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1 nt without requirement for specific signals (bulk flow).
2 high turgor to drive symplasmic unloading by bulk flow.
3 lass I molecules exit the ER by nonselective bulk flow.
4 y by enhancing its solubility and intestinal bulk flow.
5  the brain interstitium by diffusion than by bulk flow.
6 ry to the mechanics chamber while preventing bulk flow.
7 s the addition of chemicals without creating bulk flows.
8                          The new variable of bulk flow acceleration affords a new selectivity paramet
9                                              Bulk flow analyses further revealed that keratin filamen
10               The existence and magnitude of bulk flow and the so-called glymphatic system are topics
11  channels as a closed system and treated the bulk flow as in traditional capillary electrophoresis.
12 ity was too low to allow for any substantial bulk flow at physiological hydrostatic pressure gradient
13 lecting major changes in the sodium/chloride bulk flow close to term.
14 k constitutes a miniature conduit system for bulk flow delivery of soluble molecules to distinct site
15       Several lines of evidence suggest that bulk flow drainage via the glymphatic system is driven b
16 ng phagosomes have full access to the early "bulk flow" endocytic pathway.
17             The edges of this region exhibit bulk flow features without polygons.
18  data support cargo protein involvement, not bulk flow, in the formation of post-TGN vesicles and pre
19 ration of which extracts a large fraction of bulk-flow kinetic energy from the incident solar wind.
20                    Inconsistent with exit by bulk flow, loading of MHC class I molecules with high-af
21 kers in the ER, the trafficking of a soluble bulk-flow marker to the cell surface was also compromise
22 mately 20 fold in these vesicles relative to bulk flow markers.
23 o COPII vesicles as inefficiently as soluble bulk flow markers.
24 ydrocephalus as the result of obstruction to bulk flow of CSF is evolving to models that incorporate
25  Bleb growth occurs through a combination of bulk flow of lipids and delamination from the cell corte
26 ophoretic velocity of an analyte against the bulk flow of solution in the presence of a temperature g
27          This work suggests that the forward bulk flow of the cytoskeletal framework that occurs duri
28 hought to leave the ER in these vesicles by "bulk flow" or through recognition by hypothetical shuttl
29       We show X-ray radiography captures the bulk flow properties, in contrast to optical methods whi
30 on a variation of the TGF method whereby the bulk flow rate is varied over time so that a large numbe
31 her predictions were that decreasing the CSF bulk flow rate would increase AUC(C(IAR)), with 3 mL/h b
32 default pathway requiring no export signals (bulk flow; refs. 6,7).
33 y matching condition between the EDL and the bulk flow region is incomplete under spatial bulk flow v
34 ke the steady pure electroosmotic flows, the bulk flow region of time periodic electroosmotic flows a
35 ocity matches our analytical solution in the bulk flow region.
36  II-dependent endoplasmic reticulum-to-Golgi bulk flow route and that VSR-ligand interactions in the
37                    Scanning TGF uses varying bulk flow so that a large number of analytes that have l
38  permeability and simulate pressure-mediated bulk flow through 3D electron microscope (EM) reconstruc
39 by applying a pressure gradient to establish bulk flow through the brain interstitium during infusion
40                 Three independent markers of bulk flow transport out of the ER indicate that in the a
41 be oriented in the direction opposite to the bulk flow under normal transpiration conditions.
42 bulk flow region is incomplete under spatial bulk flow variations across the finite EDL.
43 mula: see text] higher permeability, but the bulk flow was still insignificant.

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