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1 nduce the invasive infection associated with bubonic plague.
2 increased virulence and the emergence of the bubonic plague.
3 contributes to disease in the mouse model of bubonic plague.
4 hese MPs significantly increased survival of bubonic plague.
5 play an important role in the progression of bubonic plague.
6 el; however, it was attenuated in developing bubonic plague.
7 med to be important for the establishment of bubonic plague.
8 with antibody therapy in the mouse model of bubonic plague.
9 mination during pneumonic plague than during bubonic plague.
10 imic transmission by fleabite, leads only to bubonic plague.
11 rat closely resembled descriptions of human bubonic plague.
12 es, ranging from food-borne illnesses to the bubonic plague.
13 been proposed to provide protection against bubonic plague.
14 se that is an essential virulence factor for bubonic plague.
15 s ranging from gastrointestinal syndromes to Bubonic Plague.
16 o its current frequency can be attributed to bubonic plague.
17 s ranging from gastrointestinal syndromes to bubonic plague.
18 significantly attenuated in a mouse model of bubonic plague.
19 ifferent stages of the infectious process of bubonic plague.
20 f infection that mimics flea transmission of bubonic plague.
21 r of Yersinia pestis, the causative agent of bubonic plague.
23 ions to study bacterial dissemination during bubonic plague and compare this model with an s.c. inocu
24 antibody responses that protect mice against bubonic plague and pneumonic plague, suggesting that rV1
25 Yersinia pestis is the causative agent of bubonic plague and possesses a set of plasmid-encoded, s
26 tigations into the molecular pathogenesis of bubonic plague and the immune response to Y. pestis at d
27 f new vaccines to prevent naturally acquired bubonic plague and to study events at the vector-host in
28 exhibited an exceptional capacity to resist bubonic plague and used it to identify immune mechanisms
29 nt resulted in an atypical, subacute form of bubonic plague associated with extensive recruitment of
30 e clusters are present in the genomes of the bubonic plague bacillus Yersinia pestis and the human an
32 superfamily, is an effector produced by the bubonic plague bacterium, Yersinia pestis, that is essen
35 , spatial metapopulation model, we show that bubonic plague can persist in relatively small rodent po
36 use provides significant protection against bubonic plague caused by an F1- strain (C12) or against
40 ia pestis is transmitted by fleas and causes bubonic plague, characterized by severe local lymphadeni
41 the more biologically relevant i.d. model of bubonic plague differs significantly from the s.c. model
45 le of Yersinia pestis YopJ in a rat model of bubonic plague following intradermal infection with a fu
49 ntly caught ill-prepared societies off-guard-Bubonic plague in medieval times, AIDS in the 1980s, and
50 ugh flea-borne transmission usually leads to bubonic plague in mice, it can also lead to primary sept
51 adermal model, suggesting a role for YopM in bubonic plague, in which acute inflammation occurs soon
54 ional human-disease models, and propose that bubonic plague is driven by the dynamics of the disease
60 s of Yersinia pestis, the causative agent of bubonic plague, is the yersiniabactin (Ybt) siderophore-
61 lence of Yersinia pestis, causative agent of bubonic plague, is the yersiniabactin (Ybt) siderophore-
64 Brown Norway rat was recently described as a bubonic plague model that closely mimics human disease.
69 ults indicate that YopJ is not essential for bubonic plague pathogenesis, even after peripheral inocu
70 large-scale food-borne illnesses, dysentery, bubonic plague, secondary hospital infections, and sexua
73 indicating a more important role for RovA in bubonic plague than pneumonic plague or systemic infecti
74 past because it provided protection against (bubonic) plague; the mutation, called CCR5Delta32, is ch
75 ases explored include tuberculosis, leprosy, bubonic plague, typhoid, syphilis, endemic and epidemic
77 The Y. pestis Ail protein is an important bubonic plague virulence factor that inhibits the innate
81 on at 8 LD50 when tested in a mouse model of bubonic plague, with infection by 10/20 of the aforement
82 LcrV in the passive transfer of immunity to bubonic plague, with multiple neutralizing epitopes in L
83 y to control malaria, typhus, body lice, and bubonic plague worldwide, until countries began restrict
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