B. bronchiseptica) and whooping cough in humans (B. pertuss
51 ive analysis of the Bordetella pertussis, B. bronchiseptica, and B. parapertussis genome assemblies p
52 t studies addressing virulence factors of B. bronchiseptica are based on isolates derived from hosts
53 rdetella pertussis, B. parapertussis, and B. bronchiseptica are closely related species associated wi
54 eport, the fhaB genes of B. pertussis and B. bronchiseptica are functionally interchangeable, at leas
55 with the idea that the O-antigen loci of B. bronchiseptica are horizontally transferred between stra
57 line augmented transferrin utilization by B. bronchiseptica, as well as siderophore function in vitro
59 Ac by various Bordetella species (Bordetella bronchiseptica, B. pertussis, and B. parapertussis) and
60 ring analog to phage display, the Bordetella bronchiseptica bacteriophage (BP) employs a highly varia
62 t, bfeA transcription in B. pertussis and B. bronchiseptica bfeR mutants was completely unresponsive
63 fusion analyses found that expression of B. bronchiseptica bfrA was increased during iron starvation
64 ctional in B. bronchiseptica, but neither B. bronchiseptica bfrD nor bfrE imparted catecholamine util
66 alyses of the extracellular components of B. bronchiseptica biofilm matrix revealed that the major su
69 espondingly, TLR4 is critical in limiting B. bronchiseptica but not B. pertussis or B. parapertussis
70 ization of the mouse respiratory tract by B. bronchiseptica, but is required for persistence of the o
71 pertussis were shown to be functional in B. bronchiseptica, but neither B. bronchiseptica bfrD nor b
73 ciated with virulence in B. pertussis and B. bronchiseptica (bvgS, fhaB, fhaC, and fimC) were identif
74 sis are predominantly differentiated from B. bronchiseptica by large, species-specific regions of dif
76 In this report, we determine that Bordetella bronchiseptica can form biofilms in vitro and that the g
77 growth phase-dependent gene regulation in B. bronchiseptica can function independently from the BvgAS
80 rmones also induce bfeA transcription and B. bronchiseptica can use the catecholamine noradrenaline f
81 ly adapted to the human body temperature, B. bronchiseptica causes infection in a broad range of anim
83 irulence factors at 24 degrees C, whereas B. bronchiseptica cells resumed the production only upon te
84 in produced by all members of the Bordetella bronchiseptica cluster, which includes B. pertussis, B.
86 on model, mutation of arnT did not affect B. bronchiseptica colonization, growth, persistence through
87 cyaA promoter or in the bvgAS alleles of B. bronchiseptica compared to B. pertussis, but appears to
88 increased in mice infected with wild-type B. bronchiseptica compared with mice infected with TTSS mut
89 y loci indicated an increased capacity in B. bronchiseptica, compared to B. pertussis, for ex vivo ad
90 rica, Pseudomonas aeruginosa, and Bordetella bronchiseptica contain an outer membrane 3-O-deacylase (
92 conjugate vaccine composed of the Bordetella bronchiseptica core oligosaccharide with one terminal tr
94 iptome and CGH analysis, we report that a B. bronchiseptica cystic fibrosis isolate, T44625, contains
100 zed that the defective persistence of the B. bronchiseptica deltapagP mutant was due to an increased
101 y, we identified an open reading frame in B. bronchiseptica, designated bcfA (encoding BcfA [bordetel
102 ry phases, we found that the adherence of B. bronchiseptica did not decrease in these later phases of
103 d invasins, deletion of this protein from B. bronchiseptica did not result in any significant defect
105 pressed during infection, confirming that B. bronchiseptica does not modulate to the Bvg(-) phase in
107 d that <100 colony-forming units (CFU) of B. bronchiseptica efficiently infected mice and displaced c
111 eria that serve as an amoeba food source, B. bronchiseptica evades amoeba predation, survives within
112 DCs) from C57BL/6 mice infected with live B. bronchiseptica exhibited high surface expression of MHCI
113 omplement killing assay demonstrated that B. bronchiseptica exhibits pagP-dependent resistance to ant
114 eria Bordetella parapertussis and Bordetella bronchiseptica express a lipopolysaccharide O antigen co
115 In the virulent state (Bvg+), Bordetella bronchiseptica expresses adhesins and toxins that mediat
116 nonmotile human pathogens, while Bordetella bronchiseptica expresses flagellin and causes disease in
117 hese findings suggest that virulent-state B. bronchiseptica expresses multiple adhesins with overlapp
118 A(Bp) was able to substitute for FHA from B. bronchiseptica (FHA(Bb)) with regard to its ability to m
121 ithelial cells, we studied the effects of B. bronchiseptica flagellin on host defense responses.
