ライフサイエンスコーパス: burn

1 sources (19 to 73% contribution from biomass burning).

2 ly enhanced corneal recovery from the alkali burn.

3 6 h directly downwind during the peak of the burn.

4 scles were studied at 7, 14 and 21 days post-burn.

5 tion was not observed in experimental alkali burns.

6 emales across all external causes except for burns.

7 r 84% of all wildfires and 44% of total area burned.

8 TSF and the units' difference in time since burning.

9 tion in regions strongly impacted by biomass burning.

10 nd to a lesser degree from increased biomass burning.

11 ate emissions worldwide from industrial coal-burning.

12 from vehicle emissions and residential fuel burning.

13 le particles present in emissions of biomass burning.

14 ning contribution comes from natural biomass burning.

15 injury sustained (818 [61.2%]), followed by burn (307 [23.0%]) and rashes (163 [12.2%]).

16 e two new species are Udranomia sallydaleyae Burns, a dry forest denizen occurring from Costa Rica to

17 We found that topical EPO treatment of the burns accelerated their healing through an AQP3-dependen

18 easing fraction of absorption as the biomass-burning aerosol aged.

19 HULIS) in particulate matter such as biomass burning aerosol chelate Fe(II), but the effect on ROS fo

20 this chloride acid displacement, the biomass-burning aerosol still converted on the order of 10% of r

21 ies, the particulate chloride in the biomass-burning aerosol was rapidly but incompletely displaced,

22 We suggest that the brownishness of biomass burning aerosols indicates the amount of BC/BrC as well

23 The regional characteristics of biomass burning aerosols were found to depend strongly on the mo

24 black carbon and larger than that of biomass burning aerosols.

25 f of them executed-either shot, beheaded, or burned alive-while the rest died on Mount Sinjar from st

26 al body surface area third degree skin flame burn and 48 breaths of cooled cotton smoke inhalation un

27 al components of retinal injury after alkali burn and explored a novel neuroprotective regimen suitab

28 positively correlated with a larger percent burn and with the development of urinary tract infection

29 rosol particles emitted per kilogram of fuel burned and the microphysical properties of those aerosol

30 , but no differences on larger trees between burned and unburned plots in the long term.

31 sions and facilitating conversion to cleaner-burning and renewable fuels.

32 OC indicator, and PC represents both biomass burning and secondary aerosol.

33 apes through deforestation, agriculture, and burning and thereby might reduce the capacity of landsca

34 own how ecosystems will respond to increased burning and warming.

35 rentiated cells are useful for treating skin burns and have potential for regenerative treatment of o

36 and fires, but also while working prescribed burns and while off-duty.

37 strates with accelerants (either burnt or un-burnt) and inhibit responses to the same substrates (eit

38 h 20% or more TBSA burn similar in age, TBSA burn, and inhalation injury.

39 ing for age, gender, total body surface area burned, and inhalation injury (hazard ratio, 1.73; 95% C

40 are converted into titanium suboxides during burning, and initial biotoxicity screening suggests that

41 exchange, gross primary production, biomass burning, and respiration to these climate anomalies by a

42 tions, specifically improved-technology wood-burning appliances or air-filtration devices, would impr

43 tracks were displaced north, and the average burn area per fire also tended to be higher in years wit

44 fuel and fine fuel structure) on the summer Burned Area (BA) across all eco-regions in Mediterranean

45 ark thickness is largely explained by annual burned area and precipitation seasonality.

46 We found that global burned area declined by 24.3 +/- 8.8% over the past 18 y

47 For the first time, we show that relative burned area depth decreases over the first four fire eve

48 The estimated decrease in burned area remained robust after adjusting for precipit

49 e rate of fire spread, fuel consumption, and burned area.

50 bark is a pervasive adaptation in frequently burned areas across savannas and forests in both tempera

51 No association was observed for burning artificial fire-logs [HR=0.98 (95% CI: 0.85, 1.1

52 attributed to primary emissions from biomass burning (BB) and fossil fuel burning (FFB).

