ライフサイエンスコーパス: burst discharge

1 pical TRN neurons that could produced strong burst discharge.

2 s contribute to production of high-frequency burst discharge.

3 re well established electrogenic drivers for burst discharge.

4 aptically mediated long-latency epileptiform burst discharges.

5 ) synchronous with the GABAergic interneuron burst discharges.

6 decreased T-channel availability as well as burst discharges.

7 ) generate single, spontaneous, synchronized burst discharges, 2) support activity spread along axes

8 e cells generates repetitive, high-frequency burst discharges, a pattern referred to as "chattering."

9 tegrating synchronous inputs and presynaptic burst discharges, allowing hilar cells to respond over a

10 tion of the depolarizing envelope underlying burst discharges and attenuated the subsequent afterhype

11 rity of neurons in the dorsal TRN (56%) lack burst discharge, and the remaining neurons (35%) show an

12 rget nuclei display synchronized oscillatory burst discharge at low frequencies, some of which correl

13 rates that synaptically dependent excitatory burst discharges can be evoked from daPC neurons without

14 in ventral TRN (82%) display a stereotypical burst discharge consisting of a transient, high frequenc

15 onic depolarizations or repetitive, rhythmic burst discharges, either as clonic or spike-wave activit

16 We now report that a single somatic burst discharge evokes large-magnitude calcium responses

17 tion time of input signals and could exhibit bursting discharge.for loosely synchronized inputs, we f

18 ures might reflect a reduction in endogenous burst discharges from that side.

19 Synaptic transmission was optimized for burst discharge >14 Hz and showed considerable short-ter

20 from adult rats, is known to cause prolonged burst discharges (i.e. several seconds vs. tens of milli

21 erneurons, including tonic firing or initial bursting discharge, Ih currents, and islet cell morpholo

22 tion of decreased interictal single neuronal burst discharge in epileptogenic structures stresses the

23 reshold membrane oscillations and rhythmical burst discharge in Mes V neurons from rats ages postnata

24 tonic periods of REM sleep, with occasional burst discharge in phasic REM.

25 A single stimulus could evoke burst discharges in infrapyramidal granule cells but not

26 PD-like locomotor deficits and increased STN burst discharges in normal rats.

27 te blockade of firing in principal cells and burst discharges in putative interneurons.

28 (2)-like receptors inhibit complex EPSCs and burst discharges in the SNR by acting within the STN to

29 microM) to the perfusate elicited repetitive burst discharges in the somatic motor outflow which were

30 channels, play a key role in the genesis of burst discharges in the subthalamic nucleus (STN) and pa

31 NMDA induced burst discharges in the sympathetic outflow.

32 We propose that the activation of burst discharges in these cell types is essential for th

33 The increased tendency of STN burst discharges may by itself serve as a direct cause o

34 The generation of high-frequency burst discharges may strongly influence the response of

35 amp recordings, SST preferentially inhibited burst discharges mediated by near-threshold corticothala

36 ogenous bursting could be due to the loss of burst discharging neurons as a product of seizure-relate

37 This decreased burst discharge of nRt neurons during CCK application re

38 Notably, oscillatory burst discharge of reticular neurons is typical for slee

39 BA(A)- and GABA(B)-receptor antagonists, the burst discharges of immature CA3 pyramidal cells were st

40 Burst discharges of thalamic neurons reflected the chang

41 spontaneous activity for stages 39-43 was a bursting discharge pattern in >75% of active neurons (33

42 The frequency of these burst discharges/rhythmic activity varied between prepar

43 ceptor antagonist DNQX blocked the remaining burst discharges, suggesting that differences in recurre

44 ile mice can generate episodes of repetitive burst discharges that may underlie the pulsatile secreti

45 TRN neurons that lack burst discharge typically did not produce low threshold

46 Burst discharge via Ca(V)3.3 channels induced long-term

47 tic currents (EPSCs), spontaneous EPSCs, and burst discharges were demonstrated in UBCs and granule c

48 d, in deep layer cells only, a short latency burst discharge which could be followed by one or more a

49 is study, we have examined the prevalence of burst discharge within TRN neurons.