ライフサイエンスコーパス: brush border

1 ve with normal human kidney proximal tubular brush border.

2 d an apical array of microvilli known as the brush border.

3 accumulated in villi and at the base of the brush border.

4 position and highly ordered structure of the brush border.

5 n of intermediate filament proteins into the brush border.

6 philic adhesion complexes help stabilize the brush border.

7 bules of endocytic compartments in the renal brush border.

8 PPIV complex in microdomains of rabbit renal brush border.

9 htly packed array of microvilli known as the brush border.

10 egalin in the intermicrovillar region of the brush border.

11 al tubules, NHE-RF was located in the apical brush border.

12 (megalin-associated) oligomers in the renal brush border.

13 mice became stunted, and enterocytes lacked brush border.

14 splaying an intact polarization with regular brush border.

15 ic protein receptors from porcine intestinal brush borders.

16 the lipid extract of rabbit small intestinal brush borders.

17 formed proteomic analysis on WT and Myo1a KO brush borders.

18 Marked destruction of the microvillous brush border adjacent to adherent organisms was observed

19 ired for ileal adherence as assayed by ileal brush border aggregation.

20 In addition, the brush-border alkaline phosphatase was reduced as was exp

21 These studies revealed that WT brush borders also contain the short-tailed class I myos

22 cal surface during differentiation to form a brush border: an array of actin-supported membrane protr

23 trols genes required to construct the apical brush border and absorb nutrients, including dietary lip

24 Uptake is slower than at the brush border and although, as in the brush border, uptak

25 diated transport systems for L-tryptophan in brush border and basal membranes.

26 to continuous FSS also acquired an extensive brush border and basolateral membrane invaginations rese

27 se found in the microvilli of the intestinal brush border and kidney.

28 gnaling molecules cosediment with megalin in brush border and microvillar fractions.

29 epitopes blocked the binding of bacteria to brush borders and of fimbriae to IMTGP.

30 by using 24-well plates coated with purified brush borders and purified microvilli.

31 duodenal enterocytes in control rats and on brush-border and basolateral membrane domains in iron-de

32 protein levels are dramatically increased in brush-border and basolateral membrane vesicles isolated

33 nterocytes, M cells lack an organized apical brush border, and are able to transcytose microparticles

34 ytoskeletal and membrane components from the brush border, and redistribution of intermediate filamen

35 ges (vacuolization of tubular cells, loss of brush border, and tubular cell swelling) were each obser

36 detected Klotho in the proximal tubule cell, brush border, and urinary lumen, where phosphate homeost

37 of microvilli in both kidney and intestinal brush borders, and loss of Myo7b in differentiating inte

38 ncrease in the NHE3 mobile fraction from the brush border; and was accompanied by a NHERF2 ezrin-radi

39 We postulate that antibodies to brush border antigens cause direct epithelial injury, ac

40 ointerstitial nephritis due to antibodies to brush border antigens of the proximal tubule has been de

41 eleton following treatment with anti-tubular brush-border antiserum (anti-Fx1A).

42 ding, we found that the abundance of AQP1 in brush border apical and basolateral membranes was augmen

43 data reveal an adhesion-based mechanism for brush border assembly and illuminate the basis of intest

44 We found that brush border assembly is driven by the formation of Ca(2

45 udy uncovers an essential role for ANKS4B in brush border assembly, reveals a hierarchy in the molecu

46 isrupts intermicrovillar adhesion and, thus, brush border assembly.

47 hrough hydrolysis of the phosphate esters by brush border-associated enzymes, leading to a high conce

48 Higher magnification revealed well-developed brush borders at the apical side of the tissue.

49 icrovillar lipid rafts, was missing from the brush border (BB) in cells expressing this fragment.

50 xchanger 3 (NHE3) and is associated with the brush border (BB) membrane of murine kidney and small in

51 or cordon bleu (COBL) promotes the growth of brush border (BB) microvilli.

52 identifies calpain as being instrumental for brush border (BB) microvillus assembly during differenti

53 The epithelial brush border (BB) Na(+)/H(+) exchanger 3 (NHE3) accounts

54 ed that approximately 25-50% of rabbit ileal brush border (BB) Na(+)/H(+) exchanger NHE3 is in lipid

55 The brush border (BB) Na(+)/H(+) exchanger NHE3 is rapidly a

56 The intestinal and renal proximal tubule brush border (BB) Na+-H+ exchanger NHE3 binds to members

57 ine, LLC-PK1-CL4 (CL4), forms a well ordered brush border (BB) on its apical surface.

