ライフサイエンスコーパス: bulimia

1 with 12 female volunteers who had never had bulimia.

2 OCD symptoms persist after recovery from bulimia.

3 elated and contribute to the pathogenesis of bulimia.

4 mptoms persist after long-term recovery from bulimia.

5 r between alcohol dependence and anorexia or bulimia.

6 se of procedural justice than did those with bulimia.

7 examined the benefits of two treatments for bulimia: 1) fluoxetine, an antidepressant medication, an

8 ine dependence but not alcohol dependence or bulimia; (2) greater number of episodes, more impairment

9 l disorders, the highest ORs being seen with bulimia and alcohol and other drug dependence.

10 therapy may also be applicable to adolescent bulimia and binge eating.

11 Women with bulimia and those who had recovered from bulimia had sim

12 such as neurodegenerative diseases, obesity, bulimia, and anorexia nervosa.

13 ssions of OCD, the binge eating behaviors of bulimia, and the self-starvation of anorexia.

14 olics after controlling for other disorders, bulimia did occur at a greater than expected rate.

15 subscales of the Eating Disorders Inventory (bulimia, drive for thinness, maturity fears, perfectioni

16 lting positive feedback cycle escalates into bulimia for many, and anorexia in a few.

17 ervosa, 29 women who had been recovered from bulimia for more than 1 year, and 19 healthy female comp

18 ith bulimia and those who had recovered from bulimia had similar Yale-Brown scale scores and endorsed

19 he Yale-Brown scale scores of the women with bulimia (mean = 13.1, SD = 10.6) and those who had recov

20 SD = 10.6) and those who had recovered from bulimia (mean = 7.9, SD = 7.0) were significantly higher

21 ong, positive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confi

22 We enrolled patients with severe bulimia nervosa (at least seven coupled binge/vomit epis

23 hotherapy in reducing behavioral symptoms of bulimia nervosa (binge eating and vomiting).

24 on in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

25 Anorexia nervosa (AN) and bulimia nervosa (BN) are disorders characterized by abno

26 ED data are presented here and compared with bulimia nervosa (BN) data in the World Health Organizati

27 Bulimia nervosa (BN) has been associated with dysregulat

28 Women with bulimia nervosa (BN) have disturbances of mood and behav

29 Bulimia nervosa (BN) is strongly familial, and additive

30 t as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Health

31 trol circuits in adolescents and adults with bulimia nervosa (BN), but less is known about the micros

32 disorders, such as anorexia nervosa (AN) and bulimia nervosa (BN), have genetic and environmental und

33 in adolescent and adult female subjects with bulimia nervosa (BN).

34 =13.9), significantly longer than for either bulimia nervosa (mean=5.8 years, SD=9.1) or anorexia ner

35 either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110) between January 1, 1987, and D

36 ficantly diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemody

37 as there were about anorexia nervosa and/or bulimia nervosa (N=169).

38 diagnoses of binge eating disorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

39 ing-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=

40 Women diagnosed with either anorexia or bulimia nervosa (N=246) completed prospective evaluation

41 with restricting anorexia nervosa (n=26) or bulimia nervosa (n=47), control women (n=44), and first-

42 5 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwis

43 to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criteria).

44 .38-9.54] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.8

45 inin release compared with participants with bulimia nervosa (t(76.44) = 2.51; P = .01) and did not d

46 Nine women who had recovered from bulimia nervosa (they had no episodes of binge eating or

47 ervosa among women might be up to 4%, and of bulimia nervosa 2%.

48 Eight women with bulimia nervosa and 10 age- and weight-matched control s

49 d, medication-free, normal-weight women with bulimia nervosa and 14 age-matched healthy female contro

50 ndent response in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjec

51 Eight women with bulimia nervosa and 8 female controls underwent brain MR

52 found empirical support for conceptualizing bulimia nervosa and binge eating disorder as discrete sy

53 y remain the most established treatments for bulimia nervosa and binge eating disorder, with stepped-

54 utrient and food group intakes of women with bulimia nervosa and binge-eating disorder during pregnan

55 to measure the prevalence and correlates of bulimia nervosa and bulimic behaviors in a sample of und

56 nal distress compared with participants with bulimia nervosa and controls.

57 ialization affects the risk for anorexia and bulimia nervosa and disordered eating in members of oppo

58 ality ratios were significantly elevated for bulimia nervosa and eating disorder not otherwise specif

59 ndardized mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specif

60 compare serotonin function in patients with bulimia nervosa and healthy controls.

61 ell-validated disorders anorexia nervosa and bulimia nervosa and likely represents a stable syndrome.