53 carbon (BrC), from laboratory-based biomass burning (BB) has been studied intensively to understand

54 Particles from smoldering biomass burning (BB) represent a major source of carbonaceous ae

55 ent of the total carbon that is emitted from burning biomass over the same period.

56 ed trial to compare the effects of a cleaner burning biomass-fuelled cookstove intervention to contin

57 ence that an intervention comprising cleaner burning biomass-fuelled cookstoves reduced the risk of p

58 ence that an intervention comprising cleaner burning biomass-fuelled cookstoves reduced the risk of p

59 sised that replacing open fires with cleaner burning biomass-fuelled cookstoves would reduce pneumoni

60 geochemical processes, including fossil fuel burning, biospheric photosynthesis and respiration, hydr

61 ition that the FOA3 is applicable to engines burning both conventional and alternative fuel blends wi

62 Initially, large biomass burning BrC molecules were rapidly photoenhanced, follow

63 d the total light absorption of aged biomass burning BrC.

64 III digestion of hybridized DNA tracks in a burnt-bridge mechanism.

65 osene were also low compared with solid fuel burning but had a relatively high F30 value of approxima

66 erra Nevada Mountains under climate and area burned by large wildfires projected by late 21(st) centu

67 areas of the northwestern United States were burned by moderate to high severity wildfires when storm

68 istance to wildfire and restoration of areas burned by wildfire through fire suppression and fuels ma

69 This study reveals that open-field biomass burning can be an important source of various semivolati

70 molecular weight BrC compounds from biomass burning can be relatively long-lived components in atmos

71 ngs illuminate the mechanism by which alkali burns cause retinal damage and may have importance in de

72 ust sensing, without electrode corrosion and burning/causing discomfort in subjects.

73 volvement), who were treated at one tertiary burn center in Hong Kong between 1999 and 2014, were ret

74 Eighteen burn centers enrolled 345 patients with 20% or more TBSA

75 Here we have discovered that burning coal also produces large quantities of otherwise

76 Yet, burning coal produces more global warming CO2 relative t

77 ved when pooling fuels, indicating that both burn conditions and fuel types may impact BB BrC charact

78 e recovery of lean tissue following a severe burn, contributing to prolonged frailty in burn survivor

79 engines onboard a NASA DC-8 aircraft as they burned conventional Jet A fuel and a 50:50 (by volume) b

80 a at cruise altitudes are sparse for engines burning conventional fuels, and no data have previously

81 rimental health and climate effects; cleaner-burning cookstoves may provide "co-benefits".

82 We found that PM2.5 from biomass burning, diesel vehicle, gasoline vehicle, and dust sour

83 nst instabilities triggered by spectral hole burning effects, and the possibility of more versatile m

84 on at midcentury reflects policies promoting burning efficiency and fuel transitions rather than regu

85 est adult residence at least once a week and burning either wood or natural gas/propane was associate

86 mospheric methane with these revised biomass burning emissions (and assuming no change to the chemica

87 We collected filter samples of biomass-burning emissions at three locations in Canada and the U

88 nocturnal chemistry using authentic biomass-burning emissions in a smog chamber.

89 Here we show that biomass burning emissions of methane decreased by 3.7 (+/-1.4) T

90 ynamometer tests, cooking emissions, biomass burning emissions, and a highway traffic tunnel.

91 s exposed to air pollution caused by biomass burning emissions.

92 investigate the influence of a major biomass burning event on the light absorption properties of atmo

93 t was observed during the night-long biomass burning event.

94 to the retina and optic nerve in chemically burned eyes.

95 n chest [aPR=1.58 (95% CI: 1.37, 1.81)], and burning eyes [aPR=1.48 (95% CI: 1.35, 1.64).

96 display an inherently increased capacity to burn fat and suppress de novo hepatic lipid synthesis.

97 ns from biomass burning (BB) and fossil fuel burning (FFB).