58 nt within the microvillus (MV) of the apical brush border (BB) where it forms lateral tethers between

59 E3) was shown to exist in three pools in the brush border (BB), including a population in lipid rafts

60 Within microvilli of the enterocyte brush border (BB), myosin-1a (Myo1a) forms an ordered en

61 sed on physiological studies, the epithelial brush-border (BB) Na+/H+ antiporter3 (NHE3) seems to ass

62 To investigate the possible role of a brush border beta-glucosidase in the hydrolysis of PNG,

63 8 in mice also disrupted the assembly of the brush border, but ezrin distributed away from the apical

64 edulla displaying cell necrosis, loss of the brush border, cast formation and tubular dilatation in W

65 in, which is found only in the microvilli of brush border cells.

66 Klf5(Delta/Delta) fetuses lacked the apical brush border characteristic of enterocytes, and a loss o

67 ity of the transporter responsible for renal brush border Cl(-)-formate exchange has yet to be elucid

68 thelial cells, leading to the formation of a brush border containing microvilli on the apical surface

69 n of villin, an actin-binding protein of the brush border cytoskeleton.

70 nal epithelium in maximizing flow-activated, brush border-dependent, transcellular salt and water rea

71 d extracellular adhesion to shape the apical brush border domain.

72 vivo or inadequate nutrient exposure to the brush border due to small intestinal dysmotility.

73 olved in Map-induced filopodia formation and brush border elongation on infected HeLa and Caco-2 cell

74 ipopolysaccharide were reduced in Caco2-BBE (brush border enterocyte) cells exposed to NP-KPV compare

75 Duodenal mucosal histology and expression of brush border enzyme dipeptidyl peptidase IV and peptide

76 ncreatic triglyceride lipase, and intestinal brush border enzyme, phospholipase B.

77 Brush-border enzyme (intestinal alkaline phosphatase [IA

78 and in vivo models, we demonstrate that the brush-border enzyme, intestinal alkaline phosphatase (IA

79 we examined the role of the small intestinal brush-border enzyme, intestinal alkaline phosphatase (IA

80 further digestion of a gliadin by intestinal brush border enzymes and is a highly specific substrate

81 The brush border enzymes DPPIV and sucrase-isomaltase still

82 design, allowing for cleavage by the kidney brush border enzymes of the CPP before uptake by proxima

83 illar membrane, contain catalytically active brush border enzymes, and are specifically enriched in i

84 ferentiated intestinal epithelium, including brush border enzymes, polarization, and tight junctions.

85 teroendocrine cells), presence of functional brush-border enzymes (lactase, sucrase-isomaltase and di

86 and SI are anchored to the small intestinal brush-border epithelial cells, and each contains a catal

87 a protein found in the actin bundles of the brush border epithelium, is of interest both as a compac

88 ignificantly reduced NHE3 basal activity and brush border expression in Caco-2BBe cells.

89 These studies indicated that, although brush border expression of NHE3 was unaffected by the lo

90 In conclusion, epithelial MLCK-activated brush border fanning by IFN-gamma promotes adherence and

91 ht chain phosphorylation, arc formation, and brush border fanning coincided with intermicrovillous ba

92 a caused MLCK-dependent TW arc formation and brush border fanning, which preceded caveolin-mediated b

93 nase (MLCK) in TW myosin phosphorylation and brush border fanning.

94 but did affect epithelial morphogenesis and brush border formation.

95 of the apical microvilli were induced as the brush border formed during polarization; as barrier func

96 distinct membrane domain at the base of the brush border, from which NHE-RF was absent, consistent w

97 tose, was accompanied by marked increases in brush border GLUT5 abundance, and was blocked by RU486.

98 oxin (STa) to its receptor on the intestinal brush border, guanylyl cyclase type C (GC-C).

99 ed the safety factor of the mouse intestinal brush-border hydrolase maltase in series with the glucos

100 The duodenal epithelial brush border IAP-P2Y-HCO(3-) surface microclimate pH reg

101 PepT1 is abundant along the small-bowel brush border in humans; expression in the colon indicate

102 e of which (NHERFs 1-3) are localized to the brush border in kidney and intestinal epithelial cells.

