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1 isease including COPD (emphysema and chronic bronchitis).
2 IgG (not IgE), and positive GM (Aspergillus bronchitis).
3 ranulomatous rhinosinusitis, and aspergillus bronchitis.
4 ay diseases, including emphysema and chronic bronchitis.
5 meningitis and one in the combined group had bronchitis.
6 ogression that are characteristic of chronic bronchitis.
7 w in primary care and in patients with acute bronchitis.
8 ons seen in chronic otitis media and chronic bronchitis.
9 f chronic obstructive pulmonary disease, and bronchitis.
10 sfunction and acute rejection or lymphocytic bronchitis.
11 tract infections, including otitis media and bronchitis.
12 e respiratory diseases such as pneumonia and bronchitis.
13 no better than low-dose vitamin C for acute bronchitis.
14 ted, FEV(1)/FVC < 90% predicted) and chronic bronchitis.
15 excess of that seen in patients with chronic bronchitis.
16 (LOD = 1.21) and 22 (LOD = 1.37) for chronic bronchitis.
17 es of physician-diagnosed asthma and chronic bronchitis.
18 eas of CD3(+) CD8(+) T cell-rich lymphocytic bronchitis.
19 in patients with smoking-associated chronic bronchitis.
20 c fibrosis (CF) causes a chronic destructive bronchitis.
21 pneumonia and acute exacerbations of chronic bronchitis.
22 y airway diseases such as asthma and chronic bronchitis.
23 treatment of adults with uncomplicated acute bronchitis.
24 pathogen of humans, and causes pneumonia and bronchitis.
25 ough, mucus production, sinusitis, and acute bronchitis.
26 ns from the airways of patients with chronic bronchitis.
27 ere written for children diagnosed as having bronchitis.
28 luded a principal diagnosis of cold, URI, or bronchitis.
29 th common colds, 46% with URIs, and 75% with bronchitis.
30 help improve MCC in smoking-related chronic bronchitis.
31 gm other than when accompanied by a cold, or bronchitis.
32 atic perfusion in most patients with plastic bronchitis.
33 ecreases in subjects with history of chronic bronchitis.
34 rapy in moderate-to-severe COPD with chronic bronchitis.
35 subphenotypes such as emphysema and chronic bronchitis.
36 illosis, chronic pulmonary aspergillosis and bronchitis.
37 g diseases such as asthma, COPD, and chronic bronchitis.
38 f chronic obstructive pulmonary disease, and bronchitis.
39 ay play a role in the development of chronic bronchitis.
40 ough, eczema, and parental history of asthma/bronchitis.
41 ith improved outcomes in patients with acute bronchitis?
43 ng/ml) and cough variant asthma/eosinophilic bronchitis (10.2 ng/ml) than in normal subjects (2.6 ng/
45 (16 mL/y [95% CI, 12-20 mL/y]), mild wheezy bronchitis (14 mL/y [95% CI, 8-19 mL/y]), wheezy bronchi
46 chitis (14 mL/y [95% CI, 8-19 mL/y]), wheezy bronchitis (16 mL/y [95% CI, 11-20 mL/y]), and persisten
47 ernatant in eight patients with eosinophilic bronchitis, 17 patients with asthma matched for sputum e
48 re significantly more likely to have chronic bronchitis (19.5% versus 6.1%) and emphysema (7.9% versu
50 II/IV, or protein-losing enteropathy/plastic bronchitis) 20 years after Fontan was 70% (95% CI, 63%-7
51 ts with cough variant asthma or eosinophilic bronchitis, 20 patients with nonasthmatic chronic cough,
52 diagnosed in 71.9% (pneumonia, 42.3%; acute bronchitis, 21.9%; chronic obstructive pulmonary disease
53 (URTIs) (24%), acute sinusitis (24%), acute bronchitis (23%), otitis media (5%), pharyngitis, laryng
54 itions for one or more ARI: pneumonia (537), bronchitis (2931), sinusitis (717) and non-specific ARI
55 RI, including the common cold (53.4%), acute bronchitis (31.3%), acute sinusitis (13.6%), or acute la
58 42); asthma: 27 +/- 40 pg/ml (n = 11); acute bronchitis: 44 +/- 112 pg/ml (n = 14); pneumonia: 55 +/-
59 age of 50 years was 64% in those with wheezy bronchitis, 47% for those with persistent asthma, and 15
61 tions of COPD were used: symptoms of chronic bronchitis (667 subjects), physician-diagnosed emphysema
63 for sinusitis (adjusted proportion, 86%) and bronchitis (85%) and varied little according to fever, a
64 or the highest quartile was 0.66 for chronic bronchitis (95 percent confidence interval, 0.52 to 0.85
66 nd 6.4-fold (1.4, 28) higher in eosinophilic bronchitis and 1.9-fold (1.3, 2.9) and 7.7-fold (1.2, 46
