ライフサイエンスコーパス: bronchoconstrictor

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1 from COX-1(-/-) mice were hyperresponsive to bronchoconstrictors.

2 causing changes in airway responsiveness to bronchoconstrictors.

3 a(2+)]i, although to a much lower level than bronchoconstrictors.

4 Originally recognized for their potent bronchoconstrictor actions, they were subsequently deter

5 nto the chemotaxin LTB4, which has no direct bronchoconstrictor activity.

6 Leukotrienes are bronchoconstrictor and vasoactive lipid mediators that a

7 fore, involvement of ADO A3 receptors in the bronchoconstrictor and/or inflammatory effects have to b

8 everses the increase in [Ca(2+)]i induced by bronchoconstrictors, and this lowering of the [Ca(2+)]i

9 of an asthmatic response to several types of bronchoconstrictor challenge.

10 Anti-leukotrienes inhibit several bronchoconstrictor challenges, including allergens, exer

11 high concentrations of inhaled NO reduce the bronchoconstrictor effect of methacholine in animal mode

12 sts that, in addition to the well-known anti-bronchoconstrictor effect, tiotropium might also display

13 is study was to determine whether the potent bronchoconstrictor endothelin-1 was coupled to the activ

14 istance in response to increasing doses of a bronchoconstrictor following OVA immunization and challe

15 Adenosine (ADO) is a potent bronchoconstrictor in allergic patients and has been sho

16 ated that Rac1 activation is responsible for bronchoconstrictor-induced increase in intracellular Ca(

17 ion with increased release of vasoactive and bronchoconstrictor mediators.

18 ed airway resistance response to the inhaled bronchoconstrictor methacholine in the TG mice.

19 aggerated airflow obstruction in response to bronchoconstrictors), mucus overproduction and chronic e

20 ntracellular selective inhibitor of multiple bronchoconstrictor receptors, may play a central role in

21 ase in isolated mast cells, and prevents the bronchoconstrictor response in subjects with exercise-in

22 sponsiveness (BHR) describes the exaggerated bronchoconstrictor response to a host of stimuli such as

23 in E(2) (PGE(2)) inhibits the early and late bronchoconstrictor response to inhaled allergen.

24 Inhaled enoxaparin failed to modify the bronchoconstrictor response to methacholine, and did not

25 cal inhibition of Rac1 in mice prevented the bronchoconstrictor response to methacholine.

26 at treatment with zafirlukast attenuates the bronchoconstrictor response.

27 Heparin inhibited the bronchoconstrictor responses to exercise by 58%, 78%, an

28 Moreover, the bronchoconstrictor responses to PGE2 and all other teste

29 Bronchoconstrictor responses were recorded from offsprin

30 ors (a) modulate the activity of cardiac and bronchoconstrictor vagal preganglionic neurones (CVPNs a

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