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1                          Similarly, in vitro burst-forming unit-erythroid and colony-forming unit-ery
2                  Deletion of Stat1 increased burst-forming unit-erythroid and reduced colony-forming
3            It has been previously shown that burst-forming units-erythroid and colony-forming units-e
4 ma-globin expression in K562 cells and human burst-forming units-erythroid and that increase prolifer
5  colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, and high proliferative pot
6                                       Normal burst forming unit-erythroid (BFU-E) growth (>30 bursts/
7 points representing the transition from late burst forming unit-erythroid (BFU-E) to basophilic eryth
8 nit-granulocyte-macrophage (CFU-GM)-derived, burst forming unit-erythroid (BFU-E)-derived, and CFU-me
9 ly supports the differentiation of wild-type burst-forming unit erythroid (BFU-e) and colony-forming
10 lf-renewal of an early erythroid progenitor, burst-forming unit erythroid (BFU-E), and increase the p
11 median slope per day, -0.57; P = .0008), and burst-forming unit-erythroid (BFU-E) (median slope per d
12 1 expression in human CD34(+) cells impaired burst-forming unit-erythroid (BFU-E) and colony-forming
13 le colony-forming unit-erythroid (CFU-E) and burst-forming unit-erythroid (BFU-E) colonies were not p
14                                              Burst-forming unit-erythroid (BFU-E) colony-formation wa
15 formation by promoting self-renewal of early burst-forming unit-erythroid (BFU-E) progenitors.
16                      Studies with progeny of burst-forming unit-erythroid (BFU-E) suggest that the FR
17  colony-forming unit-macrophage (CFU-M), and burst-forming unit-erythroid (BFU-E) was markedly decrea
18 orming unit-granulocyte/macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and CFU-erythroid
19 ming unit-granulocyte, -macrophage (CFU-GM), burst-forming unit-erythroid (BFU-E), and colony-forming
20 main (HBD), to promote adhesion of primitive burst-forming unit-erythroid (BFU-E), mature BFU-E, and
21 , levels of these factors were determined in burst-forming unit-erythroid (BFU-E)-derived cells at di
22                                              Burst-forming unit-erythroid (BFU-E)-derived colony grow
23 3 protein was also highly expressed in early burst-forming unit-erythroid (BFU-E)-derived erythroid p
24 ntirely in the SBA(+) fraction; in contrast, burst-forming unit-erythroid (BFU-E)-forming cells were
25 eous cell population, we analyzed individual burst-forming units-erythroid (BFU-E) and nonerythroid c
26                      Progeny of normal human burst-forming units-erythroid (BFU-E) contained Mpl rece
27                                       Single burst-forming units-erythroid (BFU-E) from 1 patient wer
28 , and greater than 10-fold higher numbers of burst-forming units-erythroid (BFU-E) in the Wnt-express
29                                              Burst-forming units-erythroid (BFU-E) increased in the s
30 acrophage colony-forming cells (GM-CFC), and burst-forming units-erythroid (BFU-E) per kilogram in th
31 0(4)/kg, respectively; the median numbers of burst-forming units-erythroid (BFU-e) were 0.20, 6.94, a
32 ethasone selectively increased the number of burst-forming units-erythroid (BFU-E), whereas lenalidom
33 p67(+) cells were significantly enriched for burst-forming units-erythroid (BFU-Es) and depleted of c
34 nulocyte-macrophage [CFU-GM]; 17% inhibition burst forming unit-erythroid [BFU-E]) and 3.44 micromol/
35                       Erythroid progenitors (burst-forming unit-erythroid [BFU-E]) were significantly
36 ny-forming unit-megakaryocyte [CFU-Meg], and burst-forming unit-erythroid [BFU-E]), and CD34(+) cells
37 ocyte, megakaryocyte [CFU-GEMM]), erythroid (burst-forming unit-erythroid [BFU-E]), and granulocyte-m
38 ing unit-granulocyte-macrophage [CFU-GM] and burst-forming unit-erythroid [BFU-E]).
39  and chemotherapy patient-derived erythroid (burst-forming units-erythroid [BFU-E]), myeloid (colony-
40 ently detected on red blood cell precursors, burst-forming unit-erythroid- (BFU-E) derived cells.
41 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells revealed absence of t
42 lony-forming unit granulocyte-macrophage and burst-forming unit erythroid cells with the 11q deletion
43 oid progenitors (CFU-granulocyte-macrophage, burst-forming unit-erythroid, CFU-granulocyte-erythrocyt
44 yocyte [CFU-MK], CFU-granulocyte/macrophage, burst-forming unit-erythroid/CFU-erythroid, and CFU-gran
45 -EPOR antibodies stimulated the formation of burst forming unit erythroid colonies from human CD34(+)
46  Frs. 2 and 3, greater than 70% of the total burst-forming unit erythroid colonies in CB and PB were
47                            Further, although burst-forming unit erythroid colonies of BM were distrib
48               Erythroblasts plucked from the burst-forming unit-erythroid colonies of one of these ch
49 onies, whereas Flt3low cells produced mostly burst-forming unit-erythroid colonies.
50          AT1 protein was detected in 7-d-old burst-forming units-erythroid colonies by Western blotti
51 progenitors, defined as increased numbers of burst-forming units-erythroid colonies.
52 y erythroid progenitors showed inhibition of burst-forming unit-erythroid colony formation when inter
53 y transiently delaying erythropoiesis at the burst-forming unit-erythroid/colony-forming unit-erythro
54 ing unit-granulocyte/macrophage (CFU-GM) and burst-forming unit-erythroid derived from CML over a 2-l
55 in other hematopoietic cells including human burst forming unit-erythroid-derived erythroblasts and T
56                         Fura-2 loaded day-10 burst-forming units-erythroid-derived erythroblasts were
57       We focus on the regulated expansion of burst-forming unit-erythroid erythroid progenitors by gl
58 a and promoted the formation of CFU-GEMM and burst-forming unit-erythroid in methylcellulose cultures
59 iest erythroid-restricted precursors (BFU-E [burst-forming unit-erythroid]) is also detected in the I
60 lony-forming unit-granulocyte macrophage and burst-forming unit-erythroid numbers and preferentially
61  colony-forming unit-granulocyte-macrophage, burst-forming unit-erythroid, or colony-forming unit-ery
62 479 (Y8) were capable of supporting immature burst-forming unit-erythroid progenitor development.
63 ming granulocytic-macrophage) and erythroid (burst-forming unit-erythroid) progenitor colony formatio
64                                              Burst-forming unit erythroid progenitors (BFU-Es) are so
65 r, only gp55-P induces erythroid bursts from burst-forming unit-erythroid progenitors and only gp55-P
66 g unit-granulocyte-monocytic) and erythroid (burst-forming unit-erythroid) progenitors.
67 eLV-C impairs the in vivo differentiation of burst-forming unit-erythroid to colony-forming unit-eryt
68 lony-forming unit-granulocyte macrophage and burst forming unit-erythroid were detected in the BM of
69                 Maximal numbers of CFU-E and burst-forming unit-erythroid were increased, and CFU-E d
70 lony forming unit granulocyte-macrophage and burst-forming unit erythroid, while treatment with block

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