62 n appear less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating

63 cles published about anorexia nervosa and/or bulimia nervosa and the number of articles published abo

64 t while menstrual irregularities are common, bulimia nervosa appears to have little impact on later a

65 ecovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is

66 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

67 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (

68 Anorexia nervosa and bulimia nervosa are common problems facing adolescents a

69 Both anorexia nervosa and bulimia nervosa are familial.

70 -based treatment trials for adolescents with bulimia nervosa are largely absent.

71 These results support the validity of bulimia nervosa as a diagnostic category that is distinc

72 ht to investigate the predictive validity of bulimia nervosa as a diagnostic category.

73 ivate to the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

74 Women with bulimia nervosa before and during pregnancy and those wi

75 Women with the diagnosis of bulimia nervosa between 1981 and 1987 who participated i

76 itudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa

77 er mistakes was associated with anorexia and bulimia nervosa but not with other psychiatric disorders

78 ubtypes over time; one-third crossed over to bulimia nervosa but were likely to relapse into anorexia

79 ed in participants with purging disorder and bulimia nervosa compared with controls but did not diffe

80 ognize hunger signals, whereas overeating in bulimia nervosa could represent an exaggerated perceptio

81 women who continue to meet full criteria for bulimia nervosa declines as the duration of follow-up in

82 zygosity; the prevalence of DSM-IV or broad bulimia nervosa did not differ in women from opposite- v

83 essary to resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activatio

84 tine versus placebo in preventing relapse of bulimia nervosa during a 52-week period after successful

85 howed symptoms of either anorexia nervosa or bulimia nervosa during pregnancy had a higher frequency

86 ple of individuals with anorexia nervosa and bulimia nervosa from the Price Foundation collaborative

87 a nervosa groups and on the left side in the bulimia nervosa group relative to the healthy comparison

88 educed in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to rewa

89 Women who had recovered from bulimia nervosa had a reduction of medial orbital fronta

90 mately 50% of women initially diagnosed with bulimia nervosa had fully recovered from their disorder,

91 In addition, women with bulimia nervosa had less activation in the lateral and a

92 As bulimia nervosa has been associated with a dysregulated

93 tions decrease binge eating in patients with bulimia nervosa has fueled interest in the hypothesis th

94 Recent research on Anorexia Nervosa and Bulimia Nervosa has yielded an increasingly detailed und

95 Two treatments for bulimia nervosa have emerged as having established effic

96 The treatment of patients with bulimia nervosa in a primary care setting is hampered by

97 haviours in patients with severe and chronic bulimia nervosa in a randomised, double-blind, placebo-c

98 g the usefulness of family interventions for bulimia nervosa in adolescents.

99 ervosa and possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern

100 on; and increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority

101 Bulimia nervosa in men was too rare to be assessed by zy

102 ta on outcome for individuals diagnosed with bulimia nervosa in order to better understand long-term

103 whether treatments known to be effective for bulimia nervosa in specialized treatment centers can be

104 due to all causes of death for subjects with bulimia nervosa in these studies was 0.3% (seven deaths

105 Bulimia nervosa is a common eating disorder in adolescen

106 The etiology of bulimia nervosa is complex, with biologic, psychological

107 acy of fluoxetine in the acute management of bulimia nervosa is well established; however, few contro

108 ver" among the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity o

109 r of eating that appears to be distinct from bulimia nervosa on subjective and physiological response

110 th binge eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purgin

111 ften useful as adjuncts to psychotherapy for bulimia nervosa or binge-eating disorder; in the case of

112 acial/ethnic difference in the prevalence of bulimia nervosa or bulimic behaviors; 1.5% of the whites

113 y (CBT) guided self-care in adolescents with bulimia nervosa or eating disorder not otherwise specifi

114 Eighty-five adolescents with bulimia nervosa or eating disorder not otherwise specifi

115 were randomly assigned to family therapy for bulimia nervosa or individual CBT guided self-care suppo

116 isorders, 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial

117 The participants were 80 mothers with bulimia nervosa or similar eating disorder who were atte

118 A total of 120 women with bulimia nervosa participated in a randomized, placebo-co

119 mined menstrual and reproductive function in bulimia nervosa patients retrospectively, with 10-15-yea

120 Adolescents with and without bulimia nervosa performed similarly on the task.

121 rial was conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psycho

122 Long-term outcome for women diagnosed with bulimia nervosa remains unclear.

123 outcome studies of both anorexia nervosa and bulimia nervosa show that no significant differences exi

124 ompared with control subjects, patients with bulimia nervosa showed a blunting of postprandial cholec

125 More research on bulimia nervosa specific to adolescence is needed.

126 h higher in people with anorexia nervosa and bulimia nervosa than in a nonclinical group of women in

127 in engendering improvement in patients with bulimia nervosa than IPT.

