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1 ilineage colony-forming units, and erythroid burst-forming units.
2 (+) cells (2.7-fold/kg/apheresis), erythroid burst-forming units (1.8-fold/kg/apheresis), and colony-
3                                    Erythroid burst-forming unit and colony-forming unit numbers are g
4 ononuclear cells were cultured for erythroid burst-forming unit and granulocyte-macrophage colony-for
5 eir wild-type littermates, splenic erythroid burst-forming unit and high-proliferative potential colo
6 on of late-stage erythroid progenitors-day 3 burst-forming units and colony-forming units, associated
7  normal hematopoietic progenitors, erythroid burst-forming units and granulocyte/monocyte colony-form
8 and osteoclastogenic but a low proportion of burst-forming unit (BFU)e.
9                              Using erythroid burst-forming unit (BFU-E) and CFU-E progenitors purifie
10     High-level MDR transduction of erythroid burst-forming unit (BFU-E) and colony-forming unit-granu
11 hood showed a moderate decrease in erythroid burst-forming unit (BFU-E) and erythroid colony-forming
12 e colony-forming unit (CFU-GM) and erythroid burst-forming unit (BFU-E) colonies.
13  early erythropoiesis encompassing erythroid burst-forming unit (BFU-E) differentiation to proerythro
14 e colony-forming unit (CFU-GM) and erythroid burst-forming unit (BFU-E) growth.
15 25 inhibitor reduced the number of erythroid burst-forming units (BFU-e's) but not the more different
16   A second wave of definitive-type erythroid burst-forming units (BFU-e's), erythroid colony-forming
17                                    Erythroid burst-forming units (BFU-E) were isolated from periphera
18  colony-forming units (CFU-GM) and erythroid burst-forming units (BFU-E) were performed on each harve
19 mpanied with decreased bone marrow erythroid burst-forming units (BFU-Es) and colony-forming units-er
20 f EFA to hematopoietic progenitors erythroid burst-forming units (BFU-Es) and granulocyte-macrophage
21                     V617F-positive erythroid burst-forming units (BFU-Es) were more frequent in patie
22 requency and number of both early (erythroid burst-forming unit [BFU-E]) and late erythroid progenito
23 umber and frequency of both early (erythroid burst-forming unit [BFU-E]) and late erythroid progenito
24 e reported that SP stimulates erythroid (E) (burst-forming unit [BFU]-E and colony-forming unit [CFU]
25 ells (colony-forming unit-erythroid [CFU-E], burst-forming unit [BFU]-E, and CFU-granulocyte-macropha
26 globin-containing erythroblasts in erythroid burst-forming unit (BFUe) cultures from healthy adult in
27 n in erythroid progenitors (mature erythroid burst-forming units [BFUEs]) was observed between e14.5
28 ith discordant results and also in erythroid burst-forming unit colonies but not in those with clonal
29 macrophage colony-forming unit and erythroid burst-forming unit colonies compared with plasma from ca
30 macrophage colony-forming unit and erythroid burst-forming unit colony formation compared with BM of
31 common myeloid progenitors (CMPs), erythroid burst-forming units, colony-forming units in spleen, and
32 ac or embryo body by three different assays, burst-forming units, colony-forming units, and analysis
33 res, as indicated by more numerous erythroid burst-forming unit-derived colonies in low Epo concentra
34 k and E progenitors identified as CFU-Mk and burst forming unit-E.
