ライフサイエンスコーパス: bronchoalveolar

1 nd protein levels were highly upregulated in bronchoalveolar cells and fluid after allergen challenge

2 2287 significantly decreased esophageal and bronchoalveolar eosinophilia but only when given as a th

3 ermeability, lung histology, AFC, and plasma/bronchoalveolar fluid measurements of proinflammatory cy

4 ated with sRAGE levels in the plasma and the bronchoalveolar fluid of acid-injured mice (Spearman's r

5 d anti-inflammatory cytokines (IL-10) in the bronchoalveolar fluid, and IL-2 and IFN-gamma cytokines

6 serum IgG antibodies as well as vaginal and bronchoalveolar IgA antibodies.

7 ntral and peripheral airway remodelling, and bronchoalveolar inflammation were assessed.

8 Cells recovered by bronchoalveolar lavage (BAL) after imaging were stained

9 tegrated study of human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine

10 bit AHR, increased numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen cont

11 he diagnostic yield and complication rate of bronchoalveolar lavage (BAL) and lung biopsy in the eval

12 Bronchoalveolar lavage (BAL) and lung tissue were examin

13 a methacholine test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total

14 e expression and activity were determined in bronchoalveolar lavage (BAL) and lungs of human donors a

15 We investigated bronchoalveolar lavage (BAL) and serum samples from pati

16 ort study; 72 consented to bronchoscopy with bronchoalveolar lavage (BAL) and transbronchial biopsies

17 Conventional methods to identify HBEC in bronchoalveolar lavage (BAL) and wash (BW) have throughp

18 cDNA library derived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sar

19 was to investigate the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from centr

20 Using blood and bronchoalveolar lavage (BAL) cells from normal control s

21 esponsiveness (AHR) to inhaled methacholine, bronchoalveolar lavage (BAL) cytokine levels, and lung h

22 ) and nearly 7-fold more active TGF-beta1 in bronchoalveolar lavage (BAL) fluid (BALF).

23 phil and bacterial counts were determined in bronchoalveolar lavage (BAL) fluid and blood.

24 perimental model of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were colle

25 igatus Its performance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens,

26 and production of inflammatory cytokines in bronchoalveolar lavage (BAL) fluid and the lungs.

27 ormed in nasopharyngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

28 (1,3)beta-D-glucan detection in bronchoalveolar lavage (BAL) fluid by Fungitell assay ai

29 elial growth factor (VEGF) levels in hamster bronchoalveolar lavage (BAL) fluid early after intranasa

30 A gene sequencing was performed on acellular bronchoalveolar lavage (BAL) fluid from 30 subjects infe

31 ic bead array, and L-ficolin was measured in bronchoalveolar lavage (BAL) fluid from lung transplant

32 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic

33 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines

34 We found that bleomycin injury increases the bronchoalveolar lavage (BAL) fluid levels of ATX protein

35 mesenteric lymph nodes (MLNs), jejunum, and bronchoalveolar lavage (BAL) fluid of healthy and SIV-in

36 nsus sequences of viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.

37 rometry based proteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observa

38 respiratory tract disease, 72 paired NP and bronchoalveolar lavage (BAL) fluid specimen sets, mostly

39 We analyzed 46 clinically obtained bronchoalveolar lavage (BAL) fluid specimens from sympto

40 Bronchoalveolar lavage (BAL) fluid was assessed for tota

41 Using a cutoff of >/=2% eosinophilia in bronchoalveolar lavage (BAL) fluid, chronic lung allogra

42 manifestation, so we evaluated biomarkers in bronchoalveolar lavage (BAL) fluid.

43 responses, we hypothesized that MV, found in bronchoalveolar lavage (BAL) fluids (BALF) of LTR at CLA

44 igated M. tuberculosis-specific responses in bronchoalveolar lavage (BAL) from persons with latent M.

45 We measured mtDNA concentrations in bronchoalveolar lavage (BAL) from subjects with and with

46 ALI was quantified by weight loss, bronchoalveolar lavage (BAL) inflammatory cell number, c

47 We assessed bronchoalveolar lavage (BAL) inflammatory cell numbers,

48 ls (MSCs) in the terminal airways-alveoli by bronchoalveolar lavage (BAL) of human adult lungs.

49 d their binding to TIMP-1, -2, -3, and -4 in bronchoalveolar lavage (BAL) of lung transplant recipien

50 r flow cytometry in digested lung tissue and bronchoalveolar lavage (BAL) simultaneously, 6 h after e

51 ng flow cytometry and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52

52 However, no CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia

53 Six hours later, bronchoalveolar lavage (BAL) was collected for leukocyte

54 Airway responsiveness was assessed, bronchoalveolar lavage (BAL) was performed, and lung cel

55 s of CD4(+) T and B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.