103 ti-AgALP serum localized AgALP to the apical brush border in the anterior and posterior midgut of lar

104 n caspase-3 activity, tubular apoptosis, and brush border injury (BBI) in mouse kidneys.

105 differentiation, as revealed by the lack of brush border intestinal alkaline phosphatase activity, t

106 Preliminary studies of enterocyte brush-border ion transporter proteins (sodium hydrogen e

107 AgCad1 in the larval brush border is a binding protein for Cry4Ba toxin.

108 Although the brush border is essential for physiological homeostasis,

109 e most abundant components of the enterocyte brush border is the actin-based monomeric motor, myosin-

110 e II transmembrane protein of the enterocyte brush border, is sorted directly to the apical membrane

111 +)/H(+) exchanger 3 (NHE3) is the epithelial-brush border isoform responsible for most intestinal and

112 e whether PDZK1 interaction is essential for brush border localization of NHE3 and CFEX in vivo, we e

113 ly, the treatment strategy prevented tubular brush border loss, diminished tubular iron deposition, b

114 Here we show that Fas2 is essential to brush border maintenance in renal tubules of Drosophila.

115 Brush-border maltase-glucoamylase (MGA) activity serves

116 well as epithelial markers and a PT-enriched brush border marker Ggt1.

117 tered KBrO(3) on redox status and enzymes of brush border membrane (BBM) and carbohydrate metabolism

118 lotting revealed a 56 +/- 6% decrease in the brush border membrane (BBM) expression of mURAT1 in NHER

119 The epithelial brush border membrane (BBM) Na(+)-H(+) exchanger 3 (NHE3

120 or (EGF) stimulation of Na absorption by the brush border membrane (BBM) Na(+)/H(+) exchanger NHE3.

121 chronically inflamed rabbit small intestine, brush border membrane (BBM) Na-glutamine co-transport is

122 The apical brush border membrane (BBM) of intestinal epithelial cel

123 , and ezrin, was decreased in the intestinal brush border membrane (BBM) of mice with streptozotocin-

124 nzyme activities of cortical homogenates and brush border membrane (BBM) preparations documented litt

125 nd the uptake of these lipids in vitro using brush border membrane (BBM) vesicles.

126 se from the proximal tubule lumen across the brush border membrane (BBM) via a sodium-dependent trans

127 alent metal ions across the small-intestinal brush border membrane (BBM).

128 by uptake of ferrous Fe(II) iron across the brush border membrane and culminates in transfer of the

129 B1 phenylalanine permitted it to bind to the brush border membrane and greatly enhanced its hypoglyce

130 an enzyme that is anchored to the intestinal brush border membrane and is expressed by the glutamate

131 eficiency, the enzyme fails to anchor in the brush border membrane and so is secreted into the lumen,

132 A scavenger receptor (type BI) in the brush border membrane appears to facilitate cholesterol

133 are transported across the ileal enterocyte brush border membrane by the well characterized apical s

134 single nephrotoxic dose of KBrO(3) inhibits brush border membrane enzymes, induces oxidative stress

135 The AgCad1 protein was present in brush border membrane fractions prepared from larvae, an

136 ce expression of AQP1 in the proximal tubule brush border membrane is regulated in response to flow.

137 s formed from the raft lipid mixture or from brush border membrane lipids an array of more ordered an

138 escence localized the P2Y(1) receptor to the brush border membrane of duodenal villi.

139 nriched in the small intestine and is in the brush border membrane of enterocytes.

140 by increasing NHE3 protein abundance at the brush border membrane of intestinal epithelial cells.

141 her show that native ABCG2 is located in the brush border membrane of kidney proximal tubule cells, w

142 tic lipid mixtures and lipid extracts of the brush border membrane of mouse kidney cells.

143 logue of human SLC26A6, was localized to the brush border membrane of proximal tubule cells and was d

144 expression of CFEX, but not pendrin, on the brush border membrane of proximal tubule cells.

145 ed the dynamic translocation of GLUT2 to the brush border membrane of RPTCs, and reduced glucose reab

146 rption is mediated by SGLT1 localized in the brush border membrane of small intestinal enterocytes, i

147 The intestinal brush border membrane of the canine hookworm, Ancylostom

148 immunohistochemistry localized SGLT2 to the brush border membrane of the early proximal tubule.

149 ctivity of a putative sterol permease in the brush border membrane of the enterocyte that actively fa

150 raluminal cholesterol molecules crossing the brush border membrane of the enterocyte.

