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1 ocytes, urinary epithelial cells (UECs), and buccal mucosa.
2 tion occurred across inflamed but not normal buccal mucosa.
3 lesions, but also in grossly normal adjacent buccal mucosa.
4 rboring the mutation in her blood, skin, and buccal mucosa.
5 s of the surface of the dorsal tongue or the buccal mucosa.
6 the superficial squamous cell layers of the buccal mucosa.
7 connective tissue of the facial flap to the buccal mucosa.
8 e skin biopsy, as well as from the patient's buccal mucosa.
9 ne were initiated at three sites through the buccal mucosa: 1) 6 mm apical to the cemento-enamel junc
11 and, surprisingly, also in normal-appearing buccal mucosa adjacent to HNSCC (see the related article
14 major salivary glands, tongue, gingiva, and buccal mucosa and detected beta-defensin peptides in sal
15 to have tongue malignancy (82%) followed by buccal mucosa and gingivobuccal sulcus malignancy (18%).
17 the m.14487T>C mutation in his blood, urine, buccal mucosa, and hair follicle DNA samples, while the
18 in plaque, others in keratinized gingiva or buccal mucosa, and some oligotypes were characteristic o
20 amples from the tongue, tonsils, throat, and buccal mucosa; and stimulated and unstimulated saliva.
22 s, we have investigated the potential of the buccal mucosa as an alternative delivery route for ShK b
23 ening sites included the forehead, nostrils, buccal mucosa, axilla, antecubital fossa, groin, and toe
24 ted AKT (pAKT) in 15 paired tumor, skin, and buccal mucosa biopsies (at baseline and after 1 month of
25 om LL explants compared to explants from the buccal mucosa (BM), HP, and transition zone of the lower
26 objective was to monitor lycopene changes in buccal mucosa cells (BMCs) in response to 3 vehicles for
27 ion protein Cx43 were markedly diminished in buccal mucosa cells from arrhythmogenic cardiomyopathy p
29 esmosomal protein desmoplakin was reduced in buccal mucosa cells from patients with mutations in DSP,
30 mosomal protein plakophilin-1 was reduced in buccal mucosa cells in patients with PKP2 mutations but
31 Inhibition of prelamin A farnesylation in buccal mucosa cells of patients treated with SCH66336 wa
32 , and 15 wk and 2 mo after the study, serum, buccal mucosa cells, and adipose tissue were analyzed fo
33 By comparing the DNA from leukemic cells to buccal mucosa cells, LOH was detected with markers D5S47
37 including decreases in microcirculations of buccal mucosa, cerebral microvascular flow was preserved
40 aps more germane to epithelial malignancies, buccal mucosa have provided surrogate tissues that allow
42 ups based on similar community compositions: buccal mucosa, keratinized gingiva, hard palate; saliva,
43 (+) cells equally distributed throughout the buccal mucosa of OPC(-) persons (HIV(-) or HIV(+)), irre
45 of selegiline, which is absorbed through the buccal mucosa producing higher plasma levels of selegili
47 th the best sensitivity were the leg and the buccal mucosa, respectively (82.6% and 52.2%; P = 0.003)
50 f fluorescent 5-Fam-ShK to untreated porcine buccal mucosa, there was no detectable peptide in the re
51 f the attached gingiva, alveolar mucosa, and buccal mucosa to gain insight into human tissue performa
52 f PBD-1 in the dorsal tongue surface and the buccal mucosa was estimated at 20 to 100 micrograms/ml.
53 nactivation patterns in peripheral blood and buccal mucosa were compared between monochorionic MZ (MC
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