ライフサイエンスコーパス: bronchus

1 genetic changes in cells exfoliating in the bronchus.

2 ting a 5-F balloon catheter into a secondary bronchus.

3 achea, and the other, of the left upper lobe bronchus.

4 of an organized epithelial cell layer in the bronchus.

5 onstriction and proliferation in the primary bronchus.

6 xamined in purified HLMCs and isolated human bronchus.

7 expiratory collapse was calculated for each bronchus.

8 th muscle-positive cells of human trachea or bronchus.

9 receptive fields in the trachea or main stem bronchus.

10 the distal trachea, the carina, and the main bronchus.

11 acheobronchial damage at the carina and main bronchus.

12 infiltrate occurring distal to an obstructed bronchus.

13 cally sensitive nerve endings in the trachea/bronchus.

14 ts with 54 telescoping anastomoses (30 right bronchus, 24 left bronchus) were retrospectively reviewe

15 iniferatoxin were examined in human isolated bronchus (5-12 mm o.d.).

16 irway lavage (PAL) of the isolated left main bronchus and bronchoalveolar lavage (BAL; bronchial frac

17 ed vascular compression of the proximal left bronchus and distal trachea, and the other, of the left

18 A hypoplastic bronchus and ipsilateral lung were manifest in all four

19 n the number of deaths due to cancers of the bronchus and lung (SMR = 1.3).

20 The size of the ipsilateral bronchus and lung may be estimated by comparison of radi

21 e risks were found for cancers of the colon, bronchus and lung, breast, and uterus.

22 nd cell tropism in ex-vivo cultures of human bronchus and lung.

23 ral genes being highly expressed in both the bronchus and nose.

24 y system, such as an enclosed intrapulmonary bronchus and parabronchi.

25 pase-1 expression in human airway epithelium bronchus and primary cells, (2) characterized NLRP3 infl

26 anted lung by occlusion of the contralateral bronchus and pulmonary artery.

27 revealed DeltaNp63alpha expression in normal bronchus and squamous carcinomas but not in normal lung

28 duced but findings were significant only for bronchus and trachea.

29 extrapulmonary airways (larynx, trachea and bronchus) and the lung parenchymal tissue.

30 roduced by complete obstruction of one lobar bronchus, and (3) VA/Q inequality (n = 8) created by par

31 in different airway regions (mouth, trachea, bronchus, and bronchiole) were similar.

32 several tissues, including human lung, human bronchus, and human peripheral blood leukocytes.

33 and anus (male, 3.3; female, 3.0); trachea, bronchus, and lung (male, 3.3; female, 7.5); and brain a

34 iratory tract of ferrets, including trachea, bronchus, and lung alveolus tissues.

35 these, 1.5 million (19%) were from trachea, bronchus, and lung cancer.

36 and 2014, including 5656423 due to tracheal, bronchus, and lung cancer; 2484476 due to colon and rect

37 r and biliary; pancreatic; larynx; tracheal, bronchus, and lung; malignant skin melanoma; nonmelanoma

38 the rectum, rectosigmoid, and anus; trachea, bronchus, and lung; skin; and connective tissues (all si

39 ) relax bronchiolus to a greater degree than bronchus, as seen with volatile anesthetics.

40 characterized by lymphoid hyperplasia of the bronchus-associated lymphoid tissue (BALT) and infiltrat

41 Ectopic lymphoid tissue, such as bronchus-associated lymphoid tissue (BALT) in the lung,

42 Bronchus-associated lymphoid tissue (BALT) is occasional

43 Bronchus-associated lymphoid tissue (BALT) participates

44 odels of acute rejection in lung allografts, bronchus-associated lymphoid tissue (BALT) plays a major

45 Bronchus-associated lymphoid tissue (BALT) was originall

46 athologic changes including the expansion of bronchus-associated lymphoid tissue (BALT), goblet cell

47 ymphatic growth was restricted to regions of bronchus-associated lymphoid tissue (BALT), where VEGF-C

48 ty, which resulted in lesions that resembled bronchus-associated lymphoid tissue (BALT).