98 since fire and the difference in time since burning for a focal-source pair ranging from 3 to 58 mon

99 Patients with burns, foreign body injury, toxic effects, or late compl

100 increase in the number of small stems in the burned forests up to 5-9 years after fire because of reg

101 nditions, glucose is generally assumed to be burned fully by tissues via the tricarboxylic acid cycle

102 enstrophy decreases from the unburned to the burned gas side for the cases representing the TRZ and B

103 grouped 18,155 patients, including 1476 with burns >20% total body surface area, by presence of AKI.

104 ar postburn injury in 63 patients with major burns (>/=15% total body surface area).

105 action of climate change and increasing area burned has the potential to alter forest composition and

106 Patients with major burns have major (>1 blood volume) transfusion requireme

107 used by deforestation and associated biomass burning Hg emissions.

108 ay play an important role in the recovery of burned holm-oak forests.

109 ng, we identified the main bacterial taxa of burnt holm-oak rhizosphere, then we obtained an isolate

110 ons to either the medical or surgical/trauma/burn ICU with available continuous electrocardiogram dat

111 T4T-2OD:EH-IDTBR solar cell shows negligible burn in efficiency loss.

112 mulating corneal angiogenesis with an alkali burn in Tie2-GFP fluorescent-reporter mice, we evaluated

113 ncy loss under simulated solar irradiation ("burn in" degradation) due to the trap-assisted recombina

114 and one of the oldest surviving encaustic ("burned in") paintings in the world.

115 Near Arkhangelsk (White Sea), biomass burning in mid-latitudes, surface transportation, and re

116 the OSRC, with the strongest association for burning in the nose, throat, or lungs [adjusted PR (aPR)

117 ollution focus on emissions from fossil fuel burning in urban centers.

118 wildland firefighters conducting prescribed burns in California.

119 e reported, which sustain 80% of their "post burn-in" efficiency over 3400 h under full sun illuminat

120 We have previously shown that burn increases farnesyltransferase (FTase) expression an

121 These findings indicate that these burn-induced changes, which encompass various aspects of

122 tion and that FTase inhibitor (FTI) prevents burn-induced hyperlactatemia, insulin resistance, and in

123 olvement of the mTORC1-HIF-1alpha pathway in burn-induced metabolic derangements.

124 ds functionalized with the adhesin MAM7 to a burn infected with multidrug-resistant Pseudomonas aerug

125 at vancomycin treatment of a S. aureus mouse burn infection is potentiated by the presence of a LasA-

126 In this study, we found that burn injury activated mTORC1 and hypoxia-inducible facto

127 tiple aspects of metabolic alterations after burn injury and as a novel potential molecular target to

128 al muscle stem cells, is altered following a burn injury and likely hinders regrowth of muscle.

129 neutrophil function longitudinally following burn injury and to examine the relationship between neut

130 ted to the hospital after sustaining a large burn injury are at high risk for developing hospital-ass

131 velopment of acute kidney injury (AKI) after burn injury as an independent risk factor for increased

132 Because the mouse inflammatory response to burn injury cannot account for the contribution of human

133 Burn injury caused mobilization of human inflammatory ce

134 (8 weeks of age) were randomized to sham or burn injury consisting of a dorsal scald burn injury cov

135 or burn injury consisting of a dorsal scald burn injury covering 30% of total body surface area.

136 f myofibre area and volume following a scald burn injury in mice.

137 ptosis of muscle satellite cells following a burn injury in paediatric patients.

138 Burn injury induced robust atrophy in muscles located bo

139 Additionally, burn injury induced skeletal muscle regeneration, satell

140 Severe burn injury induces satellite cell proliferation and fus

141 Severe burn injury induces skeletal muscle regeneration and myo

142 However, major burn injury is precluded from these studies.

143 The optimal transfusion strategy in major burn injury is thus needed but remains unknown.

144 Skeletal muscle regrowth following a burn injury requires satellite cell activity, underscori

145 microbiome and antimicrobial peptides after burn injury to identify potential mechanisms leading to

146 18 hospitalized with a primary diagnosis of burn injury using ICD-9 codes.

147 nificant complication of major trauma (e.g., burn injury) and include various aspects of metabolism,

148 ith a major risk of local excessive heating, burn injury, and fire.