56 ergic patients underwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hou

57 SCFA levels in anaerobic supernatants and bronchoalveolar lavage (BAL) were determined by gas chro

58 The predominant macrophages harvested by bronchoalveolar lavage (BAL), alveolar macrophages (AMs)

59 flux of neutrophils and macrophages into the bronchoalveolar lavage (BAL), and human CD45(+) cells in

60 Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood

61 D4 and CD8 T cell immune responses in blood, bronchoalveolar lavage (BAL), and tracheobronchial lymph

62 linically indicated fiberoptic bronchoscopy, bronchoalveolar lavage (BAL), endobronchial brushings, a

63 t computed tomography, and bronchoscopy with bronchoalveolar lavage (BAL).

64 ated with proinflammatory cytokines in human bronchoalveolar lavage (BAL).

65 by an accumulation of CD4(+) T cells in the bronchoalveolar lavage (BAL).

66 aeruginosa, P. aeruginosa) were analyzed in bronchoalveolar lavage (BAL); and alveolar SGLT1 was ana

67 oneal (p = 0.037), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacteria

68 in several proinflammatory cytokines in the bronchoalveolar lavage and in serum.

69 ortant cellular sources of IL-5 and IL-13 in bronchoalveolar lavage and lung tissue.

70 ion model, IL-10-producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decre

71 ition of ADAM10 reduces sEphrin-B2 levels in bronchoalveolar lavage and prevents lung fibrosis in mic

72 Bronchoalveolar lavage and pulmonary function tests were

73 Double-stranded RNA in bronchoalveolar lavage and serum samples following lung

74 We found that levels of inflammation in the bronchoalveolar lavage and the lung, as well as levels o

75 Bronchoalveolar lavage and tissues were sampled for myco

76 ermate control animals through evaluation of bronchoalveolar lavage and tissues.

77 BAFF levels were also higher in blood and bronchoalveolar lavage B cells in patients with COPD ver

78 subjects had symptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induc

79 Following silica treatment, bronchoalveolar lavage cell infiltrates decreased in fem

80 Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS

81 Ag85A-specific CD4 T cells were detected in bronchoalveolar lavage cells from both groups and respon

82 At two independent study sites, bronchoalveolar lavage cells from donors with latent tub

83 nsistent with this, 14-HDoHE was detected in bronchoalveolar lavage cells of mild to moderate asthmat

84 e marrow progenitors, blood neutrophils, and bronchoalveolar lavage cells to initiate and complete an

85 increased, and protein concentration in the bronchoalveolar lavage diminished, showing the impact of

86 This activation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as we

87 niae infection was diagnosed on the basis of bronchoalveolar lavage eosinophilia and blood findings.

88 b induced a rise in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil pero

89 Before mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3

90 tracellular vesicles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium o

91 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph n

92 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

93 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

94 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

95 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

96 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

97 -type lymphocytes were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric f

98 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell ty

99 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

100 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

101 etabolic profiling of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mo

102 identification of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas ex

103 y flow cytometry on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ve

104 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

105 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

106 ive to air-exposed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of

107 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

108 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

109 d higher levels of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an i

110 4, 17, 21, 25, or 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic anal

111 mpared with WT mice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar lev

112 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

113 utum (n = 128), tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n

114 ns and polymorphonuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

115 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

116 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

117 Higher levels of Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cel

118 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

119 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

120 ical studies and biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

121 nd primary lung monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR i

122 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transp

123 SM reduces inflammatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokin

124 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

125 No changes in bronchoalveolar lavage fluid angiotensin-converting enzy

126 l counts with increased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue,

127 n reduced the immunosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marro

128 s extracted from endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic

129 Analysis of bronchoalveolar lavage fluid collected from human patien

130 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

131 iduals with cystic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest comp

132 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a s

133 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

134 17A, neutrophil counts, and total protein in bronchoalveolar lavage fluid from acute respiratory dist

135 Bronchoalveolar lavage fluid from Ad-MD-2s mice transfer

136 In bronchoalveolar lavage fluid from human lung transplant

137 In bronchoalveolar lavage fluid from humans with ARDS, gut-

138 was consistently inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kina

139 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

140 we found significant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, an

141 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

142 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

143 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

144 F-specific IgG is elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice

145 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3

146 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

147 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

148 es in lung epithelial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte grow

149 Bronchoalveolar lavage fluid LT levels were increased in

150 Similarly, bronchoalveolar lavage fluid obtained from human volunte

151 m asthmatic and control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe an

152 cytokines, chemokines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 p

153 P-ribosyl-HNP-(ornithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathi

154 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

155 1) levels were significantly elevated in the bronchoalveolar lavage fluid of all mice infected with C

156 OSM levels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patie

157 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

158 As we found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma,

159 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

160 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

161 of desialylated MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated

162 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

163 identify host proteins contained within the bronchoalveolar lavage fluid of mice that are cleaved an

164 lenge, and eosinophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

165 HDM, elevated the levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

166 resulted in hypovirulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosi

167 istress syndrome 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48

168 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from

169 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

170 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

171 ith severe asthma had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmon

172 ther report that intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin compl

173 ressive alveolar neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha

174 Bronchoalveolar lavage fluid was analyzed for inflammato

175 gillus species, Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinica

176 Bronchoalveolar lavage fluid was collected from 23 stero

177 Bronchoalveolar lavage fluid was collected from patients

178 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

179 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

180 third MR imaging examination, eosinophils in bronchoalveolar lavage fluid were counted.