125 to 7.6, Bordetella pertussis and Bordetella bronchiseptica FtrABCD system mutants showed dramatic re
132 , in addition to the structure of Bordetella bronchiseptica GmhB bound to Mg(2+) and orthophosphate (
134 sole NAD precursor, quinolinate promoted B. bronchiseptica growth, and the ability to use it require
137 -Mulneix et al. demonstrates that Bordetella bronchiseptica has two different gene suites that are ac
138 We hypothesize that hemin is acquired by B. bronchiseptica in a BhuR-dependent manner after spontane
139 rotective immune response against Bordetella bronchiseptica in a mouse model of intranasal infection.
141 hrine could promote the growth of Bordetella bronchiseptica in iron-restricted medium containing seru
142 aprn mutant did not differ from wild-type B. bronchiseptica in its ability to adhere to epithelial an
143 ils (PMN) are critical for the control of B. bronchiseptica in mice, our data support the hypothesis
144 (FHA(Bp)) and compared it with wild-type B. bronchiseptica in several natural-host infection models.
145 the bpsABCD locus to the pathogenesis of B. bronchiseptica in swine, the KM22Deltabps mutant was com
146 bution of the T3SS to the pathogenesis of B. bronchiseptica in swine, we compared the abilities of a
147 dependent contribution to pathogenesis of B. bronchiseptica in swine, we constructed a series of isog
151 on of the pagP gene on the persistence of B. bronchiseptica in the lower respiratory tract of mice.
154 were defective in reducing the numbers of B. bronchiseptica in the upper respiratory tract compared t
155 fhaS strain was out-competed by wild-type B. bronchiseptica, indicating that fhaS is expressed in viv
158 and provided evidence that FHA-deficient B. bronchiseptica induces more inflammation in the lungs of
161 + splenocytes, and that lung tissues from B. bronchiseptica-infected mice exhibit a strong Th17 immun
162 contributes to pulmonary host defense in B. bronchiseptica infection by recruiting lymphocytes and N
163 of host cells are dephosphorylated during B. bronchiseptica infection in a TTSS-dependent manner.
164 gA response contributes to the control of B. bronchiseptica infection of the upper respiratory tract,
166 ransplant center developed severe Bordetella bronchiseptica infections within 3 days of each other.
168 monstrate that norepinephrine facilitates B. bronchiseptica iron acquisition from the iron carrier pr
176 st inflammatory response to FHA-deficient B. bronchiseptica is characterized by the early and sustain
178 huRSTUV heme utilization locus in Bordetella bronchiseptica is coordinately controlled by the global
179 athogens Bordetella pertussis and Bordetella bronchiseptica is dependent on the BfeA outer membrane r
183 including those required for motility in B. bronchiseptica, is activated and genes encoding virulenc
184 hooping cough), whereas their progenitor, B. bronchiseptica, is of variable virulence in a wide varie
185 An extensive characterization of human B. bronchiseptica isolates is needed to better understand t
186 Cross-reactivity was found only with 5 B. bronchiseptica isolates, which were positive with IS1001
187 nes, BteA is secreted through the TTSS of B. bronchiseptica, it is required for cytotoxicity towards
188 is thought to have derived from a Bordetella bronchiseptica-like ancestor, we hypothesized that growt
189 . parapertussis evolved separately from a B. bronchiseptica-like progenitor to naturally infect only
190 e model that BhuR is a hemin receptor and B. bronchiseptica likely acquires heme during infection aft
191 involved in the increased virulence of a B. bronchiseptica lineage which appears to be disproportion
193 se data are consistent with the view that B. bronchiseptica lineages can have different levels of vir
196 endotoxins using RAW cells suggests that B. bronchiseptica lipopolysaccharide (LPS) is 10- and 100-f
200 stigated Bordetella pertussis and Bordetella bronchiseptica LPS-derived core oligosaccharide (OS) pro
201 indicate that persistent colonization by B. bronchiseptica may rely on the ability of the bacteria t
202 ature adaptation between B. pertussis and B. bronchiseptica may result from selective adaptation of B
203 This investigation characterizes a new B. bronchiseptica mechanism for iron uptake from transferri
204 sults indicate a critical role for FHA in B. bronchiseptica-mediated immunomodulation, and they sugge
208 was found to aggregate and permeabilize a B. bronchiseptica mutant lacking the terminal trisaccharide