149 d its associated mortality is high following burn injury, and sepsis diagnosis is complicated by the

150 on of nociceptive primary sensory neurons by burn injury, capsaicin application or sustained electric

151 atients survive the initial 72 hours after a burn injury, infections are the most common cause of dea

152 SI, mortality, or organ dysfunction in major burn injury, these outcomes were no worse than the liber

153 ite cells during muscle recovery following a burn injury, we utilized a genetically modified mouse mo

154 ajor complications that hamper recovery from burn injury.

155 ucial to avoid the consequences of AKI after burn injury.

156 lerating action of EPO on the healing of the burn injury.

157 y of lean tissue recovery following a severe burn injury.

158 ntial for the early diagnosis of sepsis post-burn injury.

159 s are major barriers to preventing AKI after burn injury.

160 ars, the mean total body surface area (TBSA) burned is 18.5 +/- 16.4%, and the population is 74.9% ma

161 Biomass burning is a known source of brown carbon aerosol in the

162 Household air pollution from solid fuel burning is a leading contributor to disease burden globa

163 se experiments directly confirm that biomass burning is a potentially significant source of atmospher

164 Biomass burning is a source of both particulate chloride and nit

165 SUMMARY OF BACKGROUND DATA: HO in burns is an uncommon but severely debilitating problem w

166 ence of accidental and intentional acid skin burns is rising.

167 ecialized metabolic tissue that takes up and burns lipids and is linked to systemic metabolic homeost

168 ted with oxygenated species (primary biomass burning markers) increased (decreased) with MCE; fresh O

169 Seizure disorders, burn marks, and respiratory problems were important como

170 May the force be with you: Xia, Burns, Martinez, and co-workers harnessed the ring strai

171 glyoxal emissions from agricultural biomass burning may be significantly overestimated.

172 soil nitrogen that result from more frequent burning may in turn decrease the carbon that is sequeste

173 broadening, as observed using spectral hole burning measurements.

174 d or ameliorated all of these alterations in burned mice.

175 sed corneal opacity in murine corneal alkali burn model by modulating inflammatory cytokines.

176 Data were obtained from the Burn Model System National Database from 1994 to 2010 (n

177 les easily applicable and hand-held body fat burn monitoring for personalized and immediate feedback

178 Wildfires burn more than 7 million acres in the United States annu

179 nt across years, were found for residual oil burning (nickel, zinc), near-road traffic (copper, iron,

180 -like OA is largely water-insoluble, biomass burning OA and cooking OA have the largest range of wate

181 Local burning occurred at least three times in the past 4,500

182 Smoke from the burning of biomass fuels has been linked with adverse pr

183 Indoor burning of fuel for heating or cooking releases carcinog

184 The burning of LPG and alcohol had the lowest PM2.5 mass emi

185 wn-regulation after hepatectomy promotes the burning of TRAS-derived lipids to fuel hypertrophic live

186 for 39 individual SVOCs are determined from burning of various fuel types that are common in tropica

187 noscale defects left behind after the binder burnt off became effective phonon scattering centers, le

188 wing printing, the binders were subsequently burnt off via sintering and hot pressing.

189 nding to substrates with accelerants (either burnt or un-burnt) and inhibit responses to the same sub

190 bit responses to the same substrates (either burnt or un-burnt) without accelerants (S+ counterbalanc

191 ant light absorption contributors of biomass burning organic aerosol (BBOA), suggesting that night ti

192 formation of reactive compounds capable of 'burning out' the whole photosynthetic unit.

193 an ingredient in spicy peppers that produces burning pain by activating transient receptor potential

194 Severely ill burn patients admitted to our tertiary referral center b

195 peptide levels were significantly reduced in burn patients at 1 and 40 hours postadmission.

196 to develop a scoring system that stratifies burn patients at the time of hospital admission accordin

197 evere increase in morbidity and mortality in burn patients during initial hospitalization and up to 1

198 Elderly burn patients had significantly lower burn size but sign

199 HO risk scoring system identifies high-risk burn patients suitable for diagnostic testing and interv

200 ction development and several morbidities in burn patients through alterations in the urinary microbi

201 t increase in urinary microbial diversity in burn patients versus controls, which positively correlat

202 rmore, the antimicrobial peptide activity in burn patients was more effective against E. coli than E.

