ライフサイエンスコーパス: bronchoalveolar lavage fluid

1 creased the numbers of inflammatory cells in bronchoalveolar lavage fluid.

2 ed in tissue extracts, nasal secretions, and bronchoalveolar lavage fluid.

3 ung epithelial cells and readily detected in bronchoalveolar lavage fluid.

4 1) with the percentage of eosinophils in the bronchoalveolar lavage fluid.

5 rase chain reaction using RNA extracted from bronchoalveolar lavage fluid.

6 reased protein and cytokine concentration in bronchoalveolar lavage fluid.

7 release from lung epithelium as detected in bronchoalveolar lavage fluid.

8 HR or total inflammatory cell numbers in the bronchoalveolar lavage fluid.

9 Cells were counted in bronchoalveolar lavage fluid.

10 Recruited eosinophils were enumerated in bronchoalveolar lavage fluid.

11 in (TSLP), IL-9, and IL-13, but not IL-5, in bronchoalveolar lavage fluid.

12 L-13, IL-1beta, IL-33, and eotaxin levels in bronchoalveolar lavage fluid.

13 lls and 20-35% reduction in Th2 cytokines in bronchoalveolar lavage fluid.

14 s product of cellular necrosis, in cell-free bronchoalveolar lavage fluid.

15 ers were assessed in lung tissue, serum, and bronchoalveolar lavage fluid.

16 sing 454 pyrosequencing of 16S rDNA found in bronchoalveolar lavage fluid.

17 ecline in memory CD4(+) T cells in blood and bronchoalveolar lavage fluid.

18 solated from alcoholic and control patients' bronchoalveolar lavage fluid.

19 IgA, IgG2c, and IgG1 antibodies in serum and bronchoalveolar lavage fluid.

20 n and presence of lipid-laden macrophages in bronchoalveolar lavage fluid.

21 production, and cytokine accumulation in the bronchoalveolar lavage fluid.

22 the macrophage chemoattractant MCP-1 in lung bronchoalveolar lavage fluid.

23 llikrein (KLK) 5 and KLK14, were assessed in bronchoalveolar lavage fluid.

24 lung while also reducing LPA 18:2 content in bronchoalveolar lavage fluid.

25 sed the production of TNF-alpha and MIP-2 in bronchoalveolar lavage fluid.

26 of increased macrophages and eosinophils in bronchoalveolar lavage fluid.

27 , IL-5, and IL-13 measured by ELISA in their bronchoalveolar lavage fluids.

28 0.00003), and similar results were found in bronchoalveolar lavage fluids.

29 levels of sTNFR production were elevated in bronchoalveolar lavage fluid 1 month following infection

30 ix -metalloproteinase-13 accumulation in the bronchoalveolar lavage fluid (1513 +/- 337 pg/mL broncho

31 lmost 10-fold-higher bacterial burden in the bronchoalveolar lavage fluid 3 h following infection.

32 2 ligands was significantly increased in the bronchoalveolar lavage fluid 48 hours after segmental al

33 The specimens (5 cerebrospinal fluid, 7 bronchoalveolar lavage fluid, 5 plasma, 2 serum, and 1 n

34 neutrophil chemoattractant-1 accumulation in bronchoalveolar lavage fluid 5999 +/- 1267 pg/mL vs. 334

35 oss, mortality, and virus titers in lung and bronchoalveolar lavage fluid after infection with the mu

36 okines such as IL-5, IL-13, and eotaxin-1 in bronchoalveolar lavage fluid after OVA challenge.

37 chronic rhinosinusitis (CRS), as well as in bronchoalveolar lavage fluid, after segmental allergen c

38 iration caused increased ALI (as assessed by bronchoalveolar lavage fluid albumin concentration) in b

39 secretion of Th2 cytokines IL-4 and IL-5 in bronchoalveolar lavage fluid, allergen-induced inflammat

40 ere associated with sHLA-G concentrations in bronchoalveolar lavage fluid among asthmatic subjects wi

41 were associated with G-CSF concentrations in bronchoalveolar lavage fluid and blood.