151 ssays of Cry1Ac, Cry2Aa and Cry1Ca to midgut brush border membrane proteins from BPH and PWS.

152 of specific alkaline phosphatase activity in brush border membrane proteins from susceptible (YDK and

153 CaLP expressed in S2 cells or in solubilized brush border membrane proteins.

154 n assays with GST-fusion proteins and native brush border membrane proteins.

155 P(i) flux at the apical (luminal) brush border membrane represents the rate-limiting step

156 Indomethacin treatment increased renal brush border membrane vesicle NaPi-2 protein abundance i

157 g and oligomerization by western blots using brush border membrane vesicles (BBMV) from a strain of P

158 hibitory AgAPN2ta blocked Cry11Ba binding to brush border membrane vesicles (BBMV) of A. gambiae wher

159 kDa aminopeptidase N (APN) was isolated from brush border membrane vesicles (BBMV) of Anopheles quadr

160 , and Cry8Ba on western corn rootworm midgut brush border membrane vesicles (BBMV).

161 Partial purification of proteins from brush border membrane vesicles (BBMVs) by gel filtration

162 Binding assays with brush border membrane vesicles (BBMVs) prepared from L.

163 ce the structure and stability of BT-R(1) on brush border membrane vesicles (BBMVs) prepared from M.

164 e and detected a 65 kDa species on a blot of brush border membrane vesicles (BBMVs) protein prepared

165 ertion of the whole toxin into Manduca sexta brush border membrane vesicles (BBMVs).

166 ibited phosphate transport in isolated renal brush border membrane vesicles and in cultured renal pro

167 unctional activity as Cl-oxalate exchange in brush border membrane vesicles and oxalate-stimulated vo

168 By contrast, phosphate transport in brush border membrane vesicles and proximal tubule cells

169 d the level of binding of [(125)I]Cry11Ba to brush border membrane vesicles by 41%, a percentage comp

170 bits sodium-dependent phosphate transport in brush border membrane vesicles derived from hormone-trea

171 ent bile salt transport was also measured in brush border membrane vesicles from the kidney.

172 protein that is present at reduced levels in brush border membrane vesicles from YHD2 larvae.

173 Brush border membrane vesicles prepared from rabbit kidn

174 Toxin-binding assays using brush border membrane vesicles revealed that a wild-type

175 pendent uptake of (3)H-taurocholate in renal brush border membrane vesicles was decreased.

176 CR12-MPED peptide bound brush border membrane vesicles with high affinity (K(d)

177 Finally, in assays with aphid gut-derived brush border membrane vesicles, binding of CP-P-GFP comp

178 levels of gamma-secretase activity in renal brush border membrane vesicles.

179 mately 2-3-fold in these rafts compared with brush border membrane vesicles.

180 or by surface plasmon resonance to M. sexta brush border membrane vesicles.

181 rabbit PepT1 and through PepT1 in rat renal brush border membrane vesicles.

182 e fraction, of which 40-50% was localized to brush border membrane.

183 and protein levels of NHE3 and SGLT1 in the brush border membrane.

184 water transport across the human intestinal brush border membrane.

185 otonic fluid transport across the intestinal brush border membrane.

186 hanced AQP1 abundance in the proximal tubule brush border membrane.

187 ase of the GLUT2/SGLT-1 protein ratio in the brush border membrane.

188 sodium-phosphate cotransporter Npt2a at the brush border membrane.

189 reductions in NPT2a expression in the renal brush border membrane.

190 ponsible for transport across the intestinal brush border membrane; however, the carrier(s) responsib

191 all intestinal glucose absorption across the brush-border membrane (BBM) via SGLT1 and GLUT2 were ana

192 -Reg variant that downregulates SGLT1 in the brush-border membrane at high luminal glucose concentrat

193 actose are transported across the intestinal brush-border membrane by the Na+/glucose cotransporter,

194 ERF1 is specifically localized at the apical brush-border membrane in intestinal epithelial cells and

195 Enrichment of PLBs with WCRW midgut brush-border membrane material resulted in a 2000-fold r

196 -glutamyl transpeptidase (GGT) on the apical brush-border membrane of 786-O proximal tubule cells wit

197 tein was iron regulated and localized to the brush-border membrane of duodenal enterocytes in iron de

198 rane reductases, was highly expressed in the brush-border membrane of duodenal enterocytes, and induc

199 f the visceral endoderm and localized to the brush-border membrane of extraembryonic endodermal cells

200 Taurine uptake across the brush-border membrane of human intestinal Caco-2 cell mo

201 mino acid transport function measured at the brush-border membrane of intact intestinal epithelia res

202 Ac-CP-2 is expressed by the parasite in the brush-border membrane of its alimentary canal, and anti-

203 l role in folate transport across the apical brush-border membrane of the proximal small intestine es

204 n by all gastric, pancreatic, and intestinal brush-border membrane proteases.