67 cipients with viral infection and transplant bronchitis and again observed excessive epithelial p80 e
69 ficant damage to the lungs with acute, focal bronchitis and alveolitis associated with massive pulmon
74 first public hospital admission for asthma, bronchitis and bronchiolitis (International Classificati
77 -Antitrypsin (AAT) deficiency predisposes to bronchitis and emphysema associated with neutrophilic ai
78 r co-occurring respiratory diseases, chronic bronchitis and emphysema continue to have a positive ass
80 e pulmonary disease (COPD) comprises chronic bronchitis and emphysema, and is a leading cause of morb
82 lymphatic imaging, in patients with plastic bronchitis and percutaneous lymphatic intervention as a
84 causing otitis media in children and chronic bronchitis and pneumonia in patients with chronic obstru
85 e of otitis media in children and of chronic bronchitis and pneumonia in patients with chronic obstru
86 re frequent causes of repeated common colds, bronchitis and pneumonia, which often occur unpredictabl
91 variable combined those with either chronic bronchitis and/or emphysema, together considered as chro
92 onchitic symptoms (chronic cough, phlegm, or bronchitis) and of wheeze in the previous 12 months were
93 7 of 218) had pneumonia, 32% (70 of 218) had bronchitis, and 14% (31 of 218) had colonization; in-hos
95 vere airflow limitation, symptoms of chronic bronchitis, and a history of exacerbations, whose diseas
96 e tobacco smoke exposure, emphysema, chronic bronchitis, and asthma (each condition and the combined
97 r those with history of diabetes, emphysema, bronchitis, and asthma, respectively, compared with thos
98 prevalence of self-reported asthma, chronic bronchitis, and asthma-like respiratory symptoms among w
100 vere airflow limitation, symptoms of chronic bronchitis, and at least two exacerbations in the previo
101 with public hospital admissions for asthma, bronchitis, and bronchiolitis (International Classificat
102 dysfunction on acute rejection, lymphocytic bronchitis, and bronchiolitis obliterans syndrome stage
105 independent of acute rejection, lymphocytic bronchitis, and community-acquired respiratory viral inf
106 independent of acute rejection, lymphocytic bronchitis, and community-acquired respiratory viral inf
112 disease includes both emphysema and chronic bronchitis, and in the case of chronic bronchitis repres
113 ic upper respiratory tract infections, acute bronchitis, and influenza) from 18 months preinterventio
114 or acute sinusitis, acute pharyngitis, acute bronchitis, and nonspecific upper respiratory tract infe
116 of occupational exposures in asthma, chronic bronchitis, and respiratory symptoms in the Singapore Ch
118 h childhood asthma; 53 with childhood wheezy bronchitis; and 239 control subjects (of whom 57 develop
119 atasvir-voxilaprevir alone were diarrhea and bronchitis; and with sofosbuvir-velpatasvir-voxilaprevir
121 opriate antibiotic use for adults with acute bronchitis apply to immunocompetent adults without compl
122 well as bronchiolitis obliterans and chronic bronchitis are chronic lung diseases characterized by ai
123 FEV1 below 60%, FEV1 below 80%, and chronic bronchitis are each approximately three in current or ex
125 onia, atypical pneumonia, bronchiolitis, and bronchitis-are responsible for much morbidity and mortal
126 ce area (emphysema) and airway inflammation (bronchitis) as the consequence of cigarette smoke (CS) e
127 bjects and compared large-airway lymphocytic bronchitis assessed by a 0-2 "E-score" and with standard
130 upper respiratory infection, croup, asthma, bronchitis, bronchiolitis, a wheezy lower respiratory in
133 e respiratory tree and can cause tracheitis, bronchitis, bronchiolitis, diffuse alveolar damage with
134 ry illness in the first year of life (croup, bronchitis, bronchiolitis, or pneumonia) (RR = 2.25; 95%
135 acute perivascular rejection and lymphocytic bronchitis/bronchiolitis (LBB), and the grades were tota
137 L-12 was also inducible during paramyxoviral bronchitis, but in this case, initial IL-12 p70 expressi
138 ammatory diseases such as asthma and chronic bronchitis, but the mechanisms and consequences of the c
139 s suggest that antibiotic treatment of acute bronchitis can be reduced by using a combination of pati