203 Urine samples from catheterized burn patients were collected hourly for up to 40 hours.

204 rophobicity and activity between control and burn patients when urinary fractions were tested against

205 Furthermore, a multidisciplinary approach to burn patients with a team that includes an infectious di

206 Urinary tract infection-positive burn patients with altered urinary antimicrobial peptide

207 the burden AKI has on the clinical course of burn patients would highlight the need for standardized

208 act infections and associated morbidities in burn patients.

209 ice of empiric antibiotics in critically ill burn patients.

210 ns increases with hospital length of stay in burn patients.

211 ty to urinary tract infections and sepsis in burn patients.

212 Burn patients.

213 n 20% or more total body surface area (TBSA) burn patients.

214 Furthermore, burned patients with sepsis had a more profound expansio

215 Our results demonstrate that burned patients with sepsis have more profound impairmen

216 irment of monocytes and dendritic cells than burned patients without sepsis.

217 inosa is a major cause of sepsis in severely burned patients.

218 PA14) was grown in whole blood from severely burned patients.

219 of CD14/CD16 Mo when compared with nonseptic burned patients.

220 to improve the clinical outcome of severely burned patients.

221 t been thoroughly characterized over time in burns patients.

222 Here we show that diagnostic burn patterns on eggshell fragments of the megafaunal bi

223 We find that frequently burned plots experienced a decline in surface soil carbo

224 ide and formaldehyde in agricultural biomass burning plumes intercepted by the NOAA WP-3D aircraft du

225 the primary PM2.5 sources assessed, biomass burning PM2.5 was most strongly associated with respirat

226 f respiratory disease ED visits with biomass burning PM2.5; associations with diesel and gasoline PM2

227 m explained 17% of the variability in ocular burning (R = 0.41; P < .001), and PTSD score, tear break

228 articles that can accurately follow body fat burn rates in real time.

229 table biomedical sensors to monitor body fat burn rates in situ, to guide physical activity or dietar

230 cover change estimated from MODIS Normalized Burn Ratio (NBR), existing MODIS Land Cover (LC) and Veg

231 Depletion of satellite cells impaired post-burn recovery of both muscle fibre cross-sectional area

232 Burning reduced the cover of the dominant grasses by mor

233 e human lung.Solid-state emissions from coal burning remain an environmental concern.

234 trong evidence indicates that this partially burnt remnant was ejected following a subluminous Type I

235 Severe burns result in profound skeletal muscle atrophy that ha

236 ABSTRACT: Severe burns result in profound skeletal muscle atrophy; persis

237 KEY POINTS: Severe burns result in significant skeletal muscle cachexia tha

238 Consensus protocols for initial burn resuscitation and early sepsis recognition and trea

239 ering the carbon sink capacity of frequently burning savanna grasslands and broadleaf forests.

240 Other symptoms were dryness, redness, burning sensation and itching.

241 Patients with lower burn severity (total body surface area, 20-30%) express

242 ent scenarios increased C stability, reduced burn severity, and consequently emitted less C as a resu

243 mpared with the control plots, Rs in the low burning severity plots decreased by 19%, while it decrea

244 y 19%, while it decreased by 28% in the high burning severity plots.

245 e results showed that Rs decreased with fire burning severity.

246 enrolled 345 patients with 20% or more TBSA burn similar in age, TBSA burn, and inhalation injury.

247 lderly burn patients had significantly lower burn size but significantly higher Baux scores.

248 e wound-healing and reduce hair loss in acid-burn skin injury.

249 the plant-microbe interactions that occur in burned soils.

250 iptors ("garlic," "fish," "sweet," "fruit," "burnt," "spices," "flower," and "sour").

251 al or domestic waste, wich is sometimes then burned spreading toxic contaminants.