42 PM2.5 increased neutrophil numbers and KC in bronchoalveolar lavage fluid and caused slight peribronc

43 measured by changes in inflammatory cells in bronchoalveolar lavage fluid and cytokine expression in

44 rs of T cells and expression of TNF-alpha in bronchoalveolar lavage fluid and deteriorates lung funct

45 Higher levels of Th2 cytokines were found in bronchoalveolar lavage fluid and draining lymph node cel

46 ifested persistent inflammation in blood and bronchoalveolar lavage fluid and exacerbations despite h

47 pothermia did not affect bacterial growth in bronchoalveolar lavage fluid and in homogenized lungs co

48 exacerbated eosinophilic inflammation in the bronchoalveolar lavage fluid and in lung tissue, along w

49 ivity was associated with increased Gt in CF bronchoalveolar lavage fluid and increased bronchoalveol

50 augmented by 4MDM, the levels of PGP in the bronchoalveolar lavage fluid and infiltration of neutrop

51 elicited by the four immunization routes in bronchoalveolar lavage fluid and jejunal, rectal, and va

52 ikingly reduced numbers of leukocytes in the bronchoalveolar lavage fluid and lower expression of inf

53 y had lower levels of the chemokine CXCL1 in bronchoalveolar lavage fluid and lung parenchyma.

54 Bacterial cultures of bronchoalveolar lavage fluid and lung tissue samples wer

55 The percentage of bronchoalveolar lavage fluid and lung tissue samples wit

56 ine expression in ILC2s and TH2 cells in the bronchoalveolar lavage fluid and lung tissue were assess

57 uce eosinophil extracellular traps (EETs) in bronchoalveolar lavage fluid and lung tissue.

58 phil infiltration and levels of cytokines in bronchoalveolar lavage fluid and lung tissue.

59 ical studies and biochemical measurements in bronchoalveolar lavage fluid and lung tissue.

60 nd primary lung monocytes/macrophages, mouse bronchoalveolar lavage fluid and lung tissues, and AHR i

61 Bronchoalveolar lavage fluid and lungs were harvested fo

62 neutrophils, lymphocytes, and macrophages in bronchoalveolar lavage fluid and lungs; inflammatory cel

63 NETs were also measured in bronchoalveolar lavage fluid and plasma from lung transp

64 vascular tissue to extracellular fluid (eg, bronchoalveolar lavage fluid and plasma).

65 SM reduces inflammatory cell accumulation in bronchoalveolar lavage fluid and proinflammatory cytokin

66 as increased expression of galectin-3 in the bronchoalveolar lavage fluid and serum from patients wit

67 We detected the presence of PRELP in human bronchoalveolar lavage fluid and showed that PRELP can b

68 ophilic and neutrophilic inflammation in the bronchoalveolar lavage fluid and Th1, Th2, and Th17 infl

69 d polymorphonuclear leukocytes (PMNs) in the bronchoalveolar lavage fluid and the wet-to-dry lung wei

70 MMP-9 is the major MMP found in the bronchoalveolar lavage fluids and bronchial biopsies fro

71 c antibodies were detected in nasal wash and bronchoalveolar lavage fluids and in sera from mice intr

72 ed hyperoxia-induced accumulation of IL-6 in bronchoalveolar lavage fluids and NADPH oxidase (NOX) 2

73 ngly expressed in bronchi, we measured it in bronchoalveolar lavage fluids and observed that it is st

74 urement and polymorphonuclear recruitment in bronchoalveolar lavage fluid), and lethality were evalua

75 e lungs, (ii) higher amounts of IL-17 in the bronchoalveolar lavage fluid, and (iii) more neutrophils

76 d lower total cell counts and neutrophils in bronchoalveolar lavage fluid, and had earlier influx of

77 nd disaturated PC in lung tissue homogenate, bronchoalveolar lavage fluid, and lung LB was increased

78 d culture of tissue, respiratory secretions, bronchoalveolar lavage fluid, and other fluids.

79 were sensitized and challenged with OVA and bronchoalveolar lavage fluid, and the lungs were collect

80 alpha and the chemokines eotaxin and CCL2 in bronchoalveolar lavage fluid; and decreased pulmonary in

81 ive plus had greater than 20% lymphocytes in bronchoalveolar lavage fluid; and three had findings on

82 No changes in bronchoalveolar lavage fluid angiotensin-converting enzy

83 l counts with increased extracellular DNA in bronchoalveolar lavage fluid as well as in lung tissue,

84 IgG1, IgG2a, and IgE; and cytokine levels in bronchoalveolar lavage fluids as well as in supernatants

85 and nonlymphoid tissues, including lung and bronchoalveolar lavage fluid, as measured by H2-Db NP366

86 sed patients were compared to fungal load in bronchoalveolar lavage fluids assessed using quantitativ

87 ane B2 and immunoglobulin E were measured in bronchoalveolar lavage fluid at baseline and 24 h after

88 oxidative stress), and IgE were measured in bronchoalveolar lavage fluid at baseline and 24 hours af

89 The clinical utility of bronchoalveolar lavage fluid (BAL) cell analysis for the

90 -, and 2.1-fold greater drug levels in lung, bronchoalveolar lavage fluid (BAL), and plasma compared

91 tion of virus-specific memory CD8 T cells in bronchoalveolar lavage fluid (BAL), lungs, and spleen.

