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1 ter graft survival in eyes with pseudophakic bullous keratopathy.
2 formed for Fuchs' dystrophy and pseudophakic bullous keratopathy.
3 secondary corneal edema due to pseudophakic bullous keratopathy.
4 p is strongest in patients with pseudophakic bullous keratopathy.
5 from Fuchs endothelial dystrophy and 5 with bullous keratopathy.
6 Fuchs endothelial dystrophy or pseudophakic bullous keratopathy.
7 of cellular adhesion molecules is altered in bullous keratopathy.
8 tes the dominant features of contemporaneous bullous keratopathy.
9 lay a significant role in the progression of bullous keratopathy.
11 5 DMEK cases were performed for pseudophakic bullous keratopathy (2 cases, 1 in each cohort), and the
14 o be effective in symptomatic improvement of bullous keratopathy, and infectious keratitis but furthe
15 ycans from human corneas with chronic edema, bullous keratopathy, and keratoconus and from normal cor
16 st, Africa, and South America), pseudophakic bullous keratopathy/aphakic bullous keratopathy (North A
17 the most common indication for PTK is still bullous keratopathy, as PTK can be successfully used whi
21 dothelial corneal dystrophy (FECD; n = 314), bullous keratopathy (BK; n = 31), and failed previous en
22 he hypothesis that epithelial alterations in bullous keratopathy compromise the surface of the cornea
23 DNA damage was not detected in pseudophakic bullous keratopathy corneas, whereas it colocalized with
24 Fuchs endothelial dystrophy and pseudophakic bullous keratopathy, EK achieved better average best-cor
27 Fuchs endothelial dystrophy or pseudophakic bullous keratopathy from January 2006 through December 2
28 mpared with normal controls and pseudophakic bullous keratopathy (iatrogenic CE cell loss) specimens.
29 uchs' endothelial dystrophy and pseudophakic bullous keratopathy in patients without other vision-lim
30 MEK surgery in the treatment of pseudophakic bullous keratopathy in the presence of sf-IOL and if-IOL
31 ssociated with Fuchs' endothelial dystrophy, bullous keratopathy, iridocorneal endothelial syndrome o
35 gery (n = 127; 90%) followed by pseudophakic bullous keratopathy (n = 4; 4%) and regrafts (n = 9; 6.4
36 a), pseudophakic bullous keratopathy/aphakic bullous keratopathy (North America), and keratitis (Asia
41 elial dystrophy and 9 eyes with pseudophakic bullous keratopathy (PBK) that underwent DSAEK, and 17 e
42 hs endothelial dystrophy (FED), pseudophakic bullous keratopathy (PBK), infection and other indicatio
43 hs endothelial dystrophy (FED), pseudophakic bullous keratopathy (PBK), or keratoconus who had underg
44 ly a Descemet membrane disorder), but not in bullous keratopathy (primarily an endothelial depletion)
46 ll and superficial cell layers were lower in bullous keratopathy specimens (1.6 vs. 2.0; P < 0.0001)
47 ers evident in sections was increased in the bullous keratopathy specimens compared with controls (0.
48 orated with antibodies to MUC16 was lower in bullous keratopathy specimens than in controls (0.5 vs.
50 Fuchs endothelial dystrophy and pseudophakic bullous keratopathy undergoing DSAEK surgery were includ
51 monstrated in a pre-clinical rabbit model of bullous keratopathy using a tissue-engineered endothelia
52 s associated with them, such as pseudophakic bullous keratopathy, uveitis-glaucoma-hyphema syndrome,
54 The reduction of layers expressing MUC1 in bullous keratopathy was not statistically significant.
55 rformed for Fuchs' dystrophy or pseudophakic bullous keratopathy was poorer than survival of penetrat
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