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1 chemic pattern (ST-segment elevation or left bundle branch block).
2 -segment elevation or depression and/or left bundle-branch block).
3 duction system abnormalities, including left bundle branch block.
4 e branch block is far more common than right bundle branch block.
5 tivity and specificity in patients with left bundle branch block.
6 cal septal myectomy, 47 (40%) developed left bundle branch block.
7 ol septal ablation, 21 (36%) developed right bundle branch block.
8 60 patients with mild heart failure and left bundle branch block.
9 spected AMI and ST segment elevation or left bundle branch block.
10 h of symptom onset with ST elevation or left bundle branch block.
11 egree atrioventricular block with associated bundle branch block.
12 ose with QRS width 120 to 149 ms or non-left bundle branch block.
13 (1.6%) had left (n = 131) or right (n = 289) bundle branch block.
14 igraphy is decreased in patients with a left bundle branch block.
15 d pharmacologic stress in patients with left bundle branch block.
16 nts with heart failure (HF) and without left bundle branch block.
17 dent predictors of new onset persistent left bundle branch block.
18 LVP and BiVP in the failing heart with left bundle branch block.
19 ith or without systolic dysfunction and left bundle-branch block.
20 patients with systolic dysfunction and left bundle-branch block.
21 ventricular ejection fraction <35% and left bundle-branch block.
22 frequently accompanied by an apparent right bundle-branch block.
23 had chronic coronary artery disease and left bundle-branch block.
24 arction in patients with chest pain and left bundle-branch block.
25 r mortality rate than either transient or no bundle-branch block.
26 of acute infarction in the presence of left bundle-branch block.
27 ts presenting with acute chest pain and left bundle-branch block.
28 t) with acute myocardial infarction had left bundle-branch block.
29 ll mortality rate associated with persistent bundle-branch block.
30 ng could differentiate between BrS and right bundle-branch block.
31 morphology into those with and without left bundle-branch block.
32 lockade, and the presence of a complete left bundle-branch block.
33 lts: (1) ST elevation or presumably new left bundle branch block, (2) other ECG signs indicating myoc
34 1.44) versus 1.04 (0.78, 1.51), de novo left bundle-branch block (4% versus 0%) and Q waves (5.3% ver
36 ing ECG abnormalities were as follows: right bundle-branch block, 65.2%; left bundle-branch block, 43
37 icular conduction delay, or incomplete right bundle branch block, a much higher proportion than in th
38 pacing/stimulation in DCM patients with left bundle-branch block acutely enhances systolic function w
39 possibly with harm in patients without left bundle-branch block (adjusted hazard ratio for death fro
41 y 17 patients (2.0%) had new Q waves or left bundle-branch block after surgery; however, cTnT elevati
45 with Lugol's solution induced complete right bundle branch block and converted the bidirectional VT i
47 hic VT (n = 23) or PVCs (n = 47) with a left bundle branch block and inferior QRS axis morphology.
48 rietal band VAs were characterized by a left bundle branch block and left inferior (n=12) or superior
49 to have nonischemic cardiomyopathy and left bundle branch block and less likely to have renal dysfun
50 failure with QRS duration >/=150 ms and left bundle branch block and less predictable in those with Q
53 cular ventricular tachycardia, and the right bundle branch block and ST segment elevation syndrome of
54 n association with typical or atypical right bundle branch block and ST-segment elevations (Brugada s
55 in; ventricular tachycardias (VTs) with left bundle branch block and V(3) transition remain a challen
57 cluded Coronary Arteries) trial who had left bundle-branch block and acute myocardial infarction conf
58 nduction disturbances, mainly new-onset left bundle-branch block and advanced atrioventricular block
60 justment for other significant factors, left bundle-branch block and intraventricular conduction dela
61 ctors that impair the detection of ischemia (bundle-branch block and paced rhythms)-in predicting out
63 icular fibrillation characterized by a right bundle-branch block and ST elevation in the right precor
64 wo groups: group 1 (n=16) patients had right bundle-branch block and ST-segment elevation in V1 throu
66 er implantation after TAVR, with prior right bundle-branch block and transcatheter valve type and imp
67 ts reviewed presenting ECGs to identify left bundle-branch block and, in the absence of left bundle-b
69 od pressure <100 mm Hg, anterior MI, or left bundle branch block, and as "low clinical risk" if none
71 smoking, left ventricular hypertrophy, left bundle branch block, and diabetes mellitus; it had good
72 ents with left bundle branch block, non-left bundle branch block, and right bundle branch block morph
73 isease), normal QT intervals, apparent right bundle branch block, and sudden cardiac death, particula
74 le-branch block (LBBB), 228 (13%) with right bundle-branch block, and 308 (17%) with nonspecific intr
75 ent smoking, body mass index >35 kg/m2, left bundle-branch block, and left ventricular hypertrophy.