92 a-induced lung injury was evaluated by using bronchoalveolar lavage fluid (BALF) analysis and patholo

93 Bronchoalveolar lavage fluid (BALF) analysis demonstrate

94 tracellular vesicles (EVs) are detectable in bronchoalveolar lavage fluid (BALF) and culture medium o

95 Lung tissue, bronchoalveolar lavage fluid (BALF) and draining lymph n

96 im of this study was to analyze cytokines in bronchoalveolar lavage fluid (BALF) and explore predicti

97 We examined bronchoalveolar lavage fluid (BALF) and histological lun

98 infected with Streptococcus pneumoniae, and bronchoalveolar lavage fluid (BALF) and lung CFU values

99 d temporal kinetics of GT and bmGT in serum, bronchoalveolar lavage fluid (BALF) and lungs of A. fumi

100 s the overlap in metabolites between matched bronchoalveolar lavage fluid (BALF) and plasma, identifi

101 tized hosts to assess airway inflammation in bronchoalveolar lavage fluid (BALF) and Th2 responses in

102 BORT reduced bronchoalveolar lavage fluid (BALF) and tissue eosinophi

103 ransplant recipients with CARV infection had bronchoalveolar lavage fluid (BALF) available for analys

104 -type lymphocytes were assessed in lungs and bronchoalveolar lavage fluid (BALF) by multiparametric f

105 Bacterial burdens, bronchoalveolar lavage fluid (BALF) cell counts, cell ty

106 We compared lung bacterial clearance, bronchoalveolar lavage fluid (BALF) characterization, lu

107 sion, PLTP activity decreased by 80% in COPD bronchoalveolar lavage fluid (BALF) due to serine protea

108 understanding of the proinflammatory role of bronchoalveolar lavage fluid (BALF) exosomes in patients

109 We studied bronchoalveolar lavage fluid (BALF) from 36 patients wit

110 etabolic profiling of serum, lung tissue and bronchoalveolar lavage fluid (BALF) from a non-lethal mo

111 cytokine/chemokine profiles were measured in bronchoalveolar lavage fluid (BALF) from children with C

112 identification of two biomarkers present in bronchoalveolar lavage fluid (BALF) from chlorine gas ex

113 y flow cytometry on neutrophils in blood and bronchoalveolar lavage fluid (BALF) from mechanically ve

114 with human MSCs when stimulated with LPS or bronchoalveolar lavage fluid (BALF) from patients with A

115 HR, airway inflammation and cytokines in the bronchoalveolar lavage fluid (BALF) in a murine model of

116 cytomegalovirus (HCMV) DNA detection in the bronchoalveolar lavage fluid (BALF) indicates HCMV repli

117 ken together with the observation that local bronchoalveolar lavage fluid (BALF) levels of vitamin D

118 CD4(+) T cells in the bronchoalveolar lavage fluid (BALF) of Af5517-aspirated

119 recently shown an altered exosome profile in bronchoalveolar lavage fluid (BALF) of patients with sar

120 ive to air-exposed controls, ozone increased bronchoalveolar lavage fluid (BALF) protein, a marker of

121 Neutrophils in bronchoalveolar lavage fluid (BALF) served as markers of

122 Bronchoalveolar lavage fluid (BALF) was analysed for tot

123 Bronchoalveolar lavage fluid (BALF) was collected from c

124 levels and in CA04-neutralizing activity of bronchoalveolar lavage fluid (BALF) was observed followi

125 Lung tissue and bronchoalveolar lavage fluid (BALF) were analyzed for in

126 In parallel, adenosine levels in bronchoalveolar lavage fluid (BALF) were increased by ap

127 d higher levels of Th2 and Th17 cytokines in bronchoalveolar lavage fluid (BALF), accompanied by an i

128 Bronchoalveolar lavage fluid (BALF), airway inflammation

129 iveness, cytokine levels in spleen cells and bronchoalveolar lavage fluid (BALF), and cellular distri

130 4, 17, 21, 25, or 33 d after exposure, SpO2, bronchoalveolar lavage fluid (BALF), and histologic anal

131 mpared with WT mice, with fewer cells in Wsh bronchoalveolar lavage fluid (BALF), despite similar lev

132 Lung function measurements, and bronchoalveolar lavage fluid (BALF), serum, and lungs we

133 ted biomarkers of aspiration (bile salts) in bronchoalveolar lavage fluid (BALF).

