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3 letion occurred, characterized by follicular burn out and a loss of CD3 T lymphocytes, which was asso
4 ntinual rise in SBP, cannot be explained by "burned out" diastolic hypertension or by "selective surv
6 to discriminate between patients with stable burnt-out disease that is no longer metabolically active
7 ted with almost complete hepatic fat loss or burnt-out NASH (12.1 versus 7.4 mug/L, P = 0.001) on mul
8 patic fat to the point of complete fat loss (burnt-out NASH), but the mechanisms behind this phenomen
16 wders with a graphite fugitive phase that is burned out to create internal cavities and microchannels
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