128 lescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation

129 sover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR

130 of the studies linking anorexia nervosa and bulimia nervosa to substance use disorders suffer from p

131 Cognitive behavioural therapy for bulimia nervosa was assigned grade A because of the evid

132 al behavior therapy adapted for treatment of bulimia nervosa was associated with a promising decrease

133 Bulimia nervosa was more common than anorexia nervosa in

134 obands; the corresponding relative risks for bulimia nervosa were 4.2 and 4.4 for female relatives of

135 enty patients meeting DSM-III-R criteria for bulimia nervosa were allocated at random to 19 sessions

136 en partial syndromes of anorexia nervosa and bulimia nervosa were considered, relative risks fell by

137 tes of full and partial anorexia nervosa and bulimia nervosa were determined in first-degree relative

138 omen with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating

139 Antidepressants for bulimia nervosa were given grade B.

140 A total of 173 women with bulimia nervosa were interviewed an average of 11.5 year

141 ing female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a me

142 who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effect

143 Women with bulimia nervosa were unlikely to cross over to anorexia

144 th fluoxetine is useful for individuals with bulimia nervosa who do not respond to psychotherapy or r

145 Twenty-two patients with bulimia nervosa who had not responded to, or had relapse

146 e study examined relapse in 48 patients with bulimia nervosa who had responded to cognitive behaviora

147 y be a useful intervention for patients with bulimia nervosa who have not responded adequately to psy

148 d treatment with fluoxetine in patients with bulimia nervosa who responded to acute treatment with fl

149 formation exists on relapse in patients with bulimia nervosa who responded with complete abstinence f

150 194 women meeting the DSM-III-R criteria for bulimia nervosa who were treated with 18 sessions of man

151 of attrition and outcome in the treatment of bulimia nervosa with cognitive behavior therapy.

152 e recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2

153 orexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.

154 hout obsessive-compulsive features; and LC4, bulimia nervosa with self-induced vomiting as the sole f

155 (LC1) resembled restricting AN; LC2, AN and bulimia nervosa with the use of multiple methods of purg

156 Since bulimia nervosa's introduction to the psychiatric nomenc

157 (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses re

158 alized anxiety disorder, panic disorder, and bulimia nervosa), and four were "externalizing" (nicotin

159 a nervosa and higher in women recovered from bulimia nervosa).

160 male patients with eating disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 health

161 0 years after they appeared as patients with bulimia nervosa, 177 women (participation rate=79.7%) co

162 Thirty-one female patients with bulimia nervosa, 29 women who had been recovered from bu

163 s), 11% of this sample met full criteria for bulimia nervosa, and 0.6% met full criteria for anorexia

164 individuals with anorexia nervosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulim

165 tes were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherw

166 ering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating di

167 examined the prevalence of anorexia nervosa, bulimia nervosa, and binge eating disorder in a geograph

168 me prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.

169 DSM-IV classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

170 current classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

171 1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

172 y was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

173 Research into treatment has focused on bulimia nervosa, and evidence-based management of this d

174 ssion and co-twin risk for anorexia nervosa, bulimia nervosa, and obesity.

175 miological studies, short-term treatment for bulimia nervosa, and outcome in anorexia nervosa.

176 ree diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eating disorders.

177 g disorders, especially anorexia nervosa and bulimia nervosa, are more common among white women than

178 e four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disor

179 ipants with a diagnosis of anorexia nervosa, bulimia nervosa, binge eating disorder, or eating disord

180 ), attention-deficit/hyperactivity disorder, bulimia nervosa, cataplexy, dysthymic disorder, fibromya

181 rs, but not the women who had recovered from bulimia nervosa, had an age-related decline in 5-HT(2A)

182 Anorexia nervosa, but not bulimia nervosa, has one of the highest mortality rates

183 interventions are of established utility in bulimia nervosa, medications have no clear role in the t

184 a longitudinal study of anorexia nervosa and bulimia nervosa, now in its 12th year.

185 evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated wit

186 ot respond to cognitive behavior therapy for bulimia nervosa, potentially allowing early use of a sec

187 Patients who met DSM-IV criteria for bulimia nervosa, purging type, were assigned to single-b

188 For patients with bulimia nervosa, the fenfluramine-stimulated increase in

189 en substantial advances in the management of Bulimia Nervosa, the goal of offering effective treatmen

190 ed to reward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identi

191 h AN and related eating disorders, including bulimia nervosa, was performed, resulting in only modest

192 diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts

193 onwhites (n = 693) were classified as having bulimia nervosa, while 5.5% of the whites and 5.3% of th

194 ory of a desensitized dopaminergic system in bulimia nervosa.