134 ammation, and total protein and cytokines in bronchoalveolar lavage fluid (BALF).

135 rbent assay (ELISA) to measure biomarkers in bronchoalveolar lavage fluid (BALF).

136 hocyte and elevation of the CD4/CD8 ratio in bronchoalveolar lavage fluid (BALF).

137 examined the content in MMP active forms of bronchoalveolar lavage fluids (BALf) from male C57BL/6 m

138 d with the concentration of total protein in bronchoalveolar lavage fluids (BALF) from patients with

139 RP3 inflammasome activation, was detected in bronchoalveolar lavage fluids (BALF) in a macrophage- an

140 s) of the allergic response were assessed in bronchoalveolar lavage fluids (BALFs) after allergen cha

141 n H-ficolin and H-ficolin in human serum and bronchoalveolar lavage fluid bind to IAV and inhibit vir

142 damage and increased lung injury scores and bronchoalveolar lavage fluid cell numbers in control but

143 Total bronchoalveolar lavage fluid cell numbers increased from

144 resulting in increase in lung injury scores, bronchoalveolar lavage fluid cell numbers, and cellular

145 n reduced the immunosuppressive functions of bronchoalveolar lavage fluid cells, inhibited bone marro

146 generation by severe asthma whole blood and bronchoalveolar lavage fluid cells.

147 evels of interleukin-1beta, tended to reduce bronchoalveolar lavage fluid CINC-3 levels, but no effec

148 ated by quantifying cellular accumulation in bronchoalveolar lavage fluid, collagen levels, and histo

149 s extracted from endobronchial brushings and bronchoalveolar lavage fluid collected from 39 asthmatic

150 Analysis of bronchoalveolar lavage fluid collected from human patien

151 se in the injury markers was observed in the bronchoalveolar lavage fluids collected from rats treate

152 After bronchoalveolar lavage fluids collection during standard

153 eduction of eosinophil and T cell numbers in bronchoalveolar lavage fluid compared with those in dilu

154 tion and IL-17A/F production in the lung and bronchoalveolar lavage fluid compared with wild-type mic

155 y), total number of WBCs, and neutrophils in bronchoalveolar lavage fluids compared with wild-type mi

156 , bronchoconstriction, serum IgE levels, and bronchoalveolar lavage fluid cytokine and eicosanoid lev

157 Airway inflammation and function, bronchoalveolar lavage fluid cytokine levels, and flow c

158 Outcome measures included: bronchoalveolar lavage fluid cytology to assess airway e

159 d tumor-promoting cyto-/chemokine profile in bronchoalveolar lavage fluid, decreased TLR2/4 expressio

160 Quantitative microbiology applied to bronchoalveolar lavage fluid derived from infected segme

161 dies hint toward the proinflammatory role of bronchoalveolar lavage fluid-derived exosomes in asthmat

162 ed levels of cytokines and chemokines in the bronchoalveolar lavage fluid, enhanced bronchoalveolar c

163 ILC2 numbers, proliferation (Ki-67(+)), and bronchoalveolar lavage fluid eosinophil numbers were mea

164 iduals with cystic fibrosis underwent annual bronchoalveolar lavage fluid examination, and chest comp

165 Our findings validate bronchoalveolar lavage fluid exosomal shuttle RNA as a s

166 Bronchoalveolar lavage fluid exosomes were collected fro

167 fferential cell counts were performed on the bronchoalveolar lavage fluid, followed by histological a

168 We also detect Siglec-F ligand activity in bronchoalveolar lavage fluid fractions containing polyme

169 Bronchoalveolar lavage fluid from 23 lean, 12 overweight

170 Consistent with this finding, bronchoalveolar lavage fluid from A(2B)R(-/-) mice with

171 17A, neutrophil counts, and total protein in bronchoalveolar lavage fluid from acute respiratory dist