76 ude various RSR' patterns, without a typical bundle-branch block are markers of altered ventricular d
77 us persistent ST-segment elevation and right bundle branch block, as well as the effectiveness of sod
79 ricular (AV) block, bifascicular block, left bundle branch block, atrial fibrillation, and left ventr
83 versus 85 to 99 ms and >/=100 ms (excluding bundle branch block [BBB]) and QRS morphology in those w
88 io-ventricular conduction block (AVB), right bundle branch block, bradycardia, and the Brugada syndro
89 as applied in the presence of complete right bundle branch block by atrial-synchronized RV free wall
91 es were significant among patients with left bundle branch block conduction disturbance (n = 1,204, p
92 y in those with QRS >/= 150 ms and with left bundle branch block conduction disturbance, respectively
93 ctural heart disease associated with a right bundle branch block conduction pattern and ST-segment el
94 In patients with mild heart failure and left bundle branch block, decreased RWT was associated with a
95 dle-branch block and, in the absence of left bundle-branch block, definite STEMI (according to both c
97 vival benefit of CRT-D in patients with left bundle-branch block did not differ significantly accordi
99 structural heart disease, paced rhythms, and bundle branch block during sinus rhythm were excluded.
100 toms, left ventricular dysfunction, and left bundle-branch block, early intervention with CRT-D was a
102 North American population with acute MI and bundle branch block enrolled in the Global Utilization o
103 nsustained ventricular tachycardia with left bundle branch block excluding right ventricular outflow
104 and 24 patients with heart failure and left bundle branch block followed by computer simulations of
105 55; p = 0.033) and new-onset persistent left bundle-branch block following TAVR (HR: 2.26, 95% CI: 1.
107 nontetralogy control subjects (20 with right bundle-branch block [group 4] and 20 with normal ECG pat
108 left ventricular ejection fraction and left bundle-branch block; group 3 (n=20), left ventricular ej
109 We identified 12 affected individuals (right bundle-branch block, > or =1-mm STE) with an autosomal d
115 HFEs was pronounced among patients with left bundle branch block (HR: 0.38, 95% CI: 0.29 to 0.49, p <
116 was neutral, regardless of morphology, right bundle branch block (HR=1.01, P=0.975), and intraventric
118 (ii) heart with a conduction disorder (right bundle branch block) (iii) focal activation initiated by
119 n resulted in ST-segment elevation and right bundle branch block in all patients in group A and in al
123 Idiopathic ventricular arrhythmias of left bundle branch block inferior axis morphology are usually
124 hic pattern associated with this VT was left bundle branch block, inferior axis and early precordial
128 premature ventricular contractions with left bundle branch block/inferior axis pattern in 16 ARVD/C p
129 ch block/inferior axis, case 3 showed a left bundle branch block/inferior axis, and case 4 showed a n
130 h block/superior axis, case 2 showed a right bundle branch block/inferior axis, case 3 showed a left
132 t bundle branch block (LBBB; including right bundle branch block, intraventricular conduction delay)
136 pa 0.52), and Q waves (kappa 0.44), good for bundle branch block (kappa 0.78), and very good for atri
138 upraventricular complexes with left or right bundle-branch block (L/RBBB) was investigated in a canin
139 a pronounced effect among patients with left bundle branch block (LBBB) (hazard ratio [HR]: 0.58; p <
140 82], p = 0.001) and CRT-D patients with left bundle branch block (LBBB) (HR: 0.51 [95% CI: 0.35 to 0.
141 evolution in ST segment, T or Q wave or left bundle branch block (LBBB) abnormalities between the pre
142 ronization Therapy) study patients with left bundle branch block (LBBB) and 0, 1, 2, or >/=3 comorbid
143 ific syndrome characterized by isolated left bundle branch block (LBBB) and a history of progressive
144 t to separate the effects of associated left bundle branch block (LBBB) and coronary artery disease (
145 de (VAQRS), halfway between that during left bundle branch block (LBBB) and LV pacing, reflects optim
146 ata on the clinical impact of new-onset left bundle branch block (LBBB) and permanent pacemaker impla
147 t guidelines suggest that patients with left bundle branch block (LBBB) be treated with cardiac resyn
149 and death events in CRT-D patients with left bundle branch block (LBBB) enrolled in MADIT-CRT (Multic
151 rognostic value of new-onset persistent left bundle branch block (LBBB) in patients undergoing transc
154 a suspected acute coronary syndrome and left bundle branch block (LBBB) present a unique diagnostic a
155 no benefit of CRT-D in patients without left bundle branch block (LBBB) regardless of patient sex.