140 ults diagnosed as having uncomplicated acute bronchitis can be safely reduced using a combination of
141 proximately 10% of pneumonia cases and 5% of bronchitis cases are attributed to C. pneumoniae infecti
146 -years of smoking, and asthma, emphysema, or bronchitis (chronic obstructive pulmonary disease) were
147 seen in respiratory disorders (eg, asthma or bronchitis, chronic obstructive pulmonary disease (COPD)
148 nd associated diseases, e.g. asthma, chronic bronchitis, chronic obstructive pulmonary disease, and h
149 (nose, eyes, and throat irritations; cough; bronchitis; cold; wheezing; asthma attacks), medication
152 hildren diagnosed as having colds, URIs, and bronchitis, conditions that typically do not benefit fro
154 E, namely airway hyperresponsiveness (AHR), bronchitis, cough reflex hypersensitivity, damage to the
155 act infection; however, for otitis media and bronchitis, declines were only observed in the populatio
156 Patient satisfaction with care for acute bronchitis depends most on physician-patient communicati
158 xposure, atopic diseases), symptoms (chronic bronchitis, dyspnea-modified Medical Research Council sc
159 f nonmalignant respiratory diseases (asthma, bronchitis, emphysema, hay fever, and pneumonia) in 1,55
160 redicted, in males whose parents had asthma, bronchitis, emphysema, or hay fever and with increased p
162 the role of previous lung diseases (chronic bronchitis, emphysema, pneumonia, and tuberculosis) in t
163 ate lung cancer risk associated with chronic bronchitis, emphysema, tuberculosis, pneumonia, and asth
165 ns even in asthma patients with neutrophilic bronchitis, EPX-based ELISA levels are not increased in
167 pper respiratory tract infections, and acute bronchitis (for which routine antibiotic treatment is no
169 g chronic cough include asthma, eosinophilic bronchitis, gastro-oesophageal reflux disease, postnasal
170 otitis media, sinusitis, and exacerbation of bronchitis, has acquired widespread ability to produce b
171 yperplasia, histological features of chronic bronchitis, have been linked to epidermal growth factor
173 e cells induced a lymphocytic vasculitis and bronchitis, ICAM-1 mutant allogeneic BAL cells only indu
176 d increased odds of reduced FEV1 and chronic bronchitis in current or ex-smoking first-degree relativ
177 s 1.64 at chromosome 19, whereas for chronic bronchitis in smokers only, the maximum LOD was 2.08 at
181 Pulmonary emphysema, together with chronic bronchitis is a part of chronic obstructive pulmonary di
183 liary clearance associated with mild chronic bronchitis is acutely improved with minimal doses of aer
187 antibiotic treatment of uncomplicated acute bronchitis is not recommended, regardless of duration of
188 value of azithromycin for treatment of acute bronchitis is unknown, even though this drug is commonly
190 t COPD probands for reduced FEV1 and chronic bronchitis, limited to current or ex-smokers, suggests g
191 ; 95% CI: 1.47-3.65), daily intake of asthma/bronchitis medication (OR = 2.26; 95% CI: 1.42-3.58), fi
194 of UTP in chronic smokers with mild chronic bronchitis (n = 15) by measuring the clearance of (99m)T
195 n = 189,816), and for 0- to 18-year-olds for bronchitis (n = 76,243), chronic sinusitis (n = 15,745),
198 terval, 3.73-10.94), as was childhood wheezy bronchitis (odd ratio 1.81; 95% confidence interval, 1.1
200 g toxic effects were hospital admissions for bronchitis (one) and pleural effusion (one), grade 3 inc
201 ve association with lung cancer than chronic bronchitis "only." Asthma had an inverse association wit
203 uded a doctor's diagnosis of asthma, chronic bronchitis or COPD, and a history of wheezing or use of
204 14% vs. 10%), while similar rates of chronic bronchitis or emphysema (1% vs. 1%) and asthma (1% vs. 1
206 s (within 30 days of the incident visit) for bronchitis or pneumonia did not change significantly for
207 nfidence interval (CI): 1.08, 1.30), chronic bronchitis (OR = 1.26, 95% CI: 1.01, 1.57), and adult-on
208 confidence interval (CI) 1.10-3.43), chronic bronchitis (OR = 1.73, 95% CI 1.10-2.72), and the combin
211 the combined endpoint of emphysema, chronic bronchitis, or asthma (OR = 1.82, 95% CI 1.26-2.63).
215 rence in efficacy by sex, history of chronic bronchitis, oxygen use, or concomitant COPD therapy.
