ライフサイエンスコーパス: bulimia nervosa

1 a nervosa and higher in women recovered from bulimia nervosa).

2 ory of a desensitized dopaminergic system in bulimia nervosa.

3 ty factor for developing anorexia nervosa or bulimia nervosa.

4 h (1.3; 95% confidence interval, 0.0-7.2) in bulimia nervosa.

5 ome in women diagnosed as having anorexia or bulimia nervosa.

6 aminergic dysfunction in the pathogenesis of bulimia nervosa.

7 sfunction occurs frequently in patients with bulimia nervosa.

8 more inpatient treatment than did women with bulimia nervosa.

9 apy to decrease binge and purge behaviors in bulimia nervosa.

10 version of psychoanalytic psychotherapy for bulimia nervosa.

11 ies for relapse of susceptible patients with bulimia nervosa.

12 present in patients who have recovered from bulimia nervosa.

13 erations in subjects who have recovered from bulimia nervosa.

14 behavioral therapy (CBT) in the treatment of bulimia nervosa.

15 n the pathophysiology of the eating disorder bulimia nervosa.

16 he preferred psychotherapeutic treatment for bulimia nervosa.

17 st effective psychotherapeutic treatment for bulimia nervosa.

18 mptoms are common in people who are ill with bulimia nervosa.

19 ilial aggregation was independent of that of bulimia nervosa.

20 ilial vulnerability for anorexia nervosa and bulimia nervosa.

21 have been implicated in the neurobiology of bulimia nervosa.

22 on of psychiatric disorders for anorexia and bulimia nervosa.

23 independent of that of anorexia nervosa and bulimia nervosa.

24 arly 20% continued to meet full criteria for bulimia nervosa.

25 is the psychological treatment of choice for bulimia nervosa.

26 questions based on the DSM-III criteria for bulimia nervosa.

27 for males, especially for those who develop bulimia nervosa.

28 observed deficit in satiety in patients with bulimia nervosa.

29 of abnormal eating patterns in patients with bulimia nervosa.

30 0.2% of the males were classified as having bulimia nervosa.

31 current diagnostic criteria for anorexia or bulimia nervosa.

32 oral and neural reward-effort integration in bulimia nervosa.

33 ervosa and may be efficacious for youth with bulimia nervosa.

34 eating in anorexia nervosa and overeating in bulimia nervosa.

35 norexia nervosa and promising for adolescent bulimia nervosa.

36 oods and avoid weight gain that characterize bulimia nervosa.

37 and conflict resolution in adolescents with bulimia nervosa.

38 including the subtypes anorexia nervosa and bulimia nervosa.

39 49 females (0.30%), and 61 males (0.01%) had bulimia nervosa.

40 patterns were found for anorexia nervosa and bulimia nervosa.

41 focused on the treatment of adolescents with bulimia nervosa.

42 determine how purging disorder differs from bulimia nervosa.

43 ng disorders, including anorexia nervosa and bulimia nervosa.

44 l of the brain mu-opioid receptor (mu-OR) in bulimia nervosa.

45 r the compulsive eating behavior observed in bulimia nervosa.

46 entified as a common feature of anorexia and bulimia nervosa.

47 male patients with eating disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 health

48 0 years after they appeared as patients with bulimia nervosa, 177 women (participation rate=79.7%) co

49 ervosa among women might be up to 4%, and of bulimia nervosa 2%.

50 Thirty-one female patients with bulimia nervosa, 29 women who had been recovered from bu

51 ong, positive dose-response association with bulimia nervosa (adjusted hazard ratio = 1.15, 95% confi

52 Eight women with bulimia nervosa and 10 age- and weight-matched control s

53 d, medication-free, normal-weight women with bulimia nervosa and 14 age-matched healthy female contro

54 ndent response in 18 female adolescents with bulimia nervosa and 18 healthy female age-matched subjec

55 Eight women with bulimia nervosa and 8 female controls underwent brain MR

56 found empirical support for conceptualizing bulimia nervosa and binge eating disorder as discrete sy

57 y remain the most established treatments for bulimia nervosa and binge eating disorder, with stepped-

58 utrient and food group intakes of women with bulimia nervosa and binge-eating disorder during pregnan

59 to measure the prevalence and correlates of bulimia nervosa and bulimic behaviors in a sample of und

60 nal distress compared with participants with bulimia nervosa and controls.