35 locyte macrophage, but not other HPC such as burst-forming unit erythrocyte or CFU granulocyte, eryth
36 of cells in G1 phase (27.4%) was observed in burst-forming unit-erythrocytes (BFU-E)-derived erythrob
37 -EPOR antibodies stimulated the formation of burst forming unit erythroid colonies from human CD34(+)
38 ly supports the differentiation of wild-type burst-forming unit erythroid (BFU-e) and colony-forming
39 lf-renewal of an early erythroid progenitor, burst-forming unit erythroid (BFU-E), and increase the p
40 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells revealed absence of t
41 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells with the 11q deletion
42  Frs. 2 and 3, greater than 70% of the total burst-forming unit erythroid colonies in CB and PB were
43                            Further, although burst-forming unit erythroid colonies of BM were distrib
44                                              Burst-forming unit erythroid progenitors (BFU-Es) are so
45 lony forming unit granulocyte-macrophage and burst-forming unit erythroid, while treatment with block
46                                       Normal burst forming unit-erythroid (BFU-E) growth (>30 bursts/
47 points representing the transition from late burst forming unit-erythroid (BFU-E) to basophilic eryth
48 nit-granulocyte-macrophage (CFU-GM)-derived, burst forming unit-erythroid (BFU-E)-derived, and CFU-me
49 nulocyte-macrophage [CFU-GM]; 17% inhibition burst forming unit-erythroid [BFU-E]) and 3.44 micromol/
50 lony-forming unit-granulocyte macrophage and burst forming unit-erythroid were detected in the BM of
51 in other hematopoietic cells including human burst forming unit-erythroid-derived erythroblasts and T
52 median slope per day, -0.57; P = .0008), and burst-forming unit-erythroid (BFU-E) (median slope per d
53 1 expression in human CD34(+) cells impaired burst-forming unit-erythroid (BFU-E) and colony-forming
54 le colony-forming unit-erythroid (CFU-E) and burst-forming unit-erythroid (BFU-E) colonies were not p
55                                              Burst-forming unit-erythroid (BFU-E) colony-formation wa
56 formation by promoting self-renewal of early burst-forming unit-erythroid (BFU-E) progenitors.
57                      Studies with progeny of burst-forming unit-erythroid (BFU-E) suggest that the FR
58  colony-forming unit-macrophage (CFU-M), and burst-forming unit-erythroid (BFU-E) was markedly decrea
59 orming unit-granulocyte/macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and CFU-erythroid
60 ming unit-granulocyte, -macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and colony-forming
61 main (HBD), to promote adhesion of primitive burst-forming unit-erythroid (BFU-E), mature BFU-E, and
62 , levels of these factors were determined in burst-forming unit-erythroid (BFU-E)-derived cells at di
63                                              Burst-forming unit-erythroid (BFU-E)-derived colony grow
64 3 protein was also highly expressed in early burst-forming unit-erythroid (BFU-E)-derived erythroid p
65 ntirely in the SBA(+) fraction; in contrast, burst-forming unit-erythroid (BFU-E)-forming cells were
66                       Erythroid progenitors (burst-forming unit-erythroid [BFU-E]) were significantly
67 ny-forming unit-megakaryocyte [CFU-Meg], and burst-forming unit-erythroid [BFU-E]), and CD34(+) cells
68 ocyte, megakaryocyte [CFU-GEMM]), erythroid (burst-forming unit-erythroid [BFU-E]), and granulocyte-m
69 ing unit-granulocyte-macrophage [CFU-GM] and burst-forming unit-erythroid [BFU-E]).
70                          Similarly, in vitro burst-forming unit-erythroid and colony-forming unit-ery
71                  Deletion of Stat1 increased burst-forming unit-erythroid and reduced colony-forming
72               Erythroblasts plucked from the burst-forming unit-erythroid colonies of one of these ch
73 onies, whereas Flt3low cells produced mostly burst-forming unit-erythroid colonies.
74 y erythroid progenitors showed inhibition of burst-forming unit-erythroid colony formation when inter
75 ing unit-granulocyte/macrophage (CFU-GM) and burst-forming unit-erythroid derived from CML over a 2-l
76       We focus on the regulated expansion of burst-forming unit-erythroid erythroid progenitors by gl
77 a and promoted the formation of CFU-GEMM and burst-forming unit-erythroid in methylcellulose cultures
78 lony-forming unit-granulocyte macrophage and burst-forming unit-erythroid numbers and preferentially
79 479 (Y8) were capable of supporting immature burst-forming unit-erythroid progenitor development.