252 cy end points included additional VAS items (burning/stinging, itching, foreign body sensation, eye d

253 this prospective study, using an indoor wood-burning stove/fireplace in the longest adult residence a

254 Having an indoor wood-burning stove/fireplace in the longest adult residence w

255 mized trial was conducted in homes with wood-burning stoves among children with asthma.

256 icantly higher for Philips wood and charcoal-burning stoves compared to the threestone fire and coalp

257 the impact of indoor air pollution from wood-burning stoves or fireplaces on breast cancer risk.

258 ildren living with asthma in homes with wood-burning stoves.

259 ort study using human urine from control and burn subjects.

260 ure and archaeology reveal hermetic sealing, burning sulfur, desiccant dusts, and toxic botanicals as

261 ecialist and a pharmacist in addition to the burn surgeon is highly recommended.

262 e burn, contributing to prolonged frailty in burn survivors.

263 ls in the prevention of prolonged frailty in burn survivors.

264 form of steady-state phononic spectral hole burning that produces a wideband transparency window wit

265 In 2009, a lightning-caused wildfire burned through the experiment.

266 the effects of fire and recovery pathways of burned TMCFs, with a detailed analysis of carbon stocks,

267 sal strategies such as boiling, roasting, or burning to be differentiated.

268 We investigated the ability of biomass burning to produce N2O5(g) and ClNO2(g) through nocturna

269 Alkali burns to the eye constitute a leading cause of worldwide

270 etabolic alterations as patients with larger burns (total body surface area, >/= 30%).

271 values of glyoxal relative to other biomass burning trace gases, indicating that glyoxal emissions f

272 iquitous gas in the atmosphere and a biomass burning tracer.

273 radiative forcing and could serve as biomass burning tracers.

274 a were used to assign PMF factors to biomass burning, traffic, and smoking emission sources.

275 bute to the erosion of muscle mass following burn trauma, and we have previously shown concurrent act

276 response of six human inflammatory diseases (burns, trauma, infection, sepsis, endotoxin and acute re

277 Sediments from the burned tributary inputs contained higher levels of bioav

278 e infection control approach is essential in burn units.

279 when farming communities expanded and forest burning was used for land clearance.

280 alpha-particle heating expected to trigger a burn wave in the hydrogen pellet and resulting in high t

281 oncentrations observed during Amazon biomass burning were sufficient to induce severe adverse effects

282 The top two causes of burns were hot liquids (36.3%) and hot object (29.5%).

283 and number-based emission indices (/kg fuel burned) were 6.2-14.7 mg and 2.8 x 10(14) - 8.7 x 10(14)

284 Cooking-related serious adverse events (burns) were seen in 19 children; nine in the interventio

285 osta Rica to Mexico, and Udranomia tomdaleyi Burns, which occupies the junction between the rainfores

286 t direct contact with smoke near an actively burning wildland fire.

287 ction to prolonged sun exposure (for painful burn with blisters vs. practically no reaction, multivar

288 apoptosis and activation acutely following a burn, with a net reduction in satellite cell content com

289 s to the same substrates (either burnt or un-burnt) without accelerants (S+ counterbalanced).

290 itional stove/fuel combinations, the Philips burning wood or charcoal showed significant fuel and ene

291 ed growing season dryness and increased area burned would substantially decrease forested area, trans

292 s study, we utilize a panel of P. aeruginosa burn wound and cystic fibrosis (CF) lung isolates to dem

293 g represents a breakthrough in second-degree burn wound care.

294 An adhesive yet easily removable burn wound dressing represents a breakthrough in second-

295 Current second-degree burn wound dressings absorb wound exudate, reduce bacter

296 cial role in the pathogenesis of PA14 during burn wound infection, most likely by contributing to PA1

297 to the pathogenesis of P. aeruginosa during burn wound infection.

298 shown the abilities to prevent infection of burn wound, aid healing, and an anti-inflammatory dressi

299 cells (ASCs) accelerates the process of acid burn wound-healing.

300 he mechanism of EPO action on the healing of burn wounds in the skin of pigs with experimentally indu