172 Bronchoalveolar lavage fluid from Ad-MD-2s mice transfer

173 CG and NE in bronchoalveolar lavage fluid from CF patients both contr

174 ion, and are detected at increased levels in bronchoalveolar lavage fluid from cystic fibrosis patien

175 In bronchoalveolar lavage fluid from human lung transplant

176 Here we show that bronchoalveolar lavage fluid from humans heterozygous fo

177 In bronchoalveolar lavage fluid from humans with ARDS, gut-

178 erleukin 12, were significantly lower in the bronchoalveolar lavage fluid from IL-15(-/-) and NK1.1(+

179 was consistently inhibited by treatment with bronchoalveolar lavage fluid from inhibitory kappaB kina

180 cell counts were significantly higher in the bronchoalveolar lavage fluid from OVA/OVA than OVA/PBS m

181 that leptin levels would be increased in the bronchoalveolar lavage fluid from overweight/obese subje

182 vo, histone-C1INH complexes were detected in bronchoalveolar lavage fluid from patients with acute re

183 we found significant elevation of IL-17A in bronchoalveolar lavage fluid from patients with ARDS, an

184 he protein expression patterns in plasma and bronchoalveolar lavage fluid from patients with ARDS.

185 nase activities were quantified in serum and bronchoalveolar lavage fluid from patients with CF, asth

186 pulmonary eosinophilia were measured in the bronchoalveolar lavage fluid from patients with mild ast

187 his study were: 1) to quantify fibrocytes in bronchoalveolar lavage fluid from patients with or witho

188 F-specific IgG is elevated in both serum and bronchoalveolar lavage fluid from Rasgrp1-deficient mice

189 Additionally, bronchoalveolar lavage fluid from this group of hepSTAT3

190 ory cells and cytokines were compared in the bronchoalveolar lavage fluid from WT and SP-D(-/-) mice

191 ed growth factor-Ralpha were detected in all bronchoalveolar lavage fluids from acute lung injury/acu

192 Treatment of alveolar epithelial cells with bronchoalveolar lavage fluids from ARDS patients drove b

193 Bronchoalveolar lavage fluids from ozone-treated rats re

194 und significantly elevated total proteins in bronchoalveolar lavage fluid, higher parasitemia and tis

195 beta, and chemokines in lung homogenates and bronchoalveolar lavage fluid; however, PMN recruitment i

196 ase-9 and proinflammatory mediator levels in bronchoalveolar lavage fluid, ii) lung parenchymal leuko

197 ry inflammation, eosinophilia, and increased bronchoalveolar lavage fluid IL-4 and IL-5, whereas adop

198 Bronchoalveolar lavage fluid IL-9 and IL-10, serum IL-9,

199 (IL-28A/B) production was attenuated in the bronchoalveolar lavage fluid in all factor-deficient mic

200 ntrations and absolute neutrophil numbers in bronchoalveolar lavage fluid in GSTM1+ but not GSTM1null

201 s, angiotensin-converting enzyme activity in bronchoalveolar lavage fluid increased 3.2-fold in elder

202 Furthermore, a delayed (6-8 hr) increase in bronchoalveolar lavage fluid interleukin-6 concentration

203 ezolid administration sufficiently decreased bronchoalveolar lavage fluid levels of IFN-gamma at day

204 ance responses was associated with increased bronchoalveolar lavage fluid levels of IL-10 and TGF-bet

205 F bronchoalveolar lavage fluid and increased bronchoalveolar lavage fluid levels of IL-5 and IL-13.

206 Hypothermia reduced bronchoalveolar lavage fluid levels of interleukin-1beta

207 transfusion-related acute lung injury cases, bronchoalveolar lavage fluid levels of interleukin-8, in

208 es in lung epithelial cell proliferation and bronchoalveolar lavage fluid levels of keratinocyte grow

209 asthma resulted in a remarkable increase in bronchoalveolar lavage fluid levels of LPA enriched in p

210 In plasma and bronchoalveolar lavage fluid, levels of interleukin-6, i

211 Bronchoalveolar lavage fluid LT levels were increased in

212 kine and chemokine levels were determined in bronchoalveolar lavage fluid, lung homogenates and lung

213 utrophils, B and T cells, and macrophages in bronchoalveolar lavage fluid, lung Th2, Tc2, Th17, Tc17,

