ライフサイエンスコーパス: bronchoalveolar lavage

1 ed numbers of lymphocytes and neutrophils in bronchoalveolar lavage.

2 All patients underwent bronchoalveolar lavage.

3 Twenty-three ventilated patients underwent bronchoalveolar lavage.

4 derwent spirometry, chest x-ray study, and a bronchoalveolar lavage.

5 g surfactant membranes isolated from porcine bronchoalveolar lavage.

6 olating large numbers of cells by whole-lung bronchoalveolar lavage.

7 ant SP-D and captured native SP-D from human bronchoalveolar lavage.

8 r infiltration, and E. coli colony counts in bronchoalveolar lavage.

9 els of Aspergillus and total fungus in their bronchoalveolar lavage.

10 from the baseline value until 2 hours after bronchoalveolar lavage and became similar to the baselin

11 in several proinflammatory cytokines in the bronchoalveolar lavage and in serum.

12 or cigarette smoke for 22 weeks followed by bronchoalveolar lavage and lung and cardiac tissue analy

13 ortant cellular sources of IL-5 and IL-13 in bronchoalveolar lavage and lung tissue.

14 ion model, IL-10-producing CD4(+) T cells in bronchoalveolar lavage and lung were significantly decre

15 ition of ADAM10 reduces sEphrin-B2 levels in bronchoalveolar lavage and prevents lung fibrosis in mic

16 Bronchoalveolar lavage and pulmonary function tests were

17 Double-stranded RNA in bronchoalveolar lavage and serum samples following lung

18 We found that levels of inflammation in the bronchoalveolar lavage and the lung, as well as levels o

19 Bronchoalveolar lavage and tissues were sampled for myco

20 ermate control animals through evaluation of bronchoalveolar lavage and tissues.

21 the lung, proinflammatory cytokine levels in bronchoalveolar lavage, and alveolar capillary leakage.

22 ovided induced sputum, endobronchial biopsy, bronchoalveolar lavage, and blood samples.

23 Eosinophils were examined in blood, bronchoalveolar lavage, and endobronchial biopsies 48 ho

24 ects underwent phlebotomy, sputum induction, bronchoalveolar lavage, and endobronchial biopsy.

25 h assessment by means of forced oscillation, bronchoalveolar lavage, and histologic analysis.

26 Silica-induced SPP1 in lung tissue, bronchoalveolar lavage, and serum increased more in male

27 bacterial burden in the lungs, blood, liver, bronchoalveolar lavage, and spleens of mice at 24 h post

28 tinocyte chemoattractant, and neutrophils in bronchoalveolar lavage; and mortality, mucus obstruction

29 BAFF levels were also higher in blood and bronchoalveolar lavage B cells in patients with COPD ver

30 Cells recovered by bronchoalveolar lavage (BAL) after imaging were stained

31 tegrated study of human small airway tissue, bronchoalveolar lavage (BAL) and an experimental murine

32 bit AHR, increased numbers of eosinophils in bronchoalveolar lavage (BAL) and increased collagen cont

33 he diagnostic yield and complication rate of bronchoalveolar lavage (BAL) and lung biopsy in the eval

34 Bronchoalveolar lavage (BAL) and lung tissue were examin

35 a methacholine test, airway inflammation in bronchoalveolar lavage (BAL) and lung tissue, and total

36 e expression and activity were determined in bronchoalveolar lavage (BAL) and lungs of human donors a

37 We investigated bronchoalveolar lavage (BAL) and serum samples from pati

38 ort study; 72 consented to bronchoscopy with bronchoalveolar lavage (BAL) and transbronchial biopsies

39 Conventional methods to identify HBEC in bronchoalveolar lavage (BAL) and wash (BW) have throughp

40 Alveolar macrophages (AMs) obtained by bronchoalveolar lavage (BAL) are commonly used to study

41 cDNA library derived from mRNA isolated from bronchoalveolar lavage (BAL) cells and leukocytes of sar

42 was to investigate the expression of iNOS in bronchoalveolar lavage (BAL) cells and tissue from centr

43 Using blood and bronchoalveolar lavage (BAL) cells from normal control s

44 esponsiveness (AHR) to inhaled methacholine, bronchoalveolar lavage (BAL) cytokine levels, and lung h

45 ) and nearly 7-fold more active TGF-beta1 in bronchoalveolar lavage (BAL) fluid (BALF).