156 Studies in canine hearts with acute left bundle branch block (LBBB) showed that endocardial left
157 nts with CRT-D in a very wide QRSD with left bundle branch block (LBBB) versus those without LBBB.
159 ead electrocardiographic morphology was left bundle branch block (LBBB), and in 15, it was nonspecifi
160 hy (DCM); in particular, the effects of left bundle branch block (LBBB), coronary artery disease (CAD
163 synchronization therapy candidates with left bundle branch block (LBBB)-like electrocardiogram morpho
167 Therapy (MADIT-CRT), patients with non-left bundle branch block (LBBB; including right bundle branch
168 strated an adverse impact of persistent left bundle-branch block (LBBB) after surgical aortic valve r
170 ree wall differed between patients with left bundle-branch block (LBBB) and normal QRSd and if synchr
171 go exercise testing develop a transient left bundle-branch block (LBBB) during exercise, but its prog
174 d to investigate the cardiac effects of left bundle-branch block (LBBB) using myocardial contrast ech
175 at baseline, there were 1281 (70%) with left bundle-branch block (LBBB), 228 (13%) with right bundle-
176 ardial infarction (MI) in patients with left bundle-branch block (LBBB), but the clinical impact of t
177 ough 30% to 50% of patients develop new left bundle-branch block (LBBB), its effect on clinical outco
178 similarly augment systolic function in left bundle-branch block (LBBB)-failing hearts despite differ
179 ts with (1) RV dilatation, (2) multiple left bundle-branch block (LBBB)-type VTs, and (3) an abnormal
183 rformed in anesthetized dogs with acute left bundle-branch block (LBBB, n=10) and chronic LBBB with t
184 5+/-12; ejection fraction 26+/-7%) with left bundle-branch block (LBBB; QRS duration 174+/-18 ms) wer
185 tment group, age, gender, QRS duration, left bundle-branch block, left ventricular ejection fraction,
186 the 9 patients; 9 of the 11 VTs had a right bundle branch block/left-axis morphology that mimicked l
187 diothoracic ratio, higher prevalence of left bundle branch block, lower prevalence of atrial fibrilla
188 tricular arrhythmias with a predominant left bundle branch block morphology and no ECG or echocardiog
189 hallenging because they present with a right bundle branch block morphology by electrocardiography.
190 rall, patients with mild HF but without left bundle branch block morphology did not derive clinical b
191 ricular arrhythmias with a predominant right bundle branch block morphology, 13 of 27 (48%) showed EC
196 CRT was performed in dogs with chronic left bundle-branch block (n=8) or atrioventricular (AV) block
197 clinical response between patients with left bundle branch block, non-left bundle branch block, and r
198 or more in at least 2 contiguous leads, left bundle branch block not known to be old, and no absolute
200 When multivariate analyses were used, left bundle-branch block (odds ratio [OR]=0.22; 95% CI=0.20 t
205 (paced rhythms, ventricular rhythms, or left bundle-branch block) on the ECG performed before thrombo
206 ere most pronounced among patients with left bundle branch block or a QRS duration at least 150 ms an
207 d the clinical outcome of patients with left bundle branch block or intraventricular conduction delay
208 logic Q waves on the electrocardiogram, left bundle branch block or nonischemic cardiomyopathy were n
209 onged P-R interval, but not incomplete right bundle-branch block or early repolarization patterns, in
210 cardia, QRS prolongation resulting from left bundle-branch block or intraventricular conduction delay
211 patients with a non-LBBB QRS pattern (right bundle-branch block or intraventricular conduction distu
212 ry (OR 4.6; P = 0.016), the presence of left bundle branch block (OR 39; P = 0.002), and first-degree
213 2.1; 95% CI, 1.3 to 3.3), preoperative left bundle branch block (OR, 2.0; 95% CI, 1.3 to 2.9), preop
214 ration >/=150 ms (OR: 1.79; p = 0.007), left bundle branch block (OR: 2.05; p = 0.006), body mass ind
215 abnormalities, left ventricular hypertrophy, bundle branch block, or left-axis deviation) or exercise
217 > 0.2), first-degree heart block (P = 0.12), bundle-branch block (P > 0.2), and ST-segment abnormalit
219 h a higher rate of new onset persistent left bundle branch block, particularly in patients receiving
220 respectively) and nonsignificant in non-left bundle branch block patients (HR: 1.12, 95% CI: 0.77 to
221 long-term clinical outcomes of 537 non-left bundle branch block patients with mild HF enrolled in th
227 ferentiated from the A-LV summit VAs by left bundle branch block pattern, QRS duration </=175 ms, pre
228 icular fibrillation characterized by a right bundle-branch block pattern and ST elevation (STE) in th
