ライフサイエンスコーパス: buffer capacity

1 flux, pH is altered in a medium with finite buffer capacity.

2 hat M(2) currents can be limited by external buffer capacity.

3 - 0.97 and 2.24 +/- 0.34 slykes of the total buffer capacity.

4 zation by strain Z6 in media with increasing buffer capacity.

5 governed by the mobile fraction of intrinsic buffering capacity.

6 als, consistent with a decrease in glutamate buffering capacity.

7 of calcium, increasing the lysosomal Ca(2+) buffering capacity.

8 chronic hyperglycemia may alter the brain's buffering capacity.

9 and that it increases mitochondrial calcium-buffering capacity.

10 o millimolar dithionite did not increase the buffering capacity.

11 n juxtanuclear ones despite their comparable buffering capacity.

12 portant for matching local energy and Ca(2+) buffering capacity.

13 ow level of atmospheric CO2 and a high ocean buffering capacity.

14 e, limited hydraulic conductivity, and redox buffering capacity.

15 due to blunted endoplasmic reticulum Ca(2+) buffering capacity.

16 gentler droplet environment, despite its low buffering capacity.

17 a from Wld(S) mice exhibited enhanced Ca(2+) buffering capacity.

18 mitochondria, potentiating a loss of Ca(2+) buffering capacity.

19 roducible LC separation due to an acceptable buffering capacity.

20 xia, resulting in substantial loss of Ca(2+) buffering capacity.

21 thylamine or piperazine groups and potential buffering capacity.

22 ls (CGCs) while varying the extracellular pH buffering capacity.

23 be, including a proposed role of cytoplasmic buffering capacity.

24 ent manner that is correlated with predicted buffering capacity.

25 gene are weak predictors of the duplicate's buffering capacity.

26 ]SR, not to alterations in SR volume or Ca2+ buffering capacity.

27 used to obtain estimates of cellular Ca(2+) buffering capacities.

28 ator loading yielded estimates of endogenous buffering capacity (44-80) and peak [Ca(2+)] changes at

29 at the site, and the depletion of Al mineral buffering capacity after approximately 5 years.

30 t to the lung, nor a change in eosinophil pH-buffering capacity, allergen-challenged chimeric mice th

31 The very high buffering capacity allows the Mediterranean Sea waters t

32 t SOD1-mediated loss of mitochondrial Ca(2+) buffering capacity, altered mitochondrial morphology, mo

33 specific set of parameters determines model buffer capacity and buffer function, and individual diff

34 ructure, leading to loss of the interstitial buffer capacity and disproportionate interstitial fluid

35 trongly influenced by the electrolyte pH and buffer capacity and not limited to BDD or formaldehyde,

36 and comprehensiveness: proton (H(+)) efflux, buffer capacity and the contributions of glycolytic (L)

37 ondria exhibit reduced mitochondrial calcium buffering capacity and are highly sensitive to mitochond

38 oes not alter cytosolic pH but diminishes pH buffering capacity and causes poor growth at low pH in a

39 Additionally, they have an increased calcium buffering capacity and generate fewer mitochondrial reac

40 n significantly reduced saliva flow rate and buffering capacity and increased mucus acidity.

41 Tris-borate has a high-buffering capacity and is therefore widely used in elect

42 lled chemical weathering, which controls the buffering capacity and mineral content of receiving stre

43 Proteins differ widely in buffering capacity and pI and therefore the same PTMs ma

44 lusion, our findings propose that the proton buffering capacity and protein loading in PLGA MS can be

45 The buffering capacity and response of 2', 7'-bis(2-carboxye

46 sients because of a higher endogenous Ca(2+)-buffering capacity and significantly higher rates of Ca(

47 ructure with the potential to extend stretch-buffering capacity and support a revised model for the f

48 ished physiological values for intracellular buffering capacity and the permeability ratio of lactic

49 tablish a close link between nuclear calcium buffering capacity and the transcription of genes that d

50 Measurements of H(+) leak rates, buffer capacities, and estimates of surface areas and vo

51 iation in calcium influx density, endogenous buffer capacity, and calcium extrusion density contribut

52 the micelles mostly maintained their proton buffering capacity, and consequently, prevented or delay

53 ter absorption, nutrient inflow, and luminal buffering capacity, and generates testable predictions o

54 emperature sensitivity of pHi, intracellular buffering capacity, and the activity of sarcolemmal acid

55 inc ions, the thresholds of compromised zinc buffering capacity, and the mechanism of cellular homeos

56 pproximately 15 microM), high cytosolic Ca2+ buffering capacity, and the spatial separation of CRUs h

57 rmined during stimulation from pH and tissue buffer capacity, as well as the oxidative phosphorylatio

58 is a commonly used additive, despite its low buffering capacity at pH 7.

59 malian vertebrates, enrichment of retinal pH buffering capacity attenuates horizontal cell feedback,

60 That is, the conventional buffer capacity-based I(H) estimation must also take int

61 consumption) was used to estimate cytosolic buffer capacity (beta).