61 ialization affects the risk for anorexia and bulimia nervosa and disordered eating in members of oppo

62 ality ratios were significantly elevated for bulimia nervosa and eating disorder not otherwise specif

63 ndardized mortality ratios were elevated for bulimia nervosa and eating disorder not otherwise specif

64 compare serotonin function in patients with bulimia nervosa and healthy controls.

65 ell-validated disorders anorexia nervosa and bulimia nervosa and likely represents a stable syndrome.

66 n appear less likely to exhibit anorexia and bulimia nervosa and more likely to exhibit binge eating

67 cles published about anorexia nervosa and/or bulimia nervosa and the number of articles published abo

68 alized anxiety disorder, panic disorder, and bulimia nervosa), and four were "externalizing" (nicotin

69 s), 11% of this sample met full criteria for bulimia nervosa, and 0.6% met full criteria for anorexia

70 individuals with anorexia nervosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulim

71 tes were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for eating disorder not otherw

72 ering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as a specified eating di

73 examined the prevalence of anorexia nervosa, bulimia nervosa, and binge eating disorder in a geograph

74 me prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating disorder were 0.3%, 0.

75 DSM-IV classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

76 current classifications of anorexia nervosa, bulimia nervosa, and binge-eating disorder.

77 1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

78 y was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder not otherwise speci

79 Research into treatment has focused on bulimia nervosa, and evidence-based management of this d

80 ssion and co-twin risk for anorexia nervosa, bulimia nervosa, and obesity.

81 miological studies, short-term treatment for bulimia nervosa, and outcome in anorexia nervosa.

82 ree diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eating disorders.

83 ns and modifications to anorexia nervosa and bulimia nervosa; and the inclusion of binge eating disor

84 t while menstrual irregularities are common, bulimia nervosa appears to have little impact on later a

85 ecovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, respectively; little is

86 Both anorexia nervosa and bulimia nervosa are associated with increased mortality.

87 Anorexia nervosa and bulimia nervosa are common and severe eating disorders (

88 Anorexia nervosa and bulimia nervosa are common problems facing adolescents a

89 Both anorexia nervosa and bulimia nervosa are familial.

90 -based treatment trials for adolescents with bulimia nervosa are largely absent.

91 g disorders, especially anorexia nervosa and bulimia nervosa, are more common among white women than

92 These results support the validity of bulimia nervosa as a diagnostic category that is distinc

93 ht to investigate the predictive validity of bulimia nervosa as a diagnostic category.

94 ivate to the same degree in adolescents with bulimia nervosa as in healthy comparison subjects.

95 We enrolled patients with severe bulimia nervosa (at least seven coupled binge/vomit epis

96 Women with bulimia nervosa before and during pregnancy and those wi

97 Women with the diagnosis of bulimia nervosa between 1981 and 1987 who participated i

98 hotherapy in reducing behavioral symptoms of bulimia nervosa (binge eating and vomiting).

99 e four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder, and eating disor

100 ipants with a diagnosis of anorexia nervosa, bulimia nervosa, binge eating disorder, or eating disord

101 han black women (N=4, 0.4%) met criteria for bulimia nervosa; binge eating disorder also was more com

102 on in anorexia nervosa (AN) or overeating in bulimia nervosa (BN) a form of addiction?

103 Anorexia nervosa (AN) and bulimia nervosa (BN) are disorders characterized by abno

104 ED data are presented here and compared with bulimia nervosa (BN) data in the World Health Organizati

105 Bulimia nervosa (BN) has been associated with dysregulat

106 Women with bulimia nervosa (BN) have disturbances of mood and behav

107 Bulimia nervosa (BN) is strongly familial, and additive

108 t as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n = 82; Recovered AN n = 90; Health

109 trol circuits in adolescents and adults with bulimia nervosa (BN), but less is known about the micros

110 disorders, such as anorexia nervosa (AN) and bulimia nervosa (BN), have genetic and environmental und

111 in adolescent and adult female subjects with bulimia nervosa (BN).