233 entricular enlargement only, one with a left bundle branch block) presented with advanced HF and SCD,
234 and male sex, left ventricular hypertrophy, bundle branch block, previous myocardial infarction, and
235 tensive ST-segment elevation, new-onset left bundle branch block, previous myocardial infarction, Kil
236 raventricular conduction delay but not right bundle-branch block provided prognostic information abou
237 P<0.001) along with elimination of the right bundle branch block QRS morphology, increase in RV filli
238 ion (OR=0.32; 95% CI, 0.19 to 0.53) and left bundle-branch block QRS morphology (OR=0.21; 95% CI, 0.1
239 score (female sex, nonischemic origin, left bundle-branch block, QRS >/=150 milliseconds, prior hosp
240 , SA was found to increase the risk of right bundle branch block (RBBB) (pooled odds ratio [OR]: 56.3
241 aventricular tachycardia with aberrant right bundle branch block (RBBB) and left anterior hemiblock (
242 ock (LBBB) was present in 65 patients, right bundle branch block (RBBB) in 48 patients, and nonspecif
243 udy sought to test the hypothesis that right bundle branch block (RBBB) patients have larger scar siz
244 lock may be made when catheter-induced right bundle-branch block (RBBB) develops in patients with bas
248 of ventricular tachycardia (VT) with a right bundle-branch block (RBBB)-type pattern and an R-S ratio
252 nization therapy for heart failure with left bundle branch block reduces left ventricular (LV) conduc
253 e studied 383 consecutive patients with left bundle branch block referred for perfusion scintigraphy
256 excitation, as well as during right or left bundle branch block, resembled experimental measurements
257 ior hemiblock (RR: 1.62; p < 0.01), or right bundle branch block (RR: 2.89; p < 0.01) at baseline; an
258 ents with congenital heart disease and right bundle branch block, RV cardiac resynchronization therap
259 atients with systolic heart failure and left bundle-branch block scheduled for cardiac resynchronizat
260 , nonsustained ventricular tachycardia, left bundle branch block, signal-averaged electrocardiogram,
261 patients with QRS 120 to 149 ms or non-left bundle branch block, significant associations of baselin
263 eria (QRS duration of 120-149 ms or non-left bundle branch block), SSI>/=9.7% was independently assoc
264 ribed in patients with the syndrome of right bundle branch block, ST-segment elevation in leads V1 to
267 an 6 hours after the onset of symptoms, left bundle branch block, total ST-segment elevation of 6 mm
268 was relatively well preserved, and the right bundle-branch block type of BBR was frequently induced.
270 ree patients with systolic heart failure and bundle-branch block underwent implantation of biventricu
271 reverse remodeling is predicted by the left bundle branch block ventricular activation sequence, the
273 depression greater than 1 mm, complete left bundle-branch block, ventricular paced rhythm, preexcita
275 n 46% of septal myectomy patients, and right bundle branch block was evident in 58% of alcohol septal
276 in the VA interval with the development of a bundle branch block was the only tachycardia characteris
277 eath from any cause among patients with left bundle-branch block was 18% among patients randomly assi
279 an electrocardiographic pattern showing left bundle-branch block was associated with a significant re
287 th complete, partial and no reversion of the bundle branch block were 8%, 12% and 20%, respectively (
288 Mortality rates in patients with and without bundle-branch block were 8.7% and 3.5%, respectively (P
289 urface electrocardiographic patterns of left bundle-branch block were all noted during intentional an
290 prognostic information in patients with left bundle-branch block, which is incremental to clinical as
291 on-4, we studied patients with STEMI or left bundle branch block who received acute reperfusion with
292 associated with ST-segment elevation or left bundle branch block who underwent primary angioplasty.
293 dy group consisted of 245 patients with left bundle-branch block who underwent tomographic (single ph
294 associated with ST-segment elevation or left bundle-branch block who were treated with primary angiop
295 acute STEMI (including 3 patients with left bundle-branch block) who were undergoing infarct-artery
296 pidemia, and had more often a non-LBBB (left bundle branch block) wide QRS complex, and lower left ve
297 h a higher rate of new onset persistent left bundle branch block with the CoreValve (47.7% versus 35.
298 %, 55% ischemic cardiomyopathy, and 71% left bundle-branch block) with a follow-up of 29+/-11 months.
299 able adjustment, older age, female sex, left bundle branch block, worsened heart failure class, highe
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