205 rric iron is attributed to the presence of a brush-border membrane reductase activity that displays a

206 with changes in Bt-toxin binding to sites in brush-border membrane vesicles of the larval midgut, and

207 nhibit Na+/glucose transport into intestinal brush-border membrane vesicles.

208 d activation and recruitment of GLUT2 to the brush-border membrane.

209 expressed on the apical domain of the ileal brush-border membrane.

210 acilitative glucose transporter GLUT2 to the brush-border membrane; regulation involves a protein kin

211 nance of active ERM proteins at the cortical brush border membranes (BBM) of polarized epithelia.

212 Experiments using brush border membranes and cultured renal proximal tubul

213 expressed in mammalian kidney and intestinal brush border membranes and in leukocytes and certain can

214 y 1,120 kDa that bound to solubilized insect brush border membranes and induced pore formation in bla

215 onide binds specifically to a single site in brush border membranes and to human embryonic kidney 293

216 t helices alpha-4 or alpha-6 insert into the brush border membranes because of their hydrophobic natu

217 the membrane proteinase meprin beta found in brush border membranes of kidney and small intestine.

218 gomyelin or 2) natural lipids extracted from brush border membranes that are rich in sphingomyelin an

219 s study, GUVs were assembled from rat kidney brush border membranes, which included the integral memb

220 ular, the insertion of the toxin into insect brush border membranes.

221 Na(+)/H(+) exchanger isoform, NHE3, in renal brush border membranes.

222 e, ERM proteins are significantly reduced in brush-border membranes from kidney and small intestine.

223 eduction in the transporter protein in renal brush-border membranes isolated from the mutant mice.

224 tightly ordered cross-linked bundles in the brush border microvilli and in the stereocilia of inner

225 B (ANKS4B) localizes to the tips of adherent brush border microvilli and is essential for intermicrov

226 The espins are actin-bundling proteins of brush border microvilli and Sertoli cell-spermatid junct

227 ons) and the 253-amino-acid "small espin" of brush border microvilli are splice isoforms that share a

228 For decades, enterocyte brush border microvilli have been viewed as passive cyto

229 zation resulting in the morphogenesis of the brush border microvilli in epithelial cells remain unkno

230 not absolutely required for the formation of brush border microvilli or for the establishment or main

231 Rab25-knockdown cells showed disorganized brush border microvilli with decreases in villin express

232 Within brush border microvilli, Myo1a carries out a number of c

233 imately to intestinal enterocytes and efface brush border microvilli.

234 These data support our hypothesis that the "brush-border" microvilli serve a mechanosensory function

235 Brush-border mobility was determined by fluorescence rec

236 ates two distinct adhesive interactions with brush border molecules of the intestinal epithelial cell

237 inal enterocytes did not identify defects in brush border morphology or structural polarization, demo

238 dherin mislocalization and severe defects in brush border morphology.

239 led that in the in vitro context of isolated brush borders, myosin-1a (myo1a) powers the sliding of m

240 Thus, regulation of renal brush border Na(+)-H(+) exchange activity may be mediate

241 aracterize the oligomeric forms of the renal brush border Na(+)-H(+) exchanger in more detail, we per

242 es revealed an unexpected association of the brush border Na(+)-H(+) exchanger NHE3 with dipeptidyl p

243 In our studies of the intestinal brush border Na+-glucose cotransporter we have obtained

244 on due to TNF-induced internalization of the brush border Na+/H+ exchanger NHE3.

245 olutes, and water and the endocytosis of the brush border Na+/H+ exchanger, thereby inhibiting Na+ ab

246 The epithelial brush-border Na(+)/H(+) exchanger NHE3 is acutely inhibi

247 rter 3 (NHE3), which is the major intestinal brush-border Na(+)/H(+) exchanger.