49 ults in significant recruitment of inducible bronchus-associated lymphoid tissue (iBALT) as well as C

50 ukin-1alpha (IL-1alpha) and caused inducible bronchus-associated lymphoid tissue (iBALT) formation in

51 Inducible bronchus-associated lymphoid tissue (iBALT) was also for

52 tertiary lymphoid tissues, such as inducible bronchus-associated lymphoid tissue (iBALT), form in non

53 he development and organization of inducible bronchus-associated lymphoid tissue (iBALT).

54 , polyclonal LPD (n = 8), and hyperplasia of bronchus-associated lymphoid tissue (n = 1).

55 mice, but some pathologies such as enhanced bronchus-associated lymphoid tissue and bronchiolitis ob

56 ses were retained, including vaccine-induced bronchus-associated lymphoid tissue and CD8(+) effector

57 T cell proliferation, induction of inducible bronchus-associated lymphoid tissue and correlates of ba

58 Autoantibodies are produced in bronchus-associated lymphoid tissue and, when bound to p

59 ion using CCR7-deficient mice, which develop bronchus-associated lymphoid tissue early in life.

60 Bronchus-associated lymphoid tissue expands in pulmonary

61 results in lung lymphocyte accumulation and bronchus-associated lymphoid tissue formation.

62 Development of bronchus-associated lymphoid tissue has been suggested t

63 ne expression assays to evaluate the role of bronchus-associated lymphoid tissue in pulmonary hyperte

64 Bronchus-associated lymphoid tissue plays important role

65 Bronchus-associated lymphoid tissue was more numerous, l

66 As expected, no bronchus-associated lymphoid tissue was observed in M. t

67 The major responding components of the bronchus-associated lymphoid tissue were the CD8(+) T ly

68 We reasoned that activated bronchus-associated lymphoid tissue would be evident in

69 y defined cellular accumulations include the bronchus-associated lymphoid tissue, cryptopatches, and

70 y chemokines and the appearance of inducible bronchus-associated lymphoid tissue-like structures in t

71 ration clearly resembles an expansion of the bronchus-associated lymphoid tissue.

72 he epithelial and smooth muscle cells in the bronchus at the region of balloon placement.

73 y (always right upper lobe second-generation bronchus) at baseline, after 2 wk, and again after 8 wk

74 California's lung and bronchus cancer incidence is already declining at a sign

75 difference in relative survival for lung and bronchus cancer patients is examined using data from pop

76 uding bladder, prostate, female breast, lung/bronchus, endometrial, colon, non-Hodgkin lymphoma, panc

77 he dorsal and ventral aspects of the primary bronchus, especially before branch formation, inhibiting

78 MD) cell cultures compared with normal human bronchus fibroblasts (HBFs) from LAM patients.

79 ml for normal human mesenchymal cells, human bronchus fibroblasts and human airway smooth muscle cell

80 l blocker was placed into the left main stem bronchus for lung isolation and application of continuou

81 n of balloon-tip catheters into the bleeding bronchus for tamponade of the hemorrhagic artery, protec

82 ions caused by separation of the invaginated bronchus from the recipient bronchus were seen in 16 ana

83 ependent contractions of guinea pig isolated bronchus, however, thrombin (3-300 nM) was a weak spasmo

84 xpression changes that occur in the mainstem bronchus in response to smoking.

85 onchus (RMB), left main bronchus, (LMB), and bronchus intermedius (BI), and the mean percentage of ex

86 edetermined levels (aortic arch, carina, and bronchus intermedius) to confirm ECAC (>50% reduction in

87 t the levels of the aortic arch, carina, and bronchus intermedius).

88 s 30.9% with end expiration (P < .0001); and bronchus intermedius, 57.5% with dynamic expiration vers

89 nomas showed aggressive growth patterns with bronchus invasion and obstruction.