46 phil and bacterial counts were determined in bronchoalveolar lavage (BAL) fluid and blood.

47 perimental model of allergic asthma, matched bronchoalveolar lavage (BAL) fluid and plasma were colle

48 igatus Its performance has been validated on bronchoalveolar lavage (BAL) fluid and serum specimens,

49 and production of inflammatory cytokines in bronchoalveolar lavage (BAL) fluid and the lungs.

50 ormed in nasopharyngeal aspirates (NPAs) and bronchoalveolar lavage (BAL) fluid before HCT.

51 (1,3)beta-D-glucan detection in bronchoalveolar lavage (BAL) fluid by Fungitell assay ai

52 elial growth factor (VEGF) levels in hamster bronchoalveolar lavage (BAL) fluid early after intranasa

53 A gene sequencing was performed on acellular bronchoalveolar lavage (BAL) fluid from 30 subjects infe

54 ic bead array, and L-ficolin was measured in bronchoalveolar lavage (BAL) fluid from lung transplant

55 Unlike blood ILC2s, bronchoalveolar lavage (BAL) fluid ILC2s from asthmatic

56 We examined bronchoalveolar lavage (BAL) fluid leukocytes, cytokines

57 We found that bleomycin injury increases the bronchoalveolar lavage (BAL) fluid levels of ATX protein

58 mesenteric lymph nodes (MLNs), jejunum, and bronchoalveolar lavage (BAL) fluid of healthy and SIV-in

59 nsus sequences of viruses collected from the bronchoalveolar lavage (BAL) fluid of the animals.

60 Prospective real-time testing of serum and bronchoalveolar lavage (BAL) fluid pools from positive a

61 rometry based proteome analysis of acellular bronchoalveolar lavage (BAL) fluid samples on an observa

62 respiratory tract disease, 72 paired NP and bronchoalveolar lavage (BAL) fluid specimen sets, mostly

63 We analyzed 46 clinically obtained bronchoalveolar lavage (BAL) fluid specimens from sympto

64 Bronchoalveolar lavage (BAL) fluid was assessed for tota

65 Using a cutoff of >/=2% eosinophilia in bronchoalveolar lavage (BAL) fluid, chronic lung allogra

66 manifestation, so we evaluated biomarkers in bronchoalveolar lavage (BAL) fluid.

67 responses, we hypothesized that MV, found in bronchoalveolar lavage (BAL) fluids (BALF) of LTR at CLA

68 igated M. tuberculosis-specific responses in bronchoalveolar lavage (BAL) from persons with latent M.

69 We measured mtDNA concentrations in bronchoalveolar lavage (BAL) from subjects with and with

70 ALI was quantified by weight loss, bronchoalveolar lavage (BAL) inflammatory cell number, c

71 We assessed bronchoalveolar lavage (BAL) inflammatory cell numbers,

72 ls (MSCs) in the terminal airways-alveoli by bronchoalveolar lavage (BAL) of human adult lungs.

73 d their binding to TIMP-1, -2, -3, and -4 in bronchoalveolar lavage (BAL) of lung transplant recipien

74 mplicated in the pathogenesis of ACR) in the bronchoalveolar lavage (BAL) of lung transplant recipien

75 r flow cytometry in digested lung tissue and bronchoalveolar lavage (BAL) simultaneously, 6 h after e

76 ng flow cytometry and a multiplex assay with bronchoalveolar lavage (BAL) specimens (n = 68) from 52

77 However, no CMV DNA threshold exists in bronchoalveolar lavage (BAL) to differentiate pneumonia

78 Six hours later, bronchoalveolar lavage (BAL) was collected for leukocyte

79 Airway responsiveness was assessed, bronchoalveolar lavage (BAL) was performed, and lung cel

80 s of CD4(+) T and B cells in the spleens and bronchoalveolar lavage (BAL) were also observed.

