1 rized clinically by the development of tense bullae and by the presence of an antibasement membrane a

2 ay be related to the formation of epithelial bullae and microcystic changes in PBK patients.

3 r stroma, epithelial basement membrane (BM), bullae and subepithelial fibrosis (SEF) areas, and poste

4                                         Oral bullae are the most common oral finding and affect all s

5 istory of SLE who was seen with vesicles and bullae around her lips, trunk, axillae, arms, and thighs

6 s stopped, and the skin lesion progressed to bullae formation with extensive superficial erosion.

7                                   Violaceous bullae, hypotension, fever, and evidence of organ failur

8          Removing nonventilating lung (e.g., bullae) simply displaces the MEFV curve down the volume

9 ts with severe symptomatic emphysema without bullae were entered into the protocol (39 staple, 33 las

10 fter infection, after which the superior ear bullae were excised and examined by culture and microsco

11 ested by inoculating middle ear hypotympanic bullae with Streptococcus pneumoniae types 6B, 6A, 19F,