195 patterns were found for anorexia nervosa and bulimia nervosa.

196 l of the brain mu-opioid receptor (mu-OR) in bulimia nervosa.

197 r the compulsive eating behavior observed in bulimia nervosa.

198 entified as a common feature of anorexia and bulimia nervosa.

199 ty factor for developing anorexia nervosa or bulimia nervosa.

200 h (1.3; 95% confidence interval, 0.0-7.2) in bulimia nervosa.

201 ome in women diagnosed as having anorexia or bulimia nervosa.

202 sfunction occurs frequently in patients with bulimia nervosa.

203 aminergic dysfunction in the pathogenesis of bulimia nervosa.

204 more inpatient treatment than did women with bulimia nervosa.

205 apy to decrease binge and purge behaviors in bulimia nervosa.

206 ies for relapse of susceptible patients with bulimia nervosa.

207 version of psychoanalytic psychotherapy for bulimia nervosa.

208 present in patients who have recovered from bulimia nervosa.

209 erations in subjects who have recovered from bulimia nervosa.

210 behavioral therapy (CBT) in the treatment of bulimia nervosa.

211 n the pathophysiology of the eating disorder bulimia nervosa.

212 he preferred psychotherapeutic treatment for bulimia nervosa.

213 st effective psychotherapeutic treatment for bulimia nervosa.

214 mptoms are common in people who are ill with bulimia nervosa.

215 ilial aggregation was independent of that of bulimia nervosa.

216 ilial vulnerability for anorexia nervosa and bulimia nervosa.

217 have been implicated in the neurobiology of bulimia nervosa.

218 on of psychiatric disorders for anorexia and bulimia nervosa.

219 independent of that of anorexia nervosa and bulimia nervosa.

220 arly 20% continued to meet full criteria for bulimia nervosa.

221 is the psychological treatment of choice for bulimia nervosa.

222 questions based on the DSM-III criteria for bulimia nervosa.

223 for males, especially for those who develop bulimia nervosa.

224 observed deficit in satiety in patients with bulimia nervosa.

225 of abnormal eating patterns in patients with bulimia nervosa.

226 0.2% of the males were classified as having bulimia nervosa.

227 current diagnostic criteria for anorexia or bulimia nervosa.

228 ervosa and may be efficacious for youth with bulimia nervosa.

229 oral and neural reward-effort integration in bulimia nervosa.

230 eating in anorexia nervosa and overeating in bulimia nervosa.

231 norexia nervosa and promising for adolescent bulimia nervosa.

232 oods and avoid weight gain that characterize bulimia nervosa.

233 and conflict resolution in adolescents with bulimia nervosa.

234 including the subtypes anorexia nervosa and bulimia nervosa.

235 focused on the treatment of adolescents with bulimia nervosa.

236 determine how purging disorder differs from bulimia nervosa.

237 49 females (0.30%), and 61 males (0.01%) had bulimia nervosa.

238 ng disorders, including anorexia nervosa and bulimia nervosa.

239 ns and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disor

240 han black women (N=4, 0.4%) met criteria for bulimia nervosa; binge eating disorder also was more com

241 e a patient they were currently treating for bulimia or anorexia.

242 d greater acceptability for adolescents with bulimia or eating disorder not otherwise specified.

243 er, the types of OCD symptoms experienced by bulimia patients do not vary dramatically with improveme

244 Persistent OCD symptoms after recovery from bulimia raise the possibility that these behaviors are t

245 Bulimia status was assessed using criteria from the Diag

246 ng Disorder Inventory drive for thinness and bulimia subscales and depressed mood.

247 on and was operationalized using the Revised Bulimia Test.

248 ptor binding in women who had recovered from bulimia-type AN but not restricting-type AN.

249 Women recovered from bulimia-type AN had significantly (P<.05) increased [11C

250 tively, for the alcohol-dependent women, and bulimia was observed in 1.35% of the alcoholic men.

251 Lifetime rates for anorexia and bulimia were 1.41% and 6.17%, respectively, for the alco

252 of the alcohol-dependent men; the rates for bulimia were 3.46% and 0.72%, respectively.

253 tric disorders, and symptoms of anorexia and bulimia were evaluated among alcohol-dependent probands,

254 a nervosa, and 293 with anorexia nervosa and bulimia were given the Structured Clinical Interview for

255 Treatments for bulimia yield extremely high relapse rates, but cognitiv