81 ergic patients underwent SAC, and cells from bronchoalveolar lavage (BAL) were collected after 24 hou

82 SCFA levels in anaerobic supernatants and bronchoalveolar lavage (BAL) were determined by gas chro

83 The predominant macrophages harvested by bronchoalveolar lavage (BAL), alveolar macrophages (AMs)

84 flux of neutrophils and macrophages into the bronchoalveolar lavage (BAL), and human CD45(+) cells in

85 Subjects underwent bronchoalveolar lavage (BAL), and peripheral whole blood

86 D4 and CD8 T cell immune responses in blood, bronchoalveolar lavage (BAL), and tracheobronchial lymph

87 linically indicated fiberoptic bronchoscopy, bronchoalveolar lavage (BAL), endobronchial brushings, a

88 t computed tomography, and bronchoscopy with bronchoalveolar lavage (BAL).

89 ated with proinflammatory cytokines in human bronchoalveolar lavage (BAL).

90 by an accumulation of CD4(+) T cells in the bronchoalveolar lavage (BAL).

91 aeruginosa, P. aeruginosa) were analyzed in bronchoalveolar lavage (BAL); and alveolar SGLT1 was ana

92 Oral washes (OW), induced sputa (IS), and bronchoalveolar lavages (BAL) were collected from 56 par

93 6S rRNA sequencing data from oral washes and bronchoalveolar lavages (BALs) obtained from HIV-uninfec

94 subjects had symptom scores, spirometry, and bronchoalveolar lavage before and after rhinovirus-induc

95 Following silica treatment, bronchoalveolar lavage cell infiltrates decreased in fem

96 Bronchoalveolar lavage cell mRNA levels of iNOS or iNOS

97 Ag85A-specific CD4 T cells were detected in bronchoalveolar lavage cells from both groups and respon

98 At two independent study sites, bronchoalveolar lavage cells from donors with latent tub

99 nsistent with this, 14-HDoHE was detected in bronchoalveolar lavage cells of mild to moderate asthmat

100 e marrow progenitors, blood neutrophils, and bronchoalveolar lavage cells to initiate and complete an

101 At baseline and after hyperoxia exposure, bronchoalveolar lavage cytokine levels were unchanged in

102 expression of PD-1 and PD-L1 on systemic and bronchoalveolar lavage-derived cells of subjects with sa

103 increased, and protein concentration in the bronchoalveolar lavage diminished, showing the impact of

104 This activation phenotype indicated by bronchoalveolar lavage eosinophil surface markers, as we

105 niae infection was diagnosed on the basis of bronchoalveolar lavage eosinophilia and blood findings.

106 b induced a rise in circulating eosinophils, bronchoalveolar lavage eosinophilia, and eosinophil pero

107 Before mepolizumab treatment, bronchoalveolar lavage eosinophils had more surface IL-3

108 ions to perform bronchoscopic airway survey, bronchoalveolar lavage, esophageal pH monitoring, and a

109 tracellular vesicles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium o

110 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph n

111 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

112 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

113 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

114 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

115 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

116 -type lymphocytes were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric f

117 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell ty

118 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

119 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

120 etabolic profiling of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mo

121 identification of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas ex

122 y flow cytometry on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ve

123 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

124 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

125 ive to air-exposed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of

126 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

127 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

128 d higher levels of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an i

129 4, 17, 21, 25, or 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic anal

130 mpared with WT mice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar lev

131 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

132 utum (n = 128), tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n

133 ns and polymorphonuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

134 lmost 10-fold-higher bacterial burden in the bronchoalveolar lavage fluid 3 h following infection.

135 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

136 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

137 Higher levels of Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cel

138 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

139 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

140 ical studies and biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

141 nd primary lung monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR i

142 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transp

143 SM reduces inflammatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokin

144 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

145 No changes in bronchoalveolar lavage fluid angiotensin-converting enzy

146 l counts with increased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue,

147 n reduced the immunosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marro

148 s extracted from endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic

149 Analysis of bronchoalveolar lavage fluid collected from human patien

150 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

151 tion and IL-17A/F production in the lung and bronchoalveolar lavage fluid compared with wild-type mic

152 iduals with cystic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest comp

153 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a s

154 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

155 17A, neutrophil counts, and total protein in bronchoalveolar lavage fluid from acute respiratory dist

156 Bronchoalveolar lavage fluid from Ad-MD-2s mice transfer

157 In bronchoalveolar lavage fluid from human lung transplant

158 In bronchoalveolar lavage fluid from humans with ARDS, gut-

159 was consistently inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kina

160 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

161 we found significant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, an

162 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

163 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

164 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

165 F-specific IgG is elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice

166 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3

167 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

168 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

169 es in lung epithelial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte grow