248 together, these data demonstrate that renal brush border NHE3 exists in two oligomeric states: a 9.6

249 It is associated with increased brush-border NHE3 and association between ezrin and NHE3

250 n actin bundling protein found in the apical brush border of absorptive tissues, is one of the first

251 brane projections that form the basis of the brush border of enterocytes and the Drosophila melanogas

252 lin is a major actin-bundling protein in the brush border of epithelial cells.

253 scaffold to membrane proteins in the apical brush border of intestinal epithelial cells.

254 rt-tailed class I motor, myosin-1c, into the brush border of knockout enterocytes.

255 um samples from the patient reacted with the brush border of normal human kidney, in contrast with th

256 expression of NHE3 was reduced in the ileal brush border of patients with diarrhea.

257 d gamma-secretase activity were found in the brush border of proximal kidney tubules where megalin is

258 b2 (p = 0.006) were significantly reduced in brush border of syncytiotrophoblast of infected placenta

259 e neonatal Fc receptor (FcRn) located at the brush border of the apical membrane has been implicated

260 , FcRn is expressed on the podocytes and the brush border of the proximal tubular epithelium.

261 CFEX are localized to and maintained in the brush border of the proximal tubule are largely unknown.

262 th increases in the length and regularity of brush borders of epithelial cells as seen with the elect

263 ction to target a disaccharidase to rafts in brush borders of intestinal mammalian cells.

264 glycoprotein and glycolipid receptors on the brush borders of piglet enterocytes.

265 otein and a sulfatide receptor on intestinal brush borders of piglets.

266 Villin 1, a protein typically found in the brush borders of proximal tubular cells, has been detect

267 zes mainly to the podocyte cell membrane and brush borders of the proximal tubule cells.

268 segment 3 cells, but was not detected in the brush border or basolateral membranes.

269 lar membrane morphology, distinct changes in brush-border organization, loss of numerous cytoskeletal

270 The susceptibility to the brush-border peptidases and route of transepithelial tra

271 n enterocytes by 1 hour, translocated to the brush border, preceded ulceration and vascular protein l

272 croclimate overlying the duodenal enterocyte brush border protects the mucosa from luminal acid.

273 Here we demonstrate that the brush border protein Myosin Ia (MYO1A) is important for

274 and demonstrate that the loss of structural brush border proteins involved in cell polarity are impo

275 Second, FasG recognizes specific intestinal brush border proteins that migrate on a sodium-dodecyl s

276 In the enterocyte brush border, protocadherin function requires a complex

277 The intestinal brush border provides an elaborate example with tightly

278 idney tubules resulted in loss of the tubule brush border, reduced GFR, pericardial edema, and increa

279 aling pathways, are also concentrated in the brush border region of PTE and are present in megalin-ex

280 it, Galphai3, has also been localized to the brush border region of PTE.

281 thrin-coated pits (CCPs) and vesicles in the brush border region.

282 Brush-border sucrase-isomaltase (SI) activity is complem

283 y expressed in the small intestine along the brush border, suggesting that the neutral ceramidase may

284 heir association with the negatively charged brush border surfaces.

285 Preliminary characterization of the brush border target antigen excluded megalin, CD10, and

286 i, but only lactaderin-C2 expression reduced brush border targeting of Myo1a-TH1.

287 The polarized enterocytes developed brush borders, tight junctions and desmosomes, and goble

288 his case, NHERF2 directly binds cGKII in the brush border to form an NHE3 complex, with cGKII also as

289 hat are characterized by an elaborate apical brush border to increase transport efficiency.

290 ld-type mice had increased expression of the brush border transporter Dmt1.

291 l proteins involved in iron transport: DMT1 (brush border transporter of ferrous iron) in the mk/mk m

292 icularly in the developing glomeruli and non-brush border tubules in the embryonic day 20 and newborn

293 at the brush border and although, as in the brush border, uptake is sodium independent, it is less s

294 Brush border vesicle uptake of L-tryptophan is substanti

295 Physiological studies using brush border vesicles and perfused tubules have indicate

296 and purified fimbriae to porcine enterocyte brush border vesicles and purified K88 receptors, respec

297 bacterial adherence assays by using porcine brush border vesicles that are specific to either the K8

298 y to the intermicrovillar microdomain of the brush border, was enriched in the dense vesicles, implic

299 ring tubule morphogenesis, localizing to the brush border whenever the tissue is transport competent.

300 C-1 adhered specifically to small intestinal brush borders, with both sialic acid and beta-galactosyl