216 ponse by sex (P = 0.75), presence of chronic bronchitis (P = 0.19), concomitant inhaled therapy (P =
217 alue) and with an increased risk of allergic bronchitis (p = 1.77*10(-4) and p = 7.94*10(-4), respect
218 ay quantitate a key component of the chronic bronchitis pathophysiologic cascade that produces sputum
220 hogen that frequently causes ear infections, bronchitis, pneumonia, and exacerbations in patients wit
221 waterborne pathogen responsible for causing bronchitis, pneumonia, and gastrointestinal infections,
224 level and increased asthma and bronchiolitis/bronchitis rates in childhood were consistent with a pre
225 ronic bronchitis, and in the case of chronic bronchitis represents an inflammatory response of the ai
226 owing Medical Subject Headings terms: "acute bronchitis," "respiratory tract infection," "pharyngitis
228 COPD), which comprises emphysema and chronic bronchitis resulting from prolonged exposure to cigarett
230 (RR, 1.15; 95% CI, 1.00-1.32; P = .05), and bronchitis (RR, 1.18; 95% CI, 1.01-1.37; P = .04) at any
232 presumptive diagnosis of uncomplicated acute bronchitis should focus on ruling out serious illness, p
234 ified Medical Research Council >/=2, chronic bronchitis, St. George's Respiratory Questionnaire >25,
237 associations of e-cigarette use with chronic bronchitis symptoms and wheeze in an adolescent populati
239 tructive pulmonary disease (COPD)-associated bronchitis than in those from throats of healthy individ
240 on and susceptibility to recurrent infective bronchitis that may, in turn, contribute to further ster
242 kage of mild airflow obstruction and chronic bronchitis to several genomic regions; for mild airflow
243 sistent with acute rejection and lymphocytic bronchitis, to subepithelial and intraluminal fibrotic l
244 reatment-related sinusitis, pneumonia, viral bronchitis, tooth infection, stomatitis, and leukopenia
245 neric term encompasses emphysema and chronic bronchitis, two common conditions, each having distinct
246 ronic obstructive pulmonary disease, chronic bronchitis, two or more exacerbations and/or hospitaliza
247 A sample enriched for asthma and chronic bronchitis underwent a further questionnaire, pre- and p
251 eral gammacoronaviruses including infectious bronchitis virus (IBV) contain a putative open reading f
252 tein from the group 3 coronavirus infectious bronchitis virus (IBV) contains a canonical dilysine end
253 he genomic RNA of the coronavirus infectious bronchitis virus (IBV) contains a classic hairpin-type R
254 t signal of the avian coronavirus infectious bronchitis virus (IBV) contains two cis-acting signals e
255 we show that the Gammacoronavirus infectious bronchitis virus (IBV) does induce host shutoff, and we
256 r investigate the function of the infectious bronchitis virus (IBV) E protein, we developed a recombi
257 The E protein from the avian infectious bronchitis virus (IBV) has dramatic effects on the secre
258 her viruses, the gammacoronavirus infectious bronchitis virus (IBV) has evolved under evolutionary pr
259 the E protein of the coronavirus infectious bronchitis virus (IBV) is localized to the Golgi complex
260 n the present study, we show that infectious bronchitis virus (IBV) is resistant to IFN treatment and
263 The potential interactome of the infectious bronchitis virus (IBV) N protein was mapped using stable
266 gence of a nephropathogenic avian infectious bronchitis virus (IBV) with a novel genotype in India.
269 otein from the avian coronavirus, infectious bronchitis virus (IBV), contains information for localiz
270 he prototypical Gammacoronavirus, infectious bronchitis virus (IBV), induces a delayed activation of
271 strain of avian Gammacoronavirus infectious bronchitis virus (IBV), induces regions of ER that are z
272 mmacoronavirus, including chicken infectious bronchitis virus (IBV), require specific alpha2,3-linked
273 the frameshifting pseudoknot from infectious bronchitis virus (IBV), three constituent hairpins, and
277 ble to inhibit the early phase of infectious bronchitis virus and Dengue infection, in addition to th
278 aracterization of CoV N NTDs from infectious bronchitis virus and from severe acute respiratory syndr
279 tail for two other coronaviruses, infectious bronchitis virus and the severe acute respiratory syndro
280 erimental data for adenovirus and infectious bronchitis virus infections as examples, we demonstrate
281 domain targeting signal from the infectious bronchitis virus M protein and the lumenal and cytoplasm
282 acid identity similar to that of infectious bronchitis virus N protein and shares a higher level of
283 ellular localization of the avian infectious bronchitis virus N protein both in the absence and in th
286 increased with childhood asthma, and wheezy bronchitis was associated with reduced FEV1 that was evi
289 probands for airflow obstruction and chronic bronchitis was performed to determine whether significan
291 exposure to pets, doctor's diagnosed wheezy bronchitis (WB), and compositional changes in the gut mi
292 the prevalence odds ratios (ORs) for chronic bronchitis were 0.98, 0.88, and 0.69 for the second, thi
293 c prescription rates for uncomplicated acute bronchitis were similar at all 4 sites during the baseli
295 g acute otitis media, sinusitis, and chronic bronchitis, which are preceded by asymptomatic H. influe
296 ic obstructive pulmonary disease and chronic bronchitis who are at risk of frequent and severe exacer
297 rrected congenital heart disease and plastic bronchitis who presented for lymphatic imaging and inter
298 s active airway inflammation in eosinophilic bronchitis, with reduced release of important effector m
300 cin concentration and a diagnosis of chronic bronchitis yielded areas under the curve of 0.72 (95% co
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