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1 otonic fluid transport across the intestinal brush border membrane.
2 asolateral membrane and NHE2 and NHE3 on the brush border membrane.
3 ase of the GLUT2/SGLT-1 protein ratio in the brush border membrane.
4 sodium-phosphate cotransporter Npt2a at the brush border membrane.
5 reductions in NPT2a expression in the renal brush border membrane.
6 and protein levels of NHE3 and SGLT1 in the brush border membrane.
7 e fraction, of which 40-50% was localized to brush border membrane.
8 water transport across the human intestinal brush border membrane.
9 hanced AQP1 abundance in the proximal tubule brush border membrane.
10 bolic turnover rate, or translocation to the brush-border membrane.
11 d activation and recruitment of GLUT2 to the brush-border membrane.
12 expressed on the apical domain of the ileal brush-border membrane.
13 Na(+)/H(+) exchanger isoform, NHE3, in renal brush border membranes.
14 ced binding and no binding, respectively, to brush border membranes.
15 ular, the insertion of the toxin into insect brush border membranes.
16 by uptake of ferrous Fe(II) iron across the brush border membrane and culminates in transfer of the
17 B1 phenylalanine permitted it to bind to the brush border membrane and greatly enhanced its hypoglyce
18 an enzyme that is anchored to the intestinal brush border membrane and is expressed by the glutamate
19 eficiency, the enzyme fails to anchor in the brush border membrane and so is secreted into the lumen,
20 phosphorylation of PI 3-kinase in both ileal brush border membranes and Caco-2/NHE3 cells, suggesting
22 expressed in mammalian kidney and intestinal brush border membranes and in leukocytes and certain can
23 y 1,120 kDa that bound to solubilized insect brush border membranes and induced pore formation in bla
24 onide binds specifically to a single site in brush border membranes and to human embryonic kidney 293
25 the expression of iron transport in duodenal brush-border membrane and an enhanced electrical driving
27 -Reg variant that downregulates SGLT1 in the brush-border membrane at high luminal glucose concentrat
28 tered KBrO(3) on redox status and enzymes of brush border membrane (BBM) and carbohydrate metabolism
29 lotting revealed a 56 +/- 6% decrease in the brush border membrane (BBM) expression of mURAT1 in NHER
31 or (EGF) stimulation of Na absorption by the brush border membrane (BBM) Na(+)/H(+) exchanger NHE3.
32 chronically inflamed rabbit small intestine, brush border membrane (BBM) Na-glutamine co-transport is
33 rized by a rapid adaptive increase in apical brush border membrane (BBM) Na-Pi cotransport activity a
36 , and ezrin, was decreased in the intestinal brush border membrane (BBM) of mice with streptozotocin-
37 nzyme activities of cortical homogenates and brush border membrane (BBM) preparations documented litt
40 se from the proximal tubule lumen across the brush border membrane (BBM) via a sodium-dependent trans
42 nance of active ERM proteins at the cortical brush border membranes (BBM) of polarized epithelia.
43 all intestinal glucose absorption across the brush-border membrane (BBM) via SGLT1 and GLUT2 were ana
44 hen this toxin is associated with intestinal brush border membrane (BBMs), it was shown that radiolab
45 s proposing that CPE inserts into intestinal brush border membranes (BBMs) when this toxin is localiz
46 e presence of this regulatory factor in both brush-border membranes (BBMs) and basolateral membranes
47 t helices alpha-4 or alpha-6 insert into the brush border membranes because of their hydrophobic natu
48 are transported across the ileal enterocyte brush border membrane by the well characterized apical s
49 actose are transported across the intestinal brush-border membrane by the Na+/glucose cotransporter,
50 Caco-2BBe cell monolayers with expression of brush border membrane components and lectin binding site
51 single nephrotoxic dose of KBrO(3) inhibits brush border membrane enzymes, induces oxidative stress
53 ressed greater specific enzyme activities in brush border membrane fractions than the cell clone.
54 that NHE3 protein expression was greater in brush border membranes from acidotic compared with contr
55 lly all measured Na+-H+ exchange activity in brush border membranes from control and acidotic rats is
56 e, ERM proteins are significantly reduced in brush-border membranes from kidney and small intestine.
57 e across the liver sinusoidal or ileal/renal brush border membrane have been identified and share app
58 ponsible for transport across the intestinal brush border membrane; however, the carrier(s) responsib
59 ERF1 is specifically localized at the apical brush-border membrane in intestinal epithelial cells and
60 ce expression of AQP1 in the proximal tubule brush border membrane is regulated in response to flow.
61 eduction in the transporter protein in renal brush-border membranes isolated from the mutant mice.
62 al gene products used to induce this complex brush border membrane lesion and diarrhoeal disease star
63 s formed from the raft lipid mixture or from brush border membrane lipids an array of more ordered an
65 study were to estimate the fraction of renal brush border membrane Na+-H+ exchange activity mediated
66 earlier studies on phlorizin binding to the brush border membrane of duodenal biopsy specimens from
69 transport process for intact peptides in the brush border membrane of intestinal and renal absorptive
70 by increasing NHE3 protein abundance at the brush border membrane of intestinal epithelial cells.