181 Bacterial counts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranbe

182 While total and differential cell counts in bronchoalveolar lavage fluid were similar between the Sy

183 urement and polymorphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evalua

184 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

185 The specimens (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 n

186 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

187 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

188 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

189 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

190 and nonlymphoid tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366

191 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

192 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

193 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

194 ase-9 and proinflammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leuko

195 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

196 fected myeloid cells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain,

197 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

198 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

199 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

200 increased the numbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decre

201 llikrein (KLK) 5 and KLK14, were assessed in bronchoalveolar lavage fluid.

202 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

203 sed the production of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

204 of increased macrophages and eosinophils in bronchoalveolar lavage fluid.

205 creased the numbers of inflammatory cells in bronchoalveolar lavage fluid.

206 ed in tissue extracts, nasal secretions, and bronchoalveolar lavage fluid.

207 ung epithelial cells and readily detected in bronchoalveolar lavage fluid.

208 1) with the percentage of eosinophils in the bronchoalveolar lavage fluid.

209 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

210 release from lung epithelium as detected in bronchoalveolar lavage fluid.

211 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

212 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

213 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

214 d with the concentration of total protein in bronchoalveolar lavage fluids (BALF) from patients with

215 Bronchoalveolar lavage fluids from ozone-treated rats re

216 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

217 The concentration of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

218 The assay is validated for testing bronchoalveolar lavage fluids, replacing the requirement

219 0.00003), and similar results were found in bronchoalveolar lavage fluids.

220 uction compared to normal lung, as did human bronchoalveolar lavage following lipopolysaccharide inst

221 d, induced sputum, endobronchial biopsy, and bronchoalveolar lavage for flow cytometry and multiplex-

222 AMs were obtained from bronchoalveolar lavage from healthy donors or patients w

223 AMs were recovered from bronchoalveolar lavage from healthy subjects and patient

224 Bronchoalveolar lavage from mice expressing CAIKKbeta mi

225 leukocytes, macrophages, and neutrophils in bronchoalveolar lavage from O3-exposed mice.

226 Interestingly, bronchoalveolar lavage IL-6, interferon gamma-induced pr

227 L-1alpha positively correlated with elevated bronchoalveolar lavage IL-8 levels (r(2) = 0.6095, p < 0

228 We found that bronchoalveolar lavage interleukin-17A was strongly asso

229 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13,

230 Bronchoalveolar lavage macrophages were stimulated in vi

231 increases than downwind fine/ultrafine PM in bronchoalveolar lavage neutrophils, eosinophils, and lac

232 cell therapy groups, despite a reduction in bronchoalveolar lavage neutrophils.

233 ll as 16S ribosomal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF

234 rleukin-1 alpha (IL-1alpha) was increased in bronchoalveolar lavage of lung transplant recipients gro

235 des of the biculture and was also present in bronchoalveolar lavage of lung transplantation patients.

236 ive HC3-HA structures were also found in the bronchoalveolar lavage of naive mice and were observed o

237 was also demonstrated in vivo by challenging bronchoalveolar lavage of SET-M33-treated mice with LPS,

238 % male) with pulmonary nocardiosis proved by bronchoalveolar lavage or biopsy were reviewed by two ex

239 marker of PARP activation) and IL-6, in the bronchoalveolar lavage or the lung tissue, and histology

240 phenotype and function of T lymphocytes from bronchoalveolar lavage or the peripheral blood.

241 ukin-17A was strongly associated with higher bronchoalveolar lavage percent neutrophils (p < 0.001) a

242 gonist resulted in a 59% and 91% increase in bronchoalveolar lavage protein and LDH, respectively.