62 By assuming accepted values of intracellular buffering capacity (beta(i)), intracellular acidificatio

63 romal compartment are relatively low and the buffering capacity (beta) for protons of the lumen is re

64 Increasing the cytosolic Ca(2+) buffer capacity by internal perfusion with 1 mM EGTA lim

65 APTA into the red cells increased their Ca2+ buffering capacity by 300-600 mumol (340 g Hb)-1.

66 Moreover, exposure to Fe(II) increased the buffering capacity by 44%, while exposure to millimolar

67 ing CaMBP4 or increasing the nuclear calcium buffering capacity by means of expression of a nuclear t

68 ased sensitivity likely results from reduced buffering capacity by non-DA cells, leading to more PQ(+

69 f RVH rats, and (2) that a blunted ER Ca(2+) buffering capacity contributes to the altered NMDAR-Delt

70 are consistent with the hypothesis that Ca2+ buffering capacity contributes to the control of intrins

71 of MON and SAL was observed in aerobic, low-buffer capacity culture series as a result of abiotic ac

72 main transformation process in aerobic, high-buffer capacity culture series.

73 plify electrode addressing and to counteract buffering capacity depletion arising from the high elect

74 The cytosolic Ca2+ buffering capacity, determined as the ratio of the expec

75 Ryanodine also had little effect on the buffering capacity during 800-1000 ms of the depolarizin

76 ewer striatal dopamine terminals and reduced buffering capacity; fluctuations in plasma levodopa conc

77 s also indicated that algae have substantial buffering capacity for free heavy metals in their cytoso

78 ll was dialysed with a solution of increased buffering capacity for protons.

79 nsistent with alkalinization, increasing the buffering capacity from 3 to 24 mm HEPES at pH 7.4 resul

80 tween intermittent contractions and inherent buffering capacity had minimal impact on predicted fatig

81 tenuation was a function of cytoplasmic Ca2+ buffering capacity; i.e., loading increasing concentrati

82 A decrease in mitochondrial Ca2+ buffering capacity in cells affected by these lysosomal

83 cell patch-clamp dialysis and quantified its buffering capacity in murine hippocampal slices using co

84 m human brain that suggested loss of calcium buffering capacity in neurons correlated with areas of n

85 ection to achieve balanced diets and provide buffering capacity in the face of variable food quality.

86 The model also predicts that an increase in buffering capacity in the nucleoplasm would cause a peri

87 orce for equalization is a higher Ca(2+)-CaM-buffering capacity in the nucleus compared with the cyto

88 Increasing pH buffering capacity in the pipette solution with 40 mm HE

89 For example, buffering capacity increases to 23% amongst highly expre

90 Thus, we can realize a buffering-capacity independent monitoring of changes in

91 r has proven challenging because of a signal buffering capacity inherent in the functionally relevant

92 ticular, it suggests that the Class 1 Ca(2+) buffering capacity is auto-regulated by the rate at whic

93 l mg(-1) Chl of the total sequestered domain buffering capacity is contributed by lysines with anomol

94 Mitochondria also buffer Ca(2+), and their buffering capacity is dependent on DeltaPsi Here, we cha

95 that increasing neuronal mitochondrial Ca2+-buffering capacity is not beneficial in the R6/2 mouse m

96 However, a reduction in the buffer capacity leads to large deviations from the expec

97 activation, loss of glutamate and potassium buffering capacity, loss of astrocyte coupling, and chan

98 This decreased bicarbonate buffering capacity may contribute to the increased vulne

99 Ca2+ release mechanism, had little effect on buffering capacity measured over the first 200 ms of the

100 gentless pH control, acidity/alkalinity, and buffer capacity measurements in very small samples of bi

101 Together with ion substitution and buffer capacity measurements, we conclude that Cl(-) tra

102 This buffering capacity might explain the observed difference

103 gle biological cells to assess intracellular buffer capacities of different metal ions, such as Ca(II

104 unction at nonphysiological pH (at which the buffer capacity of biological samples is small) makes it

105 extracellular L(-) increased with increasing buffer capacity of extracellular compartment, (iii) the

106 solution at S/Fe = 0.112 is due to the redox buffer capacity of FeS, which is evidenced by the parall

107 reducing electrode, showing the insufficient buffer capacity of PBS to maintain a stable pH at the gi

108 characterized by sequestered domains with a buffering capacity of approximately 150 nmol H(+) mg(-1)

109 ermore, under normoxic conditions, glutamate-buffering capacity of astrocytes is increased upon cocul

110 ations include evidence that both the Ca(2+) buffering capacity of calsequestrin in solution and that

111 s in this pathway could compromise the redox buffering capacity of cells, which may in turn be relate

112 Indeed, increasing the buffering capacity of culture medium or frequency of med

113 y, and adjusting the serum concentration and buffering capacity of culture medium.