112 itudinal distinction of anorexia nervosa and bulimia nervosa but do not support the anorexia nervosa

113 er mistakes was associated with anorexia and bulimia nervosa but not with other psychiatric disorders

114 ubtypes over time; one-third crossed over to bulimia nervosa but were likely to relapse into anorexia

115 ), attention-deficit/hyperactivity disorder, bulimia nervosa, cataplexy, dysthymic disorder, fibromya

116 ed in participants with purging disorder and bulimia nervosa compared with controls but did not diffe

117 ognize hunger signals, whereas overeating in bulimia nervosa could represent an exaggerated perceptio

118 women who continue to meet full criteria for bulimia nervosa declines as the duration of follow-up in

119 zygosity; the prevalence of DSM-IV or broad bulimia nervosa did not differ in women from opposite- v

120 essary to resolve conflict, adolescents with bulimia nervosa displayed abnormal patterns of activatio

121 tine versus placebo in preventing relapse of bulimia nervosa during a 52-week period after successful

122 howed symptoms of either anorexia nervosa or bulimia nervosa during pregnancy had a higher frequency

123 ple of individuals with anorexia nervosa and bulimia nervosa from the Price Foundation collaborative

124 a nervosa groups and on the left side in the bulimia nervosa group relative to the healthy comparison

125 educed in the recovered anorexia nervosa and bulimia nervosa groups and predicted sensitivity to rewa

126 Women who had recovered from bulimia nervosa had a reduction of medial orbital fronta

127 mately 50% of women initially diagnosed with bulimia nervosa had fully recovered from their disorder,

128 In addition, women with bulimia nervosa had less activation in the lateral and a

129 rs, but not the women who had recovered from bulimia nervosa, had an age-related decline in 5-HT(2A)

130 As bulimia nervosa has been associated with a dysregulated

131 tions decrease binge eating in patients with bulimia nervosa has fueled interest in the hypothesis th

132 Recent research on Anorexia Nervosa and Bulimia Nervosa has yielded an increasingly detailed und

133 Anorexia nervosa, but not bulimia nervosa, has one of the highest mortality rates

134 Two treatments for bulimia nervosa have emerged as having established effic

135 (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any lifetime diagnoses re

136 The treatment of patients with bulimia nervosa in a primary care setting is hampered by

137 haviours in patients with severe and chronic bulimia nervosa in a randomised, double-blind, placebo-c

138 g the usefulness of family interventions for bulimia nervosa in adolescents.

139 ervosa and possibly lower incidence rates of bulimia nervosa in Caucasian North American and Northern

140 on; and increasing rates of binge eating and bulimia nervosa in Hispanic and Black American minority

141 Bulimia nervosa in men was too rare to be assessed by zy

142 ta on outcome for individuals diagnosed with bulimia nervosa in order to better understand long-term

143 whether treatments known to be effective for bulimia nervosa in specialized treatment centers can be

144 due to all causes of death for subjects with bulimia nervosa in these studies was 0.3% (seven deaths

145 Bulimia nervosa is a common eating disorder in adolescen

146 The etiology of bulimia nervosa is complex, with biologic, psychological

147 acy of fluoxetine in the acute management of bulimia nervosa is well established; however, few contro

148 ver" among the anorexia nervosa subtypes and bulimia nervosa may reflect problems with the validity o

149 =13.9), significantly longer than for either bulimia nervosa (mean=5.8 years, SD=9.1) or anorexia ner

150 interventions are of established utility in bulimia nervosa, medications have no clear role in the t

151 either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110) between January 1, 1987, and D

152 ficantly diminished and women recovered from bulimia nervosa (N=14) had significantly elevated hemody

153 as there were about anorexia nervosa and/or bulimia nervosa (N=169).

154 diagnoses of binge eating disorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

155 ing-type anorexia nervosa (N=24), women with bulimia nervosa (N=19), and healthy comparison women (N=

156 Women diagnosed with either anorexia or bulimia nervosa (N=246) completed prospective evaluation

157 with restricting anorexia nervosa (n=26) or bulimia nervosa (n=47), control women (n=44), and first-

158 5 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or eating disorder not otherwis

159 a longitudinal study of anorexia nervosa and bulimia nervosa, now in its 12th year.

160 r of eating that appears to be distinct from bulimia nervosa on subjective and physiological response

161 th binge eating and purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purgin

162 ften useful as adjuncts to psychotherapy for bulimia nervosa or binge-eating disorder; in the case of

163 acial/ethnic difference in the prevalence of bulimia nervosa or bulimic behaviors; 1.5% of the whites

164 y (CBT) guided self-care in adolescents with bulimia nervosa or eating disorder not otherwise specifi

165 Eighty-five adolescents with bulimia nervosa or eating disorder not otherwise specifi

166 were randomly assigned to family therapy for bulimia nervosa or individual CBT guided self-care suppo

167 isorders, 0.8% had partial- or full-criteria bulimia nervosa or purging disorder and 2.9% had partial

168 The participants were 80 mothers with bulimia nervosa or similar eating disorder who were atte

169 evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating was associated wit

170 A total of 120 women with bulimia nervosa participated in a randomized, placebo-co

171 mined menstrual and reproductive function in bulimia nervosa patients retrospectively, with 10-15-yea

172 to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and broad criteria).