214 Remodeling factors in murine bronchoalveolar lavage fluid, lung tissue, or human nasa

215 Mice were euthanized and bronchoalveolar lavage fluid, lungs and serum were colle

216 fected myeloid cells were detected in blood, bronchoalveolar lavage fluid, lungs, spleen, and brain,

217 L-9(+) CD4(+) T cells was increased in lung, bronchoalveolar lavage fluid, lymph nodes, and blood of

218 ded throughout the study day, and plasma and bronchoalveolar lavage fluid markers of inflammation wer

219 ergic inflammation, including cell counts in bronchoalveolar lavage fluid, mucin production, ASM mass

220 utum (n = 128), tracheal aspirates (n = 71), bronchoalveolar lavage fluid (n = 152), pleural fluid (n

221 red soluble HLA-G (sHLA-G) concentrations in bronchoalveolar lavage fluid (n = 36) and plasma (n = 57

222 Bronchoalveolar lavage fluid neutrophils, total protein,

223 Bronchoalveolar lavage fluid obtained during this bronch

224 Similarly, bronchoalveolar lavage fluid obtained from human volunte

225 m asthmatic and control lung tissue, (ii) in bronchoalveolar lavage fluid obtained from non-severe an

226 cytokines, chemokines, and growth factors in bronchoalveolar lavage fluid of 20 stable patients, 20 p

227 P-ribosyl-HNP-(ornithine) were isolated from bronchoalveolar lavage fluid of a patient with idiopathi

228 cells in lymph nodes, peripheral blood, and bronchoalveolar lavage fluid of AGMs and rhesus macaques

229 1) levels were significantly elevated in the bronchoalveolar lavage fluid of all mice infected with C

230 OSM levels were also increased in bronchoalveolar lavage fluid of allergic asthmatic patie

231 Consistently, the IL-22 expression in bronchoalveolar lavage fluid of asthmatic patients inver

232 mal human bronchial epithelial cells and the bronchoalveolar lavage fluid of asthmatic subjects by ul

233 we report that SOCS3 protein was elevated in bronchoalveolar lavage fluid of both virus- and bacteria

234 icantly reduced the number of neutrophils in bronchoalveolar lavage fluid of C3aR-deficient mice.

235 Exosomes were isolated and detected from bronchoalveolar lavage fluid of control and asthmatic mi

236 g immunohistochemistry and Luminex assays in bronchoalveolar lavage fluid of healthy and asthmatic pa

237 As we found ILC3-like cells in the bronchoalveolar lavage fluid of individuals with asthma,

238 -promoting cytokines and chemokines into the bronchoalveolar lavage fluid of Lipa(-/-) mice.

239 oxidase were more frequently detected in the bronchoalveolar lavage fluid of lung transplant patients

240 of desialylated MUC1-ED were elevated in the bronchoalveolar lavage fluid of mechanically ventilated

241 f RAGE was determined in protein, serum, and bronchoalveolar lavage fluid of mice and lungs and serum

242 5-HT levels are increased in bronchoalveolar lavage fluid of mice and people with ast

243 F) in the medium of B20-treated cells and in bronchoalveolar lavage fluid of mice exposed to B20 were

244 crophages, neutrophils, and cytokines in the bronchoalveolar lavage fluid of mice than its mutant cou

245 identify host proteins contained within the bronchoalveolar lavage fluid of mice that are cleaved an

246 lenge, and eosinophils were increased in the bronchoalveolar lavage fluid of wild-type mice.

247 HDM, elevated the levels of chemerin in the bronchoalveolar lavage fluid of WT mice.

248 ncreased ATP concentrations were reported in bronchoalveolar lavage fluids of asthmatic patients.

249 ar species and the level of ATX exoenzyme in bronchoalveolar lavage fluids of human patients with ast

250 5-HT levels were measured in bronchoalveolar lavage fluid or serum of animals with AA

251 d with CA-MHB supplemented with serum, mouse bronchoalveolar lavage fluid, other macrolides, and othe

252 ns and polymorphonuclear cell recruitment in bronchoalveolar lavage fluid (p<0.05 for both).