114 These findings highlight the buffering capacity of developmental systems, allowing ma

115 The buffering capacity of duplicates appears to be independe

116 g/lysis were accounted for by the greater H+ buffering capacity of endosomes containing PEI or PAM ve

117 ng evidence suggests that changes in the ion buffering capacity of glial cells can give rise to neuro

118 (+)-H+ antiport, intracellular pH (pHi), and buffering capacity of hepatocytes obtained from rats aft

119 The pHi and the buffering capacity of hepatocytes were not different in

120 ty is important for cell uptake and that the buffering capacity of histidine facilitates endosomal me

121 eir virulence varied depending on the pH and buffering capacity of host tissue.

122 er, in a physiological situation, the Ca(2+)-buffering capacity of mitochondria was found not to be e

123 parison, diminishment of the endogenous Ca2+ buffering capacity of nerve endings by treatment with th

124 that may play a key role in setting the Ca2+ buffering capacity of Purkinje cells.

125 -producing S. gordonii is dominant while the buffering capacity of saliva is valid ( approximately pH

126 vironment can vary depending on the diet and buffering capacity of saliva, materials testing in const

127 ification in the surface ocean decreases the buffering capacity of seawater for CO2, whilst photosynt

128 f biodiesel wastewater and by increasing the buffering capacity of the anode medium.

129 Here we show that the calcium buffering capacity of the cell nucleus in mouse hippocam

130 f the system on its pH environment (e.g. the buffering capacity of the colon) and the pattern of inco

131 Adding 100 microM fluo-3, which doubles the buffering capacity of the cytoplasm, reduces peak averag

132 Simulation results also suggest that the buffering capacity of the ER, and not restricted diffusi

133 n a more significant optimality in the error-buffering capacity of the genetic code when compared to

134 obal temperature change based on the thermal buffering capacity of the germination phenotype.

135 antitative imaging experiments show that the buffering capacity of the nerve terminal is markedly low

136 in terrestrial biogeochemical cycles, in the buffering capacity of the oceans, and in the containment

137 urthermore, the decrease in C(c) reduces the buffering capacity of the other actin monomer binding pr

138 ion efficiency, presumably by increasing the buffering capacity of the polymer.

139 e second is an exploitation of the extensive buffering capacity of the turtle's shell and skeleton to

140 The mechanical stability and buffering capacity of this hydrogel can be adjusted by w

141 Furthermore, we show that the buffering capacity of torsinA is greatly diminished by t

142 We propose that the enhanced Ca(2+) buffering capacity of Wld(S+) mitochondria leads to incr

143 n the bacterial cells and facilitated by the buffering capacity of yogurt.

144 Manipulating Hsp90's buffering capacity offers a tool for harnessing cryptic

145 high-capacity immobile Ca(2+) buffer, with a buffer capacity on the order of 1000 and appropriate aff

146 ardium, and the role of extracellular pH and buffer capacity on this relationship.

147 In addition, increased cytosolic pH buffering capacity or elevated [Ca(2+)](i) reduces the r

148 Together they lead to a poor acid buffering capacity, severe acidification and increased c

149 of absolute Ca(2+) concentration and of rod buffering capacity suggest that the slower components of

150 ering or increasing the intracellular Ca(2+) buffering capacity, suggesting that this current was reg

151 alized structures with low endogenous Ca(2+) buffer capacity that allows large and extremely rapid [C

152 to be associated with the intracellular Ca2+ buffering capacity that could regulate the sensitivity t

153 d be facilitated by maximizing intracellular buffering capacity through the presence of HCO(3)(-), HC

154 in releasing caged Ca(2+) but also increases buffer capacity to reduce [Ca(2+)](i) rises caused by Ca

155 ency inner hair cells must have a low Ca(2+) buffer capacity to sustain exocytosis, thus making them

156 ribution of the loss of mitochondrial Ca(2+)-buffering capacity to disease mechanism(s) by eliminatin

157 s of this neurocircuitry maintain sufficient buffering capacity to resist an effect on motivated beha

158 ng a mechanism to spatially match energy and buffering capacity to the demands imposed by transport.

159 In sharp contrast, enrichment of retinal pH-buffering capacity, to attenuate negative feedback from

160 contribution to glutathione-dependent redox-buffering capacity under ex vivo conditions in brain cel

161 The non-Pi, non-CO2 buffer capacity was calculated to be 27.0 +/- 6.2 slykes

162 Cytosolic buffer capacity was quantified for the first time non-in

163 BCs was enhanced when the intraterminal Ca2+ buffer capacity was reduced.

164 calcium, phosphate, acetate, propionate, and buffer capacity were not affected by the different amoun

165 Increasing retinal buffering capacity with HEPES attenuated the LM-ON surro

166 Enrichment of the extracellular buffering capacity with HEPES selectively attenuates sur

167 Reducing HSP90's buffering capacity with inhibitors or febrile temperatur

168 d were blocked by increasing cellular Ca(2+) buffering capacity with Quin-2.

169 (approximately 11 mM) of the total intrinsic buffering capacity within the myocyte (the other half be