170 Bronchoalveolar lavage fluid LT levels were increased in

171 ded throughout the study day, and plasma and bronchoalveolar lavage fluid markers of inflammation wer

172 Similarly, bronchoalveolar lavage fluid obtained from human volunte

173 m asthmatic and control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe an

174 cytokines, chemokines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 p

175 P-ribosyl-HNP-(ornithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathi

176 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

177 1) levels were significantly elevated in the bronchoalveolar lavage fluid of all mice infected with C

178 OSM levels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patie

179 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

180 As we found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma,

181 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

182 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

183 of desialylated MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated

184 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

185 identify host proteins contained within the bronchoalveolar lavage fluid of mice that are cleaved an

186 lenge, and eosinophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

187 HDM, elevated the levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

188 resulted in hypovirulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosi

189 istress syndrome 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48

190 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from

191 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

192 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

193 ith severe asthma had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmon

194 ther report that intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin compl

195 ressive alveolar neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha

196 Bronchoalveolar lavage fluid was analyzed for inflammato

197 gillus species, Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinica

198 Bronchoalveolar lavage fluid was collected from 23 stero

199 Bronchoalveolar lavage fluid was collected from patients

200 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

201 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

202 third MR imaging examination, eosinophils in bronchoalveolar lavage fluid were counted.

203 Bacterial counts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranbe

204 While total and differential cell counts in bronchoalveolar lavage fluid were similar between the Sy

205 urement and polymorphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evalua

206 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

207 The specimens (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 n

208 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

209 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

210 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

211 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

212 and nonlymphoid tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366

213 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

214 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

215 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

216 ase-9 and proinflammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leuko

217 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

218 fected myeloid cells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain,

219 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

220 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

221 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

222 increased the numbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decre

223 llikrein (KLK) 5 and KLK14, were assessed in bronchoalveolar lavage fluid.

224 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

225 sed the production of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

226 of increased macrophages and eosinophils in bronchoalveolar lavage fluid.

227 creased the numbers of inflammatory cells in bronchoalveolar lavage fluid.

228 ed in tissue extracts, nasal secretions, and bronchoalveolar lavage fluid.

229 ung epithelial cells and readily detected in bronchoalveolar lavage fluid.

230 1) with the percentage of eosinophils in the bronchoalveolar lavage fluid.

231 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

232 release from lung epithelium as detected in bronchoalveolar lavage fluid.

233 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

234 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

235 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

236 d with the concentration of total protein in bronchoalveolar lavage fluids (BALF) from patients with

237 Bronchoalveolar lavage fluids from ozone-treated rats re

238 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

239 The concentration of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

240 The assay is validated for testing bronchoalveolar lavage fluids, replacing the requirement

241 0.00003), and similar results were found in bronchoalveolar lavage fluids.

242 uction compared to normal lung, as did human bronchoalveolar lavage following lipopolysaccharide inst

243 d, induced sputum, endobronchial biopsy, and bronchoalveolar lavage for flow cytometry and multiplex-

244 AMs were obtained from bronchoalveolar lavage from healthy donors or patients w

245 AMs were recovered from bronchoalveolar lavage from healthy subjects and patient

246 Bronchoalveolar lavage from mice expressing CAIKKbeta mi

247 leukocytes, macrophages, and neutrophils in bronchoalveolar lavage from O3-exposed mice.

248 ial alarmins were measured longitudinally in bronchoalveolar lavages from lung transplant recipients

249 sed eosinophil and neutrophil populations in bronchoalveolar lavages from mice with asthma.

250 Interestingly, bronchoalveolar lavage IL-6, interferon gamma-induced pr

251 L-1alpha positively correlated with elevated bronchoalveolar lavage IL-8 levels (r(2) = 0.6095, p < 0

252 We found that bronchoalveolar lavage interleukin-17A was strongly asso

253 Blood, bronchoalveolar lavage, large proximal and small distal

254 Blood, bronchoalveolar lavage, large proximal, and small distal

255 arance from the lungs, cytokine secretion in bronchoalveolar lavage, lung antimicrobial peptide expre

256 llergic airway inflammation was evaluated by bronchoalveolar lavage, lung histology, serology, gene e

257 , PAR, lactate dehydrogenase and proteins in bronchoalveolar lavage, lung weight gain, perivascular e

258 OVA-induced increases in bronchoalveolar lavage lymphocytes, eosinophils, IL-13,

259 Bronchoalveolar lavage macrophages were stimulated in vi

260 increases than downwind fine/ultrafine PM in bronchoalveolar lavage neutrophils, eosinophils, and lac

261 cell therapy groups, despite a reduction in bronchoalveolar lavage neutrophils.