213 Vaccination with heat-killed whole-cell B. bronchiseptica or B. pertussis inhibited shedding of B.
218 identification of a novel gene in Bordetella bronchiseptica, plrS, the product of which shares sequen
219 tranasal inoculation of mice with Bordetella bronchiseptica produces a transient pneumonia that is cl
220 losely related zoonotic pathogen, Bordetella bronchiseptica, raising important questions about the co
221 Bordetella pertussis Tohama I and Bordetella bronchiseptica RB50 differ in the number of 90-amino-aci
224 iderably lesser extent when compared with B. bronchiseptica Remarkably, B. pertussis maintained the p
225 nd myoglobin as sources of nutrient Fe by B. bronchiseptica requires expression of BhuR, an outer mem
226 sing cloned alcS genes of B. pertussis or B. bronchiseptica restored the wild-type phenotype to the a
228 phase-dependent gene regulation occurs in B. bronchiseptica, resulting in prominent temporal shifts i
230 sis of Bvg regulation in B. pertussis and B. bronchiseptica revealed a relatively conserved Bvg(+) ph
231 immunization strategies aimed at inducing B. bronchiseptica-specific IgA may be beneficial to prevent
232 ion of vaccines, we constructed a Bordetella bronchiseptica strain (LPaV) that does not express the a
234 versus chronic disease, we constructed a B. bronchiseptica strain expressing FHA from B. pertussis (
236 CVs) from the lungs of mice infected with B. bronchiseptica strain RBX9, which contains an in-frame d
239 eady-state manner by constructing Bordetella bronchiseptica strains in which the bvgAS promoter was r
240 Multilocus sequence typing analysis of 49 B. bronchiseptica strains was used to build a phylogenetic
242 infection, we found that the virulence of B. bronchiseptica strains, as measured by the mean lethal d
243 t are protective against highly divergent B. bronchiseptica strains, preventing colonization in the l
245 hen compared to Bvg+ or Bvg- phase-locked B. bronchiseptica strains, single-knockout strains lacking
248 pneumonia after inoculation with Bordetella bronchiseptica, suggesting that TLR4 is required for exp
249 -gamma production by the TTSS facilitates B. bronchiseptica survival in the lower respiratory tract.
250 series of isogenic mutants in a virulent B. bronchiseptica swine isolate and compared each mutant to
252 ne, is activated substantially earlier in B. bronchiseptica than B. pertussis following a switch from
253 parapertussis are more closely related to B. bronchiseptica than they are to each other, they share t
257 lar characterization of ZIPB from Bordetella bronchiseptica, the first ZIP homolog to be purified and
261 family sensor kinases and is required for B. bronchiseptica to colonize and persist in the lower, but
262 ically, FHA(Bb), but not FHA(Bp), allowed B. bronchiseptica to colonize the lower respiratory tracts
263 , FhaS was unable to mediate adherence of B. bronchiseptica to epithelial cell lines in vitro and was
264 trating a stable relationship that allows B. bronchiseptica to expand and disperse geographically via
265 es are involved in the ability of Bordetella bronchiseptica to grow and disseminate via the complex l
266 of Paraburkholderia phytofirmans allowed B. bronchiseptica to grow in the absence of supplied pyridi
267 results suggest that pagP is required for B. bronchiseptica to resist antibody-mediated complement ly
268 y its catalytic activity, is required for B. bronchiseptica to resist phagocytic clearance but is nei
269 hypothesized that the ability of Bordetella bronchiseptica to undergo phenotypic modulation is requi
270 o mouse studies, we hypothesized that the B. bronchiseptica type III secretion system (T3SS) would be
272 an immunomodulation involving the Bordetella bronchiseptica type III secretion system (TTSS) which co
275 t studies addressing virulence factors of B. bronchiseptica utilize isolates derived from hosts other
279 usly for B. pertussis, bfrD expression in B. bronchiseptica was also dependent on the BvgAS virulence
280 to demonstrate that the rate of growth of B. bronchiseptica was directly correlated with the rate at
286 med by the sequenced laboratory strain of B. bronchiseptica We hypothesized that swine isolates would
287 ing the broad host range pathogen Bordetella bronchiseptica We recently discovered an additional sens
288 g mice that are natural host's of Bordetella bronchiseptica, we determined the effects of vaccination
289 ng the mouse respiratory pathogen Bordetella bronchiseptica, we examined the mechanisms of Ab-mediate
290 ll death, type III-secreted proteins from B. bronchiseptica were analyzed using matrix-assisted laser
292 nal regulators that were Bvg regulated in B. bronchiseptica were deleted, inactivated, or unregulated
294 gh, is a human-adapted variant of Bordetella bronchiseptica, which displays a broad host range and ty
295 w that the cyaA genes of B. pertussis and B. bronchiseptica, which encode adenylate cyclase toxin (AC
296 d that the fhaB genes of B. pertussis and B. bronchiseptica, which encode filamentous hemagglutinin (
297 a genetically engineered double mutant of B. bronchiseptica, which lacks adenylate cyclase and type I
299 4 (TLR4) in immunity to B. pertussis and B. bronchiseptica, while no role for TLR4 during B. paraper
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