253 Bronchoalveolar lavage fluid PGE(2) concentrations were

254 Although bronchoalveolar lavage fluid phosphatidylcholine was mod

255 l infiltration, lung tissue malondialdehyde, bronchoalveolar lavage fluid protein concentration and l

256 d by reductions in pulmonary cell influx and bronchoalveolar lavage fluid protein levels compared wit

257 Removal of ficolins from human serum or bronchoalveolar lavage fluid reduces their antiviral act

258 Th2 cytokines including IL-4 and IL-5 in the bronchoalveolar lavage fluid relative to control mice.

259 The assay is validated for testing bronchoalveolar lavage fluids, replacing the requirement

260 ay and analyses of the injury markers in the bronchoalveolar lavage fluid, respectively.

261 resulted in hypovirulence, while analysis of bronchoalveolar lavage fluid revealed that tumor necrosi

262 s with that of conventional culture by using bronchoalveolar lavage fluid samples from patients with

263 istress syndrome 1, we used paired serum and bronchoalveolar lavage fluid samples obtained within 48

264 Endobronchial biopsies and bronchoalveolar lavage fluid samples were collected from

265 tained pulmonary or systemic health effects, bronchoalveolar lavage fluid, serum metabolic and inflam

266 ynthase similarly attenuated the increase in bronchoalveolar lavage fluid SOCS3 noted in lungs of mic

267 The effect of bronchoalveolar lavage fluids stimulating migration was

268 ption factor GATA3 and intracellular IL-4 in bronchoalveolar lavage fluid T cells, but expression of

269 ith severe asthma had increased HA levels in bronchoalveolar lavage fluid that correlated with pulmon

270 ity in lymphoid tissues and Th2 responses in bronchoalveolar lavage fluid), they also accumulate func

271 ther report that intra-alveolar coagulation (bronchoalveolar lavage fluid thrombin-antithrombin compl

272 rs of inflammation (i.e., neutrophils in the bronchoalveolar lavage fluid, TNF-alpha).

273 ficantly reduced histopathologic changes and bronchoalveolar lavage fluid total protein (endothelial

274 In serum and bronchoalveolar lavage fluid, total anti-IAV IgG and IgA

275 ressive alveolar neutrocytosis and increased bronchoalveolar lavage fluid tumor necrosis factor-alpha

276 yte count (p = 0.03) and a minor increase in bronchoalveolar lavage fluid tumor necrosis factor-alpha

277 CINC-3 levels, but no effect was observed on bronchoalveolar lavage fluid tumor necrosis factor-alpha

278 choalveolar lavage fluid (1513 +/- 337 pg/mL bronchoalveolar lavage fluid vs. 230 +/- 19 pg/mL, shock

279 Bronchoalveolar lavage fluid was analyzed for inflammato

280 gillus species, Streptococcus pneumoniae) in bronchoalveolar lavage fluid was associated with clinica

281 Bronchoalveolar lavage fluid was collected from 23 stero

282 Bronchoalveolar lavage fluid was collected from 68 atopi

283 Bronchoalveolar lavage fluid was collected from healthy

284 Bronchoalveolar lavage fluid was collected from patients

285 One day after treatment, bronchoalveolar lavage fluid was collected to determine

286 Mouse bronchoalveolar lavage fluid was harvested for cell coun

287 had a separate donor; however, pretransplant bronchoalveolar lavage fluid was only available from the

288 The concentration of CXCL12 in plasma and bronchoalveolar lavage fluids was quantified by ELISA.

289 Elevated levels of Cif in bronchoalveolar lavage fluid were correlated with lower

290 third MR imaging examination, eosinophils in bronchoalveolar lavage fluid were counted.

291 Bacterial counts in homogenized lungs and bronchoalveolar lavage fluid were decreased after cranbe

292 1) , and IgG(2a) levels and cell counts from bronchoalveolar lavage fluid were determined.

293 Additionally, Th1 cytokine levels in bronchoalveolar lavage fluid were increased.

294 Levels of cytokines and chemokines in bronchoalveolar lavage fluid were measured by enzyme-lin

295 Concentrations of PGE(2) in bronchoalveolar lavage fluid were measured by using ELIS

296 Aliquots of blood, sputum, and bronchoalveolar lavage fluid were obtained from asthma s

297 While total and differential cell counts in bronchoalveolar lavage fluid were similar between the Sy

298 reased numbers of T cells and neutrophils in bronchoalveolar lavage fluid, whereas expression of Foxp

299 trophil apoptosis and reduce neutrophilia in bronchoalveolar lavage fluid, while IL-6 increases rapid

300 increased the numbers of eosinophils in the bronchoalveolar lavage fluid, while simultaneously decre