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1 in partial-thickness articular (48%) than in bursal (13%) tears (P<.001).
2 on of PE prolonged survival of VLR(PE)Tmu(+) bursal and peripheral B cells.
3 riant GLS or virulent IM strain caused rapid bursal atrophy or hemorrhagic lesions in the bursa, as s
4  role of BCR ligation in the later stages of bursal B cell lymphopoiesis remains elusive.
5 conclude that Ag-mediated ligation of BCR in bursal B cells acts to positively select bursal B cells
6 the chB1 molecules inhibits proliferation of bursal B cells and the DT40 cell line.
7  in bursal B cells acts to positively select bursal B cells into both short-lived and long-lived peri
8                                  We screened bursal B cells with a panel of Abs and lectins to identi
9 l surface molecules selectively expressed on bursal B cells.
10 disrupted the MEKK1 kinase domain in chicken bursal B-cell line DT40 by homologous recombination and
11 l myristate acetate, two known inhibitors of bursal cell apoptosis, induced Nr13 expression.
12 examined to identify the genes that regulate bursal cell apoptosis.
13 lly expressed in cultured chicken spleen and bursal cells after mitogen stimulation.
14                             The abundance of bursal cells carrying these integrations increases rough
15 emarkably common, with an estimated 1 in 350 bursal cells having proviral c-myc integrations.
16                                   Spleen and bursal cells of IBDV-infected chickens had upregulated g
17                       At 7 days p.i., 65% of bursal cells were T cells and 7% were B cells.
18                                              Bursal cells with proviral c-myc integrations also arise
19    Although BCR expression is sufficient for bursal colonization, the role of BCR ligation in the lat
20  attachments of the abductor muscles and the bursal complex of the greater trochanter.
21  target cell population present during early bursal development and progresses through preneoplastic
22                                   Infectious bursal disease (IBD) is of economic importance to the po
23 d on synthetic transcripts of the infectious bursal disease virus (IBDV) genome, was recently develop
24                                   Infectious bursal disease virus (IBDV) is an avian lymphotropic vir
25 Here, we analyzed the role of the infectious bursal disease virus (IBDV) VP3, a major component of IB
26 a replicating nonpathogenic avian infectious bursal disease virus (IBDV) was explored.
27                    In this study, infectious bursal disease virus (IBDV) was used to investigate the
28  VP2 capsid protein (pVP2) of the infectious bursal disease virus (IBDV), a double-stranded RNA virus
29 ost proteins in the life cycle of infectious bursal disease virus (IBDV), a double-stranded RNA virus
30 endent RNA polymerase of IPNV and infectious bursal disease virus (IBDV), another birnavirus, can sup
31                                   Infectious bursal disease virus (IBDV), the causative agent of a hi
32 the host response to infection by infectious bursal disease virus (IBDV).
33  (chIFN-alpha) and the attenuated infectious bursal disease virus vaccine strain PBG98.
34                                   Infectious bursal disease viruses (IBDVs), belonging to the family
35  results in a normal pattern of posthatching bursal emigration in resistant transformed follicles, wh
36 that these cells hyperproliferate within the bursal environment.
37 tions are preferentially expanded within the bursal environment.
38 of sheep are regarded as a type of mammalian bursal equivalent where B cells diversify their repertoi
39 ated gammaglobulin and complement identified bursal-equivalent or bone marrow-derived (B) lymphocytes
40 ign, joint fluid, and subacromial-subdeltoid bursal fluid.
41               VLR(PE)Tmu expression supports bursal follicle colonization, clonal expansion, and Ig V
42 ood agreement with the number of transformed bursal follicles arising with these integrations.
43 s to an oligoclonal population of developing bursal follicles, showing a variable propensity to form
44  myc-overexpressing progenitors into ablated bursal follicles, suggesting that these angiogenic chang
45 ed early angiogenesis within myc-transformed bursal follicles, which persisted in lymphomas and metas
46 verexpressing lymphocytes within transformed bursal follicles.
47      Hence, GALT may function as a mammalian bursal homologue.
48 th the bursal medial zone (P = .002) and the bursal lateral zone (P = .003) were observed.
49 est (ROIs): bursal medial, articular medial, bursal lateral, and articular lateral.
50                       Tendon attachments and bursal localization were related to the facets of the gr
51 induced, whereas Nr13 levels diminished when bursal lymphoblasts were induced to apoptosis by dispers
52 t with the expression signature of embryonic bursal lymphocytes.
53               Overexpression of Nr13 in DT40 bursal lymphoma cells protected them from low serum-indu
54           Avian leukosis virus (ALV) induces bursal lymphoma in chickens after proviral c-myc gene in
55           Avian leukosis virus (ALV) induces bursal lymphoma in tumor-susceptible chicken strains aft
56 e to avian leukosis virus (ALV) induction of bursal lymphoma, involving proviral integration within t
57 ens; these arise with a shorter latency than bursal lymphomas caused by deregulation of c-myc.
58 an, and only partially shared with, those of bursal lymphomas caused by Myc or Rel oncogenes.
59 Avian leukosis virus (ALV) infection induces bursal lymphomas in chickens after proviral integration
60  1, in a pattern similar to that observed in bursal lymphomas.
61 tween the articular medial zone and both the bursal medial zone (P = .002) and the bursal lateral zon
62 erived from four regions of interest (ROIs): bursal medial, articular medial, bursal lateral, and art
63 lls can therefore occur independently of the bursal microenvironment and is dependent on signaling do
64 interactions between B lineage cells and the bursal microenvironment, we sought to identify genes enc
65 at negative selection was independent of the bursal microenvironment.
66 s alter recognition of dormant articular and bursal monosodium urate monohydrate (MSU) crystal deposi
67 ediated overexpression of c-myc in embryonic bursal precursors induces multi-staged tumorigenesis beg
68 y indicates that c-myb mutation in embryonic bursal precursors leads to an oligoclonal population of
69 al program of differentiation and subsequent bursal retention of lymphocytes accounts for most of the
70 revent the in vivo programmed elimination of bursal stem cells after hatching, suggesting that Nr13 p
71 esting that Nr13 plays a role in maintaining bursal stem cells.
72 scopy revealed 21 full-thickness tears, five bursal surface partial-thickness tears, 10 articular sur
73 n and location of partial tear (articular or bursal surface) were recorded.
74        After virus infection, the numbers of bursal T cells expressing activation markers Ia and CD25
75                    In addition, IBDV-induced bursal T cells produced elevated levels of interleukin-6
76                   In IBDV-infected chickens, bursal T cells proliferated in vitro upon stimulation wi
77 c analysis of single-cell suspensions of the bursal tissue revealed that T cells were first detectabl
78                                   Spleen and bursal tissue were examined from control and infected bi
79                                              Bursal transplantation studies demonstrated that Nr13 co

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