243 ression resulted in reduced edema formation (bronchoalveolar lavage protein concentration and lung hi

244 Incubating control cells with disease bronchoalveolar lavage recapitulated the aberrant functi

245 ially in patients with severe asthma in whom bronchoalveolar lavage regulatory T-cell numbers were al

246 Blood, bone marrow, and bronchoalveolar lavage sample analyses from juvenile and

247 l or immunocompromised or fail to improve, a bronchoalveolar lavage sample FARP (BAL FARP) is perform

248 y evaluating both upper airway and acellular bronchoalveolar lavage samples from 49 subjects from thr

249 have previously demonstrated that acellular bronchoalveolar lavage samples from half of the healthy

250 and BAFF expression in B cells in blood and bronchoalveolar lavage samples from the same subject gro

251 Bronchoalveolar lavage samples from Ugandan patients wit

252 pression of FXIII mRNA and protein levels in bronchoalveolar lavage samples obtained before and after

253 tes for the galactomannan assay in serum and bronchoalveolar lavage samples were 61.3% and 57.1%, res

254 terium tuberculosis (MTB) mRNA in sputum and bronchoalveolar lavage samples, in a substantial proport

255 Serial bronchoalveolar lavage specimens were ultracentrifuged t

256 We used bronchoalveolar lavage to identify and characterize huma

257 r lavage percent neutrophils (p < 0.001) and bronchoalveolar lavage total protein (p < 0.01) in acute

258 r (CD95) and programmed death-1, but similar bronchoalveolar lavage viral loads as control subjects.

259 Three days after instillation, bronchoalveolar lavage was performed and plastic-adheren

260 asis of chest CT findings, bronchoscopy with bronchoalveolar lavage was performed.

261 E. coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy

262 Notably, Ag-specific T cells in the bronchoalveolar lavage were sustained at approximately 5

263 the lung, proinflammatory cytokine levels in bronchoalveolar lavage, and alveolar capillary leakage.

264 ovided induced sputum, endobronchial biopsy, bronchoalveolar lavage, and blood samples.

265 Eosinophils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 ho

266 ects underwent phlebotomy, sputum induction, bronchoalveolar lavage, and endobronchial biopsy.

267 h assessment by means of forced oscillation, bronchoalveolar lavage, and histologic analysis.

268 Silica-induced SPP1 in lung tissue, bronchoalveolar lavage, and serum increased more in male

269 bacterial burden in the lungs, blood, liver, bronchoalveolar lavage, and spleens of mice at 24 h post

270 ions to perform bronchoscopic airway survey, bronchoalveolar lavage, esophageal pH monitoring, and a

271 Blood, bronchoalveolar lavage, large proximal and small distal

272 Blood, bronchoalveolar lavage, large proximal, and small distal

273 arance from the lungs, cytokine secretion in bronchoalveolar lavage, lung antimicrobial peptide expre

274 llergic airway inflammation was evaluated by bronchoalveolar lavage, lung histology, serology, gene e

275 , PAR, lactate dehydrogenase and proteins in bronchoalveolar lavage, lung weight gain, perivascular e

276 ximal and distal airways (bronchial wash and bronchoalveolar lavage, respectively), as well as mucosa

277 is factor-alpha concentrations were lower in bronchoalveolar lavage, whereas the concentrations of th

278 was associated with IFN-gamma expression in bronchoalveolar lavage, while inducing its expression in

279 expression of PD-1 and PD-L1 on systemic and bronchoalveolar lavage-derived cells of subjects with sa

280 ed numbers of lymphocytes and neutrophils in bronchoalveolar lavage.

281 All patients underwent bronchoalveolar lavage.

282 Twenty-three ventilated patients underwent bronchoalveolar lavage.

283 derwent spirometry, chest x-ray study, and a bronchoalveolar lavage.

284 g surfactant membranes isolated from porcine bronchoalveolar lavage.

285 olating large numbers of cells by whole-lung bronchoalveolar lavage.

286 ant SP-D and captured native SP-D from human bronchoalveolar lavage.

287 r infiltration, and E. coli colony counts in bronchoalveolar lavage.

288 els of Aspergillus and total fungus in their bronchoalveolar lavage.

289 tinocyte chemoattractant, and neutrophils in bronchoalveolar lavage; and mortality, mucus obstruction

290 Oral washes (OW), induced sputa (IS), and bronchoalveolar lavages (BAL) were collected from 56 par

291 6S rRNA sequencing data from oral washes and bronchoalveolar lavages (BALs) obtained from HIV-uninfec

292 ial alarmins were measured longitudinally in bronchoalveolar lavages from lung transplant recipients

293 sed eosinophil and neutrophil populations in bronchoalveolar lavages from mice with asthma.

294 NA and host total RNA were isolated from 203 bronchoalveolar lavages obtained from 112 patients post-

295 NA and host total RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post

296 A similar pattern was seen for bronchoalveolar lymphocytes, but with quadruple the magn

297 Furthermore, obese mice had fewer bronchoalveolar macrophages and regulatory T cells durin

298 Only fluticasone/salmeterol decreased bronchoalveolar neutrophilia (p = 0.03) to the same exte

299 Mif(-/-) mice demonstrated an increase in bronchoalveolar protein (48%) and lactate dehydrogenase

300 platelet-leukocyte complex formation in the bronchoalveolar space.