262 ll as 16S ribosomal RNA sequencing data from bronchoalveolar lavage obtained as part of the COMET-IPF

263 NA and host total RNA were isolated from 203 bronchoalveolar lavages obtained from 112 patients post-

264 NA and host total RNA were isolated from 189 bronchoalveolar lavages obtained from 116 patients post

265 rleukin-1 alpha (IL-1alpha) was increased in bronchoalveolar lavage of lung transplant recipients gro

266 des of the biculture and was also present in bronchoalveolar lavage of lung transplantation patients.

267 ive HC3-HA structures were also found in the bronchoalveolar lavage of naive mice and were observed o

268 was also demonstrated in vivo by challenging bronchoalveolar lavage of SET-M33-treated mice with LPS,

269 % male) with pulmonary nocardiosis proved by bronchoalveolar lavage or biopsy were reviewed by two ex

270 ; 54.3% male) with idiopathic PAP (proved by bronchoalveolar lavage or biopsy) were reviewed by two e

271 marker of PARP activation) and IL-6, in the bronchoalveolar lavage or the lung tissue, and histology

272 phenotype and function of T lymphocytes from bronchoalveolar lavage or the peripheral blood.

273 oneal (p = 0.037), systemic (p = 0.019), and bronchoalveolar lavage (p = 0.011) quantitative bacteria

274 ukin-17A was strongly associated with higher bronchoalveolar lavage percent neutrophils (p < 0.001) a

275 gonist resulted in a 59% and 91% increase in bronchoalveolar lavage protein and LDH, respectively.

276 ression resulted in reduced edema formation (bronchoalveolar lavage protein concentration and lung hi

277 Incubating control cells with disease bronchoalveolar lavage recapitulated the aberrant functi

278 ially in patients with severe asthma in whom bronchoalveolar lavage regulatory T-cell numbers were al

279 ximal and distal airways (bronchial wash and bronchoalveolar lavage, respectively), as well as mucosa

280 Blood, bone marrow, and bronchoalveolar lavage sample analyses from juvenile and

281 l or immunocompromised or fail to improve, a bronchoalveolar lavage sample FARP (BAL FARP) is perform

282 y evaluating both upper airway and acellular bronchoalveolar lavage samples from 49 subjects from thr

283 have previously demonstrated that acellular bronchoalveolar lavage samples from half of the healthy

284 and BAFF expression in B cells in blood and bronchoalveolar lavage samples from the same subject gro

285 Bronchoalveolar lavage samples from Ugandan patients wit

286 pression of FXIII mRNA and protein levels in bronchoalveolar lavage samples obtained before and after

287 tes for the galactomannan assay in serum and bronchoalveolar lavage samples were 61.3% and 57.1%, res

288 The genome yield from tracheal aspirates and bronchoalveolar lavage samples were similar (P = .1174).

289 terium tuberculosis (MTB) mRNA in sputum and bronchoalveolar lavage samples, in a substantial proport

290 Serial bronchoalveolar lavage specimens were ultracentrifuged t

291 After bronchoalveolar lavage, the value of indexed extravascul

292 We used bronchoalveolar lavage to identify and characterize huma

293 r lavage percent neutrophils (p < 0.001) and bronchoalveolar lavage total protein (p < 0.01) in acute

294 r (CD95) and programmed death-1, but similar bronchoalveolar lavage viral loads as control subjects.

295 Three days after instillation, bronchoalveolar lavage was performed and plastic-adheren

296 asis of chest CT findings, bronchoscopy with bronchoalveolar lavage was performed.

297 E. coli colony-forming units in bronchoalveolar lavage were reduced in both cell therapy

298 Notably, Ag-specific T cells in the bronchoalveolar lavage were sustained at approximately 5

299 is factor-alpha concentrations were lower in bronchoalveolar lavage, whereas the concentrations of th

300 was associated with IFN-gamma expression in bronchoalveolar lavage, while inducing its expression in