80 r, only gp55-P induces erythroid bursts from burst-forming unit-erythroid progenitors and only gp55-P
81 eLV-C impairs the in vivo differentiation of burst-forming unit-erythroid to colony-forming unit-eryt
82                 Maximal numbers of CFU-E and burst-forming unit-erythroid were increased, and CFU-E d
83 ming granulocytic-macrophage) and erythroid (burst-forming unit-erythroid) progenitor colony formatio
84 g unit-granulocyte-monocytic) and erythroid (burst-forming unit-erythroid) progenitors.
85  colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, and high proliferative pot
86 oid progenitors (CFU-granulocyte-macrophage, burst-forming unit-erythroid, CFU-granulocyte-erythrocyt
87  colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, or colony-forming unit-ery
88 ently detected on red blood cell precursors, burst-forming unit-erythroid- (BFU-E) derived cells.
89 yocyte [CFU-MK], CFU-granulocyte/macrophage, burst-forming unit-erythroid/CFU-erythroid, and CFU-gran
90 y transiently delaying erythropoiesis at the burst-forming unit-erythroid/colony-forming unit-erythro
91 iest erythroid-restricted precursors (BFU-E [burst-forming unit-erythroid]) is also detected in the I
92 eous cell population, we analyzed individual burst-forming units-erythroid (BFU-E) and nonerythroid c
93                      Progeny of normal human burst-forming units-erythroid (BFU-E) contained Mpl rece
94                                       Single burst-forming units-erythroid (BFU-E) from 1 patient wer
95 , and greater than 10-fold higher numbers of burst-forming units-erythroid (BFU-E) in the Wnt-express
96                                              Burst-forming units-erythroid (BFU-E) increased in the s
97 acrophage colony-forming cells (GM-CFC), and burst-forming units-erythroid (BFU-E) per kilogram in th
98 0(4)/kg, respectively; the median numbers of burst-forming units-erythroid (BFU-e) were 0.20, 6.94, a
99 ethasone selectively increased the number of burst-forming units-erythroid (BFU-E), whereas lenalidom
100 p67(+) cells were significantly enriched for burst-forming units-erythroid (BFU-Es) and depleted of c
101  and chemotherapy patient-derived erythroid (burst-forming units-erythroid [BFU-E]), myeloid (colony-
102            It has been previously shown that burst-forming units-erythroid and colony-forming units-e
103 ma-globin expression in K562 cells and human burst-forming units-erythroid and that increase prolifer
104          AT1 protein was detected in 7-d-old burst-forming units-erythroid colonies by Western blotti
105 progenitors, defined as increased numbers of burst-forming units-erythroid colonies.
106                         Fura-2 loaded day-10 burst-forming units-erythroid-derived erythroblasts were
107 oietic cells and completely blocks erythroid burst-forming unit formation in normal human bone marrow
108 use granulocyte-macrophage CFU and erythroid burst-forming units from STAT1(-/-) mice were resistant
109 ing units were also inhibited, but erythroid burst-forming units grew normally.
110 acrophage colony forming unit, and erythroid burst forming unit growth in rats subjected to hemorrhag
111 macrophage colony forming unit and erythroid burst forming unit) hematopoietic progenitor cell growth
112 forming unit-megakaryocytes and occasionally burst-forming unit-megakaryocytes, with a plating effici
113                      No inhibition of either burst-forming unit-MK- or colony-forming unit-MK-derived
114 cells (19-39% growth inhibition of erythroid burst-forming units, multilineage colony-forming units,
115 t significantly alter steady-state erythroid burst-forming unit numbers.
116 lization of BMP4-responsive stress erythroid burst-forming units; therefore, new stress progenitors m
117 can generate BMP4-dependent stress erythroid burst-forming units when cultured under stress erythropo

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