71 her show that native ABCG2 is located in the brush border membrane of kidney proximal tubule cells, w
73 logue of human SLC26A6, was localized to the brush border membrane of proximal tubule cells and was d
75 ed the dynamic translocation of GLUT2 to the brush border membrane of RPTCs, and reduced glucose reab
76 rption is mediated by SGLT1 localized in the brush border membrane of small intestinal enterocytes, i
79 ctivity of a putative sterol permease in the brush border membrane of the enterocyte that actively fa
81 analysis, the kinase is concentrated in the brush border membranes of epithelial cells, throughout t
82 the membrane proteinase meprin beta found in brush border membranes of kidney and small intestine.
83 7, inhibited (37.6 +/- 4.7%) NHE activity in brush border membranes of normotensive F2s (systolic blo
85 -glutamyl transpeptidase (GGT) on the apical brush-border membrane of 786-O proximal tubule cells wit
86 tein was iron regulated and localized to the brush-border membrane of duodenal enterocytes in iron de
87 rane reductases, was highly expressed in the brush-border membrane of duodenal enterocytes, and induc
88 f the visceral endoderm and localized to the brush-border membrane of extraembryonic endodermal cells
90 mino acid transport function measured at the brush-border membrane of intact intestinal epithelia res
91 Ac-CP-2 is expressed by the parasite in the brush-border membrane of its alimentary canal, and anti-
92 l role in folate transport across the apical brush-border membrane of the proximal small intestine es
94 These receptors are detected in intestinal brush border membrane preparations from pigs with adhesi
95 preserved for extended periods when purified brush border membranes prepared in hypotonic media are l
98 of a 2.5-kilobase RNA transcript and 58-kDa brush-border membrane protein detected by folate-based a
100 of specific alkaline phosphatase activity in brush border membrane proteins from susceptible (YDK and
103 (Pept-1), which is located in the intestinal brush border membrane, provides a major mechanism for pr
104 rric iron is attributed to the presence of a brush-border membrane reductase activity that displays a
105 acilitative glucose transporter GLUT2 to the brush-border membrane; regulation involves a protein kin
107 ied a 45-kDa protein purified from rat renal brush border membrane that binds short single-stranded n
108 gomyelin or 2) natural lipids extracted from brush border membranes that are rich in sphingomyelin an
109 s further support the concept that the ileal brush border membrane transporter is regulated by the av
111 hanced binding affinity to gypsy moth midgut brush border membrane vesicles (BBMV) approximately 4-fo
112 g and oligomerization by western blots using brush border membrane vesicles (BBMV) from a strain of P
113 hibitory AgAPN2ta blocked Cry11Ba binding to brush border membrane vesicles (BBMV) of A. gambiae wher
114 kDa aminopeptidase N (APN) was isolated from brush border membrane vesicles (BBMV) of Anopheles quadr
119 ce the structure and stability of BT-R(1) on brush border membrane vesicles (BBMVs) prepared from M.
120 e and detected a 65 kDa species on a blot of brush border membrane vesicles (BBMVs) protein prepared
122 molitor), binding to receptors on T. molitor brush border membrane vesicles (Tm-BBMV) and insertion i
123 ibited phosphate transport in isolated renal brush border membrane vesicles and in cultured renal pro
124 unctional activity as Cl-oxalate exchange in brush border membrane vesicles and oxalate-stimulated vo
126 uced the rate of phosphate uptake into renal brush border membrane vesicles and the expression of NaP
127 d the level of binding of [(125)I]Cry11Ba to brush border membrane vesicles by 41%, a percentage comp
128 bits sodium-dependent phosphate transport in brush border membrane vesicles derived from hormone-trea
131 bodies (mAbs) to intact right-side-out renal brush border membrane vesicles in the absence of deterge
135 lls and I100 immunoprecipitates of rat ileal brush border membrane vesicles reveals that it has dipep
140 f functional (taurocholate uptake into crude brush border membrane vesicles) and molecular assays (No
141 es less binding to M. sexta and H. virescens brush border membrane vesicles, although the off-rate of
142 Finally, in assays with aphid gut-derived brush border membrane vesicles, binding of CP-P-GFP comp
149 ospholipids across two biological membranes: brush border membranes vesicles (BBMV) from rabbit intes
150 nion transport were determined in apical (or brush border) membrane vesicles isolated from bovine cho
151 to substantially reduced binding affinity to brush-border membrane vesicles (BBMV) prepared from the
152 with changes in Bt-toxin binding to sites in brush-border membrane vesicles of the larval midgut, and
154 f hypoxia on iron uptake across the duodenal brush-border membrane, we have measured the membrane pot
155 xchanger located on the intestinal and renal brush border membrane, where it functions in transepithe
156 s study, GUVs were assembled from rat kidney brush border membranes, which included the integral memb
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