90 [IRR], 1.70; P < .01), carcinoma of the lung/bronchus (IRR, 1.58; P < .01), non-Hodgkin lymphoma (IRR

91 for the right main bronchus (RMB), left main bronchus, (LMB), and bronchus intermedius (BI), and the

92 Main bronchus (MB) and tracheal (TR) diameters and lung area

93 TBN involved the left bronchus (n = 17; 85%), the carina (n = 10; 50%), the su

94 supracarinal trachea (n = 9; 45%), the right bronchus (n = 4; 20%), and the cervical trachea (n = 3;

95 nary arteries before and after left mainstem bronchus occlusion (LMBO) in mice with and without a con

96 nd during hypoxia produced by left main stem bronchus occlusion (LMBO) in mice with and without a tar

97 tance (LPVR) before and during left mainstem bronchus occlusion (LMBO) was measured in mice with and

98 did not impair the ability of left mainstem bronchus occlusion to increase left pulmonary vascular r

99 vascular resistance induced by left mainstem bronchus occlusion, was markedly impaired in animals ven

100 ologically normal epithelium of the mainstem bronchus of smokers with lung cancer.

101 ic acid was instilled into the left mainstem bronchus of TLR4-defective (both C3H/HeJ and congenic C.

102 Subsuperior bronchus or bronchi were found on the right side in 44%

103 of significant size discrepancy in the main bronchus (P < 0.05).

104 significantly higher at the carina and main bronchus (p <.01; Kruskal-Wallis test followed by Dunn's

105 In an ex vivo innervated trachea/bronchus preparation, BK (1 microM) consistently produce

106 roptic bronchoscopy, with isolated left main bronchus proximal airway lavage (PAL) and bronchoalveola

107 decreased ability to release virus from the bronchus rather than the lung.

108 aller mean right upper lobe apical segmental bronchus (RB1) lumen volume (LV) in comparison with heal

109 created by partial obstruction of one lobar bronchus resulting in a bimodal VA/Q distribution with 1

110 rced-expiratory CT images for the right main bronchus (RMB), left main bronchus, (LMB), and bronchus

111 ved in patients with cancers of the lung and bronchus (standardized mortality ratio [SMR] = 5.74; 95%

112 ously defined smoking-induced genes from the bronchus suggested that smoking had a similar effect on

113 scopy was performed using the superDimension/Bronchus system consisting of electromagnetic board, pos

114 navigation bronchoscopy using superDimension/Bronchus System is a novel method to increase diagnostic

115 y larger in diameter than the left main-stem bronchus, though the latter was longer and straighter.

116 collected from the normal-appearing mainstem bronchus to assess the probability of lung cancer.

117 Incidence of cancers of the pancreas, bronchus, trachea, bladder, and lymphatic and haemopoiet

118 ammac(-/-) mice undergoing heterotopic human bronchus transplantation and reconstitution with allogen

119 zed mouse models of heterotopic subcutaneous bronchus transplantation imitate the in vivo development

120 Heterotopic bronchus transplantation was performed in 33 NOD.rag(-/-

121 deficient mice receiving heterotopic porcine bronchus transplants, and major histocompatibility compl

122 erges from the dorsal surface of the primary bronchus via monopodial branching to form the conducting

123 was oversewn and a fistula to the right main bronchus was closed by means of an autologous pericardia

124 In the other three patients, the left main bronchus was compressed.

125 specimens, we found that the right main-stem bronchus was consistently larger in diameter than the le

126 tient, stent placement in the left main stem bronchus was needed to relieve left lung atelectasis.

127 ic ganglia neurons on the guinea pig primary bronchus were analyzed, and the procedure for localizing

128 ontralateral right main pulmonary artery and bronchus were ligated.

129 the invaginated bronchus from the recipient bronchus were seen in 16 anastomoses (30%).

130 ping anastomoses (30 right bronchus, 24 left bronchus) were retrospectively reviewed.

131 pithelial cells of airways, particularly the bronchus, where high expression of SCGB3A2 is found.