173 Adolescents with and without bulimia nervosa performed similarly on the task.

174 ot respond to cognitive behavior therapy for bulimia nervosa, potentially allowing early use of a sec

175 Patients who met DSM-IV criteria for bulimia nervosa, purging type, were assigned to single-b

176 rial was conducted in which 70 patients with bulimia nervosa received either 2 years of weekly psycho

177 Long-term outcome for women diagnosed with bulimia nervosa remains unclear.

178 Since bulimia nervosa's introduction to the psychiatric nomenc

179 outcome studies of both anorexia nervosa and bulimia nervosa show that no significant differences exi

180 ompared with control subjects, patients with bulimia nervosa showed a blunting of postprandial cholec

181 More research on bulimia nervosa specific to adolescence is needed.

182 .38-9.54] vs adjusted, 2.67 [1.78-4.01]) and bulimia nervosa (suicide attempts: crude, 6.26 [5.73-6.8

183 inin release compared with participants with bulimia nervosa (t(76.44) = 2.51; P = .01) and did not d

184 h higher in people with anorexia nervosa and bulimia nervosa than in a nonclinical group of women in

185 in engendering improvement in patients with bulimia nervosa than IPT.

186 lescents and adults with anorexia nervosa or bulimia nervosa that, together, implicate dysregulation

187 For patients with bulimia nervosa, the fenfluramine-stimulated increase in

188 en substantial advances in the management of Bulimia Nervosa, the goal of offering effective treatmen

189 ed to reward-processing deficits in remitted bulimia nervosa, the purpose of this study was to identi

190 Nine women who had recovered from bulimia nervosa (they had no episodes of binge eating or

191 sover longitudinally in anorexia nervosa and bulimia nervosa to inform the validity of the DSM-IV-TR

192 of the studies linking anorexia nervosa and bulimia nervosa to substance use disorders suffer from p

193 Cognitive behavioural therapy for bulimia nervosa was assigned grade A because of the evid

194 al behavior therapy adapted for treatment of bulimia nervosa was associated with a promising decrease

195 Bulimia nervosa was more common than anorexia nervosa in

196 h AN and related eating disorders, including bulimia nervosa, was performed, resulting in only modest

197 obands; the corresponding relative risks for bulimia nervosa were 4.2 and 4.4 for female relatives of

198 enty patients meeting DSM-III-R criteria for bulimia nervosa were allocated at random to 19 sessions

199 en partial syndromes of anorexia nervosa and bulimia nervosa were considered, relative risks fell by

200 tes of full and partial anorexia nervosa and bulimia nervosa were determined in first-degree relative

201 omen with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7 years; weekly eating

202 Antidepressants for bulimia nervosa were given grade B.

203 A total of 173 women with bulimia nervosa were interviewed an average of 11.5 year

204 ing female patients with anorexia nervosa or bulimia nervosa were interviewed every 6 months for a me

205 who had recovered from anorexia nervosa and bulimia nervosa were studied to avoid confounding effect

206 Women with bulimia nervosa were unlikely to cross over to anorexia

207 diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize intrapsychic conflicts

208 onwhites (n = 693) were classified as having bulimia nervosa, while 5.5% of the whites and 5.3% of th

209 th fluoxetine is useful for individuals with bulimia nervosa who do not respond to psychotherapy or r

210 Twenty-two patients with bulimia nervosa who had not responded to, or had relapse

211 e study examined relapse in 48 patients with bulimia nervosa who had responded to cognitive behaviora

212 y be a useful intervention for patients with bulimia nervosa who have not responded adequately to psy

213 d treatment with fluoxetine in patients with bulimia nervosa who responded to acute treatment with fl

214 formation exists on relapse in patients with bulimia nervosa who responded with complete abstinence f

215 194 women meeting the DSM-III-R criteria for bulimia nervosa who were treated with 18 sessions of man

216 of attrition and outcome in the treatment of bulimia nervosa with cognitive behavior therapy.

217 e recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2

218 orexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia nervosa with no history of anorexia nervosa.

219 hout obsessive-compulsive features; and LC4, bulimia nervosa with self-induced vomiting as the sole f

220 (LC1) resembled restricting AN; LC2, AN and bulimia nervosa with the use of multiple methods of purg